ライフサイエンスコーパス: tubular

1 reduced renal acid excretion in distal renal tubular acidosis (dRTA) may lead to nephrocalcinosis and

2 f the human ATP6V1B1 gene cause distal renal tubular acidosis (dRTA; OMIM #267300) often associated w

3 clinical diagnosis of inherited distal renal tubular acidosis has no identified causative mutations i

4 nset sensorineural deafness and distal renal tubular acidosis.

5 lyps (SSA/P), traditional serrated adenomas, tubular adenomas >/=10 mm or with high-grade dysplasia,

6 uals with low-risk adenomas (LRAs; 1-2 small tubular adenomas, < 1 cm) every 5-10 years for colorecta

7 nd bioaccessibility of zeaxanthin from these tubular aggregates in goji berries as compared to protei

8 tigate the role of MIOX in cisplatin-induced tubular AKI, we generated conditional MIOX-overexpressin

9 Acute tubular and glomerular renal injury was accompanied by n

10 standard for in vivo assessment of proximal tubular and loop of Henle sodium handling, to assess sod

11 Tubular and toroid shapes, among others, are transcribed

12 e essential roles in the regulation of renal tubular and vascular function.

13 f apoptosis contributes significantly to the tubular apoptosis and renal interstitial fibrosis in kid

14 f the films geometrically, we can achieve 3D tubular architectures with controllable diameters.

15 umbrella architectures associated with nano-tubular arrangements enabled to tailor NA biosensor desi

16 emonstrate the formation of RNA lattices and tubular assemblies from double crossover (DX) tiles, a c

17 ish an atomic resolution model of the RSV CA tubular assembly using molecular dynamics flexible fitti

18 damage, defined by interstitial fibrosis and tubular atrophy (IF/TA), is a leading cause of allograft

19 Interstitial fibrosis and tubular atrophy (IFTA) associated with interstitial infl

20 0.001) and presence of interstitial fibrosis/tubular atrophy (P=0.003) at diagnosis and changes in GF

21 Interstitial fibrosis (IF) and tubular atrophy (TA) was present in 7 (28%) of 25 ABOi c

22 Albuminuria and tubular atrophy are among the highest risks for CKD prog

23 al damage of which interstitial fibrosis and tubular atrophy are dominant features.

24 ot the pronephric ducts, consistent with the tubular atrophy observed in the affected individuals.

25 ified, what drives interstitial fibrosis and tubular atrophy progression in individual patients is of

26 omerulosclerosis, interstitial fibrosis, and tubular atrophy) all increase with age.

27 common changes such as glomerular sclerosis, tubular atrophy, and interstitial fibrosis.

28 lomerulosclerosis, interstitial fibrosis and tubular atrophy, and vascular disease; specimens with a

29 for some of them, near foci of fibrosis and tubular atrophy.

30 (FoxO3) in mediating injury-induced proximal tubular autophagy in mice with unilateral ureteral obstr

31 damage, IgG-positive immune deposits in the tubular basement membrane, and circulating antibodies re

32 peptide nanotubes rivals some of the largest tubular biomolecular assemblies, such as GroEL and micro

33 der that could use its helens to elevate its tubular body above the sea floor.

34 bone fragility, and diaphyseal sclerosis of tubular bones.

35 ditionally, the treatment strategy prevented tubular brush border loss, diminished tubular iron depos

36 s reactive with normal human kidney proximal tubular brush border.

37 for MFM-185a due to selective elongation of tubular cages.

38 At higher concentrations, tubular capture is saturated, and they reach the urine.

39 podocyte glycocalyx, together with saturable tubular capture, determines which macromolecules reach t

40 The anterograde and retrograde tubular carriers are both largely free of the classical

41 es while in parallel recycling receptors via tubular carriers back to the Golgi.

42 in the other domain exit into predominantly tubular carriers shared with plasma membrane proteins, i

43 crom), thin ( approximately 110 nm diameter) tubular carriers.

44 ncomycin aggregates represents a new mode of tubular cast formation, revealing the hitherto unsuspect

45 sy specimen, we ascertained that obstructive tubular casts composed of noncrystal nanospheric vancomy

46 metallocavitand structures with very defined tubular cavities and are able to selectively host linear

47 p2(+))3]) reveals accessible one-dimensional tubular cavities, and variable-temperature electron para

48 ion of epithelial cytokeratin 8.18, proximal tubular CD10, distal tubular cytokeratin 7, and endothel

49 Mif deletion also resulted in reduced tubular cell apoptosis after UUO.

50 hemic acute kidney injury through regulating tubular cell apoptosis and inflammation suggesting PTEN

51 d Bak from proximal tubules attenuated renal tubular cell apoptosis and suppressed renal interstitial

52 PTEN inhibition enhanced tubular cell apoptosis in kidneys with IRI, which was as

53 water and sodium reabsorption via increased tubular cell cAMP levels, we hypothesized the ET would a

54 In LPS-stimulated tubular cell cultures, Mif deletion led to enhanced G2/M

55 evulinic acid (ALA) accumulates and promotes tubular cell death and tubulointerstitial damage.

56 sed histologic injury, oxidative stress, and tubular cell death in this model.

57 y MANF excretion concurrent with podocyte or tubular cell ER stress preceded clinical or histologic m

58 ce C3b deposition on a mouse kidney proximal tubular cell line (TEC) and a human retinal pigment epit

59 rats (Han:SPRD Cy/+), demonstrating obvious tubular cell morphological abnormalities.

60 Furthermore, MIF inhibition reduced tubular cell proliferation in vitro In all three in vivo

61 e early stages of kidney repair and promotes tubular cell survival via IL-13 receptor alpha2 (IL13Ral

62 Immortalized rat renal proximal tubular cells (IRPTCs) and kidneys from humans with T2D

63 analysis revealed Pals1 expression in renal tubular cells and podocytes of human kidneys.

64 Microarrays of proximal tubular cells and podocytes with stable HIF1alpha and/or

65 may cause mitochondrial dysfunction in renal tubular cells and reprogramming of glucose metabolism.

66 endoplasmic reticulum (ER) stress in kidney tubular cells and the expression of RTN1A correlates wit

67 Renal proximal tubular cells constantly recycle nutrients to ensure min

68 ecrosis (necroptosis), which occurs in renal tubular cells during AKI.

69 stochemistry localized MAP3K14 expression to tubular cells in acute folate nephropathy and human AKI.

70 Renal MIF expression was reduced in tubular cells in fibrotic compared with healthy murine a

71 ects of kaempferol and esculetin using renal tubular cells in vitro and in vivo in a mouse Unilateral

72 Recombinant MIF exerted opposing effects on tubular cells in vitro and in vivo Our data identify ren

73 y cultures treated with cyclosporin A, renal tubular cells isolated from Nupr1-deficient mice exhibit

74 transporter 2 (PEPT2) expressed by proximal tubular cells mediates the reabsorption of ALA, and vari

75 Pi transport in primary cultures of proximal tubular cells or in freshly isolated renal tubules revea

76 itu We now show that EV from adult rat renal tubular cells significantly improved renal function when

77 HIF-2alpha activation in renal tubular cells upregulated mRNA and protein expressions o

78 Treatment of tubular cells with dasatinib reduced the expression of C

79 as concentrated along the apical membrane of tubular cells with ET but not PA, and urine aquaporin 2

80 As with tubular cells with HIF-2alpha activation, those under hy

81 cell cycle pathways was seen in murine renal tubular cells with NOTCH overexpression, and molecular s

82 sis and abolished proliferation in wild-type tubular cells, but only reduced proliferation in Nupr1-d

83 cally found in the brush borders of proximal tubular cells, has been detected in urine of patients wi

84 In cultured tubular cells, MAP3K14 small interfering RNA targeting d

85 In proximal tubular cells, mRNA levels of the amino acid transporter

86 nd attenuated the expression of cyclin B1 in tubular cells.

87 otection required both macrophages and renal tubular cells.

88 -1, RANTES, and CXCL10 as MAP3K14 targets in tubular cells.

89 ligand 8 (CXCL8)/CXCL1 expression by injured tubular cells.

90 rofibrotic effect in cultured renal proximal tubular cells.

91 tion of NF-kappaB in cultured renal proximal tubular cells.

92 albumin-induced profibrotic effects in renal tubular cells.

93 demonstrated high levels of CtsD in damaged tubular cells.

94 ork for nutrient transport in renal proximal tubular cells.

95 talyst consisting of immobilized rennin on a tubular cellulose/starch gel (TC/SG) composite, which ha

96 structure shows an unprecedented helicoidal tubular chain resulting from the periodic alternation of

97 e developed an in vitro model whereby molded tubular channels inside a synthetic hydrogel are seeded

98 ng-induced transformation of chloroplasts to tubular chromoplasts was accompanied by an accumulation

99 ssion depletion imaging shows that ARF1-rich tubular compartments fall into two distinct classes cont

100 from animal models that indicate that distal tubular compensatory sodium reabsorption is a primary dr

101 PDAC tissues showed that AGR2 was present in tubular complexes (TC) and early pancreatic intraepithel

102 s of CCRCCs and matched microdissected renal tubular controls revealed overexpression of NOTCH ligand

103 ality crystallinity, which outperforms their tubular counterparts, delivering a superior load-bearing

104 iral recognition strategy to a new family of tubular covalent cages to create both 1D porous nanotube

105 thesis that TNFRs also have a direct role in tubular crystal deposition and progression of hyperoxalu

106 MP signaling marks dedifferentiated proximal tubular cystic segments.

107 e embryonic kidney explants induced proximal tubular cystogenesis and p-Creb expression; these effect

108 okeratin 8.18, proximal tubular CD10, distal tubular cytokeratin 7, and endothelial von Willebrand fa

109 ury which exhibited severe renal failure and tubular damage at 24 hours.

110 ) exacerbated renal dysfunction and promoted tubular damage in mice with IRI compared with vehicle-tr

111 Acute tubular damage is a major cause of renal failure, especi

112 so lacked the intrarenal CaOx deposition and tubular damage observed in wild-type mice.

113 y, EV treatment significantly improved renal tubular damage, 4-hydroxynanoneal adduct formation, neut

114 17 or 12 MBq (213)Bi-IMP288 showed signs of tubular damage, indicating nephrotoxicity.

115 ant and, presumably, in other forms of acute tubular damage.

116 rmal stroma function is essential for normal tubular differentiation.

117 pse of juxtamedullary glomeruli, microcystic tubular dilation, and tubulointerstitial fibrosis.

118 n 43+/- mice showed less crescent formation, tubular dilation, monocyte infiltration, and interstitia

119 = 5.8; 95% CI = 3.7-9.0), and proximal renal tubular dysfunction (aOR = 7.0; 95% CI = 4.9-10.2]).

120 initially affected with generalized proximal tubular dysfunction (renal Fanconi syndrome), then the d

121 nine ratio >/=3 mg/mmol), and proximal renal tubular dysfunction (retinol-binding protein/creatinine

122 owing cystine accumulation and late signs of tubular dysfunction but lacking the glomerular phenotype

123 renal tubule exhibited generalized proximal tubular dysfunction indicative of Fanconi syndrome, char

124 ment, and signs of pronephric glomerular and tubular dysfunction mimicking the early phenotype of hum

125 impairment, albuminuria, and proximal renal tubular dysfunction.

126 Its ear canal has a fully ossified tubular ectotympanic, a derived feature linking the spec

127 indicate that SGLT2 inhibitor elicits direct tubular effects in non-diabetic rats with normal renal f

128 ons involving increased GFR and vascular and tubular effects.

129 nd potassium homeostasis by regulating renal tubular electrolyte transport.

130 ree-dimensional architecture with thin-shell tubular elements, resulting in favorable modulus-density

131 Axons contain a smooth tubular endoplasmic reticulum (ER) network that is thoug

132 he plasma membrane (PM), contain a strand of tubular endoplasmic reticulum (ER), and the space betwee

133 SNX6 and serve to regulate the biogenesis of tubular endosomal sorting profiles.

134 on of RidL with retromer-coated vacuolar and tubular endosomes.

135 Overexpression of the proximal tubular enzyme myo-inositol oxygenase (MIOX) induces oxi

136 PA) is expressed in glomerular podocytes and tubular epithelia and metabolizes angiotensin II (AngII)

137 stin-1 (PC1) or polycystin-2 (PC2), in which tubular epithelia form fluid-filled cysts.

138 reated with unilateral renal IRI, persistent tubular epithelial cell damage was determined in the IRI

139 otein 13 (TRIP13) is a critical modulator of tubular epithelial cell repair following ischemia-reperf

140 Ca(2+) ion channels, respectively, result in tubular epithelial cell-derived renal cysts.

141 In cultured human kidney tubular epithelial cells (HK-2), TGF-beta1 treatment ind

142 an hepatic epithelial cells (L-02) and human tubular epithelial cells (HK-2).

143 Impaired albumin reabsorption by proximal tubular epithelial cells (PTECs) has been highlighted in

144 Previous studies revealed that tubular epithelial cells (TEC) show a limited response t

145 wever, Mif gene deletion restricted to renal tubular epithelial cells aggravated these effects.

146 nuria caused a decrease in the proportion of tubular epithelial cells and an increase in the proporti

147 CYP3A5 protein expression was detected in tubular epithelial cells and inflammatory cells within t

148 Damage to renal tubular epithelial cells by genetic, environmental, or b

149 However, podocytes and renal tubular epithelial cells do not express CD4 receptors, a

150 Studies have shown that podocytes and renal tubular epithelial cells from patients with HIV-associat

151 adhesion molecules, CD44 and annexin II, in tubular epithelial cells in vitro and in vivo, and treat

152 reased expression of IL-36alpha in the renal tubular epithelial cells of a mouse model of unilateral

153 be overexpressed in the proximal and distal tubular epithelial cells of murine and human kidneys aft

154 a low level of regenerative competence, the tubular epithelial cells of the nephrons can proliferate

155 VTEA enabled us to discover a population of tubular epithelial cells that expresses CD11C, a marker

156 IP13 increased the susceptibility of damaged tubular epithelial cells to progress towards apoptotic c

157 TAM expression and shedding by tubular epithelial cells were investigated by PCR and en

158 ansmembrane TNF-alpha in cultured CD4- renal tubular epithelial cells, 293T cells, and HeLa cells ena

159 VHL led to dysplastic hyperproliferation of tubular epithelial cells, confirming the procarcinogenic

160 olves numerous different cell types, such as tubular epithelial cells, endothelial cells, and podocyt

161 tigen was detected in less than 5% of VSMCs, tubular epithelial cells, interstitial endothelium, inte

162 tated NLRP3 inflammasome activation in renal tubular epithelial cells, macrophages, and dendritic cel

163 either globally or conditionally in proximal tubular epithelial cells, protected mice from the develo

164 nd Hmox1 in centrilobular hepatocytes and in tubular epithelial cells, respectively.

165 In human proximal tubular epithelial cells, stimulation by fluvoxamine or

166 ssion and activity at 24 h in renal proximal tubular epithelial cells, which was inhibited by sodium

167 of vascular smooth muscle cells (VSMCs) and tubular epithelial cells, with a median positivity of 20

168 ng on glomerular endothelial cells and renal tubular epithelial cells.

169 A induced Nupr1 expression in cultured human tubular epithelial cells.

170 ns target host reticulon 4 (Rtn4) to control tubular ER dynamics, resulting in tubule rearrangements

171 emical pathway to control ubiquitination and tubular ER function independently of the host ubiquitin

172 nce (defined as intestinal metaplasia in the tubular esophagus) and dysplastic BE recurrence among pa

173 albuminuric NZB/W mice, indicating enhanced tubular exposure and potential for enhanced tubular upta

174 Mice lacking distal tubular expression of CLDN10, the gene encoding the tigh

175 ohistochemical analysis revealed upregulated tubular expression of TNFR1 and TNFR2 in human and murin

176 lioration of hypoxia through increased renal tubular expression of VEGF and its isoforms.

177 somes, which then send the enzyme to LDs via tubular extensions.

178 , focusing on ultrastructural, vascular, and tubular factors.

179 osine kinase-2 (SPHK2) on the progression of tubular fibrosis by using a mouse unilateral ureteral ob

180 ional arrays (i.e., crystals), and linear or tubular filaments.

181 r zebrafish pronephric podocyte and proximal tubular function and that the ctns-mutant can be used fo

182 les, continuity of the lumen is paramount to tubular function, yet how tubules generate lumen continu

183 y that these miRNAs could modulate key renal tubular functions in a paracrine manner.

184 (SGLT2) inhibitor, on renal hemodynamics and tubular functions in anesthetized non-diabetic Sprague D

185 The tubular-glomerular feedback response and/or direct effec

186 mplicate a series of genes involved in renal tubular handling of lithogenic substrates, such as calci

187 In conclusion, late-stage renal tubular HIF-2alpha activation has protective effects on

188 nct sulfide semiconductors into hierarchical tubular hybrids with homogeneous interfacial contacts an

189 ointerstitial histopathology and the role of tubular hypoxia in the pathogenesis of chronic kidney di

190 RP2 specifically colocalized with IgG in the tubular immune deposits on the ABBA biopsy specimen but

191 the interstitium and circulation, to inhibit tubular inflammatory signaling.

192 congestion and improved histologic scores of tubular injury 4 days after IRI.

193 In wild-type mice, failure to resolve tubular injury after unilateral ischemia-reperfusion inj

194 up had significantly reduced scores of acute tubular injury and acute tubular necrosis.

195 urea, creatinine, and KIM-1 levels and more tubular injury and apoptosis, but these effects were att

196 Histological evidence of tubular injury and cell death was minimal.

197 AKI leads to tubular injury and interstitial inflammation that must b

198 ggest that YKL-40 is produced in response to tubular injury and is independently associated with reco

199 cyte loss is sufficient to trigger transient tubular injury and permanent peritubular capillary raref

200 prevented the increased excretion of urinary tubular injury biomarkers.

201 Furthermore, tubular injury in kidneys subjected to bilateral renal i

202 ssues showed a significant decrease of acute tubular injury in the CD47mAb-treated group compared to

203 nal histologic changes (mesangial expansion, tubular injury, and fibrosis) over time.

204 ause vascular and renal calcification, renal tubular injury, and premature death in multiple animal m

205 logy showed severe damage (thrombosis, acute tubular injury, capillaritis) and infiltration of many S

206 miR-146a(-/-) mice exhibited more extensive tubular injury, inflammatory infiltrates, and fibrosis t

207 e mice, IRAK-M-deficient mice showed reduced tubular injury, leukocyte infiltration, and inflammation

208 on also led to hypoxic focal and subclinical tubular injury, reflected by transient expression of Kim

209 tients displayed lambda-restriction or acute tubular injury.

210 ws potential as an early marker for proximal tubular injury/necrosis and warrants further investigati

211 tion, and remote organ injury and maintained tubular integrity after renal I/R injury.

212 the cell frequently includes deep, branching tubular invaginations that form a dynamic nucleoplasmic

213 The 1D tubular ionic polymer observed in the single crystals sh

214 vented tubular brush border loss, diminished tubular iron deposition, blocked the development of inte

215 salt treatment significantly increased renal tubular lesions from day 2 and mRNA expression of fibros

216 , Nupr1-deficient mice exhibited worse renal tubular lesions than wild-type mice.

217 nanotubes (CNTs), being pre-dispersed into a tubular level of dispersions, were used as the starting

218 Understanding the tubular location of diuretic resistance (DR) in heart fa

219 and its immediately adjacent tissue, in the tubular lumina of the epididymides, and in foci of histi

220 nionless and Dab2, which are partners in the tubular machinery.

221 In particular, nephron formation, tubular maturation, and the differentiation of smooth mu

222 Herein, a tubular MDC was operated under a wide range of salt conc

223 reported in dynamin-independent scission of tubular membrane necks, the cutting mechanism has yet to

224 Here we reconstitute a dynamic tubular membrane network with purified endoplasmic retic

225 Both domains are connected by narrow, tubular membrane segments called cristae junctions (CJs)

226 e surface sarcolemma, but also on transverse-tubular membranes in ventricular myocardium.

227 t both at surface-sarcolemmal and transverse-tubular membranes.

228 ents revealing how protein scaffolds may cut tubular membranes.

229 xchange nutrients and macromolecules through tubular membranous structures called nanotubes.

230 Rolled-up diamond tubular microcavities exhibit pronounced defect-related

231 -prepared rGO/platinum nanoparticles (PtNPs) tubular micromotors were synthesized rapidly and inexpen

232 in vitro and in vivo Our data identify renal tubular MIF as an endogenous renoprotective factor in pr

233 d levels of cardiolipin were associated with tubular mitochondria and enhanced oxidative phosphorylat

234 direct visualization of exciton diffusion in tubular molecular aggregates by transient absorption mic

235 ffusion constants of 3-6 cm(2) s(-1) for the tubular molecular aggregates, which are 3-5 times higher

236 Mathematical modelling predicted that tubular Na(+) reabsorption decreased in the proximal tub

237 Mathematical modelling predicted that tubular Na(+) reabsorption increased in the proximal tub

238 kinase (AMPK) was related to the changes in tubular Na(+) reabsorption.

239 take on glomerular filtration rate (GFR) and tubular Na(+) reabsorption.

240 The open tubular nature of the MCRs allowed for repeatable within

241 Renal transplant biopsies with acute tubular necrosis demonstrated high levels of CtsD in dam

242 sted in eight additional patients with acute tubular necrosis in the absence of hypovolemia.

243 ary YKL-40 concentration (P<0.001) and acute tubular necrosis on procurement biopsies (P=0.05).

244 phase of IRI had no impact on organ atrophy, tubular necrosis, or fibrosis.

245 sic graft failure comprised rejection, acute tubular necrosis, urinary tract infection/pyelonephritis

246 ced scores of acute tubular injury and acute tubular necrosis.

247 hookeri and removed nectar from their unique tubular nectary extensions.

248 ed with control littermates, inducible renal tubular NEDD4-2 knockout (Nedd4L(Pax8/LC1) ) mice exhibi

249 n Yop1p from Saccharomyces cerevisiae form a tubular network upon addition of GTP.

250 roanatomy, the tuft cells' cytospinules, and tubular network, might facilitate the exchange of molecu

251 dietary Na(+) intake and induces changes in tubular O2 consumption and sodium transport efficiency.

252 Tubular O2 consumption and the efficiency of sodium reab

253 The tear-producing lacrimal gland is a tubular organ that protects and lubricates the ocular su

254 s in vitro However, the relevance of MIOX to tubular pathobiology remains enigmatic.

255 tosis of filamentous bacteria occurs through tubular phagocytic cups (tPCs) and takes many minutes to

256 e cortex and papilla and display an immature tubular phenotype.

257 ngomyelin balance was shown to induce narrow tubular plasma membrane invaginations enriched with sphi

258 ilomicelles and vesicular, multilamellar and tubular polymersomes from poly(ethylene glycol)-bl-poly(

259 d p-type silicon wafers can be prepared with tubular pores imbedded in a silicon matrix.

260 )-azolate metal-organic framework (MOF) with tubular pores undergoes a reversible single crystal to s

261 low molecular weight proteins, suggestive of tubular production.

262 ating the CI-MPR dependency of SNX1/2-SNX5/6 tubular profile formation, we provide a mechanism for co

263 ent cargo recognition with the biogenesis of tubular profiles required for endosome-to-TGN transport.

264 Tail tubular protein A (TTPA) is a structural tail protein of

265 zed a high molecular weight protein in renal tubular protein extracts that we identified as LDL recep

266 s repository and tested for seven markers of tubular proteinuria.

267 The latter mode is supported by thin tubular protrusions, called nanotunnels, that contact ot

268 at changes in glomerular permselectivity and tubular reabsorption account, at least in part, for the

269 actor 23 (FGF23) axis, creatinine, and renal tubular reabsorption of phosphate (TRP).

270 We find that acute and pathological tubular remodeling significantly affect TATS electrical

271 ago, it was proposed that the regulation of tubular repair in the kidney might involve the recapitul

272 that miR-146a is a key mediator of the renal tubular response to IRI that limits the consequences of

273 ch is responsible for the majority of sodium tubular reuptake following filtration.

274 Collecting ducts make up the distal-most tubular segments of the kidney, extending from the corte

275 Salt-losing tubulopathies can affect all tubular segments, from the proximal tubule to the collec

276 To accomplish the task, a tubular shaped potentiometric sensor selective to perchl

277 structed a number of DNPs of rectangular and tubular shapes with varied dimensions using the modular

278 t suggest that calcineurin inhibitor-induced tubular SNAI1 protein cytoplasmic accumulation, possibly

279 retics develop DR due to compensatory distal tubular sodium reabsorption, but whether this translates

280 Distal tubular sodium retention is a potent driver of hypertens

281 as potentially microtubules alignment, inter-tubular spacing, and, by extension, axonal transport.

282 Additionally, the tubular structure causes a self-enhancing effect in cond

283 ls reorganization of the genome, growth of a tubular structure from a portal vertex and release of th

284 The single tubular structure of sweat glands has a lower secretory

285 objects apart through a burst of their rigid tubular structure.

286 ny legumes, bacterial uptake is mediated via tubular structures called infection threads (ITs).

287 Tubular structures can exceed 1 mum in length, suggestin

288 mplex involved in the formation of endosomal tubular structures that mediates the sorting of protein

289 tes that Troy(+) cells clonally give rise to tubular structures that persist for up to 2 y after indu

290 current approaches is the printing of hollow tubular structures.

291 rts of the brain without forming endothelial tubular structures.

292 letion and disorganization of the transverse tubular system (t-system) in cardiomyocytes.

293 l invaginations, called the transverse-axial tubular system (TATS), propagates membrane potential cha

294 the membrane network inside the fibres, the tubular (t-) system, causing the loss of its predominant

295 e networks, built from interconnected hollow tubular tetrapods of multilayer graphene, are ultra-ligh

296 A tubular thermal exchanger was used for a rapid cooling t

297 Because of the central nature of sodium in tubular transport physiology, disorders of sodium handli

298 sTyro3 and sMer was associated with loss of tubular Tyro3 and Mer expression in diabetic nephropathy

299 tubular exposure and potential for enhanced tubular uptake following filtration.

300 diagnosis of invasive mammillary carcinoma, tubular variant, strongly positive for estrogen and prog

301 abundant electroactive sites in the interior tubular vessels and outer surfaces for ultrasensitive de