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1 sulphated HS remained predominant within the tubular basement membrane.
2 talloproteinase-2 that specifically degraded tubular basement membrane.
3 sence of positively stained cells within the tubular basement membrane.
4 an extracellular matrix protein expressed in tubular basement membranes.
5 and/or heavy chains along the glomerular and tubular basement membranes.
6 matrix protein and is expressed in the renal tubular basement membranes.
7 y showed deposits in the glomeruli and along tubular basement membranes.
8 les, peritubular capillaries, glomeruli, and tubular basement membranes.
10 , glomerular basement membranes (GBM) (42%), tubular basement membranes (85%), and vessel walls (96%)
11 MMP-2 leads to structural alterations in the tubular basement membrane, a process that triggers tubul
12 that epithelial cells migrate outside of the tubular basement membrane and differentiate into interst
13 iated with the ability of E. coli to bind to tubular basement membranes and Bowman's capsule and to b
14 matrix and thickening of the glomerular and tubular basement membranes and the number of dividing ce
15 n and actin reorganization, 3) disruption of tubular basement membrane, and 4) enhanced cell migratio
16 damage, IgG-positive immune deposits in the tubular basement membrane, and circulating antibodies re
17 direct epithelial injury, accumulate in the tubular basement membrane, and elicit an interstitial in
18 ies stained positive for C5b-9 in glomeruli, tubular basement membranes, and vessel walls, albeit at
19 and actin reorganization; (3) disruption of tubular basement membrane; and (4) enhanced cell migrati
20 s including beta1 chain in the mesangium and tubular basement membranes at 1, 2, 3, and 4 mo of diabe
21 mal kidney, HS was largely restricted to the tubular basement membrane; chondroitin-4-sulphate and ch
22 All pretreatment biopsies had glomerular and tubular basement membrane deposits that stained exclusiv
24 IgG and C3 deposits were conspicuous in the tubular basement membrane of proximal tubules, correspon
25 the dra region, affecting E. coli binding to tubular basement membranes, prevented renal interstitial
26 ulointerstitial nephritis antigen (TINag), a tubular basement membrane protein, in the UUO model of t
28 lar frequency in cases and controls, whereas tubular basement membrane staining was less frequently o
29 stitial space, tubular epithelial cells, the tubular basement membrane (TBM), and vascular structures
31 d quantitative changes in the composition of tubular basement membranes (TBMs) and interstitial matri
33 n alpha5 chain content of the glomerular and tubular basement membranes was increased, with marked ex
35 tein-positive cells were confined within the tubular basement membrane, were not found in the renal i
36 estricted distribution to the glomerular and tubular basement membranes with scant expression in the
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