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1 ly been identified in primary cilia in renal tubular epithelia.
2 thelial cells, is produced by glomerular and tubular epithelia.
3 n, capillary formation, and proliferation of tubular epithelia.
4 in conferring apical character in Drosophila tubular epithelia.
5 n both the MDCK cyst model and in Drosophila tubular epithelia.
6 berrantly increased AGS3 expression in renal tubular epithelia affected by PKD and epithelial cell pr
7 t postischemic NF-kappaB activation in renal tubular epithelia aggravates tubular injury and exacerba
8 ced widespread NF-kappaB activation in renal tubular epithelia and in interstitial cells that peaked
9 PA) is expressed in glomerular podocytes and tubular epithelia and metabolizes angiotensin II (AngII)
10 Tip47-positive lipid droplets in glomeruli, tubular epithelia, and macrophages was accompanied by th
14 ogical studies localized polycystin to renal tubular epithelia, hepatic bile ductules, and pancreatic
15 In vivo, ILK was markedly induced in renal tubular epithelia in mouse models of chronic renal disea
17 apical surfaces of epidermal cells and some tubular epithelia, including the excretory duct and pore
19 d immature glomeruli, and the renal proximal tubular epithelia lacked proper localization of adhesion
21 intensity of immunostaining was detected in tubular epithelia of the proximal tubule, thick ascendin
22 with tissue-specific deletion of Adora2b in tubular epithelia or vascular endothelia implicated endo
23 ad involution, and broaden and shorten other tubular epithelia (salivary glands, tracheae, and hindgu
24 mammalian airways are branching networks of tubular epithelia that deliver oxygen to the organism.
26 d into a developing kidney and contribute to tubular epithelia, the ability to generate renal precurs
27 that the alpha8 integrin chain is induced in tubular epithelia undergoing dedifferentiation and contr
28 ell line, glomerulogenesis was abolished but tubular epithelia were expanded (0 glomeruli, 47 tubules
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