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1 asma concentrations as a result of saturable tubular reabsorption.
2 at changes in glomerular permselectivity and tubular reabsorption account, at least in part, for the
3 megalin and cubilin, mediates renal proximal-tubular reabsorption and is decreased in Dent disease be
4 dly increase renal excretion or reduce renal tubular reabsorption and thus blunt large increases in t
5 ntact by glomerular filtration, with partial tubular reabsorption and transient translocation into th
6 lation of megalin, a protein involved in the tubular reabsorption of albumin and lipid-bound proteins
7 sease indicators were the best predictors of tubular reabsorption of beta2-microglobulin (R2 = 0.53)
8 n of beta2-microglobulin (R2 = 0.53) whereas tubular reabsorption of beta2-microglobulin and RBP were
14 n with more severe disease also had impaired tubular reabsorption of low-molecular-weight proteins be
16 e exhibited physiologic functions, including tubular reabsorption of macromolecules, that gained acce
18 Angiotensin II (ANG II) stimulates renal tubular reabsorption of NaCl by targeting Na(+)/H(+) exc
19 l [95% CI], 0.59 to 0.96; P=0.02), decreased tubular reabsorption of phosphate (OR, 0.41; 95% CI, 0.2
20 ffect of vitamin D (VITD) supplementation on tubular reabsorption of phosphate (TRP), parathyroid hor
22 ls mineral metabolism by directly modulating tubular reabsorption of phosphate and calcium and by act
23 r 23, a hormone that inhibits proximal renal tubular reabsorption of phosphate and down-regulates ren
24 tration, plasma phosphate concentration, and tubular reabsorption of phosphate increased during the p
25 evaluating the fractional sodium excretion, tubular reabsorption of phosphate, and gamma-glutamyltra
26 tamin B(12)-intrinsic factor complex and the tubular reabsorption of protein in the proximal tubule.
27 n II (AngII) helps to regulate overall renal tubular reabsorption of salt and water, yet its effects
28 ion were associated with decreases in distal tubular reabsorption of sodium despite a mild decrease i
29 droxycortisol by CYP3A may mediate increased tubular reabsorption of sodium, leading to a state of mi
30 e studies examine the role of NHERF-1 in the tubular reabsorption of uric acid and regulation of mous
31 F-1 exerts a significant effect on the renal tubular reabsorption of uric acid in the mouse by modula
33 to the treatment of hyponatremia by blocking tubular reabsorption of water by binding to V2 receptors
34 The multiple effects of AngII to increase tubular reabsorption provide a powerful mechanism to pro
37 Conceptually, modest inhibition of renal tubular reabsorption should provide effective relief for
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