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1 xic acid is at least partially eliminated by tubular secretion.
2 h protein-binding affinity and dependence on tubular secretion.
3 ds to duplicate the efficacy of clearance by tubular secretion.
4 distribution of loop diuretics, reduce their tubular secretion, and enhance the inactivation of furos
8 on-GFR factors (e.g., creatinine production, tubular secretion) conservatively biased associations wi
10 rter (hOAT1) is a key component in the renal tubular secretion of negatively charged molecules includ
12 d 2j remained unchanged upon blockage of the tubular secretion pathway with probenecid, a necessary c
15 Tc-99m MAG3 is cleared from the kidney by tubular secretion, whereas Tc-99m DTPA is cleared by glo
16 ce rates of four solutes normally cleared by tubular secretion with the plasma concentrations and cle
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