ライフサイエンスコーパス: tubuloglomerular feedback

1 he GFR supranormal through the physiology of tubuloglomerular feedback.

2 whereas glomeruli respond to tubules through tubuloglomerular feedback.

3 absorption of NaCl in the TALH, and impaired tubuloglomerular feedback.

4 macula densa, providing the error signal for tubuloglomerular feedback.

5 omerular hemodynamics, including the role of tubuloglomerular feedback.

6 Furthermore, we found that tubuloglomerular feedback, a mechanism that links proxim

7 On the other hand, tubuloglomerular feedback activity is often directly pro

8 tion via its effects on renal arterioles and tubuloglomerular feedback, but effects of adenosine bloc

9 flow past the macula densa, thus minimizing tubuloglomerular feedback-dependent influences on affere

10 Tubuloglomerular feedback function curve as determined b

11 omerular feedback responses were normal, but tubuloglomerular feedback function curves were right-shi

12 rular apparatus was still able to respond to tubuloglomerular feedback in isolated perfused juxtaglom

13 s, we studied NaCl-dependent GFR regulation (tubuloglomerular feedback) in mice with targeted deletio

14 Although tubuloglomerular feedback is normal in these animals, th

15 This tubuloglomerular feedback mechanism plays an important r

16 consisting of the myogenic response and the tubuloglomerular feedback mechanism, the myogenic respon

17 tribute to the enhanced basal vascular tone, tubuloglomerular feedback, monocyte/macrophage infiltrat

18 Activation of tubuloglomerular feedback, previously thought to contrib

19 NKCC2 may permit transport and Cl-dependent tubuloglomerular feedback regulation to occur over a wid

20 tubule, consistent with a role of MD nNOS in tubuloglomerular feedback resetting.

21 rms (MD-NOS1KO) had a significantly enhanced tubuloglomerular feedback response and after acute volum

22 lt sensitivity of BP and the significance of tubuloglomerular feedback response in long-term control

23 The tubuloglomerular feedback response, the change in affere

24 ressed in the macula densa and regulates the tubuloglomerular feedback response, the natriuretic resp

25 nsin-aldosterone system (RAAS) and the renal tubuloglomerular feedback response.

26 Angiotensin II (AngII) enhances tubuloglomerular feedback responses and is considered to

27 Furthermore, whereas tubuloglomerular feedback responses did not change signi

28 AT1 receptor blockade by candesartan reduced tubuloglomerular feedback responses to a flow rate step

29 sence of NKCC2A in the macula densa, maximum tubuloglomerular feedback responses were normal, but tub

30 nd salt-sensitive hypertension by decreasing tubuloglomerular feedback responsiveness.

31 e (NO) is an important negative modulator of tubuloglomerular feedback responsiveness.

32 e via AT1 receptors and therefore may affect tubuloglomerular feedback signal transmission, at least

33 vide compelling new evidence indicating that tubuloglomerular feedback signals are coupled to autoreg

34 receptors by candesartan on the exaggerated tubuloglomerular feedback (TGF) activity in 7-wk-old, eu

35 total autoregulatory efficiency, reflecting tubuloglomerular feedback (TGF) and possibly one or two

36 Initiation of tubuloglomerular feedback (TGF) depends on Na-K-2Cl co-t

37 Tubuloglomerular feedback (TGF) describes the negative r

38 densa nitric oxide generation and actions on tubuloglomerular feedback (TGF) during salt restriction.

39 he pathogenesis of diabetic nephropathy, and tubuloglomerular feedback (TGF) has been suggested to pl

40 ngement suggested that a marked reduction in tubuloglomerular feedback (TGF) in Cx40-ko mice was resp

41 studies demonstrated that stimulation of the tubuloglomerular feedback (TGF) mechanism by increasing

42 ion, and failure to regulate GFR through the tubuloglomerular feedback (TGF) mechanism.

43 Direct measurements of maximal tubuloglomerular feedback (TGF) responses were made from

44 Tubuloglomerular feedback (TGF) stabilizes nephron funct

45 The tubuloglomerular feedback (TGF) system was used as a too

46 after an overshoot at 40 s (second response, tubuloglomerular feedback (TGF)).

47 -transporter may be a site for regulation of tubuloglomerular feedback (TGF), and recently angiotensi

48 terioles has been suggested to contribute to tubuloglomerular feedback (TGF), the vasoconstriction el

49 stments in GFR through the normal actions of tubuloglomerular feedback (TGF).

50 al reabsorption which reduces the signal for tubuloglomerular feedback (TGF).

51 Nephron function is stabilized by tubuloglomerular feedback (TGF).

52 n tubules and afferent arterioles (so-called tubuloglomerular feedback [TGF]).

53 proximal tubular natriuresis activates renal tubuloglomerular feedback through increased macula densa

54 the TALH was reduced but not eliminated, and tubuloglomerular feedback was severely impaired.

55 Tubuloglomerular feedback, which is an angiotensin II-de