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1 and the hamsters that survived showed severe tubulointerstitial nephritis.
2 some forms of immunologically mediated human tubulointerstitial nephritis.
3 nt of the changes characteristically seen in tubulointerstitial nephritis.
4 polyendocrine syndrome type 1 who developed tubulointerstitial nephritis and ESRD in association wit
5 ains, develop ESRD associated with prominent tubulointerstitial nephritis and fibrosis within 3 month
6 ic interstitial nephropathy characterized by tubulointerstitial nephritis and formation of enlarged n
7 ed glomerular filtration rate (GFR), chronic tubulointerstitial nephritis and ultrastructural changes
11 467 aa was 46% identical with that of human tubulointerstitial nephritis antigen (TIN-ag), and there
13 he pathogenesis of leukocyte infiltration in tubulointerstitial nephritis associated with glomerular
14 s of monocyte/macrophage infiltration in the tubulointerstitial nephritis associated with PAN nephros
15 athy is characterized by rapidly progressive tubulointerstitial nephritis culminating in end-stage re
18 from infantile polycystic kidneys to chronic tubulointerstitial nephritis, fibrosis, and cortical mic
20 ficant histological changes corresponding to tubulointerstitial nephritis including interstitial infl
21 trogen levels, more severe histologic GN and tubulointerstitial nephritis, increased glomerular cresc
23 phronophthisis (NPH), an autosomal-recessive tubulointerstitial nephritis, is the most common cause o
24 a 36-year-old man with AIDS showed a severe tubulointerstitial nephritis with intranuclear inclusion
25 for immune and genetic causes of microcystic tubulointerstitial nephritis with little attention to it
26 ted animals showed renal dysfunction and had tubulointerstitial nephritis with nuclear inclusions, ap
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