ライフサイエンスコーパス: tuft

1 tive activity in the distal apical dendritic tuft.

2 occurrence of bleeding from an iris vascular tuft.

3 ruitment of labeled PECs onto the glomerular tuft.

4 can tune spatio-temporal integration in the tuft.

5 neration of NMDA spikes in the distal apical tuft.

6 lus-evoked synaptic input onto the dendritic tuft.

7 tial contribution on output signaling by the tuft.

8 process invasion of the glomerular capillary tuft.

9 y form cellular adhesions with the capillary tuft.

10 veral types of neurons as well as the apical tuft.

11 for enrichment of ion channels in dendritic tufts.

12 h of the two ears through separate dendritic tufts.

13 led PECs exclusively invaded segments of the tuft affected by glomerulosclerosis, consistent with our

14 arians runs from the oral pole to the apical tuft and defines the major body axis of both the planula

15 CPI203, exhibit greater than 90% decrease in tuft and enteroendocrine cells in both crypts and villi

16 domains defines a novel pathway required for tuft and enteroendocrine differentiation and provides an

17 pt progenitors to promote differentiation of tuft and goblet cells, leading to increased frequencies

18 Reduced proliferation in glomerular tuft and increased apoptosis in perivascular mesenchyme

19 ]i resulted in contraction of the glomerular tuft and increased capillary albumin permeability.

20 he generation and spread of apical dendritic tuft and trunk regenerative activity.

21 Simultaneous apical dendritic tuft and trunk whole-cell current-clamp recordings revea

22 ub-bands have been hypothesized to represent tufted and mitral cell networks, respectively.

23 ll classes in layers 2/3, 4, and the slender-tufted and thick-tufted pyramidal cells of layer 5 using

24 nic vascular processes including neovascular tufts and blind-ended capillary sprouts.

25 ion mainly occupy layer 5B, do not display a tuft, and exhibit regular action potential firing and li

26 age of excitatory inputs mediated by mitral, tufted, and external tufted cells, and, in turn, they in

27 hared with those of infantile hemangioma and tufted angioma of children, but features of the clinical

28 mall vessels within the dermis, resembling a tufted angioma.

29 ge; other vascular tumors include congenital tufted angiomas (TAs), kaposiform hemangioendotheliomas

30 Synapses onto distal dendritic tufts are believed to function by modulating time-locked

31 Iris vascular tufts are rare iris stromal vascular hamartomas.

32 Transient, spindle-like "REM beta tufts" are described in the EEG of healthy subjects, whi

33 process invasion of the glomerular capillary tuft as an initiation mechanism of Alport glomerular pat

34 phthalmologists to be aware of iris vascular tufts as a cause for spontaneous hyphaema, independent o

35 thy mice, about two-thirds of the glomerular tufts became LacZ positive during the regenerative phase

36 dendritic Ca(2+) spikes on different apical tuft branches of individual layer V pyramidal neurons in

37 nal Institute on Drug Abuse and the Lifespan/Tufts/Brown Center for AIDS Research from the National I

38 summary, detection of PECs on the glomerular tuft by immunostaining improves the differentiation betw

39 n conclusion, repopulation of the glomerular tuft by parietal cells may represent a compensatory resp

40 li in murine kidneys and sized the capillary tufts by combining in vivo fluorescence labeling of endo

41 neurons, with prominent h-current, and thin-tufted, callosally projecting type B neurons, which lack

42 we ask whether a non-human primate species, tufted capuchin monkeys (Cebus apella), socially evaluat

43 an anointing resource given to two groups of tufted capuchins, we tested predictions derived from the

44 Volume rendering also revealed within the tuft cell an elegant network of interconnected tubules.

45 metaplasias from the mice were analyzed for tuft cell and biliary progenitor markers, including SOX1

46 ine interleukin-25, which indirectly induces tuft cell expansion by promoting interleukin-13 producti

47 These cytospinules project from the tuft cell into the nuclei of neighboring epithelial cell

48 The tuft cell is a chemosensory cell that responds to signal

49 ing is sufficient to induce expansion of the tuft cell lineage, and ectopic stimulation of this signa

50 nase 1 protein (DCLK1) is a gastrointestinal tuft cell marker that has been proposed to identify quie

51 ow-level EE gene expression but co-expressed tuft cell markers, Lgr5 and Ascl2, reminiscent of label-

52 Thus, our data define an intestinal DCLK1(+) tuft cell population that is long lived, quiescent, and

53 e find that the Pou2f3 gene is essential for tuft cell specification.

54 ld be derived from a complete account of the tuft cell ultrastructure.

55 s, and distinguished between two subtypes of tuft cell, one of which expresses the epithelial cytokin

56 ve distinct cellular lineages, including the tuft cell, whose function is unclear.

57 ibuted to the differences between mitral and tufted cell activity.

58 or neuron nerve terminals (input) and mitral/tufted cell apical dendrites (output).

59 stricted to the soma and proximal portion of tufted cell apical dendrites.

60 ns in naive animals leads to an expansion of tufted cell axons that is identical to the changes cause

61 forebrain, which led to a mixture of mitral/tufted cell excitation and suppression.

62 ly, bulbar cholinergic enhancement of mitral/tufted cell odorant responses was robust and occurred in

63 Cholinergic stimulation increased mitral/tufted cell spiking in the absence of inhalation-driven

64 ptic processing at the reciprocal mitral and tufted cell-granule cell microcircuit, the most abundant

65 liable, short-latency firing consistent with tufted cell-mediated excitation.

66 After helminth infection, tuft-cell-derived IL-25 further activates ILC2s to secre

67 al a novel function of intestinal epithelial tuft cells and demonstrate a cellular relay required for

68 es (enterocytes, goblet cells, Paneth cells, tuft cells and enteroendocrine cells), presence of funct

69 Pou2f3(-/-) mice lack intestinal tuft cells and have defective mucosal type 2 responses t

70 estinal organoids were stimulated with IL-4, tuft cells and IL-25 were induced in both WT and Raptor

71 We find that Dclk1(+) tuft cells and nerves are the main sources of acetylchol

72 After massive small bowel resection, tuft cells and Tm were diminished due to the diet used p

73 Dclk1 is a marker of differentiated Tuft cells and, when coexpressed with Lgr5, also marks i

74 Commonly found in the biliary tract, tuft cells are absent from normal murine pancreas.

75 Once referred to as "peculiar," tuft cells are enigmatic epithelial cells.

76 DCLK1(+) tuft cells are increased in inflammation-induced carcino

77 Intestinal tuft cells are one of 4 secretory cell linages in the sm

78 Tuft cells are the primary source of the parasite-induce

79 Using the aberrant appearance of tuft cells as an indicator, we tested if pancreatic meta

80 Our results identify intestinal tuft cells as critical sentinels in the gut epithelium t

81 Here we show that tuft cells constitutively express IL-25 to sustain ILC2

82 tic ablation of Dclk1 revealed that DCLK1(+) tuft cells contribute to recovery following intestinal a

83 Tuft cells express a number of tumorigenic factors that

84 Tuft cells have a Th2-related gene expression signature

85 eatic tissue arrays revealed the presence of tuft cells in metaplasia and early-stage tumors, along w

86 reatobiliary ductal systems of mice revealed tuft cells in the biliary tract but not the normal pancr

87 his signalling cascade obviates the need for tuft cells in the epithelial cell remodelling of the int

88 ers of podocytes, nonepithelial cells (NECs; tuft cells other than podocytes), and parietal epithelia

89 These findings suggest that intestinal tuft cells play an important role in regulating the ATM

90 We determined that Dclk1 expressing tuft cells regulate the whole intestinal epithelial cell

91 The function of Dclk1 expressing tuft cells regulating intestinal epithelial DNA damage r

92 Moreover, tuft cells secrete IL-25, thereby regulating type 2 immu

93 Tuft cells were frequently found in areas of pancreatic

94 ased on their location and microanatomy, the tuft cells' cytospinules, and tubular network, might fac

95 Here we show that tuft cells, a rare epithelial cell type in the steady-st

96 new papers now identify a critical role for tuft cells, an epithelial cell type involved in percepti

97 the loss of TRMP5 abrogates the expansion of tuft cells, goblet cells, eosinophils, and type 2 innate

98 in Tritrichomonas muris (Tm) infected mice, tuft cells, IL-25 in epithelium and IL-13 in the mesench

99 Tuft cells, ILC2s and epithelial progenitors therefore c

100 Here we show that tuft cells, which are taste-chemosensory epithelial cell

101 aplasias with a biliary phenotype containing tuft cells.

102 l and molecular features of gastrointestinal tuft cells.

103 d an increase in the abundance of goblet and tuft cells.

104 lution (4-5 nm/pixel) of specific intestinal tuft cells.

105 SACs release both GABA and DA onto external tufted cells (ETCs) in other glomeruli.

106 both mitral/tufted cells (MTCs) and external tufted cells (ETCs), the two major excitatory neurons th

107 lfactory bulb projection neurons, mitral and tufted cells (M/T), is modulated by pairs of reciprocal

108 naptic targets of OSNs, including mitral and tufted cells (M/TCs) and juxtaglomerular cells, form glo

109 evoked responses has been reported in mitral/tufted cells (M/TCs).

110 ation of nAChRs directly excites both mitral/tufted cells (MTCs) and external tufted cells (ETCs), th

111 tuning was heterogeneous in both mitral and tufted cells (MTCs) and GCs but relatively constant with

112 ically inhibits the OB output neurons mitral/tufted cells (MTCs) by GABA release from SACs: (2) gap j

113 reaching the cortex via inhibition of mitral/tufted cells (MTs).

114 f olfactory (OB) bulb mitral cells (MCs) and tufted cells (TCs) are known to depend on prior odor exp

115 use olfactory system, mitral cells (MCs) and tufted cells (TCs) comprise parallel pathways of olfacto

116 f olfactory bulb (OB) mitral cells (MCs) and tufted cells (TCs) is linked to a variety of computation

117 n olfactory bulb, the mitral cells (MCs) and tufted cells (TCs), differ markedly in physiological res

118 Surprisingly, mitral and tufted cells also showed firing mode differences.

119 7) was equivalent across mitral and external tufted cells and could be explained by a single pool of

120 ic and dendrodendritic circuitry in external tufted cells and mitral cells, respectively, tunes the p

121 modulation adds an excitatory bias to mitral/tufted cells as opposed to increasing response gain or s

122 ion of the raphe nuclei led to excitation of tufted cells at rest and potentiation of their odor resp

123 influence relatively large groups of MCs and tufted cells belonging to clusters of at least 15 glomer

124 ncreases the number of associated mitral and tufted cells by 40% and 100%, respectively.

125 ic excitability and more irregular firing in tufted cells can combine to drive distinct responses of

126 By contrast, MOB external tufted cells contained two alpha subunit types (alpha1 a

127 sponse profiles in mitral cells and external tufted cells could be attributed to slow dendrodendritic

128 This stronger response of tufted cells could be partially attributed to synaptic d

129 lar stuttering of action potential clusters, tufted cells demonstrated a greater propensity to stutte

130 IFICANCE STATEMENT Olfactory bulb mitral and tufted cells display different odor-evoked responses and

131 OB projection neurons, mitral and tufted cells exhibit both spiking and subthreshold membr

132 sion of voltage-gated potassium currents, as tufted cells exhibited faster action potential repolariz

133 odor discrimination learning, mitral but not tufted cells exhibited improved pattern separation, alth

134 Compared to mitral cells, tufted cells exhibited twofold greater excitability and

135 sistent with previous studies, we found that tufted cells fire with higher probability and rates and

136 ges induces a strong increase in Ng-positive tufted cells from P10 to P20, whereas no changes have be

137 Although external tufted cells had a 4.1-fold larger peak EPSC amplitude,

138 esynaptic afferents onto mitral and external tufted cells had similar quantal amplitude and release p

139 llular recordings from identified mitral and tufted cells in anesthetized rats demonstrate that nasal

140 s VPAC2R is expressed in mitral and external tufted cells in the OB.

141 ract with the apical dendrites of mitral and tufted cells inside glomeruli at the first stage of olfa

142 An OB circuit with tufted cells intermediate between OSNs and MCs suggests

143 Ng expression in developing tufted cells is also modulated at the cellular level: at

144 d the parallel pathways formed by mitral and tufted cells of the olfactory system in mice and charact

145 ially attributed to synaptic differences, as tufted cells received stronger afferent-evoked excitatio

146 ith sustained transmission, whereas external tufted cells responded transiently.

147 n with sustained responses, whereas external tufted cells responded transiently.

148 endent lateral inhibition between mitral and tufted cells that likely reflect newly described differe

149 ne to drive distinct responses of mitral and tufted cells to afferent-evoked input.

150 e circuit-level differences allow mitral and tufted cells to best discriminate odors in separate conc

151 he distinct responses of mitral and external tufted cells to high frequency stimulation did not origi

152 esting that the larger peak EPSC in external tufted cells was the result of more synaptic contacts.

153 ostsynaptic responses of mitral and external tufted cells within the glomerulus may involve both dire

154 on of spontaneous and odor-driven mitral and tufted cells' firing activity.

155 factory bulb (OB), principal neurons (mitral/tufted cells) make reciprocal connections with local inh

156 tains excitatory principal cells (mitral and tufted cells) that project to cortical targets as well a

157 uts mediated by mitral, tufted, and external tufted cells, and, in turn, they indiscriminately releas

158 ncy responses in mitral cells, compared with tufted cells, are due to weaker excitation and stronger

159 Cs of the nerve layer, as well as mitral and tufted cells, but was excluded from GABAergic interneuro

160 In individual mitral and tufted cells, inhibition was larger at specific respirat

161 Compared to external tufted cells, mitral cells have a prolonged afferent-evo

162 amic, 2-D, optogenetic stimulation of mitral/tufted cells, that virtual odors that differ by as littl

163 The activity of mitral and tufted cells, the principal neurons of the olfactory bul

164 n and feedforward inhibition onto mitral and tufted cells, the principal neurons.

165 Mitral and tufted cells, the two classes of principal neurons in th

166 ayers in the olfactory bulb (OB), mitral and tufted cells, using chronic two-photon calcium imaging i

167 g the calcium indicator GCaMP2 in the mitral/tufted cells, we investigated the effect of ACh on the g

168 naptic inputs that were targeted mainly onto tufted cells, which act as intermediaries in the excitat

169 rons in the mouse olfactory bulb, mitral and tufted cells, which send olfactory information to distin

170 through feedforward excitation from external tufted cells.

171 ity of mitral cells but had little impact on tufted cells.

172 cortex but not in olfactory bulb mitral and tufted cells.

173 asing the spike output of presumptive mitral/tufted cells.

174 and intrinsic properties between mitral and tufted cells.

175 in 36-mediated gap junctions on MCs, but not tufted cells.

176 hemical features of superficial and external tufted cells.

177 s), sparing the other principal neurons, the tufted cells.

178 volving another glutamatergic cell type, the tufted cells.

179 ctive output onto interneurons and principal tufted cells.

180 nsient response profile, typical of external tufted cells.

181 asts with the transient response in external tufted cells.

182 rons are similar between mitral and external tufted cells.

183 nearly eliminated spiking in mitral, but not tufted, cells.

184 sequent wrinkling of glomerular capillaries, tuft collapse, and periglomerular fibrosis.

185 rea and developed equal or fewer neovascular tufts compared to littermate controls, depending on the

186 OIR mice had a reduced number of neovascular tufts compared to vehicle-treated animals and a signific

187 ns, although minor differences in glomerular tuft contractility and macula densa cell calcium handlin

188 endritic integration in the apical dendritic tuft could be altered by the levels of network activity

189 g from the dental-enamel junction and enamel tufts, crack deflections, and the initiation of new crac

190 uce steady-state cytoplasmic [Ca(2+)] in the tuft dendrite without reducing action potential evoked C

191 that during subthreshold activity, basal and tuft dendrites exhibit spatially localized, small-amplit

192 annels that regulate synaptic integration in tuft dendrites have, however, not been thoroughly invest

193 ent of HCN1 and GIRK1 channels in the distal tuft dendrites of both hippocampal CA1 and neocortical l

194 We found that local NMDA spikes occurred in tuft dendrites of layer 2/3 pyramidal neurons both spont

195 Recent evidence in vitro suggests that the tuft dendrites of pyramidal neurons are capable of evoki

196 the neocortex, where they make synapses with tuft dendrites of pyramidal neurons.

197 signals were observed throughout the apical tuft dendrites when active touch occurred at particular

198 ic contacts from L1 INs target distal apical tuft dendrites, whereas PNs primarily innervate basal an

199 vated Cav3.3 channels on their distal apical tuft dendrites.

200 ctly drives asynchronous release from distal tuft dendrites.

201 -receptor-dependent calcium spikes in apical tuft dendrites.

202 uts located in distantly separated basal and tuft dendrites.

203 The Developmental FunctionaL Annotation at Tufts (DFLAT) project aims to improve the quality of ana

204 ioles, LacZ-positive cells in the glomerular tuft did not express renin.

205 stalk and are recruited onto the glomerular tuft during infancy to adolescence in mice and humans.

206 SD matrices are available from http://dsd.cs.tufts.edu/capdsd

207 s SMURFLite is available at: http://smurf.cs.tufts.edu/smurflite/

208 ed glomerular podocyte number and glomerular tuft enlargement.

209 Congenital tufting enteropathy (CTE) is a severe autosomal recessiv

210 , an effect that is mediated by the external tufted (ET) cells coupled to DAT+ cells via chemical and

211 es of neurons: periglomerular (PG), external tufted (ET), and short-axon (SA) cells.

212 f the Bowman's capsule invade the glomerular tuft exclusively in proliferative disorders.

213 ubset of labeled cells within the glomerular tuft expressed the podocyte markers Wilms tumor protein

214 c retinal angiogenesis, reducing neovascular tuft formation and increased avascular area, in a dose-d

215 upy layer 5A and display an apical dendritic tuft; functionally, they fire broad, adapting action pot

216 Patients with iris vascular tufts generally remain asymptomatic until presenting wit

217 the soma or at the border of L1/L2, near the tuft, generates a local sADP.

218 ry, failure of podocytes to match glomerular tuft growth in response to growth signaling through the

219 PEC markers were detected on the tuft in 87% of the biopsies of patients diagnosed as pri

220 nd total podocyte volume (number x size) per tuft in relation to weight gain and nephrectomy.

221 rong and specific staining of the glomerular tufts in a distribution that mimicked that of the immune

222 Areas of avascular retina and neovascular tufts in injected (treated) eyes and noninjected fellow

223 ed in a significant reduction of neovascular tufts in oxygen-induced retinopathy, supporting the feas

224 l as the pathologic formation of neovascular tufts in postnatal retinal microvascular networks.

225 vel of Cavin-2 expression in the neovascular tufts in the mouse model of oxygen-induced retinopathy,

226 ssels of patient-derived FVMs and angiogenic tufts in the retina of mice with oxygen-induced retinopa

227 cent protein-positive area in the glomerular tufts increased after mesangial injury.

228 of frequency-modulation of somatic output by tuft input and (simulated) calcium-channel blockage func

229 incidence detection between basal and apical tuft inputs by controlling the frequency of spike output

230 integration throughout the apical dendritic tuft is highly compartmentalized by voltage-gated potass

231 The apical tuft is the most remote area of the dendritic tree of ne

232 Here, we show that one-third of the thick-tufted layer 5 pyramidal neurons have an axon originatin

233 dendritic arbors in both simple- and complex-tufted layer 5 Tg1 pyramidal neurons have more branches

234 ughout the OB to cause suppression of mitral/tufted (M/T) cell firing, an effect that is mediated by

235 Mitral/tufted (M/T) cells form dendrodendritic synapses on gran

236 Mitral/tufted (M/T) cells of the main olfactory bulb transmit o

237 Here we show that individual mitral/tufted (M/T) cells sum inputs linearly across odors and

238 mitted to the olfactory cortex by mitral and tufted (M/T) cells.

239 us samples analyzed, 4683 (81.64%) were from Tufts Medical Center (TMC), 955 (16.65%) were from Bosto

240 ren-Lawrence grades 2 or 3, were enrolled at Tufts Medical Center beginning February 11, 2013; all pa

241 related to hematologic malignancies from the Tufts Medical Center Cost-Effectiveness Analysis Registr

242 of 1558 consecutive patients followed at the Tufts Medical Center Hypertrophic Cardiomyopathy Institu

243 Patients were enrolled at Tufts Medical Center in Boston between March 2006 and Ju

244 th EFE referred to New England Eye Center at Tufts Medical Center, a tertiary care ophthalmology prac

245 ts from the outpatient dermatology center of Tufts Medical Center, enrolled from August 18, 2009, to

246 l cystoid degeneration (TCD), cystic retinal tuft, meridional fold, lattice and cobblestone degenerat

247 lium, ora serrata pearl, TCD, cystic retinal tuft, meridional fold, lattice, and cobblestone degenera

248 errata pearl, ora tooth, TCD, cystic retinal tuft, meridional fold, retinal hole, and typical degener

249 one centrally and new vessels with capillary tufts midperipherally in older mice.

250 one centrally and new vessels with capillary tufts midperipherally.

251 and short axon (SA) cells, as well as mitral/tufted (MT) cells carrying OB output to piriform cortex.

252 on [SA] cells) and OB output neurons (mitral/tufted [MT] cells) projecting to the piriform cortex.

253 suppression in OB output neurons (mitral and tufted, MT cells).

254 ike activity, whereas calcium signals in the tuft occur unreliably.

255 te its distal location, the apical dendritic tuft of layer 5 pyramidal neurons receives substantial e

256 -localization signal in the apical dendritic tuft of layer 5B cortical pyramidal neurons during senso

257 on of spine synapses in the apical dendritic tuft of layer V pyramidal neurons in the mPFC.

258 In bilaterians they are characterised by a tuft of long cilia and receptor cells and they are assoc

259 o gamma oscillations generated at the apical tuft of pyramidal cells.

260 idal structures), BVNs in 26 eyes (55.3%, 26 tufts of BVNs), and stalks of origin from the choroid in

261 vestigate dendritic spine dynamics in apical tufts of GFP-expressing layer 4 (L4) pyramidal neurons o

262 omotes the formation of membrane ruffles and tufts of microvilli, whereas expression of ezrin and Eps

263 summarize evidence that input to the apical tufts of neocortical pyramidal cells modulates their res

264 ocalized to retinal glia and pathological NV tufts of the OIR retinas.

265 caused marked regression of the new vascular tufts on the vitreal side with normal organization and t

266 long Bowman's capsule, within the glomerular tuft, or in both locations.

267 ETCs transmit sensory signals to mitral/tufted output neurons and drive intraglomerular inhibiti

268 = 0.04) and reduced formation of neovascular tufts (P < 0.001), as compared with WT controls.

269 l cells (PECs) migrating onto the glomerular tuft participate in the formation of focal segmental glo

270 y focal scarring of the glomerular capillary tuft, podocyte injury, and nephrotic syndrome.

271 We identified CPMs in the Tufts Predictive Analytics and Comparative Effectiveness

272 rter 1, a presynaptic protein, in mitral and tufted projection neurons, and 5T4 in granule cells.

273 r V pyramidal neurons that have thick apical tufts, prominent h-current, and subcortical projections.

274 pses between rat somatosensory layer 5 thick-tufted pyramidal cells is mediated by a higher and more

275 ers 2/3, 4, and the slender-tufted and thick-tufted pyramidal cells of layer 5 using a combination of

276 ding to previously described thin- and thick-tufted pyramidal neurons, respectively.

277 nd focused dendritic arbors of layer 5 "tall-tufted" pyramids, all "nontufted" cells had sparse, but

278 cyte loss, activated parietal cells mediated tuft re-epithelialization by two distinct mechanisms.

279 receptors have a vital role in coupling the tuft region of the layer 2/3 pyramidal neuron to the cel

280 e of spontaneous hyphaema from iris vascular tuft related to a documented supratherapeutic Internatio

281 structure and orientation of apical dendrite tufts remained stable over a two-month period, both in c

282 t induce revascularization of the hyalinized tuft, resulting in hypoxic cell death and irreversible d

283 rominent hematologist Dr William Dameshek of Tufts School of Medicine, Blood has published many paper

284 cised from distal apical dendritic trunk and tuft sites.

285 This species has a reticulate, tufted skeleton of minute monaxon spicules, characterist

286 Trichodesmium forms macroscopic, fusiform (tufts), spherical (puffs) and raft-like colonies that pr

287 eurons comprise at least two subtypes: thick-tufted, subcortically projecting type A neurons, with pr

288 risingly, we found that activation of apical tuft synapses alone resulted in heterosynaptic potentiat

289 Carolina chickadees (Poecile carolinensis), tufted titmice (Baeolophus bicolor), and white-breasted

290 ted the interaction between apical dendritic tuft, trunk, and axosomatic integration zones to control

291 who underwent lateral sinus augmentation at Tufts University School of Dental Medicine, Boston, Mass

292 elected from patients receiving treatment at Tufts University School of Dental Medicine.

293 kshop on Cancer Systems Biology sponsored by Tufts University, Boston, Massachusetts, in July 2012.

294 ed by an independent evidence review team at Tufts University.

295 capable of shifting the balance of principal tufted versus mitral cell activity across large expanses

296 a 1.6 mm hyphaema and multiple iris vascular tufts visible around the entire pupil.

297 is confirmed the mismatch between glomerular tuft volume and total podocyte volume (number x size) pe

298 ophy prevented a change in the proportion of tuft volume occupied by podocytes.

299 The central avascular area and neovascular tufts were measured after 5 days in room air (21% oxygen

300 MP3 or erlotinib developed fewer neovascular tufts when compared to untreated littermates.