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1 d glucuronide metabolite AUC correlated with tumor lysis syndrome.
2 ity using this novel schedule was hyperacute tumor lysis syndrome.
3 e aminotransferase, febrile neutropenia, and tumor lysis syndrome.
4 f management in diseases other than gout and tumor lysis syndrome.
5 ssociated with thrombocytopenia and, rarely, tumor lysis syndrome and cytokine release reactions; the
6 l monitoring and aggressive intervention for tumor lysis syndrome and hyperkalemia is necessary for s
7 s the use of urate oxidase for prevention of tumor lysis syndrome and the associated uric acid nephro
9 self, but complications such as leukostasis, tumor lysis syndrome, and disseminated intravascular coa
10 ti-CD19-CAR T cells, another CLL patient had tumor lysis syndrome as his leukemia dramatically regres
12 ts to the dose-escalation schedule, clinical tumor lysis syndrome did not occur in any of the 60 pati
19 ccur in four patients, as a result of severe tumor lysis syndrome; three of these patients required h
20 malignancy and were in danger of developing tumor lysis syndrome (TLS) and subsequent acute uric aci
22 c malignancies at risk for hyperuricemia and tumor lysis syndrome (TLS) were randomly assigned to ras
23 yndrome was not observed, whereas laboratory tumor lysis syndrome was documented in three patients.
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