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1 n by steroids, thiopurines, methotrexate, or tumor necrosis factor inhibitors.
2 ransplant recipients, and patients receiving tumor necrosis factor inhibitors.
3 in combination with certain biologic agents (tumor necrosis factor inhibitors and rituximab).
4 halidomide, cyclophosphamide, hemoperfusion, tumor necrosis factor inhibitors, and granulocyte colony
5  lung disease may respond to cyclosporine or tumor necrosis factor inhibitors, and that tumor necrosi
6 along with oral retinoids, methotrexate, and tumor necrosis factor inhibitors as most helpful.
7 d in 20 of 106 patients (18.9%) treated with tumor necrosis factor inhibitors, but did not lead to di
8  drug was more likely with methotrexate than tumor necrosis factor inhibitors, but having 1 or more i
9 hown that in several clinical circumstances, tumor necrosis factor inhibitors can indeed have an incr
10 2.1 (95% CI, 0.55-8.4) in patients receiving tumor necrosis factor inhibitors compared with controls.
11        The introduction of the macromolecule tumor necrosis factor inhibitors etanercept, infliximab,
12                          The introduction of tumor necrosis factor inhibitors for patients with ankyl
13  other inflammatory arthritides treated with tumor necrosis factor inhibitors frequently undergo orth
14  The introduction of biologic agents such as tumor necrosis factor inhibitors has changed how we appr
15                       The notable success of tumor necrosis factor inhibitors has not only changed th
16                               Treatment with tumor necrosis factor inhibitors has not only resulted i
17                                  As a class, tumor necrosis factor inhibitors have arguably been the
18                                    Recently, tumor necrosis factor inhibitors have become available f
19                 Biologic agents, notably the tumor necrosis factor inhibitors, have effected dramatic
20 paper reviews data from clinical trials with tumor necrosis factor inhibitors in ankylosing spondylit
21 uld be performed to elucidate the effects of tumor necrosis factor inhibitors in Still's disease.
22                        Early experience with tumor necrosis factor inhibitors indicates that they may
23 ies of a co-stimulation blocker (abatacept), tumor necrosis factor inhibitor (infliximab), and interl
24 te of malignancy among children treated with tumor necrosis factor inhibitors is worrisome.
25      By contrast, it has been suggested that tumor necrosis factor inhibitors may induce some manifes
26       Information regarding the influence of tumor necrosis factor inhibitors on the risk for postope
27 ble side effects associated with one or more tumor necrosis factor inhibitors, other biologic DMARDs,
28 Medication-related AEs occur less often with tumor necrosis factor inhibitors than with methotrexate.

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