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1 n by steroids, thiopurines, methotrexate, or tumor necrosis factor inhibitors.
2 ransplant recipients, and patients receiving tumor necrosis factor inhibitors.
4 halidomide, cyclophosphamide, hemoperfusion, tumor necrosis factor inhibitors, and granulocyte colony
5 lung disease may respond to cyclosporine or tumor necrosis factor inhibitors, and that tumor necrosi
7 d in 20 of 106 patients (18.9%) treated with tumor necrosis factor inhibitors, but did not lead to di
8 drug was more likely with methotrexate than tumor necrosis factor inhibitors, but having 1 or more i
9 hown that in several clinical circumstances, tumor necrosis factor inhibitors can indeed have an incr
10 2.1 (95% CI, 0.55-8.4) in patients receiving tumor necrosis factor inhibitors compared with controls.
13 other inflammatory arthritides treated with tumor necrosis factor inhibitors frequently undergo orth
14 The introduction of biologic agents such as tumor necrosis factor inhibitors has changed how we appr
20 paper reviews data from clinical trials with tumor necrosis factor inhibitors in ankylosing spondylit
21 uld be performed to elucidate the effects of tumor necrosis factor inhibitors in Still's disease.
23 ies of a co-stimulation blocker (abatacept), tumor necrosis factor inhibitor (infliximab), and interl
27 ble side effects associated with one or more tumor necrosis factor inhibitors, other biologic DMARDs,
28 Medication-related AEs occur less often with tumor necrosis factor inhibitors than with methotrexate.
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