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1 the cytoplasmic tail, constitutively engages tumor necrosis factor receptor-associated factors.
2 urthermore, expression of the anti-apoptotic tumor necrosis factor receptor associated factor 1 (TRAF
4 activate NF-kappaB, and thereby (iv). induce tumor necrosis factor receptor-associated factor 1 expre
5 ant to chronic inflammation: TRAF1 (encoding tumor necrosis factor receptor-associated factor 1) and
6 K do not differ in their in vitro binding to tumor necrosis factor receptor-associated factor 1, 2, 3
8 R domains of c-IAP1 and c-IAP2 interact with tumor necrosis factor receptor-associated factors 1 and
12 through a common scaffolding protein termed tumor necrosis factor receptor-associated factor 2 (TRAF
14 IKKepsilon interacts with and phosphorylates tumor necrosis factor receptor-associated factor 2 (TRAF
16 eus to mitochondria, where it interacts with tumor necrosis factor receptor-associated factor 2 (TRAF
17 Consistent with its ability to interact with tumor necrosis factor receptor-associated factor 2 (TRAF
18 n ligases in the human genome and identified tumor necrosis factor receptor-associated factor 2 (TRAF
19 on of API2-MALT1 as well as interaction with tumor necrosis factor receptor-associated factor 2 (TRAF
20 at receptor-interacting protein 1 (RIP1) and tumor necrosis factor receptor-associated factor 2 (TRAF
22 3, or 6 or in their in vivo association with tumor necrosis factor receptor-associated factor 2 and d
23 receptor (IL-1R)-independent kinases such as tumor necrosis factor receptor-associated factor 2, were
24 poptosis 1 and 2 (c-IAP1/2) proteins and the tumor necrosis factor receptor-associated factors 2 and
25 lves NF-kappaB-inducing kinase (NIK) and the tumor necrosis factor receptor-associated factor-2 (TRAF
26 udy, we report the identification of a novel tumor necrosis factor receptor-associated factor 3 (TRAF
27 s essential for TLR9-dependent activation of tumor necrosis factor receptor-associated factor 3 (TRAF
30 is through its dynamic interaction with the tumor necrosis factor receptor-associated factor 3 (TRAF
31 activation of the downstream effector TRAF3 (tumor necrosis factor receptor-associated factor 3).
32 enable cytoplasmic C terminus engagement of tumor necrosis factor receptor-associated factor 3, (iii
34 DLBCL induces recruitment and degradation of tumor necrosis factor receptor-associated factor 3, whic
36 lymphotoxin-beta receptor (LTbetaR) recruits tumor necrosis factor receptor-associated factor-3 (TRAF
37 e show that Smurf1 induces ubiquitination of tumor necrosis factor receptor-associated factor 4 (TRAF
38 L-1 receptor-associated kinase 1 (IRAK1) and tumor necrosis factor receptor-associated factor 6 (TRAF
39 h receptor interacting protein-2 (RIP2)- and tumor necrosis factor receptor-associated factor 6 (TRAF
44 1 and M2 subsets was critically dependent on tumor necrosis factor receptor-associated factor 6 (TRAF
47 m specifically induces the ubiquitination of tumor necrosis factor receptor-associated factor 6 (TRAF
48 62, we found that Ajuba also interacted with tumor necrosis factor receptor-associated factor 6 (TRAF
50 signaling competence, specifically an intact tumor necrosis factor receptor-associated factor 6 (TRAF
51 , IL-1R-associated protein kinase (IRAK) and tumor necrosis factor receptor-associated factor 6 (TRAF
52 endent interaction with IRAK4, IRAK, and the tumor necrosis factor receptor-associated factor 6 (TRAF
53 erleukin 1 receptor-associated kinase 1, and tumor necrosis factor receptor-associated factor 6 are r
54 appaB at the level of (or downstream of) the tumor necrosis factor receptor-associated factor 6 compl
55 d low levels of Cathepsin K, TRAP, RANK, and tumor necrosis factor receptor-associated factor 6 mRNAs
56 interleukin receptor-associated kinase 2 and tumor necrosis factor receptor-associated factor 6 to su
57 ling to nuclear factor kappaB by suppressing tumor necrosis factor receptor-associated factor 6 ubiqu
59 s modification of the signaling intermediary tumor necrosis factor receptor-associated factor 6 with
60 xpression via a MyD88 --> IRAK1-IRAK4-TRAF6 (tumor necrosis factor receptor-associated factor 6) -->
61 include a PB1 dimerization domain, a TRAF6 (tumor necrosis factor receptor-associated factor 6) bind
63 ximal signaling components (MyD88, IRAK, and tumor necrosis factor receptor-associated factor 6) upon
64 ignaling through the ubiquitin ligase TRAF6 (tumor necrosis factor receptor-associated factor 6).
65 binding to, but impaired ubiquitination of, tumor necrosis factor receptor-associated factor 6, a vi
66 icantly enhanced Lys63 polyubiquitination of tumor necrosis factor receptor-associated factor 6, acti
67 n vitro, and this requires IL-17 receptor A, tumor necrosis factor receptor-associated factor 6, and
72 attenuates the molecular interaction between tumor necrosis factor receptor-associated factor-6 and I
73 specific interaction between p75 and TRAF6 (tumor necrosis factor receptor-associated factor-6) afte
74 e receptors, IL-1 receptor-associated kinase/tumor necrosis factor receptor-associated factor-6, IL8/
75 NF-kappaB activation through an IKKbeta- and tumor necrosis factor receptor-associated factor-6-depen
76 ys, functions reminiscent of those noted for tumor necrosis factor receptor-associated factor adaptor
77 h, we examined the role of six RANK putative tumor necrosis factor receptor-associated factor-binding
78 The tumor necrosis factor receptor-associated factor-binding
79 t Asn-234 and Asn-270, as well as one in the tumor necrosis factor receptor-associated factor domain
80 phosphatase mu) domain and one in the TRAF (tumor necrosis factor receptor-associated factor) domain
81 etween p75(NTR) and the six members of TRAF (tumor necrosis factor receptor-associated factors) famil
82 nd its homologs can physically interact with tumor necrosis factor receptor-associated factor family
83 has been shown to interact with proteins of tumor necrosis factor receptor-associated factor family,
84 One such interactor, TRAF6, a member of the tumor necrosis factor receptor-associated factor family,
85 Here, we demonstrate that the TRAF (tumor necrosis factor receptor-associated factor) family
86 diated by Math-BTB/POZ (for Meprin and TRAF [tumor necrosis factor receptor associated factor] homolo
87 ociates with the cell cytoskeleton through a tumor necrosis factor receptor-associated factor-interac
88 the common variant D10N, located in the ACT1 tumor necrosis factor receptor-associated factor-interac
90 e, and it is associated and colocalizes with tumor necrosis factor receptor-associated factor (TRAF)
91 eraction of its cytoplasmic (ct) domain with tumor necrosis factor receptor-associated factor (TRAF)
93 iates signaling by recruiting members of the tumor necrosis factor receptor-associated factor (TRAF)
94 8 are now found to be stably associated with tumor necrosis factor receptor-associated factor (TRAF)
96 o induces EGFR expression, interact with the tumor necrosis factor receptor-associated factor (TRAF)
97 ctivation is a downstream signaling event of tumor necrosis factor receptor-associated factor (TRAF)
98 ediate the binding of CD30 to members of the tumor necrosis factor receptor-associated factor (TRAF)
99 been found to associate with members of the tumor necrosis factor receptor-associated factor (TRAF)
100 ed multiple independent cDNAs encoding human tumor necrosis factor receptor-associated factor (TRAF)
102 receptor superfamily member CD40, utilizing tumor necrosis factor receptor-associated factor (TRAF)
104 tent membrane protein 1 (LMP1) share several tumor necrosis factor receptor-associated factor (TRAF)
105 , plasma membrane patching, oligomerization, Tumor Necrosis Factor Receptor-associated factor (TRAF)
106 ric ligand and propagated by interactions of tumor necrosis factor receptor-associated factor (TRAF)
107 hat interferes with the assembly of the CD40-tumor necrosis factor receptor-associated factor (TRAF)
108 -1) receptor-associated kinase (IRAK)-2, and tumor necrosis factor receptor-associated factor (TRAF)-
110 that the same region of CD30 interacts with tumor necrosis factor receptor-associated factor (TRAF)1
111 onomethylarginine (NMMA) and an inhibitor of tumor necrosis factor receptor-associated factor (TRAF)2
115 omo- and heterotrimeric association of human tumor necrosis factor receptor-associated factors (TRAF)
117 we report that STP-A interacts with cellular tumor necrosis factor receptor-associated factors (TRAF2
118 5 residues include a domain that engages the tumor necrosis factor receptor associated factors (TRAFs
119 Tumor necrosis factor receptor-associated factors (TRAFs
120 rns the interaction between LMP-1 CTAR-1 and tumor necrosis factor receptor-associated factors (TRAFs
121 Tumor necrosis factor receptor-associated factors (TRAFs
122 emonstrate here the interaction of RANK with tumor necrosis factor receptor-associated factors (TRAFs
123 smic domain to adaptor proteins, such as the tumor necrosis factor receptor-associated factors (TRAFs
124 The recruitment of tumor necrosis factor receptor-associated factors (TRAFs
125 t (CTAR1) or indirect (CTAR2) recruitment of tumor necrosis factor receptor-associated factors (TRAFs
126 the cytoplasmic adapter protein family, the tumor necrosis factor receptor-associated factors (TRAFs
127 Tumor necrosis factor receptor-associated factors (TRAFs
128 via binding, as a constitutive oligomer, to tumor necrosis factor receptor-associated factors (TRAFs
129 s through downstream adaptor proteins called tumor necrosis factor receptor-associated factors (TRAFs
130 gnaling via the recruitment of intracellular tumor necrosis factor receptor-associated factors (TRAFs
131 Tumor necrosis factor receptor-associated factors (TRAFS
132 Studies examining tumor-necrosis factor receptor-associated factors (TRAFs
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