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1 xenograft tumors, suggesting the presence of tumor-associated fibroblasts.
2 oblast growth factor in normal compared with tumor-associated fibroblasts.
3 lating mesenchymal precursors as a source of tumor-associated fibroblasts.
4 tochemistry confirm that MSF is expressed by tumor-associated fibroblasts and additionally indicate t
5 on protein (FAP) is selectively expressed on tumor-associated fibroblasts and pericytes in epithelial
6 asts) and adjacent cervical cancer biopsies (tumor-associated fibroblasts) and from primary keratinoc
7 infection of primary human dendritic cells, tumor-associated fibroblasts, and colorectal carcinoma c
8 owth on three-dimensional matrix produced by tumor-associated fibroblasts, and in formation of tumors
10 We have investigated the role of Tiam1 in tumor-associated fibroblasts as a modulator of tumor cel
11 o-like three-dimensional system derived from tumor-associated fibroblasts at diverse stages of tumor
13 on from Th2 to Th1 type while remodeling the tumor-associated fibroblasts, collagen, and blood vessel
15 product which is preferentially expressed in tumor-associated fibroblasts, could function as a tumor
16 that cells of mesenchymal origin, including tumor-associated fibroblasts, degrade substantial amount
17 ncluding mesenchymal stem cells, adipocytes, tumor associated fibroblasts, endothelial cells, and imm
21 lial cells to initiate a paracrine loop with tumor-associated fibroblasts involving TGFbeta and HGF,
22 e involving host blood vessels, lymphocytes, tumor-associated fibroblasts, macrophages, dendritic cel
23 timulatory effects of conditioned media from tumor-associated fibroblasts on keratinocyte penetration
26 conclusion, our data indicate that EREG and tumor-associated fibroblasts play a crucial role in cont
27 epsin L to be predominantly expressed by the tumor-associated fibroblasts surrounding the invading me
28 e matrix-modifying hormone relaxin increased tumor-associated fibroblast (TAF) interaction with colla
31 erapeutic NP to distribute into, and deplete tumor-associated fibroblasts (TAFs) for improved therape
33 geted immunotherapy to antigens expressed in tumor-associated fibroblasts, the predominant component
34 tor/scatter factor or conditioned media from tumor-associated fibroblasts to the Matrigel resulted in
35 the contribution of hypoxic signaling within tumor-associated fibroblasts to tumorigenesis remains un
36 ion protein (FAP), a product up-regulated in tumor-associated fibroblasts, using dendritic cells tran
38 e the functional relevance of EREG-producing tumor-associated fibroblasts, we developed a novel syste
40 eveals that p53 mutations sometimes occur in tumor-associated fibroblasts, which correlate with an in
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