ライフサイエンスコーパス: turn off

1 resented the highest fluorescence quenching (turn-off).

2 during the first min after the treadmill was turned off).

3 participates in G protein-mediated signaling turn-off.

4 s for limiting the rate of phototransduction turn-off.

5 n DNA intercalation, and Eu-DOTA-Phen, which turns off.

6 events expansion of slp stripes after Eve is turned off.

7 s that was reversed after the high light was turned off.

8 voltaic effect to be reversed in polarity or turned off.

9 ein is expressed and the wild-type Calpha is turned off.

10 oving through the x-ray beam or the beam was turned off.

11 re to light that persists after the light is turned off.

12 process is reversed, and KLF2 expression is turned off.

13 magnetic relaxation signal when the field is turned off.

14 from solution and the conjugate addition is turned OFF.

15 oA/ROCK is turned on, endothelial cells when turned off.

16 es' as a travelling wave when the tweezer is turned off.

17 pray emitters when the ionization voltage is turned off.

18 upstream DNA damage checkpoint signaling is turned off.

19 ermanently in G2 phase when Ku expression is turned off.

20 part the spindle poles when poleward flux is turned off.

21 expression of the Nrg1-ntfbeta transgene was turned off.

22 mechanism that engages when poleward flux is turned off.

23 iomarkers be nonexistent, the signal remains turned-off.

24 nd mRNA levels is transient and begins to be turned off 3-6 h after growth stimulation of cultured ce

25 Hence, rather than serving to turn off a biological response, reversible desensitizati

26 desensitization is a universal mechanism to turn off a biological response; in this process, the abi

27 Depletion of amino acids from the medium turns off a signaling pathway involving the Ragulator co

28 t in this process by acting as a switch that turns off a somatic lineage pathway.

29 n the electrostatic protein-water forces are turned off, a dewetting transition occurs in the interdo

30 If as few as three inappropriate genes are turned off, a normal cell can be converted into a cancer

31 Turning off ABA response requires turnover and/or inacti

32 sient rise in chlorophyll fluorescence after turning off actinic light reflects nonphotochemical redu

33 dent chlorophyll fluorescence increase after turning off actinic light.

34 he protein RibR counteracts the FMN-induced "turn-off" activities of both FMN riboswitches in Bacillu

35 In contrast, how transcription is turned off after activation is unclear.

36 occurs only in subconfluent cultures and is turned-off after a polarized epithelium has formed.

37 ow this response gets turned on, how it gets turned off again when no longer needed is just as import

38 fides it receives from 2-Cys Prx to APS1 and turns off AGPase.

39 ells from being fully activated, CTLA4Ig can turn off already activated effector T cells by an NO/reg

40 ion; the silencing persisted after p15AS was turned off, although methylation and heterochromatin inh

41 When STN DBS was turned off, an initial fast but partial loss of benefit

42 phosphorylates key regulatory substrates and turns off anabolic biosynthetic pathways.

43 These results reveal a potential to turn off and turn on the motor by attaching or removing,

44 ADP and AMP, we have developed fluorescence turn-off and turn-on assays that monitor the catalytic a

45 kinetochores come under tension, the SAC is turned off and APC/C(Cdc20) degrades cyclin B and securi

46 rment was ameliorated when the transgene was turned off and D2R levels were normalized.

47 c supramolecular receptors for fullerenes is turned off and on by changing the solvent, whereas the s

48 amphiphilicity of the copolymer that can be turned off and on by varying the polarity of the solvent

49 e alphaHL channel, the DC driving voltage is turned off and the conductance of the channel is monitor

50 ever, the mechanism of how the pEMT phase is turned off and the redifferentiation phase is initiated

51 Then, the metronome is turned off and the subject continues at the established

52 ment was maintained when the stimulation was turned off and was accompanied by a higher number of tyr

53 rate that genes turn on, the rate that genes turn off, and the rate of transcription.

54 ed circulating current, accelerated response turn-off, and diminished intracellular calcium.

55 x and cornea-transparency-related genes were turned off, and a repertoire of macrophage genes were sw

56 spontaneously when calcineurin activity was turned off, and expression of fetal genes reverted to ba

57 s, can be stable for hours after the beam is turned off, and ions that screen charges on the filament

58 -anaphase transition, the spindle checkpoint turns off, and MCC disassembles to allow anaphase onset.

59 those associated with body column tissue are turned off as the tentacle zone expands.

60 ndifferentiated embryonic stem cells but was turned off as these cells differentiate in vitro and in

61 When attractive van der Waals forces are turned off as well, the dewetting in the interdomain reg

62 Furthermore, when P3H2 expression was turned off, as seen naturally in cultured SAOS-2 osteosa

63 tein phosphatase type 5 (PP5), which acts to turn off ASK-1/MKK-4/JNK signaling via two mechanisms.

64 signal transducer, a real-time fluorescence turn-off assay for the enzyme alkaline phosphatase (ALP)

65 The fluorescence turn-on and turn-off assays monitor the forward and reverse transpho

66 ell genes" being highly expressed at 0 h and turned off at 48 h, while "cell division" genes were tur

67 mining gene Sox9 in mutant Sertoli cells was turned off at embryonic day 14.5 after Wt1 ablation, sug

68 iven by the distal enhancer can no longer be turned off at late stages in lin-1 and lin-31 mutants, e

69 specifically seen in progenitor cells and is turned off at the onset of neuronal differentiation.

70 When transgenic human RTN3 expression was turned off at young age, formation of RIDNs was essentia

71 was found that reserpine oxidation could be "turned off" at flow rates as low as 2.5 microL/min as we

72 spo0A cells, yloA expression was completely turned off because of upregulation of AbrB throughout gr

73 s was dependent on whether the transgene was turned off before or after the formation of RTN3 aggrega

74 ll mutants were similar to wild type showing turned-off behaviors.

75 This sheath or coaxial fluid was turned off between analysis of cells and on during cell

76 its fully reduced 4Cu(I) state, the cluster turns off binding of stronger donor ligands while enabli

77 andcuffing") between pairs of replicons that turn off both origins.

78 ined stable even when the electric field was turned off but readily redissolved when counterions were

79 fire full Ca2+ spikes, which are eventually turned off by a D1/D5-Ca2+-dependent PKC mechanism.

80 by a brief light pulse at one wavelength and turned off by a pulse at a second wavelength.

81 system and allows the dynamic exchange to be turned off by addition of 18-crown-6, which binds the eq

82 ealthy cells, the SR-BI apoptotic pathway is turned off by eNOS and HDL which prevents inappropriate

83 The surge can be turned off by fixing the rate of fluid flow through the

84 Galpha subunits are turned off by GTP hydrolysis.

85 Turned off by inhibited anion mobility, that is, proton

86 ism by which NF-kappaB-mediated signaling is turned off by its removal from the DNA in the presence o

87 Rh* is turned off by phosphorylation by rhodopsin kinase [G-pro

88 the triple-layer complex and then completely turned off by reforming it through the abstraction of Cl

89 sporters require PIP2 to function and can be turned off by signaling pathways that deplete PIP2.

90 Signaling is instead turned off by the endosomal retromer complex.

91 nthesis in both asexual and sexual spores is turned off by the NF-kB like fungal regulators VosA and

92 istinct points in the cycle, one of which is turned off by the presence of P(i).

93 turned on" by acute Gq-protein signaling or "turned off" by sustained Gq-protein signaling.

94 detection period or a slow excitation source turn off can be mitigated by using an offset time prior

95 tually leads to release from DNA damage thus turning off catalytic activity, although the effects of

96 In contrast, SpTm ultimately turns off Cdc12p-mediated elongation by "trapping" Cdc12

97 d nuclear orientation checkpoints and delays turning off Cdc14 in G1.

98 f cyclin B is the major known mechanism that turns off Cdk1 during mitotic exit.

99 n localizes Evi5 to centriolar appendages to turn off centrosomal Rab11 activity.

100 PDL pancreas were impaired when ERalpha was turned off chemically or genetically (ERalpha(-/-)), whe

101 esence of doxycycline, and the gene could be turned off completely by removal of doxycycline.

102 s information is essential to initiate or to turn off complex signaling pathways.

103 nging the catalyst to a Echavarren's gold(I) turned off cyclization to seven member ring and turned o

104 used to monitor cytoplasmic partitioning to turn off cytokinesis signaling.

105 ease of the signal from the individual MNPs (turn-off detection strategy), whereas the techniques pro

106 n immature CD4- CD8- CD44+ CD25- thymocytes, turned off during further stages of thymocyte developmen

107 When the current is turned off during phases of the domain wall motion when

108 ng through, even when the signal is actually turned off during the interruption-an effect known as th

109 aive host, the activity of the M promoter is turned off early, preventing excessive synthesis of meth

110 After the electric field is turned off, electrical current is measured and observed

111 and autophagy in response to low ATP, while turning off energy consumption mediated by mammalian tar

112 eroidal antiestrogens selectively turn on or turn off estrogen target tissues throughout the body.

113 s transduce light information to immediately turn off ethylene signaling for de-etiolation initiation

114 PhoP-activated genes in low Mg(2+) but could turn off expression in response to high Mg(2+).

115 successful infection demands that pathogens turn off expression of products that might interfere wit

116 nt with doxycycline for 2 months effectively turned off expression of the human RTN3 transgene, confi

117 pertrophic markers, which were normalized on turning off expression of the mutant protein.

118 Established phenotypes in HCM reverse upon turning off expression of the mutant protein.

119 p unfolds, allowing for mRNA degradation and turning off expression.

120 etermining transcription factors to promptly turn off extrinsic inhibitory Wnt signaling mediated by

121 ing serine 381, a covalent modification that turns off F-actin bundling activity.

122 and density functional theory (DFT)) showing turn off fluorescence behavior.

123 -metal interactions lead to both turn-on and turn-off fluorescence sensing mechanisms, enhancing the

124 Thus, "turn-off" fluorescence method was applied for ultrasensi

125 NMDAR-induced FMRP dephosphorylation, which turns off FMRP suppression of Kv4.2.

126 Accordingly, miR-150 repression was turned off following treatment with the DNMT inhibitor,

127 expressed, replaced by a second product, or turned-off following exposure to ligand.

128 nical status when deep brain stimulation was turned off for 2 days, suggesting that deep brain stimul

129 When the smoke evacuator was turned off for 30 seconds, there was a more than 26-fold

130 from this "memory" after the illumination is turned off for an extended period of time.

131 When the transgene was turned off from conception to 3 months of age, no phenot

132 ished in the host, these virulence genes are turned off, fully attenuating the vaccine strain.

133 r into CCPs where Galphai3 and GAIP meet and turn off G protein signaling.

134 (GPCRs) by GPCR kinases (GRKs) functions to turn off G-protein signaling and turn on arrestin-mediat

135 otein signaling 4 (RGS4), a protein known to turn off G-protein signaling.

136 ncorporation of [3H]palmitate and ability to turn-off Gi and Gq signaling and significantly inhibited

137 ls may acquire differentiated status as they turn off Gli3 expression during neurogenesis.

138 When spikes were turned off GOM recoveries returned to 60 +/- 19% of perm

139 ibed, but the pathway whereby the levator is turned off have not.

140 When the stimulus was turned off, his body partially tilted back at an interme

141 Thus, TTP plays an important role in turning off IFN-gamma expression at the appropriate time

142 rate-limiting chemistry of phototransduction turn-off in vivo.

143 r findings indicate that the RASSF1A gene is turned off in a significant number of melanomas and that

144 In adults, Brg1 (also known as Smarca4) is turned off in cardiomyocytes.

145 g antisense agents that can be turned on and turned off in mammalian tissue culture.

146 ation: miR-211 is a molecular switch that is turned off in melanoma cells, raising the hope that in t

147 emonstrate that (independent of EBV) TET2 is turned off in normal and malignant germinal center (GC)

148 wnstream target of the GTPase Rheb, which is turned off in response to AMPK via the tuberous sclerosi

149 side by the mid-gastrula stage, and is then turned off in the dorsal ectoderm, the neural plate, at

150 xpression of episome maintenance proteins is turned off in the predominant long-term latency reservoi

151 The current rapidly turns off in the dark as the PTL relaxes thermally back

152 MCC that takes place when the checkpoint is turned off involves the conversion of C-Mad2 back to O-M

153 The addition of ligand turns off IRF3-dependent transcription and activates ERK

154 del, the transcription factor UNC-55/COUP-TF turns off IRX-1, thus maintaining high levels of OIG-1 t

155 rsed, if expression of the mutant protein is turned off, is unknown.

156 dation is necessary for the yeast to quickly turn off its hypoxic response when oxygen is restored.

157 K-bZIP is involved in a feedback circuit to turn off its own expression and possibly the expression

158 ts reveal a mechanism that enables CCaMK to 'turn off' its function through autophosphorylation.

159 re also demonstrate that hysteresis and slow turn off kinetics are the only two system properties ass

160 e LTi developmental pathway, but needs to be turned off later to avoid diversion to the T cell fate.

161 By turning off Lck, YopH blocks T cell antigen receptor sig

162 a leading kinetochore abruptly disengage or turn off, leading to a direction reversal.

163 in which all interspecific interactions are turned off leads to analytical results that are in agree

164 The coupling field is then turned off, leaving the atoms in the first condensate in

165 st participants mentioned curtailment (e.g., turning off lights, driving less) rather than efficiency

166 urthermore, ligated Notch-2 also efficiently turns off LMP1 expression from the ED-L1 promoter in LCL

167 H3K9me3 demethylation, and fast feedback to turn off Lsd1.

168 When the checkpoint signal is turned off, MCC is disassembled and the checkpoint is in

169 When the checkpoint signal is turned off, MCC is disassembled, a process required for

170 When the light was turned off motoneuron firing and locomotor frequency bot

171 We showed that bacteria that failed to turn off MSHA biosynthesis were unable to colonize the i

172 t models of phase precession, we transiently turned off neuronal discharges for up to 250 ms and rese

173 ransition indicates that osteoblasts neither turn off nor slow down their matrix secreting genes--a w

174 differentiation at least partly because they turn off Notch3.

175 s of a ligand at T3beta, but not at T3alpha, turns off O(2) reactivity, indicating that T3beta and T2

176 proteins) are responsible for the subsecond turn off of G protein signaling and are inhibitors of si

177 G protein signaling (RGS proteins) speed the turn-off of G protein signals and inhibit signal transdu

178 beta-Arrestins, proteins involved in the turn-off of G protein-coupled receptor (GPCR) activation

179 crease in maximal rate of release and faster turn-off of release (such that integrated SR Ca(2+) rele

180 ong negative feedback that allows for a fast turn-off of the NF-kappaB response, whereas IkappaBbeta

181 tly more robust than the analysis based on a turn-off of the signal due to free magnetic nanobeads us

182 acute restraint stress followed by a delayed turn-off of the stress response.

183 n to their well-known role in regulating the turning off of Rh*, can also modulate the decay of light

184 context-contamination" leads to voluntarily "turn off" of negative states.

185 The XIST allele must be turned off on one X chromosome to maintain its activity

186 How the SAC signal is turned off once all the chromosomes have successfully at

187 Mammalian X inactivation turns off one female X chromosome to enact dosage compen

188 rformed in real-time and without the need to turn off operating room lights.

189 ut how the tectum's saccade-related activity turns off OPNs is not known.

190 r phosphatase activity and is monitored as a turn-off or turn-on signal, respectively.

191 -spin ions such as Cu and Ni can be entirely turned off or on, respectively, whereas the magnetic con

192 hanisms whereby inflammation can be reduced, turned off or perhaps prevented, both in the context of

193 osphorylation-sensitive molecular switch for turning off or on RP mRNA translation and subsequent rib

194 urified DNA fragments are released by simply turning off or reversing the electric field.

195 Interaction with tetracycline turns off or turns on the tTA and rtTA molecules, respec

196 g the AP axis, using transgenes that rapidly turn "off" or "on" BMP signaling.

197 Either receptors reach full methylation and turn off, or receptors become saturated and cease to res

198 on-bound replication initiator (Rep) protein turns off origin function.

199 signals and pathways that turn on ospC also turn off ospA.

200 that Nkx2.2 functions as a major 'switch' to turn off Pdgfra signaling in OPCs and initiate the intri

201 erved when the SPDa insulin infusion was not turned off (peak change within 5-10 min = 44 +/- 85 pg/m

202 ), showing that in Schizosaccharomyces pombe turning off peroxide defenses preserves the pool of redu

203 the antioxidant enzymes may function in the "turn-off" phase of signal transduction by ROS.

204 Hh stimulation, thus maximizing signaling by turning off PKA activity and hence Gli3 repressor format

205 in cellular grafts after NT-3 expression was turned off possibly via reduction in L1 and laminin expr

206 e after-images of the stimuli when they were turned off ('prime-sight').

207 A fluorescence "turn-off" probe has been designed and successfully appli

208 This turns off quenching, increases emission intensity, and s

209 hase, when an active p53 program needs to be turned off quickly.

210 otential progenitors of the distal lung from turning off RA signaling by locally expressing constitut

211 Ethers and chloride salts dampen or turn off reactivity, leading to three distinct catalyst

212 n just clathrin adaptor proteins involved in turning off receptor signals; they are actually capable

213 cience under an evolutionary framework risks turning off researchers who have their own theoretical p

214 Both induction and turn-off responses were fast.

215 hereas the Rh5 photoreceptors switch fate by turning off Rh5 and then turning on Rh6 expression.

216 izes kinetochore microtubule attachments and turns off SAC activity.

217 it displays a ratiometric response, while a "turn-off" sensing is observed specifically in the presen

218 discovery that MBBI can serve not only as a turn-off sensor via the simultaneous inclusion of a Trp

219 In the second approach, a fluorescence turn-off sensor was developed that relies on a peptide-d

220 Taking advantage of this phenomenon, a "turn-off" sensor was developed, which is easy to control

221 expression), induce Shh expression, and then turn off Shh responsiveness.

222 asome-mediated degradation through ERK1/2 to turn off signaling.

223 i subunits of heterotrimeric G-proteins that turns off signaling by G-protein coupled receptors.

224 othing is known about Smad phosphatases that turn off Smad activity.

225 H(+) work arises because that chemical clock turns off sooner than the H2D(+) clock, but both results

226 e that inactivates Lck in the TCR signalling turn-off stage, leading to suppression of T-cell-mediate

227 e), but typically are more luminous than the turn-off stars and therefore long ago should have evolve

228 nt behavior that occurs after the forcing is turned off suggests that the double knocks of Campephilu

229 Here, we report a "turn-off" surface enhanced Raman scattering (SERS)-based

230 acity in adipocytes and participates in the "turn-off" switch that limits adipocyte fat accumulation

231 By turning off SynCAM 1 overexpression in transgenic brains

232 In the turn-off system enzyme-catalyzed peptide hydrolysis is s

233 MicroRNAs turn off target gene expression within cells by binding

234 l cells, by downregulating 14-3-3sigma, thus turning off TGF-beta's tumor suppression function.

235 the alpha expression, and it simultaneously turns off that of the beta and thus coordinates alpha an

236 ules that turn on the enzyme (activator) and turn off the activation (blocker).

237 Small molecules that can be used to turn off the activities of specific cellular proteins ar

238 ibited by GABA(A) receptors is sufficient to turn off the brainstem REM sleep-generating network; the

239 Once activated, the events that subsequently turn off the CARMA1 signalosome are unknown.

240 transdetermine to another fate, they fail to turn off the cell cycle and continue dividing long after

241 Thus Blimp1 probably acts to turn off the default pathway that allows epiblast cells

242 recombination, Srs2p appears to be needed to turn off the DNA damage checkpoint.

243 much-sought-after feature is the ability to turn off the dynamic exchange of the system, in order to

244 osa has evolved tight regulatory networks to turn off the energy-expensive T3SS when striving for sur

245 vo, and that this association is required to turn off the ethylene-signaling pathway.

246 n of the yeast poly(A) binding protein would turn off the expression of the PAB1 gene.

247 ressively attacks other coral-reef fish, can turn off the mimetic colours that disguise it as the ben

248 en at a particular time, and, in some cases, turn off the protein as development progresses or the an

249 Doxycycline administration can turn off the transgene expression allowing the restorati

250 Moreover, this challenge simultaneously turned off the IgE enhancers among the Vgamma1(+) cells.

251 with the Set8 induction being important for turning off the activation of Smad2.

252 ntiangiogenic switch that is responsible for turning off the antiangiogenic activity of the native se

253 progression (a process called "recovery") by turning off the checkpoint.

254 assembly factors (CAFs), Asf1 and CAF-1, in turning off the DNA damage checkpoint in budding yeast.

255 ed genes associated with cytotoxic stress by turning off the expression of IFNbeta.

256 hat LCUFAs inhibit PhoQ autokinase activity, turning off the expression of the PhoP-dependent regulon

257 We show that in addition to turning off the immune/eIF2alpha phosphorylation-depende

258 (PTEN), a phosphatase critically involved in turning off the insulin signal transduction cascade, to

259 ns, the second enzyme outcompetes the first, turning off the latter's response.

260 By rapidly turning off the mitochondria using depolarization of the

261 Obstructive apnea was induced by turning off the respirator during end expiration for 2 m

262 ge also destroys the tension responsible for turning off the SAC, cells must possess a mechanism to p

263 otecting cohesin, plays an important role in turning off the SAC, in promoting the congression and bi

264 Therefore, these residues may play a role in turning off the signal transmitter domain of the recepto

265 Surprisingly, we found that turning off the transgene expression before the onset of

266 Turning off the transgene expression for 3 weeks reverse

267 d curtailment (e.g., taking shorter showers, turning off the water while brushing teeth) rather than

268 segment morphology is not achieved merely by turning off the wing and mesothoracic development progra

269 bulb in a series of Christmas lights, which turns off the entire string.

270 d by a combination of gene repression, which turns off the genes associated with most possible lineag

271 GTP hydrolysis turns off the GTPase switch by converting it to the inac

272 This step turns off the IKK/NF-kappaB/SOD2 antioxidant pathway.

273 This substitution turns off the intrinsic TL-dependent hydrolytic activity

274 Light beam illumination on a phototransistor turns off the trap and releases the trapped cell, which

275 against a series of metal cations to first "turn-off" the fluorescence.

276 e, we show that larval zebrafish essentially turn off their visual system at night when they are not

277 er nTregs can convert into effector cells by turning off their Foxp3 expression and, if so, whether T

278 ploited and it is shown how to spontaneously turn off these dipolar capillary interactions by exceedi

279 Mechanisms for turning off this enzyme once released, preventing unwant

280 A point mutation turns off this motor's contribution to phagocytosis, sug

281 o turn on anion transport, which can then be turned off through addition of the blocker dodecyl sulfa

282 "Turn-ON" and "turn-OFF" time course experiments were performed with th

283 apter) by Act1/CIKS (IL17R adapter), thereby turning off TLR2 signaling to restore homeostasis.

284 n on (to increase their firing rate) than to turn off (to reduce their rate).

285 ce a DNA-bound holo-CueR with apo-CueR, thus turning off transcription promptly and facilely.

286 Turning off transcription promptly is important, however

287 nderstood how genes in the first wave become turned off transcriptionally and how their precise timin

288 0.001 to 100000ng/mL (R(2) = 0.9339) by the turn off-turn on principle.

289 Importantly, in distant sites, turning off Twist1 to allow reversion of EMT is essentia

290 ivation limits the rate of phototransduction turn-off under these conditions.

291 A reproducible shift in the turn-off voltage of the semiconducting SWNT FETs was see

292 res VPS34, indicates that MTMs do not simply turn off VPS34 but unexpectedly also function as positiv

293 (ryanodine receptors or RyRs) can be made to turn off when their activity is sustained by the Ca2+ re

294 a de novo assembly pathway that is normally turned off when centrioles are present.

295 However, how this pathway is turned off when hepatocyte proliferation is no longer re

296 d by the integrated fluorophores, which are "turned off" when the particles become destructed and dye

297 nificantly declined when NT-3 expression was turned off, whereas continued NT-3 expression sustained

298 ngth, remain on in the dark for minutes, and turned off with millisecond long pulses at the other wav

299 subsequent peeling performance when AFF was turned off, with reductions in average force, SD of forc

300 ts expressed in resting T cells were rapidly turned off within 30 min after activation.