ライフサイエンスコーパス: twin study

1 in a large epidemiologic study (the Missouri Twin Study).

2 Prospective, classic twin study.

3 nd 606 males) from the Brisbane Longitudinal Twin Study.

4 ed from the Arthritis and Rheumatism Council Twin Study.

5 a from a prospective, four-wave longitudinal twin study.

6 ve National Heart, Lung, and Blood Institute Twin Study.

7 t for NHL, identified from the International Twin Study.

8 shown a substantial heritability of AF in a twin study.

9 d heart rate and movement sensing in a large twin study.

10 easons for different outcomes of these large twin studies.

11 is also consistent with the results of adult twin studies.

12 evidence of a genetic basis in familial and twin studies.

13 support for this is derived from family and twin studies.

14 major consequences for the interpretation of twin studies.

15 d by familial aggregation (lambdas = 20) and twin studies.

16 These five findings arose primarily from twin studies.

17 A demethylation in SLE was confirmed through twin studies.

18 mesial temporal lobe epilepsy has come from twin studies.

19 genotypes, using methods analogous to human twin studies.

20 In the International Twin Study, a case-control study of melanoma risk factor

21 ted from the Environmental-Risk Longitudinal Twin Study, a nationally representative 1994-1995 birth

22 the Environmental Risk (E-Risk) Longitudinal Twin Study, a nationally representative birth cohort of

23 In the Environmental Risk Longitudinal Twin Study, a nationally representative study of 1,106 f

24 ata from the Environmental Risk Longitudinal Twin Study, a representative British sample of 1116 twin

25 ar models to analyze data from a Children of Twins Study, a quasiexperimental design, to explore the

26 Twin studies, adoption studies, and studies of familial

27 This large twin study aimed to explore the role of shared environme

28 Twin studies allow us to estimate the relative contribut

29 A large twin study also points away from genetic influences as i

30 We reinterpret twin studies and demonstrate the use and advantages of a

31 alian families in the Tasmanian and Brisbane twin studies and from 661 participants in the 20-year fo

32 lizing liability than previously detected in twin studies and indicates that sibling rather than pare

33 Familial aggregation, twin studies and segregation analyses suggest that there

34 Twin studies and variations in the outcome of tuberculos

35 ported by analyses of ethnic predisposition, twin studies, and familial aggregation.

36 es of migraine, large-scale population-based twin studies, and linkage and association studies of fam

37 e Study in Tasmania, the Brisbane Adolescent Twin Study, and the Western Australian Pregnancy Cohort

38 Family and twin studies, animal models and gene association studies

39 Human monozygotic twin studies, animal studies, and genetic models demonst

40 Twin studies are a classic tool for assessing the influe

41 tive effects of common DNA variants, whereas twin studies are not.

42 Family and twin studies attest to the importance of genetic factors

43 A large twin study based on 458 pairs of monozygotic and 1099 pa

44 etween ASD and ADHD symptoms is supported by twin studies, but the genetic overlap between clinically

45 from the social environment of rearing, and twin studies can often give clues to the importance of b

46 t with estimates of heritability obtained in twin studies carried out in the same population.

47 xamination II of the Kaiser Permanente Women Twins Study carried out in 1989-1990 in Oakland, Califor

48 Twin studies circumvent some of these problems, because

49 y has been indicated by familial clustering, twin studies, complex segregation analyses and human leu

50 Family and twin studies confirm that chronic insomnia can have a ge

51 This twin study confirms the existence of an inverse relation

52 es; (b) cross-sectional human studies; (c) a twin study; (d) studies of changes in cognition with abs

53 Twin studies demonstrated genetic covariance (pleiotropy

54 Here, our classical twin study demonstrated that the variance of FC levels i

55 nary heart disease (CHD) using a prospective twin study design and objective measures of CHD.

56 nalysis of 2301 twin pairs confirms that the twin study design results in a very low estimate of shar

57 The power of the twin study design to detect these differences was examin

58 In this report, we used a classic twin study design to examine the role of genetic and env

59 We used a cross-sectional twin study design to examine the role of heredity in lik

60 A twin study design was used to examine conduct problem sc

61 we combine the power of a within-monozygotic twin study design with longitudinal neuroimaging methods

62 We estimated the heritability of GGT using a twin-study design and examined the genetic covariance be

63 and colleagues combine structural MRI and a twin-study design to investigate the influence of geneti

64 (MZ) twins were selected from an adolescent twin study designed to investigate the interplay of gene

65 This means twin study designs have more power to detect heritable e

66 Sibling and twin study designs provide control for confounding facto

67 The Environmental Risk Longitudinal Twin Study (E-Risk) comprised 2163 twin children from En

68 In contrast to the substantial twin study estimates of heritability which ranged from 0

69 A recent classic twin study examined systematically the role of genetic a

70 ctors for DR, including heritability scores, twin studies, family studies, candidate gene studies, li

71 e authors identify more than one large-scale twin study for meta-analysis.

72 In a twin study, GGT shared genetic codetermination with trai

73 Twin studies have also found high heritabilities for man

74 Twin studies have been either very small or based on par

75 Family and twin studies have clearly established a genetic componen

76 Molecular genetic studies and twin studies have confirmed important genetic influences

77 Twin studies have demonstrated a high heritability for r

78 Although family studies and twin studies have demonstrated genetic influences on the

79 Twin studies have established that both genes and enviro

80 Twin studies have established that genes and aspects of

81 Recent twin studies have found the ability to memorize and reco

82 Numerous large twin studies have indicated a significant genetic contri

83 Twin studies have indicated that this has a heritability

84 Twin studies have indicated that tuberculosis susceptibi

85 Genetic effects demonstrated in twin studies have led to molecular analyses to elucidate

86 Twin studies have played an essential role in estimating

87 While twin studies have previously demonstrated high heritabil

88 Twin studies have provided evidence that both genetic an

89 Data from twin studies have shown a genetic contribution to the de

90 Twin studies have shown that a significant proportion of

91 Twin studies have shown that anxiety in a general popula

92 Twin studies have shown that genetic factors contribute

93 Familial clustering and twin studies have suggested a genetic component for type

94 uggesting a genetic contribution to EoE, and twin studies have suggested a powerful contribution from

95 studies have suggested a genetic component, twin studies have suggested that little genetic contribu

96 Twin studies have suggested that many 'sporadic' cancers

97 Previous twin studies have supported a genetic contribution to th

98 Twin studies have underscored the contribution of additi

99 the gene responsible for the disorder in the twins studied here.

100 The gap between GCTA and twin study heritabilities will make it challenging to id

101 ry low for CU in our study, despite its high twin study heritability estimate.

102 However, GCTA heritability, not twin study heritability, is the ceiling for GWA studies

103 NA variants identified in GWA studies versus twin study heritability.

104 Furthermore, although twin studies in nontransplant patients with NMSC suggest

105 cal debates and reinterpretation of adoption/twin studies in the 1980s regarding intelligence suggest

106 atologic variables, we conducted a classical twin study in unselected twins.

107 This cross-sectional, population-based twin study included 34781 Danish twins, 20 to 71 years o

108 While twin studies indicate a significant role for genetic fac

109 Although twin studies indicate clear genetic bases of autism spec

110 Family and twin studies indicate substantial overlap of genetic inf

111 Twin studies indicate that 35% of all colorectal cancers

112 Twin studies indicate that ASD susceptibility has a larg

113 on, and severity of this disease, family and twin studies indicate that genetic variation also influe

114 Twin studies indicate that half of the variation in this

115 Twin studies indicate that the ability to quit smoking i

116 These DNA results confirm the results of twin studies indicating strong genetic stability but inc

117 This co-twin study investigated monozygotic twins who were disco

118 A population-based twin study is a useful design for quantification of the

119 sing genetically informative designs such as twin studies, it is possible to show that the causes of

120 Five twin studies met the inclusion criteria, and their stati

121 detailed response to several defenses of the twin study methodology; and in (4) I conclude with sever

122 d examination of the Kaiser Permanente Women Twins Study (Oakland, California, 1989-1990).

123 Twin studies of adolescent anxiety demonstrate both gene

124 iew presents selected recent developments in twin studies of adult psychiatric disorders.

125 esent study are comparable to those found in twin studies of adult substance use and substance use di

126 transmission, gene-environment interaction, twin studies of anxiety and eating disorders, the so-cal

127 Twin studies of bipolar affective disorder (BPD) have ei

128 ependent twin pairs, virtually all published twin studies of complex traits.

129 family environment, special issues raised by twin studies of drug use and abuse, and gene-environment

130 Three large scale population-based twin studies of headache have revealed that approximatel

131 ducted meta-analyses of data from family and twin studies of panic disorder, generalized anxiety diso

132 hese meta-analytic results from 12 published twin studies of schizophrenia are consistent with a view

133 There are many published twin studies of schizophrenia.

134 1 pubertal male twin pairs from the Virginia Twin Study of Adolescent Behavioral Development.

135 asions spanning 28 y in the Swedish Adoption/Twin Study of Aging and parental social class based on t

136 ained in a separate twin sample (Vietnam Era Twin Study of Aging), but birth weight in the child samp

137 children (N=2,620 children) from the Swedish Twin Study of Child and Adolescent Development reported

138 in Sweden) (n = 2309) and the TCHAD (Swedish Twin Study of Child and Adolescent Development) (n = 124

139 We performed a classical twin study of CML in healthy nondiabetic female twins, 3

140 The only large, registry-based twin study of depression using diagnostic criteria asses

141 In a twin study of face recognition, we found that the correl

142 We performed a classical twin study of HbA(1c) in healthy nondiabetic female twin

143 We conducted the first twin study of language delay to evaluate whether genetic

144 Pairs were selected from the Virginia Adult Twin Study of Psychiatric and Substance Use Disorders fo

145 ale and female twins from the Virginia Adult Twin Study of Psychiatric and Substance Use Disorders, w

146 f Schizophrenia Franz Kallmann's influential twin study of schizophrenia in 691 twin pairs was the la

147 In a population-based twin study of schizophrenia, heritability was estimated

148 TCHAD study and in the Child and Adolescent Twin Study of Sweden (CATSS) (n = 1771), the association

149 tic twin pairs from the Child and Adolescent Twin Study of Sweden (CATSS).

150 autoantibodies were determined in a classic twin study of twin pairs discordant for type 1 diabetes

151 ast cancer susceptibility genes arising from twin studies, pedigree analysis and studies of phenotype

152 Twin studies provide compelling evidence that alcohol an

153 and guilt) (1) ; heritability estimated from twin studies ranges from 30 to 50% (2) , and SNP-based h

154 Twin studies remain a vibrant part of the field of psych

155 discordant twin pairs from the International Twin Study returned questionnaires (70% response).

156 Comparing GCTA results to the results of twin studies reveals important insights into the genetic

157 Twin studies routinely estimate the heritability of CU a

158 Twin studies show the concordance in monozygotic twins t

159 Twin studies suggest a definite but relatively weak gene

160 amily studies in clinical samples as well as twin studies suggest a familial liability and consequent

161 defects account for up to 2-6% of cases, but twin studies suggest a hereditary component in 35%.

162 Twin studies suggest a heritable component to circulatin

163 is not fully understood, but population and twin studies suggest a large heritable component to the

164 Epidemiological and twin studies suggest that ADHD frequently co-occurs with

165 Twin studies suggest that alcohol-consumption patterns a

166 Twin studies suggest that conduct disorder (CD) is under

167 Twin studies suggest that economic outcomes and preferen

168 Twin studies suggest that expressive vocabulary at ~24 m

169 Family and twin studies suggest that genes play a role in male sexu

170 Family and twin studies suggest that juvenile-onset major depressiv

171 Family and twin studies suggest that liability for suicide attempts

172 While family and twin studies suggest that retrospectively reported preme

173 Prior evidence from twin studies suggested genetic moderation of the depress

174 Evidence from twin studies suggests a genetic component to caries.

175 Although twin studies support the heritability of VUR, specific g

176 ritability is more akin to those of previous twin studies than previous family studies.

177 e basis of a review of family, adoption, and twin studies that met specific inclusion criteria, the a

178 dications from epidemiological and identical-twin studies that MS is infectious, no virus or other in

179 In the twin study, the heritability of perinatal depression was

180 We did a classic twin study to assess heritabilities of these haemostatic

181 In the largest twin study to date, we confirmed that heritability for A

182 We performed a classical twin study to evaluate the relative contributions of gen

183 We used a twin study to investigate the genetic and environmental

184 els in the 3 cohorts and performed a classic twin study to partition variation in CUVAF.

185 Increasing knowledge from family and twin studies using contemporary diagnostic methods will

186 Twin studies using selected samples have shown high heri

187 A cross-sectional twin study was conducted from August 27, 2014, to March

188 A classic twin study was conducted using COMP levels in serum obta

189 This classic twin study was designed to determine the heritability of

190 A classical twin study was performed to establish the relative impor

191 A classical twin study was performed to examine the relative importa

192 The twin studies were conducted between 2001 and 2009, and t

193 Among these cancers, previous twin studies were only able to show heritability for pro

194 The aims of the present twin study were to: (i) assess the heritability of HCC;

195 ere from the Environmental Risk Longitudinal Twin Study, which follows 2,232 twin children and their

196 IGN, SETTING, AND PARTICIPANTS: Multivariate twin study with BPD criteria assessed by personal interv