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1 1.27), oral 0.34 degrees C (-0.86 to 1.54), tympanic 0.62 degrees C (-0.40 to 1.64) and mode not sta
2 Clinical examination revealed a distended tympanic abdomen with generalized tenderness but no evid
4 na, mediolateral abbreviation of the lateral tympanic, and a shortened, trapezoidal basioccipital ele
5 ed that it showed evidence of this stylohyal-tympanic articulation, from which they concluded that O.
7 that supports the throat and larynx) and the tympanic bone, which forms the floor of the middle ear.
8 helium is terminally differentiated, whereas tympanic border cells (TBCs) beneath the sensory epithel
9 se of temperature measurement with the oral, tympanic (both core and oral equivalence modes were used
10 on of the limbic cortex against the adjacent tympanic bulla and subsequent neuronal cytoskeletal coll
11 olesteatoma according to its location in the tympanic cavity (T); extension into the mastoid (M); and
12 omica cranial nerve VII was accessed via the tympanic cavity and injected with dextran coupled to Tex
13 unctional properties of the ossicles and the tympanic cavity and make comparisons with recent and ext
14 d Neandertals, leading to differences in the tympanic cavity and, consequently, the shape and spatial
15 icles (malleus, incus, stapes) housed in the tympanic cavity of the temporal bone play an important r
17 ee methods (oral-PA core, -0.15 [SD = 0.36]; tympanic core-PA core, -0.11 [SD = 0.57], tympanic oral-
21 within the catchment area and presented with tympanic (>/=38.0 degrees C) or axillary temperature (>/
22 chlear adenosine A1 receptor (A1AR) by trans-tympanic injections of the agonist R-phenylisopropyladen
24 oid the systemic treatment side-effects, the tympanic membrane (TM) represents an impenetrable barrie
26 he placement of a small drainage tube in the tympanic membrane (TM), is the most common surgical proc
28 es examined diagnosis; otoscopic findings of tympanic membrane bulging (positive likelihood ratio, 51
30 itis media unlikely whereas a distinctly red tympanic membrane increases the likelihood significantly
32 bias, a cloudy, bulging, or clearly immobile tympanic membrane is most helpful for detecting AOM.
33 ly immobile (adjusted LR, 31; 95% CI, 26-37) tympanic membrane on pneumatic otoscopy are the most use
38 e response to pyrogen, decreasing integrated tympanic membrane temperatures from 7.5+/-2.2 degrees C
39 the probe tube is sufficiently close to the tympanic membrane to capture the highest frequency of in
43 ization of middle ear structures through the tympanic membrane, including the ossicular chain, promon
45 ates disease in the external auditory canal, tympanic membrane, or middle ear; CT with thin bone algo
49 proportions of children with healing of the tympanic membranes by 16 weeks were 15% (10-21) in the d
51 speaker in free space were delivered to the tympanic membranes of barbiturized cats via sealed and c
52 were resolution of otorrhoea and healing of tympanic membranes on otoscopy by 8, 12, and 16 weeks af
57 33 (SD = 0.89); oral, x = 37.18 (SD = 0.92); tympanic oral, x = 36.80 (SD = 0.93); and tympanic core,
58 ]; tympanic core-PA core, -0.11 [SD = 0.57], tympanic oral-PA core, -0.52 [SD = 0.53]), indicating th
59 icating that non-specific stimulation of the tympanic plexus, an intervening neural structure with va
60 s and malleus) and two membranous bones, the tympanic ring and the gonium, which act as structural co
64 Gsc-null cells had the capacity to form the tympanic ring condensation in the presence of wild-type
65 The participation of Gsc-null cells in the tympanic ring condensation of chimeras may be an epigene
67 xternal acoustic meatus and formation of the tympanic ring, a first arch-derived membrane bone that a
68 sc is required for development of the entire tympanic ring, the role of Bapx1 is restricted to the sp
73 between +0.25 and -1.4 MegaPascal (MPa) for tympanic rupture, +3 and -1 MPa for lung damage, and +20
74 and orbit apex for pressures known to cause tympanic rupture, lung damage, and 50% chance of mortali
75 derived condition associated with changes in tympanic shape and the extent of its contact with the pe
79 induction of profound cerebral hypothermia (tympanic temperature 10 degrees C) by aortic flush of co
81 a venovenous extracorporeal shunt cooling to tympanic temperature 27 degrees C; in group 3 (n = 6, 2
82 bypass, postcardiac arrest mild hypothermia (tympanic temperature 34 degrees C) to 12 hrs, controlled
84 group 2 but with mild hypothermia, that is, tympanic temperature 34 degrees C; and in group 4 (n = 5
85 ective of this study was to compare oral and tympanic temperature measurements (in both the oral and
86 ons of up to 90 mins, perhaps 120 mins, at a tympanic temperature of 10 degrees C and complete recove
88 at 2 degrees C (at a rate of 1 L/min), until tympanic temperature reached 20 degrees C (for 60 mins o
90 iac arrest of up to 90 min no-flow at brain (tympanic) temperature of 10 degrees C, with functionally
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