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1 helial growth factor, PDGF, TGF-beta(1), and type 1 collagen).
2 sion of the major component of fibrotic ECM, type 1 collagen.
3 utes to their increased production of FN and type 1 collagen.
4 duced by vitronectin and fibronectin but not type 1 collagen.
5 1 also can detach cells from fibronectin and type-1 collagen.
6 dronate: 55.6% (P<.001) for C-telopeptide of type 1 collagen, 59.5% (P < .001) for serum n = propepti
7 the constitutively exposed binding motif of type 1 collagen and its receptor integrin alpha2 was sur
8 s through a microfluidic channel coated with type 1 collagen and observe the rate at which platelets
9 phatase (ALP), osteocalcin (OCN), alpha 2(1)(type 1) collagen and osteonectin (ON), were performed.
10 59.5% (P < .001) for serum n = propeptide of type 1 collagen, and 28.1% (P<.001) for bone-specific al
11 Markers of bone turnover, N-telopeptides of type 1 collagen, and bone alkaline phosphatase decreased
12 er, it remains unclear how fibronectin (FN), type 1 collagen, and their receptor integrin subtypes di
13 A and protein expressions of fibronectin and type 1 collagen associated with the activation of p38 mi
14 ally distinct collagenases (Western blot), a type-1 collagen breakdown product/bone resorption marker
15 composed of 5 to 8 lamellae of predominantly type-1 collagen bundles arranged in transverse, longitud
17 ven platelet deposition in vitro on purified type 1 collagen by video phase-contrast microscopy at 22
18 These results demonstrate that fibrillar type 1 collagen can actively disrupt cell cycle progress
20 ) and resorption (C-terminal telopeptides of type 1 collagen (CTX)), and the K59N polymorphism in the
23 also inhibits osteoblast differentiation and type 1 collagen expression in a Runx-2- and osterix-inde
24 vitro, CLDH exhibited increased vimentin and type 1 collagen expression within cellular extensions co
25 oxia also upregulated VEGF, fibronectin, and type 1 collagen expressions associated with HIF-1alpha a
27 nd cultured monocyte/derived Mphi adhered to type 1 collagen gels, but not to nongelled collagen-, fi
28 atic stellate cells as well as expression of type 1 collagen genes and tissue inhibitor of matrix met
29 inal propeptide (PICP), amino-telopeptide of type 1 collagen (ICTP), collagen VI, desmosine, matrix m
30 switching NIH3T3 fibroblasts from growth on type 1 collagen in monolayer to a collagen gel might inf
31 addition, mixing recombinant periostin with type 1 collagen in solution caused a dramatic increase i
32 ited increased deposition of fibronectin and type 1 collagen, increased chemotaxis, and decreased pro
33 rms, if their increased expression of FN and type 1 collagen is under autocrine TGF beta control.
34 oculture with chloroquine or by adherence to type 1 collagen matrices was not reversed by bafilomycin
35 monocyte-derived macrophages that adhered to type 1 collagen matrices, but not to fibronectin, vitron
36 oth alpha-smooth muscle actin expression and Type 1 collagen mRNA levels in livers of mice fed a West
37 depositing basement membrane materials onto type 1 collagen nanofibers only in a region adjacent to
39 ne (fDPD/Cr), serum N-terminal propeptide of type 1 collagen, or beta C-terminal telopeptide, althoug
40 r = 0.003) as well as FN (P < or = 0.04) and type 1 collagen (P < or = 0.03) (measured by specific EL
41 alone, the neutralizing antibodies decreased type 1 collagen production by the HGF fibroblasts by up
44 colorectal cancer cells in three-dimensional type 1 collagen reverts the invasive phenotype and resto
45 ment, mitochondrial dysfunction, and reduced type 1 collagen secretion and alkaline phosphatase activ
46 3 ng/mL; P=0.22) and C-terminal crosslink of type 1 collagen (soy-fed: 0.944 +/- 0.06 and 0.89 +/- 0.
47 oterminal propeptide of type III procollagen/type 1 collagen telopeptide ratio </=1 (odds ratio [95%
48 oterminal propeptide of type III procollagen/type 1 collagen telopeptide ratio </=1, measured 1 month
49 oterminal propeptide of type III procollagen/type 1 collagen telopeptide ratio (</=1) at 1 month is p
50 s well as biomarkers of myocardial fibrosis (type 1 collagen telopeptide, aminoterminal propeptide of
51 s of the ECM components fibronectin (FN) and type 1 collagen than normal human gingival (GN) fibrobla
52 of alpha smooth muscle actin (alpha-SMA) and type 1 collagen, the markers of HSCs activation and prol
53 o of urinary cross-linked N:-telopeptides of type 1 collagen to creatinine with alcohol consumption,
54 protein, cytochrome P4502E1, cytokeratin-18, type-1 collagen, transforming growth factor-beta 1, matr
55 rosslinks and serum N-terminal propeptide of type 1 collagen) were measured, 3199 women completed a f
56 tion, FimC and FimD bound to fibronectin and type 1 collagen, whereas FimE failed to interact with th
57 when it is responsible for the production of type 1 collagen, which causes scar formation in liver fi
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