ライフサイエンスコーパス: type A

1 is uncommon for conventional photocatalysis (type A).

2 type B, but this region was absent from PhV1 type A.

3 rmed the presence of Clostridium perfringens type A.

4 er the implicated pruno yielded C. botulinum type A.

5 centrations independent of serum osmolality (type A); 14% had copeptin concentrations that increased

6 94), followed by type C (22% [21 of 94]) and type A (20% [19 of 94]).

7 D independently of sentinel dissection type (type A: 4.3 cm [3.5-5.6 cm] versus 3.3 cm [2.9-3.7 cm],

8 consecutive patients with aortic dissection type A, 74 with Marfan syndrome (58% men; median age, 37

9 Toxin type A accounted for half of outbreaks, and these outbre

10 romoter-lacZ transcriptional fusions in wild-type A. actinomycetemcomitans and DeltaihfA and Deltaihf

11 binds to its intended pharmacologic target (type A ADR) or an unintended target (type B ADR).

12 ence of the C- allele compared with the wild type A-allele.

13 e distinct polypeptide patterns of 6 porcine type A and 6 bovine type B gelatines at molecular weight

14 Ts, we detected the GTF2I mutation in 82% of type A and 74% of type AB thymomas but rarely in the agg

15 When type A and B curves were included, the sensitivity was 1

16 features of the defibrillators, although the type A and B defibrillators accounted for a significantl

17 recapitulated in ASM-deficient Neimann-Pick type A and B fibroblasts.

18 Interactions of Type A and B flavan-3-ol dimers (procyanidins) and sever

19 Since type A and B subspecies are closely related, we hypothes

20 ously, we profiled the antibody responses in type A and B tularemia cases in the United States using

21 tar is at the dividing line between stars of type A and B, and we measure the dayside temperature of

22 a virus samples determined positive for both type A and B.

23 zed whether NanI is similarly important when type A and C human intestinal disease strains attach to

24 All type A and C human intestinal strains bound to Caco-2 ce

25 Type A and C terminals contacted CAMKIIalpha-positive pr

26 arted in 16 neonates diagnosed with MoCD (11 type A and five type B) and continued in eight type A pa

27 rst effective therapy for patients with MoCD type A and has a favourable safety profile.

28 Four patients had type A and nine had type B dissection.

29 genotypes of a novel virus, designated PhV1 type A and PhV1 type B.

30 adverse prognosis; clinical outcomes of non-type A and type A patients with wild-type KIT were simil

31 rties of CBM_E1 are at the interface between type A and type B CBMs.

32 and descending aorta have been classified as type A and type B dissections, respectively.

33 was started in neonates diagnosed with MoCD (type A and type B) following a standardised protocol.

34 imultaneous extraction of 12 trichothecenes (type A and type B) from baby foods, followed by gas chro

35 t an unprecedented 'double K-turn' module in type A and type M archaeal RPR variants.

36 evolution or introduction of high-titre RSV type-A and B infections that seeded HiT clades in the su

37 RSV type-A and B infections were most closely related to RSV

38 rapidly and severely than illness caused by types A and B botulinum neurotoxin.

39 ed more than 2,000 confirmations for each of types A and B over the study period.

40 m cases worldwide are due to botulinum toxin types A and B.

41 and have been linked to Niemann-Pick disease types A and B.

42 Budding yeast cells exist in two mating types, a and alpha, which use peptide pheromones to comm

43 The sample titres between RSV types-A and B were not significantly different.

44 lt angles of 29 +/- 4 degrees (monolayers of type A) and 38 +/- 4 degrees (type B).

45 , Ciona robusta (formerly Ciona intestinalis type A) and C. intestinalis (formerly Ciona intestinalis

46 tularensis: F. tularensis subsp. tularensis (type A) and F. tularensis subsp. holarctica (type B).

47 ition through GABAA (gamma aminobutyric acid type A) and glycine receptors depends on the presence of

48 Nine patients had acute, 2 had chronic type A, and 3 had chronic type B aortic dissections befo

49 onic anhydrase, Sushi, Von Willebrand factor type A, and chitin binding, were identified from all the

50 , serotonin type 3, gamma-amminobutyric acid type A, and glycine receptors are major players of human

51 and enables transmission in pigs; (ii) wild-type A/Anhui/1/2013 (H7N9) shows modest replication, vir

52 gnosis is urgently needed for acute Stanford type A aortic dissection (AAAD) patients due to its high

53 is study was to compare the results of acute type A aortic dissection (ATAAD) repair before and after

54 Data on outcomes after Stanford type A aortic dissection in patients with Marfan syndrom

55 tic root diameter prior to or at the time of type A aortic dissection tended to be smaller in patient

56 ed forty-one patients (13%) had AAS: 125 had type A aortic dissection, 53 had type B aortic dissectio

57 sely affects outcomes in patients with acute type A aortic dissection, but reliable quantitative data

58 ch as "complicated" and uncomplicated" acute type A aortic dissection, might help predict individual

59 ied from cardiogenic shock on day 50 after a type A aortic dissection, not related to treatment.

60 mes in patients undergoing surgery for acute type A aortic dissection.

61 pt abundance of a clade of the CK-responsive type-A Arabidopsis response regulator (ARR) genes increa

62 ngs is suppressed by loss of function of the type A ARR family member ARR5.

63 ARRs): type B ARRs (response activators) and type A ARRs (negative-feedback regulators).

64 of cytokinin-regulated genes, including the type-A ARRs, although it does not impair the cytokinin i

65 es not impair the cytokinin induction of the type-A ARRs.

66 open surgical repair is optimal for treating type A (ascending aorta) AAS, whereas thoracic endovascu

67 Botulinum toxin type A at 2 different preparations and doses (dose A: 3

68 s strains, detecting this toxin gene in some type A, B, and C strains but not in any type D or E stra

69 ite detection of botulinum neurotoxin (BoNT) types A, B, and E in complex matrixes, which is innovati

70 spanning protein channel (comprising subunit types a, b, b', and c) and a peripheral domain (subunits

71 Toxin types A, B, E, and F were identified as the causative ag

72 001 for both), but not in patients with only type A/B1 lesions (4.6% vs. 4.8%, p = 0.87, and 7.4% vs.

73 th high and low zinc levels compared to wild-type A. baumannii.

74 multiple CBFB-MYH11 fusion transcripts, with type A being most frequent.

75 etection of Clostridium botulinum Neurotoxin Type A (BoNT/A) in complex, real-world media.

76 Botulinum neurotoxin type A (BoNT/A) is a highly potent neurotoxin that elici

77 combinant derivative of Botulinum neurotoxin Type A (BoNT/A).

78 serum samples from all patients (11/11) with type A botulism within 5h.

79 en the non-pathogenic abpf2 mutants and wild-type A. brassicicola.

80 t season only; susceptibility titers against type A but not type B were consistent with this observat

81 ative to the thyroid, lesions categorized as type A by readers had mean attenuation difference (+/- s

82 In contrast, chondroitin sulfate types A, C, D, and E did not stimulate NF-kappaB activat

83 ), apoptotic debris, and chondroitin sulfate types A, C, D, and E.

84 The postsynaptic elements in type A-C synapses were identified with immunocytochemist

85 ion of two previously unidentified structure types: a C2-symmetric M(II)4L6 assembly with meridionall

86 -2,3 linkage in H1 subtype viruses; the wild-type A/California/07/2009 isolate, like most circulating

87 Modular cellulases contain non-catalytic type A carbohydrate-binding modules (CBMs) that specific

88 reported to be the principal constituent of type A, cardiac amyloid fibrils formed from wild-type TT

89 t improve despite a strict gluten-free diet (type A cases) and previously diagnosed coeliac patients

90 Although type A CBMs play a critical role in cellulose recycling,

91 with an N-terminal GFP domain, revealed that type A CBMs possess the ability to recognize different c

92 e ligand interacting platform illustrate how type A CBMs target their appended plant cell wall-degrad

93 th the beta-strands, is a typical feature of type A CBMs, although the expected affinity for bacteria

94 in the number of action potentials evoked in type A cells by AF test stimulation and a concomitant in

95 Type A cells were readily detected only in animals in wh

96 those driven synaptically by AF stimulation (type A cells) were concentrated in the deep half of the

97 assimilation capacity of canopies with wild type, a Chl-deficient mutant (Y11y11), and 67 other muta

98 area of 53 +/- 3 A(2)/molecule (monolayer of type A), compatible with the anticipated triangular pack

99 tudies are informative for individual cancer types, a comprehensive comparative study of tumorigenic

100 Ligands of type A, containing a small substituent at N-1' atom, and

101 n (DAR), which potently blocks Edn1 receptor type A, could benefit cell engraftment.

102 nd some irregular cuts on their surface with type-A crystallinity pattern, similar to cereals starche

103 The sensitivity of type A curve for detection of malignancy was 40%, and sp

104 In many cell types, a cytoskeletal structure called the ciliary rootl

105 nents reviewed their acute aortic dissection type A databases, which contained 1,821 patients.

106 Eight patients with type A disease rapidly improved under treatment and conv

107 lications, in-hospital mortality, retrograde type A dissection and follow-up mortality appeared lower

108 Emergency surgery for type A dissection in patients with Marfan syndrome is as

109 d blood pressure was higher in patients with type A dissections that were immediately fatal than in t

110 For patients undergoing surgical repair for type A dissections, the observed 30-day mortality decrea

111 hat VWF interacts with C3b through its three type A domains and initiates AP activation, although ass

112 Two varieties of dsRBD exist: canonical Type A dsRBDs interact with dsRNA, while non-canonical T

113 ndothelial cells via paracrine EDN1 receptor type A (EDNRA) activation.

114 h increased glomerular endothelin-1 receptor type A (Ednra) expression and increased circulating endo

115 The endothelin receptor type A (EDNRA) signaling pathway is essential for the es

116 ess consequences of replacing the endogenous Type-A Escherichia coli RNase P RNA with a Type-B homolo

117 angiotensin (Ang) II via endothelin receptor type A (ETAR) and Ang receptor type-1 (AT1R) activation

118 The lack of robust type A evidence with hard clinical outcomes on the benef

119 >10-fold protective effects against virulent type A F. tularensis challenge.

120 cacy against aerosol challenge with virulent type A F. tularensis in a species other than a rodent si

121 ability during intracellular infections with type A F. tularensis.

122 in anchoring method was used to produce wild-type a-factor that contains a C-terminal methyl ester al

123 e noncovalent interactions between component types, a finding that may aid the rational design of fun

124 d that the nanI gene was absent from typical type A food poisoning (FP) strains carrying a chromosoma

125 rointestinal (GI) symptoms of C. perfringens type A food poisoning and CPE-associated non-food-borne

126 Sporulation is critical for C. perfringens type A food poisoning since spores contribute to transmi

127 ion in SM101, a derivative of C. perfringens type A food-poisoning strain NCTC8798.

128 jor virulence determinant for C. perfringens type-A food poisoning, the second most common bacterial

129 , respectively, reported 4 possible cases of type A foodborne botulism to the US Centers for Disease

130 be dispensable for the acute pathogenesis of type A FP or type C enteritis necroticans.

131 We conclude that non-type A fusions associate with distinct clinical and gene

132 ontaining nucleotides, varying in nucleobase type (A, G, C, U, m(7)G), phosphate chain length (from m

133 om the light chain coding sequences of toxin types A-G.

134 equences of the 7 known bont genes for toxin types A-G.

135 nal botulinum antitoxins raised against BoNT types A-G.

136 Hyperpolarizing inhibition mediated by type A GABA (GABAA) receptors is dependent on chloride e

137 GluA1 synaptic levels after 48 h blockade of type A GABA receptor (GABAA R)-mediated inhibition with

138 rtant determinant of gamma-aminobutyric acid type A (GABA(A)) receptor (GABA(A)R)-mediated inhibition

139 odiazepines modulate gamma-aminobutyric acid type A (GABA(A)) receptors throughout the brain.

140 l application of the gamma-aminobutyric acid type A (GABA-A) agonist muscimol increased GSWD occurren

141 ovo mutations in the gamma-aminobutyric acid type A (GABAA ) receptor beta3 subunit gene GABRB3 and o

142 between the EC50 for activation of the GABA type A (GABAA) receptor by the transmitter GABA and basa

143 ssociated protein-interacting domain of GABA type A (GABAA) receptor subunit gamma2 (TAT-GABAgamma2)

144 e substituted mutant gamma-aminobutyric acid type A (GABAA) receptor with unique characteristics allo

145 Inhibitory GABA type A (GABAA) receptors critically regulate brain funct

146 tasis maintenance of gamma-aminobutyric acid type A (GABAA) receptors dictates their function in cont

147 same binding site on gamma-aminobutyric acid type A (GABAA) receptors was evaluated for its ability t

148 r affinity for brain gamma-aminobutyric acid type A (GABAA) receptors.

149 erpolarizing synaptic inhibition mediated by type A gamma-aminobutyric acid (GABAA) receptors, which

150 Type-A gamma-aminobutyric acid receptors (GABAARs) are t

151 rase gene designated csaA (capsule synthesis type a gene A).

152 In addition, in the absence of filamin, type-A glutamate receptor subunits are lacking at the po

153 pecifically correlated with the postsynaptic type-A glutamate receptors.

154 the activities of STN, Type-I GP (GP-TI) and Type-A GP (GP-TA) neurons in anaesthetised Parkinsonian

155 ffectiveness was 65% (95% CI, 44-79) against type A (H1N1) pdm09 but only 39% (95% CI, 23-52) against

156 Manufacturing Practice (GMP)-produced, wild-type A(H1N1)pdm09 virus was administered intranasally.

157 ) pdm09 but only 39% (95% CI, 23-52) against type A (H3N2).

158 tion that recapitulates critical features of type A hepatitis in humans.

159 f 2 cases of invasive Haemophilus influenzae type a (Hia) disease in Italy.

160 obligate multicellularity and had more cell types, a higher likelihood of sterile cells, and a trend

161 Compared to wild-type, a hip1 mutant strain of M. tuberculosis induced en

162 This increase was more prominent in type A ICs than in type B ICs.

163 The QAV was type A in 32% and type B in 32% (Hurwitz and Roberts cla

164 h patient'sforehead received botulinum toxin type A in saline injected with a 32-gauge needle; the ot

165 presses the most common human NPM1 mutation (type A) in the hematopoietic compartment.

166 Among patients with type A incident events, 18 (48.6%) died before hospital

167 gyrophilic grain disease); FTLD-TDP (55 nine type A including one with motor neuron disease, 27 type

168 ignificantly at 1 week after botulinum toxin type A injection (P = .003), and the effect persisted at

169 testes, p53R172H was expressed in gonocytes, type A, Int, B spermatogonia as well as in pre-Sertoli c

170 H(+) secretion in type A intercalated cells (A-ICs) is regulated by apical

171 secretion is mediated by highly specialized type A intercalated cells (A-ICs), which contain vacuola

172 Aortic dissection type A is a disease with high mortality.

173 polysaccharidosis IIIA or Sanfilippo disease type A is a progressive neurodegenerative disorder prese

174 uraminidase (NA) genes derived from the wild-type A/Japan/305/1957 (H2N2) (Jap/57), A/mallard/6750/19

175 Considering both cell types, a large number of transcripts showed significant

176 Type A lesions were higher in attenuation than the thyro

177 gists to categorize such arch dissections as type A lesions, thus making them an indication for surge

178 domain coding sequence and a nonneutralizing type A-like Hc binding domain coding sequence.

179 agnostic product ions (M) resulted mainly in type A (M - DMA) and B fragment ions (M - HO-B(N(CH3)2)2

180 of mitochondria is also observed in the wild-type: a majority of axon fragments containing a mitochon

181 Surprisingly, in all three cell types, a majority of Gag peptides derived from p15 rathe

182 t1 induces mating-type switching from mating type a (MATa) to MATalpha in the yeast K. lactis.

183 we further identified the scavenger receptor type A member I (SR-AI) to be a macrophage-specific rece

184 eroxidase is, by sequence analysis, a hybrid type A member of class I heme peroxidases [TcAPx-cytochr

185 contrast agent solution (perflutren protein-type A microspheres) was injected via the nephrostomy tu

186 BMs representative of the known diversity of type A modules.

187 mphoma (DLBCL) of the activated B-cell (ABC) type, a molecular subtype characterized by adverse outco

188 The mammalian ureter contains two main cell types: a multilayered water-tight epithelium called the

189 The possibility of MoCD type A needs to be urgently explored in every encephalop

190 Type A neurons increased their discharge rates across co

191 These differences may enable type A neurons to detect salient inputs that are focused

192 ostatin interneurons, preferentially inhibit type A neurons, leading to greater feedforward inhibitio

193 ypes: thick-tufted, subcortically projecting type A neurons, with prominent h-current, and thin-tufte

194 aller excitatory responses in type B but not type A neurons.

195 Callosal inputs also elicit more spikes in type A neurons.

196 is type IV (ML4) (TRPML1-F408), Niemann-Pick type A (NPA) and Fabry disease.

197 ed three specific genes [natriuretic peptide type A (Nppa), sarcolipin (Sln), and myosin light polype

198 In the present study, type A O-polysaccharide (OPS) and manno-heptose capsular

199 fficient to drive gene expression for either type A or B influenza virus with its cognate or heteroty

200 rnatant sialidase activity compared to other type A or C human intestinal strains.

201 Only patients with type A or O blood were included in the primary analysis,

202 imary analysis included 20,858 patients with type A or O blood.

203 athogen modifies its flagellin with either a type A or type B O-linked glycosylation system, which ha

204 pe A and five type B) and continued in eight type A patients for up to 5 years.

205 ognosis; clinical outcomes of non-type A and type A patients with wild-type KIT were similar.

206 urned to almost normal concentrations in all type A patients within 2 days, and remained normal for u

207 length of bouts was similar to that of wild types, a pattern that suggests diminished food motivatio

208 The starch and flour samples showed type A-pattern with strong reflection at 15, 18, and 23.

209 ubstrates, mutated transporter of the mating type a pheromone, Ste6* (sterile), and cystic fibrosis t

210 porcine alveolar macrophages (PAMs) and wild-type A. pleuropneumoniae (5b WT) or an Adh-deletion stra

211 ontaining Ephs and ephrins by different cell types, a process requiring endosomal sorting complex req

212 neurons of Ciona resemble an ancestral cell type, a progenitor to the complex neuronal circuit that

213 xtrasynaptic alpha-5 gamma-aminobutyric acid type A receptor (alpha5-GABAAR) regulates neuronal excit

214 tensin type 1 receptor (AT1R) and endothelin type A receptor (ETAR) is associated with allograft reje

215 acious modulators of gamma-aminobutyric acid type A receptor (GABA(A)) receptor function.

216 Gamma-amminobutyric acid type A receptor (GABAA-R) binding in the cerebellum was

217 The gamma-aminobutyric acid type A receptor (GABAA-R) is a major inhibitory neurorec

218 Modulation of the GABA type A receptor (GABAAR) function by cholesterol and oth

219 show reduced surface gamma-aminobutyric acid type A receptor (GABAAR) levels and impaired GABAAR-medi

220 alpha1, beta1, and beta2 gamma-aminobutyric type A receptor (GABAAR) subunits using deglycosylation

221 TETS is a noncompetitive blocker of the GABA type A receptor (GABAAR), but its molecular interaction

222 rts its pro-tumorigenic activity through its type A receptor (IL-17RA).

223 Two predominant genes, ephrin type A receptor 6 (EPHA6) and folliculin (FLCN), with mu

224 uncta and dendritic spine formation via GABA type A receptor activation and voltage-gated calcium cha

225 ts developed anti-AT1R and/or antiendothelin type A receptor antibodies (non-HLAabs group), 9 did not

226 or antibodies (anti-AT1R) and antiendothelin type A receptor antibodies associated with the clinical

227 sin II type-1 receptor and anti-endothelin-1 type A receptor autoantibodies are associated with an in

228 iotensin II type-1 receptor and endothelin-1 type A receptor autoantibodies pre-LT, and year 1 post-L

229 and impaired spinal gamma-aminobutyric acid type A receptor function, indicative of spinal inhibitor

230 sists despite either gamma-aminobutyric acid type A receptor or N-methyl-D-aspartate receptor inhibit

231 evidence implicates gamma-aminobutyric acid type A receptor subunits on chromosome 15q12 as candidat

232 ntly increased by blockade of the endothelin type A receptor with ABT-627 (0.116 +/- 0.006 mM vs. 0.1

233 injection treatments that inhibited the Edn type a receptor, Edn type b receptor, angiotensin type 1

234 ral disinhibition of gamma-aminobutyric acid type A receptor- and glycine receptor-mediated signaling

235 Here, we demonstrate that the ephrin type-A receptor 2 (EphA2) is an oral epithelial cell PRR

236 The receptor tyrosine kinase ephrin type-A receptor 2 (EphA2) was identified as a SOCS2-inte

237 Altering type-A receptor activities via protein kinase A (PKA) re

238 Alpha-5 gamma-aminobutyric acid type A receptors (alpha5-GABAARs) are located extrasynap

239 id calcium transients mediated by endothelin type A receptors (ETARs) and endothelin type B receptors

240 ncreased activity of gamma-aminobutyric acid type A receptors (GABA(A)Rs), and it is assumed that onc

241 receptors (GlyRs) or gamma-aminobutyric acid type A receptors (GABA(A)Rs), which are implicated in ca

242 postsynaptic clustering of glycine and GABA type A receptors (GABA(A)Rs).

243 GABA type A receptors (GABAAR), the brain's major inhibitory

244 eptors and modulates gamma-aminobutyric acid type A receptors (GABAAR).

245 ceptors (nAChRs) and gamma-aminobutyric acid type A receptors (GABAARs) are members of the pentameric

246 The gamma2 subunit of GABA type A receptors (GABAARs) is thought to be subject to p

247 ts and inhibitors of gamma-aminobutyric acid type A receptors (GABAARs) rather than potentiators.

248 osteric modulator of gamma-aminobutyric acid type A receptors (GABAARs), an interaction necessary for

249 Extrasynaptic gamma-aminobutyric acid type A receptors (GABAARs),which contribute generalized

250 ers glycine as well as major subsets of GABA type A receptors (GABAARs).

251 smission mediated by gamma-aminobutyric acid type A receptors (GABAARs).

252 nhibitory action is largely mediated by GABA type A receptors (GABAARs).

253 tein Gephyrin and of gamma-aminobutyric acid type A receptors at inhibitory neuronal synapses is crit

254 reversed following blockade of GABAARs (GABA Type A receptors), but not GABABRs (GABA Type B receptor

255 diazepines acting at gamma-aminobutyric acid type A receptors.

256 otransmission mediated by gamma-aminobutyric type-A receptors (GABAARs).

257 ivity and not the net levels of postsynaptic type-A receptors.

258 ylcholine and GABAA (gamma-aminobutyric acid type A) receptors into extrasynaptic clusters, whereas n

259 In any given cell type a relatively few active L1 loci contribute to the '

260 In order to study this tumor type, a reliable model system exhibiting the molecular f

261 and a corn field) and three flooded land-use types (a rice paddy and two restored wetlands) to assess

262 and differentiate into different blood cell types, a robust up-regulation of energy metabolism is ex

263 For detection of HPV types, a Roche Linear Array test was performed.

264 pression of cytokinin response genes and the type-A RRs (RRAs) that are encoded by primary cytokinin

265 In contrast, the type-A RRs seemed to be limited to land plants.

266 ordingly, Deltacpa and DeltapfoA mutants, in type A (S13) or type C (CN3685) backgrounds, were unable

267 ins, since a nanI null mutant constructed in type A SD strain F4969 had lower Caco-2 cell adhesion th

268 of BT3686 showed that the enzyme displayed a type A seven-bladed beta-propeller fold.

269 , constant and rapid cell turnover, few cell types, a simple body plan, and the fact that the germ li

270 (+) cells exist primarily as a subset of the type A(single) pool, and their frequency is greatest in

271 xpressed in common in two hematopoietic cell types, a stem cell-like precursor and primary mast cells

272 We conclude that antigens from the type A strain Schu S4 are suitable for detection of anti

273 of 1,741 different proteins derived from the type A strain Schu S4.

274 n the highly virulent Francisella tularensis type A strain SchuS4 are required for proper intracellul

275 mbinant attenuated derivatives of a virulent type A strain, SCHU S4, were evaluated in New Zealand Wh

276 Unlike wild type, a strain lacking all six DGCs did not exhibit a lo

277 nt Francisella tularensis subsp. tularensis (type A) strain Schu S4 in hypervesiculating E. coli cell

278 esent in type A strains from healthy humans, type A strains causing CPE-associated antibiotic-associa

279 However, the nanI gene was present in type A strains from healthy humans, type A strains causi

280 Despite there being no type A strains in Spain, we confirmed the responses agai

281 C. perfringens type A strains producing C. perfringens enterotoxin (CPE

282 is mostly conserved between influenza virus type A strains.

283 h3-null mutant plants were crossed with wild-type A. thaliana.

284 ntified two main types of pulmonary LFs: (1) type A, the predominant type in GOLD stages I-II COPD an

285 c.74146970T>A) in GTF2I at high frequency in type A thymomas, a relatively indolent subtype.

286 aling receptor, natriuretic peptide receptor type A, to the clearance receptor, nprc, was increased a

287 sure of haploid yeast cells, carrying mating type "a," to "alpha pheromone" stimulates polarized grow

288 e strabismus angles, also received botulinum type A toxin injections in the antagonist muscle at the

289 sed as early as 1 week after botulinum toxin type A treatment for lateral canthal rhytids, and the ef

290 t lacks the keto group at C-8 and hence is a type A trichothecene.

291 ABSTRACT: The type A trimeric intracellular cation channel (TRIC-A) is

292 three different enterotypes, referred as "P-type", "A-type "and "F-type" which were highly abundant

293 surface selectivity, solutions of O-type, B-type, A-type and AB-type red blood cells (RBCs) were seq

294 neuraminidases, the N2 enzyme from influenza type A virus and the enzyme from Micromonospora viridifa

295 natal and adult mice infected with influenza type A virus.

296 ld and peaked late, with circulation of both type A viruses and both lineages of type B.

297 e demonstrate that the von Willebrand factor type A (VWA) domain within the cleaved CLCA1 N-terminal

298 German Registry for Acute Aortic Dissection Type A were analyzed.

299 ected with the LVS rather than F. tularensis type A, while IL-23p19 mRNA expression was found to be c

300 (German Registry for Acute Aortic Dissection Type A) who underwent surgery between 2006 and 2010, of

301 th average thicknesses close to that of wild-type A/WSN/33.