ライフサイエンスコーパス: type B

1 type A) and F. tularensis subsp. holarctica (type B).

2 (monolayers of type A) and 38 +/- 4 degrees (type B).

3 gainst H3N2, H1N1pdm09, H1N1 (pre-2009), and type B.

4 of both type A viruses and both lineages of type B.

5 , however, was much higher in type A than in type B.

6 versus 18%, P=0.06); 44% of dissections were type B.

7 novel virus, designated PhV1 type A and PhV1 type B.

8 l disorders, including peeling skin syndrome type B.

9 ay whereby membrane-bound scavenger receptor type B-1 (SR-B1) in parent cells becomes incorporated in

10 Scavenger receptor type B-1 (SR-B1), found in lipid rafts, is a receptor fo

11 This combination of scavenger receptor type B-1 binding and relative cholesterol starvation sel

12 PD-L2 mAb blockade of wild-type B-1 cells in culture significantly increased CD138

13 biomimetic HDL-NPs target scavenger receptor type B-1, a high-affinity HDL receptor expressed by lymp

14 and less frequently with dendritic profiles (type B, 11-17%).

15 y but had abnormally low osmotic thresholds (type B); 44% had normal copeptin concentrations independ

16 5.6 cm] versus 3.3 cm [2.9-3.7 cm], P<0.001; type B: 5.0 cm [3.9-6.0 cm] versus 4.0 cm [3.5-4.8 cm],

17 )=44.4) for H3N2, 54% (46-61; I(2)=61.3) for type B, 61% (57-65; I(2)=0.0) for H1N1pdm09, and 67% (29

18 on of resurgent A(H1N1)pdm09 and late-season type B activity.

19 fore, in mainland China, safety for TEVAR of type B AD appeared better between 2008 and 2015 than in

20 estern data (2006-2013) on acute complicated type B AD, stroke, paraplegia, in-hospital mortality and

21 53.9 years, and acute (70%) or chronic (30%) type B AD.

22 nists and antagonists of endothelin receptor type B administered in slice cultures promoted and inhib

23 target (type A ADR) or an unintended target (type B ADR).

24 Immunologically mediated type B ADRs, such as drug hypersensitivity syndrome, dru

25 lations at risk for immunologically mediated type B ADRs.

26 athway and suggests that endothelin receptor type B agonists represent a promising therapeutic approa

27 Antagonists of endothelin receptor type B also inhibited remyelination of experimentally-ge

28 g that, like beta toxin, TpeL contributes to type B and C infections in hosts with decreased trypsin

29 ines during diseases caused by TpeL-positive type B and C strains, as a toxin whose cytotoxicity decr

30 Type B and C synapses were 2.4-2.6 times more frequent i

31 th groups received the combined H influenzae type b and capsular group C Neisseria meningitidis tetan

32 in is a Cl(-)/HCO3(-) exchanger expressed in type B and non-A, non-B intercalated cells in the distal

33 nt Cl(-)/HCO3(-) exchanger that localizes to type B and non-A, non-B intercalated cells, which are ex

34 ation in patients with peeling skin syndrome type B and other diseases related to epidermal barrier d

35 invasive pathogen as Haemophilus influenzae type b and pneumococcal vaccine use in Mali has diminish

36 m was defined as time-signal intensity curve type B and showed a significant association with incompl

37 of vaccines targeting Haemophilus influenzae type b and Streptococcus pneumoniae have dramatically al

38 ates diagnosed with MoCD (11 type A and five type B) and continued in eight type A patients for up to

39 sis; hepatitis B; and Haemophilus influenzae type b) and pneumococcal vaccine.

40 2)=17.6) for H3N2, 63% (33-79; I(2)=0.0) for type B, and 62% (36-78; I(2)=0.0) for H1N1pdm09.

41 licy, candidates with a CPRA>20%, with blood type B, and aged 18-49 years were more likely to undergo

42 , tetanus, pertussis, Haemophilus influenzae type b, and hepatitis B) at 6, 10, and 14 weeks of age.

43 roup B Streptococcus, Haemophilus influenzae type b, and meningococcus vaccines.

44 tion against H1N1pdm09, H1N1 (pre-2009), and type B, and reduced protection against H3N2.

45 Netherton syndrome, peeling skin syndrome type B, and skin dermatitis--multiple severe allergies--

46 Anti-H influenzae type b anti-polyribosylribitol phosphate IgG geometric m

47 The outcome of patients with acute type B aortic dissection (ABAD) is strongly related to t

48 racic endovascular aortic repair (TEVAR) for type B aortic dissection (AD).

49 he endovascular era, the management of acute type B aortic dissection (ATBAD) is undergoing dramatic

50 identify clinical parameters associated with type B aortic dissection and to develop a risk model to

51 In the risk model, the 10-year occurrence of type B aortic dissection in low-, moderate-, and high-ri

52 ction and to develop a risk model to predict type B aortic dissection in patients with Marfan syndrom

53 f uncomplicated acute, subacute, and chronic type B aortic dissection is managed with close image mon

54 A total of 140 patients with stable type B aortic dissection previously randomized to optima

55 ing identify patients with complicated acute type B aortic dissection requiring urgent aortic repair.

56 uary 1, 2007, through December 31, 2013, for type B aortic dissection were analyzed.

57 Independent variables associated with type B aortic dissection were prior prophylactic aortic

58 AS: 125 had type A aortic dissection, 53 had type B aortic dissection, 35 had intramural aortic hemat

59 In chronic (after 6 weeks) type B aortic dissection, 5-year survival of 60% to 80%

60 h thoracic aortic aneurysm, 195 with chronic type B aortic dissection, and 114 with acute type B aort

61 nt-years for thoracic aortic aneurysm, acute type B aortic dissection, and chronic type B aortic diss

62 pe B dissection or the combined end point of type B aortic dissection, distal aortic surgery, and dea

63 c aortic surgery are at substantial risk for type B aortic dissection, even when the descending aorta

64 acute type B aortic dissection, and chronic type B aortic dissection, respectively.

65 In this study of survivors of type B aortic dissection, TEVAR in addition to optimal m

66 For acute (first 2 weeks) type B aortic dissection, the pooled early mortality rat

67 (TEVAR) represents a therapeutic concept for type B aortic dissection.

68 type B aortic dissection, and 114 with acute type B aortic dissection.

69 or blocker therapy was associated with fewer type B aortic dissections (hazard ratio: 0.3; 95% confid

70 ute, 2 had chronic type A, and 3 had chronic type B aortic dissections before surgery.

71 of the thoracic aorta occurs after TEVAR for type B aortic dissections in patients with thoracic FLT

72 Between 1998 and 2013, 54 type B aortic dissections occurred in 600 patients with

73 erapy may be protective in the prevention of type B aortic dissections.

74 s in patients who underwent acute or chronic type B aortic dissections.

75 c repair (TEVAR) is used in the treatment of type B aortic dissections.

76 nt of initially uncomplicated acute Stanford type-B aortic dissection is associated with a high rate

77 3 patients with acute uncomplicated Stanford type-B aortic dissection, followed over a median of 850

78 erse events after an initially uncomplicated type-B aortic dissection.

79 ne KINASE2 (AHK2) and AHK3 receptors and the type B Arabidopsis response regulator1 (ARR1) and ARR12.

80 analysis indicated an overrepresentation of type-B Arabidopsis response regulator binding elements,

81 the transcriptional network initiated by the type-B ARABIDOPSIS RESPONSE REGULATORs (ARRs) that media

82 ponse to cytokinin is regulated by action of type-B Arabidopsis response regulators (ARRs).

83 inding elements, consistent with the role of type-B Arabidopsis response regulators as primary mediat

84 ulating the cytokinin response, mechanism of type-B ARR activation, and basis by which cytokinin regu

85 Three type-B ARR DNA-binding motifs, determined by use of prot

86 owth and root meristem size correlating with type-B ARR expression levels.

87 in ligase complex and directly interact with type-B ARR proteins.

88 s of Arabidopsis response regulators (ARRs): type B ARRs (response activators) and type A ARRs (negat

89 the LAX2 gene in vivo, which indicates that type B ARRs directly regulate genes that are repressed b

90 se to cytokinin requires ARR1 and ARR12, two type B ARRs that mediate the primary transcriptional res

91 Loss-of-function KMD mutants stabilize type-B ARRs and exhibit an enhanced cytokinin response.

92 To determine which type-B ARRs can functionally substitute for the subfamil

93 embers ARR1 or ARR12, we expressed different type-B ARRs from the ARR1 promoter and assayed their abi

94 Our results indicate that the type-B ARRs have diverged in function, such that some, b

95 shed light on the physiological role of the type-B ARRs in regulating the cytokinin response, mechan

96 R1, as well as a subset of other subfamily 1 type-B ARRs, restore the cytokinin sensitivity to arr1 a

97 ies endothelin 2 and the endothelin receptor type B as a regenerative pathway and suggests that endot

98 investigated using Candida antarctica lipase type B as biocatalyst.

99 Cockayne syndrome type B ATPase (CSB) belongs to the SwItch/Sucrose nonfer

100 fant botulism, produced botulinum neurotoxin type B (BoNT/B) and another BoNT that, by use of the sta

101 While wild-type B. bronchiseptica was shed from colonized mice and

102 ure-detectable spirochetemia induced by wild-type B. burgdorferi (WT), indicating that VlsE was likel

103 same conditions, the swimming speed of wild-type B. burgdorferi slowed by approximately 15%, with on

104 was significantly higher than parental wild-type B. burgdorferi strains, suggesting that OspC has an

105 In wild-type B. burgdorferi, bb0449 transcript and BB0449 protei

106 tadial transmission relative to that of wild-type B. burgdorferi.

107 rogressively smaller excitatory responses in type B but not type A neurons.

108 rmidium had 100% nucleotide identity to PhV1 type B, but this region was absent from PhV1 type A.

109 ating blood type A2 and A2B kidneys to blood type B candidates.

110 -matrix ratio (p < 0.001), an 8% decrease of type B carbonate substitution (p < 0.001), and a 2% incr

111 Nasopharyngeal H influenzae type b carriage was detected in one (0.2%) of 623 childr

112 Type B cases presented an increased survival rate compar

113 iet but then relapsed despite a strict diet (type B cases).

114 M_E1 are at the interface between type A and type B CBMs.

115 able identification of three lymphocyte cell types (B, CD4+ T, and CD8+ T cells) with high sensitivit

116 However, the maintenance of type B cell populations was apparently unaffected by the

117 of the NF-kappaB inhibitor, JSH-23, to wild-type B cells 15 h after LPS plus IL-4 stimulation select

118 p40, IL-10, TNF-alpha, and IgM than did wild-type B cells after TLR stimulation.

119 led heterogeneous expression of Ifnb in wild-type B cells and distinct gene expression patterns assoc

120 RNase activity and XBP-1s expression in wild type B cells and human mantle cell lymphoma cell lines.

121 anced the proliferation of B1- as well as B2-type B cells and promoted the production of IgM, IgG1, I

122 ation of T cells in vitro compared with wild-type B cells from the same tumor.

123 CD40 ligation during LPS stimulation of wild-type B cells is sufficient to inhibit PC generation.

124 Type B cells mediate Cl(-) absorption and HCO3(-) secret

125 peak firing during the CS-US interval, while type B cells presented a second peak during US presentat

126 CNTF was identified in a subset of type B cells that label with acute BrdU administration.

127 ed Ab responses, a finding confirmed in wild-type B cells transfected with a MiR-210 mimic.

128 include radial glia comparable to Type E and Type B cells, and a neuronal-like population of cerebros

129 nced in Abp1(-/-) B cells compared with wild-type B cells, including Ca(2+) flux and phosphorylation

130 cotransfer of CD40LTg B cells, but not wild-type B cells, significantly reduced IL-17 response and r

131 In wild-type B cells, UNG2 gene expression and enzymatic activit

132 g and cytokine production compared with wild-type B cells.

133 comparable to that seen with stimulated wild-type B cells.

134 sponses to anti-IgM as mCD22-expressing wild-type B cells.

135 ion and insulin resistance, compared to wild type B cells.

136 n and degradation dynamics of different cell types (B cells, T cells, naive, memory) is governed by a

137 ediated translocations in two different cell types, B cells and mouse embryonic fibroblasts (MEFs).

138 Similarly, wild-type B. cellulosilyticus DSM 14838, but not a close rela

139 Compared to wild-type B. cereus G9241, spores with a deletion of the pBCX

140 ussis-inactived polio-Haemophilus influenzae type b combined vaccine (DTaP-IPV-Hib) at 2, 3, and 4 mo

141 in a simple mixture of 'stacked' (S) and 'B-type' (B) conformers, whereas the N-acetylated FAAF also

142 vaccine (PsACWY); or Haemophilus influenzae type b conjugate vaccine (Hib-TT).

143 ar aortic repair may be optimal for treating type B (descending aorta) AAS.

144 The incidence of invasive H influenzae type b disease in children younger than 5 years declined

145 -14), only one case of invasive H influenzae type b disease was detected in a child younger than 5 ye

146 emical or clinical response in patients with type B disease.

147 sustained reduction in invasive H influenzae type b disease.

148 typing was developed to predict the risk for type B dissection in patients with Marfan syndrome.

149 or a median of 6 years for the occurrence of type B dissection or the combined end point of type B ao

150 Type B dissection was strongly associated with previous

151 In stable type B dissection with suitable anatomy, preemptive TEVA

152 nt in patients treated for acute and chronic type B dissection, and when the endograft is significant

153 Four patients had type A and nine had type B dissection.

154 .9%; 95% CI, 2.5-6.3) for surgical repair of type B dissection.

155 ing aorta have been classified as type A and type B dissections, respectively.

156 of the endonuclease, encode a protein-primed type B DNA polymerase (PolB) and hence break this patter

157 In the Rhine, type B does not show evidence of isolation by distance,

158 lves a region of the protein that contains a Type B dsRBD.

159 BDs interact with dsRNA, while non-canonical Type B dsRBDs lack RNA-binding residues and instead inte

160 he protein/protein interaction properties of Type B dsRBDs.

161 pertussis, polio, and Haemophilus influenzae type b (DTaP-IPV-Hib) administered at ages 3, 5, and 12

162 ely GABAb (gamma amino butyric acid receptor-type b) duration.

163 type A, n = 239, 45%) or bile duct injuries (types B-E, n = 289, 55%).

164 chemokine receptor 2 (CXCR2), is involved in type B EAE development, and type B EAE is ameliorated by

165 Remission was minimal in type B EAE due to neuronal damages induced by semaphorin

166 , is involved in type B EAE development, and type B EAE is ameliorated by antagonizing these receptor

167 erferon-beta (IFNbeta)-resistant EAE (termed type B EAE), whereas EAE induced by weak activation of i

168 EDN3), its receptor (the endothelin receptor type B [EDNRB]), and the transcription factors SRY-box 1

169 Type B enhancement was most common, with a prevalence of

170 e cells highly expressed endothelin receptor type B (ETB(R)) and Jagged1, a Notch1 receptor ligand.

171 r detection of antibodies from patients with type B F. tularensis infections and that these can be us

172 ional role of glycosyltransferases modifying type B flagellin in the 023 and 027 hypervirulent C. dif

173 in neonates diagnosed with MoCD (type A and type B) following a standardised protocol.

174 lysed sterile site cultures for H influenzae type b from children (aged </=12 years) admitted to the

175 extraction of 12 trichothecenes (type A and type B) from baby foods, followed by gas chromatography-

176 gamma-Aminobutyric acid type B (GABAB) receptor autoantibodies were identified i

177 utoantibodies to the gamma-aminobutyric acid type B (GABAB) receptor have recently been identified as

178 The gamma-aminobutyric acid type B (GABAB) receptor undergoes splicing and is an alc

179 ition is mediated by gamma-aminobutyric acid type B (GABAB) receptors, which are heterodimeric G-prot

180 de patterns of 6 porcine type A and 6 bovine type B gelatines at molecular weight ranged from 50 to 2

181 nd characteristics of Haemophilus influenzae type b genogroup strains isolated from genitourinary tra

182 e present the functional characterization of type B Ggamma subunit (SlGGB1) in tomato (Solanum lycope

183 We conclude that the type B Ggamma subunit SlGGB1 mediates auxin and ABA sign

184 Type B Ggamma subunits, lacking a carboxyl-terminal isop

185 C. intestinalis (formerly Ciona intestinalis type B), globally distributed and sympatric in Europe.

186 ents (amino-terminal pro-natriuretic peptide type B >8500 ng/L) had lower response rates (42%, >/= VG

187 te vaccine containing Haemophilus influenzae type b (Hib) and group C meningococcal polysaccharides,

188 Encapsulated H. influenzae type b (Hib) and type f (Hif) are the most common seroty

189 (S. pneumoniae), and Haemophilus influenzae type b (Hib) are three most common pathogens accounting

190 poliovirus (IPV), and Haemophilus influenzae type b (Hib) conjugate vaccine (DTaP-IPV-Hib) among chil

191 Haemophilus influenzae type b (Hib) conjugate vaccine, delivered as a three-dos

192 incidence of invasive Haemophilus influenzae type b (Hib) disease has significantly decreased since t

193 uring introduction of Haemophilus influenzae type b (Hib) vaccination and the rollout of antiretrovir

194 valent MMR vaccine and Haemophilus influenza type B (HiB) vaccine.

195 Protection against Haemophilus influenzae type b (Hib), a rapidly invading encapsulated bacteria,

196 arides extracted from Haemophilus influenzae type b (Hib), and the corresponding glycoconjugate made

197 s Type-A Escherichia coli RNase P RNA with a Type-B homolog derived from Bacillus subtilis, and show

198 (HR = 7.1), and time-signal intensity curve type B (HR = 4.3) were associated with earlier recurrenc

199 ase was more prominent in type A ICs than in type B ICs.

200 The development of FID in the acute phase of type B IMH has a poor prognosis owing to the high risk o

201 ere were 107 consecutive patients with acute type B IMH were included prospectively in a multicenter

202 evolution of medically treated patients with type B IMH with and without FID.

203 The QAV was type A in 32% and type B in 32% (Hurwitz and Roberts classification).

204 revealed high levels of endothelin receptor type B in oligodendrocyte lineage cells.

205 he BDNF receptor tropomyosin-receptor-kinase type B in rats and mice, we observed that chronic opiate

206 including one with motor neuron disease, 27 type B including 21 with motor neuron disease, eight typ

207 Immune tolerance to alpha-Gal in blood type B individuals might reduce the risk to this allergy

208 type C infections and is also implicated in type B infections, but little is known about the CPB str

209 gs also suggest that prior exposure to H1 or type B influenza may differentially affect cross-reactiv

210 ed from human postvaccination sera with only type B influenza preexposure consistently showed good co

211 those derived from sera with neither H1 nor type B influenza preexposure, and the correlation lessen

212 those derived from sera with neither H1 nor type B influenza preexposure.

213 rs) other medications (eg, monoamine oxidase type B inhibitors [MAOBIs], amantadine, anticholinergics

214 R607H knockin did not affect type B intercalated cells.

215 ns are consistent with a recent expansion of type B into the Rhine drainage.

216 sruption (FID) has been described in >20% of type B intramural hematomas (IMH), with unclear prognosi

217 monstrate that after wounding or ultraviolet type B irradiation, melanocyte stem cells (McSCs) in the

218 e discovered that EDNRB (Endothelin receptor type B) is a candidate gene involved in HA adaptation.

219 sis type IIIB (MPS IIIB, Sanfilippo syndrome type B) is a lysosomal storage disease characterized by

220 irus infection who all presented with severe type B lactic acidosis shortly after starting treatment

221 r-paritaprevir-ritonavir-dasabuvir may cause type B lactic acidosis.

222 ) of 39 HU +/- 13 in the arterial phase, and type B lesions had a difference of -58 HU +/- 26 in the

223 tion than the thyroid in the arterial phase, type B lesions were not higher in attenuation than the t

224 imates of vaccine effectiveness against both type B lineages were similar (overall, 58%; 95% CI, 35-7

225 o different F. tularensis subsp. holarctica (type B) live vaccine strains, thereby demonstrating the

226 of TRPM7 with NS8593 or waixenicin A in wild-type B lymphocytes results in a significant decrease in

227 pa, dopamine agonists, and monoamine oxidase type B (MAO-B) inhibitors.

228 Whereas type B modification is not required for flagellar assemb

229 as enthalpically driven, which is typical of type B modules.

230 levels of intracellular replication of wild-type B. neotomae were significantly stimulated by coinfe

231 ts that are focused in space and time, while type B neurons integrate across these dimensions.

232 rent, and thin-tufted, callosally projecting type B neurons, which lack prominent h-current.

233 difies its flagellin with either a type A or type B O-linked glycosylation system, which has a contri

234 Aortic dissection, commonly type B, occurs in an appreciable proportion of Marfan pa

235 nstitute of Standards and Technology (NIST) "Type B On Bias" approach and yielded consensus values in

236 Sanfilippo syndrome Type B or Mucopolysaccharidosis IIIB (MPS IIIB) is a neu

237 Forty-eight hours after infection, wild-type B. parapertussis bacteria but not the O antigen-def

238 ass spectrometry analysis revealed that wild-type B. parapertussis lipid A consists of a heterogeneou

239 ron disease is more commonly associated with type B pathology, but has also been reported with type A

240 The type B pattern is most common and could be diagnosed wit

241 hil extracellular traps (NETs), whereas wild-type B. pertussis does not, suggesting that ACT suppress

242 e syndromes caused by Haemophilus influenzae type b, pneumococcus, rotavirus, and early infant influe

243 DeltabcaA and Bp340DeltabcaB mutants to wild-type B. pseudomallei in vitro demonstrated similar level

244 tective immunity against challenge with wild-type B. pseudomallei, suggesting that the genes identifi

245 istically affecting the MAPK pathway in wild-type B-Raf cells.

246 , they induce conformational changes to wild-type B-Raf kinase domain leading to heterodimerization w

247 In tumors with wild-type B-Raf, vemurafenib paradoxically activates downstre

248 idepressants, alters gamma-aminobutyric acid type B receptor (GABABR) expression and signalling, to i

249 Interestingly, the ephrin type B receptor 2 (EphB2), a tyrosine kinase receptor as

250 Accumulating evidence suggests that ephrin type B receptor 4 (EphB4) plays a key role in the progre

251 gamma-Aminobutyric acid type B receptor autoimmunity deserves consideration in p

252 that inhibited the Edn type a receptor, Edn type b receptor, angiotensin type 1 receptor, mineraloco

253 tional consequences of circuit-specific GABA type-B receptor (GABABR) manipulation.

254 Here we show that ephrin type-B receptor 1 (EphB1) is upregulated in injured moto

255 The protein tyrosine kinase Ephrin type-B receptor 3 (EPHB3) counteracts tumor-cell dissemi

256 Published data show Ephrin type-B receptor 4 IC50 values obtained via tip-based ser

257 The gamma-aminobutyric acid type B-receptor agonist lesogaberan (AZD3355) has been d

258 elin type A receptors (ETARs) and endothelin type B receptors (ETBRs).

259 ABA Type A receptors), but not GABABRs (GABA Type B receptors).

260 ne kinase activity and mutants involving the type B response regulators.

261 In Arabidopsis (Arabidopsis thaliana), type-B response regulators (ARABIDOPSIS RESPONSE REGULAT

262 Type-B response regulators, acting as transcriptional ac

263 ich reflects the transcriptional activity of type-B response regulators.

264 response to cytokinin being mediated by the type-B response regulators.

265 f Arabidopsis response regulators (RRs): the type-B RR (RRB) transcriptional activators that promote

266 Farther down the signaling pathway, the type-B RRs were found across all plant clades, but many

267 t that endogenous CNTF receptor signaling in type B stem cells inhibits adult neurogenesis, and furth

268 We found that type B "stem" cells are highly responsive, whereas type

269 We find that norspermidine is absent in wild-type B. subtilis biofilms at all stages, and higher conc

270 present in both pellicle and planktonic wild-type B. subtilis cells and in strains with deletions in

271 Here, colonies of wild-type B. subtilis formed a spreading population that indu

272 bunits are lacking at the postsynapse, while type-B subunits cluster correctly.

273 AMKIIalpha-positive principal cells, whereas type B synapses contacted presumed inhibitory neurons.

274 type IIIB syndrome (also known as Sanfilippo type B syndrome) is a lysosomal storage disease resultin

275 pectrometry to compare the secretome of wild-type B. thailandensis to that of a mutant harboring a no

276 , and it is proposed that in this monolayer (type B), the molecular axes are tilted by 40-45 degrees

277 AFF-positive cells (mostly B cells); and (2) type B, the main type in GOLD stage IV COPD, characteriz

278 onventional cooperative/dual photocatalysis (type B), this new class of unconventional PCs operates v

279 WNT1), trimeric intracellular cation channel type b (TRIC-B), and old astrocyte specifically induced

280 sarium graminearum, a fungus able to produce type B trichothecenes on cereals, including deoxynivalen

281 ough the cognate tropomyosin receptor kinase type B (trkB) receptor occurs in substantia nigra pars c

282 oform of tropomyosin-related receptor kinase type B (trkB) receptors expressed on astrocytes.

283 ng downstream of tropomyosin receptor kinase type B (trkB), namely, phosphorylation of Akt and riboso

284 probed the Schu S4 array with sera from 241 type B tularemia cases in Spain.

285 igens would also have utility for diagnosing type B tularemia caused by strains from other geographic

286 e anti-A surface selectivity, solutions of O-type, B-type, A-type and AB-type red blood cells (RBCs)

287 an result in interruption of the aortic arch type B, typically found in DiGeorge syndrome.

288 tivated intestinal symbiont Epulopiscium sp. type B using fluorescent in situ hybridization.

289 mographic history of two of them (type A and type B) using microsatellite markers and asked whether t

290 hesized vitamin D in response to ultraviolet type B (UVB) light.We tested the hypothesis that, in vit

291 ter the introduction of routine H influenzae type b vaccination.

292 measles, rubella, and Haemophilus influenzae type b vaccine antigens were comparable between the 2 gr

293 nactivated poliovirus/Haemophilus influenzae type b vaccine; age 6/10/ 14 weeks) and 13-valent pneumo

294 South Carolina patients with B/I221V vs wild-type B virus infection (B/WT).

295 uman and monkey cells as efficiently as wild-type B virus.

296 ter was attributed to the complete growth of type B viruses in MDCK cells before day three post-infec

297 all influenza virus infections are caused by type B viruses, and these infections can be severe, espe

298 e detection of seasonal H1N1pdm09, H3N2, and type B viruses, as well as highly pathogenic H5 and H7 v

299 susceptibility titers against type A but not type B were consistent with this observation.

300 nfigured beta-nitroalcohol, while ligands of type B, with the reversed location of small and large su

301 epatitis B virus, and Haemophilus influenzae type b), yellow fever, measles, and tuberculosis.