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1 tion (IIT) in the treatment of patients with type I diabetes mellitus.
2 ntigens targeted by self-reactive T cells in type I diabetes mellitus.
3 te to diabetic nephropathy susceptibility in type I diabetes mellitus.
4 tion is becoming an accepted therapy to cure type I diabetes mellitus.
5 ed treatment option for select patients with type I diabetes mellitus.
6 nery and as major targets of autoimmunity in type I diabetes mellitus.
7  an attractive alternative for patients with type I diabetes mellitus.
8 gerhans to human patients in efforts to cure type I diabetes mellitus.
9 e thyroid disease, rheumatoid arthritis, and type I diabetes mellitus.
10 ation and arise spontaneously in autoimmune (type I) diabetes mellitus.
11 ion and disease course of insulin-dependent (type I) diabetes mellitus.
12 we describe our experience with two cases of type I diabetes mellitus and glycogen hepatopathy.
13  IDDM8, a linkage and association signal for type I diabetes mellitus and rheumatoid arthritis.
14               Two thirds of the patients had type I diabetes mellitus, and approximately 50% were men
15 arthritis, Sjogren syndrome, celiac disease, type I diabetes mellitus, and systemic lupus erythematos
16 lupus erythematosus, multiple sclerosis, and type I diabetes mellitus, as indicated by the efficacy o
17 for association with diabetic nephropathy in type I diabetes mellitus, by using both case-control and
18 in a streptozocin (STZ)-induced rat model of type I diabetes mellitus (DM) and to understand the path
19  of insulin-secreting beta-cells that causes type I diabetes mellitus (DM) occurs in part by apoptosi
20 r patient survival was 36%, 49%, and 65% for type I diabetes mellitus (DM), type II DM, and nondiabet
21                                              Type I diabetes mellitus (IDDM) is a T cell-dependent au
22 pment in rodent models of insulin-dependent (type I) diabetes mellitus (IDDM).
23 ning porcine islets as long-term therapy for type I diabetes mellitus in Biobreeding/Worcester (BB/Wo
24 mechanism by which TNF and anti-TNF modulate type I diabetes mellitus in NOD mice.
25                    During the development of type I diabetes mellitus in nonobese diabetic (NOD) mice
26  factor-alpha (TNF) differentially modulates type I diabetes mellitus in the nonobese diabetic (NOD)
27                                              Type I diabetes mellitus is an autoimmune disease charac
28               Poorly controlled or untreated type I diabetes mellitus is characterized by hyperglycem
29                                           In type I diabetes mellitus, islet transplantation provides
30  months) and 108 patients in five studies of type I diabetes mellitus (mean length of follow-up, 9 to
31                PIT in severely insulinopenic type I diabetes mellitus primates resulted in restoratio
32 The SUMO-4M (Met) variant is associated with type I diabetes mellitus susceptibility in families (p =
33 ecause LBH gene variants are associated with type I diabetes mellitus, systemic lupus erythematosus,
34 c T cells prevented the adoptive transfer of type I diabetes mellitus (T1DM) to NOD/scid mice.
35 ials of multiple sclerosis (alemtuzumab) and type I diabetes mellitus (teplizumab, otelixizumab).
36                                              Type I diabetes mellitus (TIDM) is an autoimmune disorde
37  results offer an exciting new potential for type I diabetes mellitus treatment.
38                                              Type I diabetes mellitus was induced in male rats by all

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