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1 plasia on day 14 postinfection than the wild-type strain.
2 A moiety compared to that found in the wild-type strain.
3 red more efficiently, compared with the wild-type strain.
4 apsule production when expressed in the wild-type strain.
5 point is present and functional in the wild-type strain.
6 with the black spore suspension of the wild-type strain.
7 r when using the RARE strain versus the wild-type strain.
8 ows at a rate comparable to that of the wild-type strain.
9 oteins within the complex compared to a wild-type strain.
10 and performed competitions against the wild-type strain.
11 model of GAS invasive disease than the wild-type strain.
12 e to oxidative stress than the parental wild-type strain.
13 algZ mutant was 40% of the level of the wild-type strain.
14 howed a lower electro-activity than the wild-type strain.
15 ded protection against infection by the wild-type strain.
16 and biomass than those of the parental wild-type strain.
17 in the DeltaartA strain but not in the wild-type strain.
18 nt strain compared to expression in the wild-type strain.
19 e resistant to LpxC inhibitors than the wild-type strain.
20 old lower in the hfq mutant than in the wild-type strain.
21 tivity and cytotoxicity compared to the wild-type strain.
22 d on plastic surfaces compared with the wild-type strain.
23 low ribosome content, compared with the wild-type strain.
24 erences between the yfiR mutant and the wild-type strain.
25 o infection than mice infected with the wild-type strain.
26 ased in the ccpE mutant relative to the wild-type strain.
27 ics comparable to those observed in the wild-type strain.
28 ed carriage, exceeding even that of the wild-type strain.
29 tant S. exfoliatus ZD27 compared to the wild-type strain.
30 ytic activities similar to those of the wild-type strain.
31 s selected for longer chains within the wild-type strain.
32 n at nearly the same levels as from the wild-type strain.
33 ncrease in chain length relative to the wild-type strain.
34 ical sites since the recent isolation of the type strain.
35 oxidative stress similar to that of the wild-type strain.
36 fective in colonization compared to the wild-type strain.
37 d reduced dissemination compared to the wild-type strain.
38 ntly higher in mice inoculated with the wild-type strain.
39 cute infection when coinoculated with a wild-type strain.
40 mutant were as virulent as those of the wild-type strain.
41 llular concentration as compared with a wild-type strain.
42 produced less pyruvate and H2S than the wild-type strain.
43 increase BAFF at levels similar to the wild type strain.
44 allenge with the B. mallei lux (CSM001) wild-type strain.
45 pat mutant reaching the spleen than the wild type strain.
46 IN-1 both induced Ohr expression in the wild-type strain.
47 n were similar compared to those of the wild-type strain.
48 ions the mutant did not differ from the wild-type strain.
49 is type strain, and 83.8% to the S. ictaluri type strain.
50 r lipid production from phenol than the wild-type strain.
51 tly more biomass than those formed by a wild-type strain.
52 sor strains tested as compared with the wild-type strain.
53 e and cold treatments than those of the wild-type strain.
54 ewer, smaller lesions compared with the wild-type strain.
55 ut strains and patients infected with a wild-type strain.
56 sease at a similar rate of onset as the wild type strain.
57 , and Tunisia, were sequenced along with the type strain.
58 hese matched the stress proteome of the wild type strain.
59 es to a level comparable to that of the wild-type strain.
60 ility upon exposure to NO compared with wild-type strain.
61 initiate growth was comparable with the wild-type strain.
62 ungal susceptibility testing of clinical and type strains.
63 % (38-97) against single-antigen non-vaccine type strains.
64 % (47-88) against single-antigen non-vaccine type strains.
65 ival rates comparable to those found in wild-type strains.
66 ve cheats in vivo and cannot outcompete wild-type strains.
67 onization of BALB/cByJ mice compared to wild-type strains.
68 host cells compared with their isogenic wild-type strains.
69 sible to the steady-state phenotypes of wide-type strains.
70 more pyruvate in the growth medium than wild-type strains.
71 higher Chl a/b ratio than corresponding wild-type strains.
72 C1q recognition when compared with the wild-type strains.
73 ave normally in crosses with opposite mating-type strains.
74 higher Chl a/b ratio than corresponding wild-type strains.
75 ed the lipidome of loa1Delta mutant and wild-type strains.
76 of all currently recognized bifidobacterial type strains.
77 not accumulate in detectable amounts in wild-type strains.
78 mants were similar to the corresponding wild-type strains.
79 ical to those of spores from a biochemically typed strain.
82 -essential ICP4 and ICP8 genes of HSV-1 wild-type strain 17syn+ were brought under the control of a d
84 s 70% (58-78) against single-antigen vaccine type strains, 37% (10-56) against partly heterotypic str
86 , 82% (70-89) against single-antigen vaccine type strains, 82% (70-89) against partly heterotypic str
87 gene was 98.8% similar to the S. halichoeri type strain, 84.6% to the S. canis type strain, and 83.8
90 usceptible to early host clearance than wild-type strains after intravenous infection, but impaired i
92 mif(-/-) L. major, when compared to the wild-type strain, also showed a 3-fold reduction in parasite
93 y 10-fold higher when compared with the wild-type strain, although the expression levels of genes enc
95 f two Caenorhabditis elegans strains, a wild-type strain and a strain containing two complex rearrang
96 tome-sequencing (RNA-seq) analysis of a wild-type strain and an isogenic fabT deletion mutant strain
97 d strain grows at a similar rate to the wild-type strain and does not exhibit any major growth defect
99 red expression compared to the isogenic wild-type strain and included transcriptional regulators, tra
100 n (CDT) were evaluated first by using a wild-type strain and its corresponding cdtB isogenic mutant a
101 ntrations that gave derepression of the wild-type strain and retained sufficient ligation activity fo
103 provement in FA titer compared with the wild-type strain and the strain carrying the uncontrolled met
104 These genomes double the number of existing type strains and expand their overall phylogenetic diver
106 kin 8 expression was evaluated by using wild-type strains and their corresponding CdtB isogenic mutan
108 alichoeri type strain, 84.6% to the S. canis type strain, and 83.8% to the S. ictaluri type strain.
109 FtsH2 also interacts with FtsH3 in the wild-type strain, and a mutant depleted in FtsH3, like ftsH2(
110 bactericidal activity than against the wild-type strain, and the IgG1 MAbs had similar or greater ac
111 ed biofilm architecture relative to the wild-type strain, and these phenotypes were partially complem
112 ation of macrophages, compared with the wild-type strain, and with delayed inflammatory stimuli as co
115 embrane proteins, TraK and TraB, in the wild-type strain as well as in overexpression strains and in
116 iculation phenotype of DeltanlpA in the wild-type strain as well as in the degP deletion strain was f
117 differentiation in THP-1 cells than the wild type strain, as determined by carboxyfluorescein diaceta
118 ly attenuated virulence relative to the wild-type strain, as manifested by prolonged survival, reduce
119 t compared with those infected with the wild-type strain, as well as significantly greater expression
120 ch differs by a point mutation from the wild-type strain, assembles into straight filaments in which
122 ely measure toxin production by C. difficile type strain ATCC 9689 under 768 culture conditions.
123 eases in intracellular C. jejuni 11168H wild-type strain bacteria were observed after 24-h coculture
126 attenuated virulence as compared to the wild-type strain but did not significantly affect bacterial g
127 ficantly up-regulated in the irradiated wild-type strain but not in the irradiated wdpks1 mutant, imp
129 pressing PR1 enhanced resistance to the wild-type strain, but not to the Sscp1 knockout strain of S.
131 ns were more sensitive to acid than the wild-type strain, but the Deltahyc strains were like the viru
132 mutant strains compared to that in the wild-type strain, but the secretion of virulence proteins in
133 te mitochondrion, even outcrossing with wild-type strains cannot facilitate spread of resistance.
136 both classes of promoters, peak KOA in wild-type strains coincided late in the circadian cycle near
138 first episodes of infections due to vaccine-type strains, community-acquired pneumonia occurred in 4
139 d its isogenic choline-treated parental wild-type strain D39 degrade extracellular DNA readily, sugge
144 of cytochromes c(1) and c(2) than did a wild-type strain due to their restricted Ccm capabilities.
146 rt, we demonstrate that the majority of wild-type (strain EGDe) and mouse-adapted (InlA(m)-expressing
148 and bactericidal assays using the infecting-type strain, emm cluster-related strains, and nonrelated
149 inal metaplasia in subjects infected with i2-type strains, even in a vacA s1, cagA(+) background.
150 sequence of the field strain ILRI181 and the type strain F38 and that was further evidenced in 10 fie
151 act that the dislocation, besides the phonon-type strain field analogous to dislocations in ordinary
152 acking all three genes behaved like the wild-type strain for all phenotypes mentioned above, but all
154 ed four strains of Bacillus subtilis and the type strains for two closely related species, Bacillus v
160 e per generation; (ii) mutations in the wild-type strain have the expected mutational bias for G:C >
161 nts in defined competitions against the wild-type strain identified nine mutants that exhibited a rep
165 3, and the ure mutant was used with the wild-type strain in competition experiments in mouse models t
169 f human fH/ml permitted survival of the wild-type strain in up to 60% infant rat serum, whereas >/=33
173 Rapid differentiation of vaccine from wild-type strains in suspect measles cases is a valuable epid
174 in exflagellation EC50 relative to the wild-type strains in the presence of compound 1294, providing
175 ind fibronectin relative to that of the wild-type strain; in situ reconstitution of fnm in the deleti
176 tress, and in response to H(2)O(2), the wild-type strain increases superoxide radical production to a
177 omes of an rppH deletion mutant and the wild-type strain incubated at 20 degrees C and 30 degrees C.
179 resistance to quinolone compared to the wild-type strain, indicating that fur functions as a positive
180 ccumbed to infection with a more recent wild-type strain, indicating that immune responses to the mor
181 cells exposed to the isogenic S. aureus wild-type strain, indicating that PSMs inhibit the production
182 hly induced under oxidative stress in a wild-type strain, indicating the critical role of Cys-25 in r
183 djustment (MOMA) from the corresponding wild-type strains instead of having maximal growth rates afte
186 olates were phenotypically distinct from the type strain isolated from a seal; comparative whole-geno
191 orcine lungs and was outcompeted by the wild-type strain (median competitive index of 2 x 10(-5)).
193 We present the draft genome sequence of the type strain Moritella marina MP-1 (ATCC 15381), having 4
195 ion was also noticed in an M. smegmatis wild-type strain (MSWt) induced with cumene hydroperoxide (CH
196 Here we present the genomic background of type strain NRRL Y-12632 and its transcriptomic response
198 ta indicate that murine death caused by wild-type strains occurs by a mechanism different from that b
200 were purified and used to show that the wild-type strain of Cba. tepidum can grow on biogenic S(0) gl
201 Archaea by sequencing, where available, the type strain of each species with a validly published nam
202 revealed a 9-bp deletion in the gene in the type strain of genomic species 9 (ATCC 9957) relative to
203 ipt in host macrophages infected with a wild-type strain of M. tuberculosis or an attenuated mutant s
205 ng were performed on the human isolates, the type strain of S. halichoeri, and type strains of closel
206 cal isolates that most closely resembled the type strain of Streptococcus halichoeri isolated from a
212 lopment of reverse genetics systems for wild-type strains of CDV and the use of the resulting recombi
217 nidia (asexual spores) of two different wild-type strains of N. fumigata and three different ergot al
218 V13) in preventing first episodes of vaccine-type strains of pneumococcal community-acquired pneumoni
219 a mechanistic explanation for how different types (strains) of rhinoviruses may elicit different cel
220 umber of disease lesions incited by the wild-type strain on bean was also significantly higher than t
221 d higher rates of survival than the S. mitis type strain or the capsule-switching mutant, except in t
225 its greater genetic diversification than the type strain PA01, despite its lower per base mutation ra
227 that OMVs generated from P. aeruginosa wild-type strain PAO1 were more cytotoxic to alveolar epithel
228 s significantly reduced compared to the wild-type strain PH-1, while 10 Group 2 mutants grew normally
229 ease upon subsequent challenge with the wild-type strain PI1428, which appears to be driven by a Th17
230 n model, 1 week following infection the wild-type strain produced significantly more widespread lesio
231 ent the genome sequence of the P. fermentans type strain R7 (DSM 17108) and genome sequences for two
232 nuated in the mouse and equine, whereas wild-type strain RacL11 induces severe inflammation of the lu
233 ore, impairment of the Sec pathway in a wild-type strain reduced Ag43 production but did not signific
234 Cse4, transient induction of PSH1 in a wild-type strain resulted in phenotypes similar to a pat1 str
235 ficient S. aureus and the corresponding wild-type strain reveal that activation of acid sphingomyelin
236 The average genome configuration of the wild-type strain revealed strong intra- and inter-chromosomal
237 a leaky Escherichia coli strain and the wild-type strain reveals the contribution of the formidable o
239 ving forced heterokaryons of opposite mating-type strains show that presence of one receptor and its
240 lated with the formyl peptide-producing wild-type strain showed a significantly increased frequency a
242 e accurate diagnosis among other prevalent B-type strains, simultaneous detections of four conserved
243 rved in domesticated and undomesticated wild-type strains sporulating in liquid and on solid media.
244 rease in virulence as compared with the wild-type strain, suggesting that LOS-IV plays a role in the
245 as still less virulent in vivo than the wild-type strain, suggesting that SecA2 function was still re
246 bserved between the luxS mutant and the wild-type strain, suggesting that the defect in virulence fol
247 n adhesion level similar to that of the wild-type strain, suggesting that the gene does not direct at
248 t spread in tomato stems as well as the wild-type strain, suggesting that these exDNases facilitate b
250 nt in eliminating SIC compared with the wild-type strain, terminal half-lives being 6 and 1.5 h, resp
252 Thus, at lower epitope density in the wild-type strain, the IgG3 anti-fHbp MAbs had the greatest ba
253 l1951 strain was similar to that of the wild-type strain, the viability of the Deltasll1951 strain wa
254 uring amino acid stress, whereas in the wild-type strain these levels declined under the same growth
256 e generated through seeds by crossing a wild-type strain to a transgenic strain with altered centrome
257 ta mutant was less susceptible than the wild-type strain to noniron metalloporphyrins, further indica
258 with altered biofilm formation and the wild-type strain to predict drug targets against P. aeruginos
260 gnotobiotic mice together with 11 other wild-type strains to generate a 15-member artificial human gu
261 molecular clock for VP1 of circulating wild-type strains to infer that the mean time from the initia
263 comparative genome sequence analysis of wild-type strain TX82 and TX6051 and found a single mutation,
264 ce were inoculated intramuscularly with wild-type strain type 3 Dearing (T3D) and T3D-sigma1R202W, a
265 ing medium and grew slower than did the wild-type strain under aerobic and anaerobic conditions, at l
267 the Deltaspx strain grew as well as the wild-type strain under anaerobic conditions, the mutant strai
270 lted in the inability to compete with a wild-type strain under selective conditions that required spo
271 o, ylxY and ylyA, were outcompeted by a wild-type strain under sporulation-inducing conditions, but n
275 oid tissues and reveals the protective (wild-type strain) versus harmful (yopP-deficient strain) cons
276 iet did not affect the outcome that the wild-type strain was better able to persist and colonize.
277 n static conditions, the fitness of the wild-type strain was higher under fluctuating humidity condit
282 tions, a pair of congenic a and alpha mating type strains was generated by a series of 11 backcrosses
283 dependent growth arrest, and unlike the wild-type strain, was susceptible to fatty acid synthesis inh
284 from five single gene knockouts and the wild type strain, we decrease the mean squared error of predi
286 Edmonston vaccine/laboratory and IC323 wild-type strains were equally affected by the inhibitors.
288 is Tn library, and in various C. jejuni wild type strains, were compared and correlated to detect gen
289 tant was the same as that of the parent wild-type strain when cultured in nutrient-rich media with or
292 P as well as stimulates swarming in the wild-type strain, while overexpression of MotA from a plasmid
294 the initial interaction of the ATCC 19606(T) type strain with A549 human alveolar epithelial cells is
297 more-severe pulmonary infection by the wild-type strain (with a 30-fold increase in the number of co
299 using a virulent C. perfringens type D wild-type strain (WT), an isogenic ETX null mutant (etx mutan
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