ライフサイエンスコーパス: typhimurium

1 h Salmonella enterica serovar Typhimurium (S Typhimurium).

2 erichia coli and Salmonella enterica serovar Typhimurium).

3 hila, Pseudomonas aeruginosa, and Salmonella typhimurium.

4 ty of Zur, ZntR, RcnR and FrmR in Salmonella Typhimurium.

5 e model pathogen Salmonella enterica serovar Typhimurium.

6 bic expansion of Salmonella enterica serovar Typhimurium.

7 ompared to that of the nontyphoidal serovarS Typhimurium.

8 including in the enteric pathogen Salmonella typhimurium.

9 ainst the drug-resistant bacteria Salmonella typhimurium.

10 munoassay of Escherichia coli and Salmonella typhimurium.

11 serotypes Enteritidis, Javiana, Panama, and Typhimurium.

12 n and exhibit increased susceptibility to S. Typhimurium.

13 l biosensor suitable for the detection of S. Typhimurium.

14 know the concentration of serovar Salmonella typhimurium.

15 oducing Klebsiella pneumoniae and Salmonella typhimurium.

16 ic separation (IMS) for detecting Salmonella typhimurium.

17 enzae, and Salmonella enterica serovar Typhi/Typhimurium.

18 ing infection withSalmonella entericaserovar Typhimurium.

19 ) compared to infections with S. Hadar or S. Typhimurium.

20 ellular pathogen Salmonella enterica serovar Typhimurium.

21 thogens, Francisella novicida and Salmonella typhimurium.

22 iving MET-1 or control, then gavaged with S. typhimurium.

23 ed at significantly lowers levels than in S. Typhimurium.

24 gen-CRM197 glycoconjugate vaccine against S. Typhimurium.

25 Enteritidis and 152 (43.3%) were Salmonella Typhimurium.

26 mucosa of mice infected with DsRed-labeled S typhimurium.

27 nvasive pathogen Salmonella enterica serovar Typhimurium.

28 ared with enterocolitis-causing strains of S Typhimurium.

29 s from Salmonella Choleraesuis in Salmonella Typhimurium.

30 particularly E. coli and Salmonella enterica Typhimurium.

31 t conducive to the intracellular growth of S Typhimurium.

32 s with the intracellular pathogen Salmonella typhimurium.

33 om primary human monocytes in response to S. typhimurium.

34 s, 40% (41) were Salmonella enterica serovar Typhimurium, 10% (10) were Salmonella enterica serovar E

35 5 toxin encoded by the broad-host Salmonella Typhimurium (15) .

36 other foodborne pathogens such as Salmonella Typhimurium, (7%) Listeria monocytogenes (3%) and Escher

37 FrmR from Salmonella enterica serovar typhimurium (a CsoR/RcnR-like transcriptional de-repress

38 Typhimurium), a common cause of food-borne gastroenterit

39 olated at higher frequencies than Salmonella Typhimurium, a common cause of human illness.

40 uccumb to systemic infection with Salmonella Typhimurium, a pathogenic bacterium that multiplies with

41 To directly detect S. typhimurium after IMS, a sandwich immunoassay was implem

42 Typhimurium, allowing the bacteria to compete with the m

43 6.1%), of which 8017 (79.1%) were Salmonella Typhimurium and 1608 (15.8%) were Salmonella Enteritidis

44 A majority (88/114 [77%]) of Salmonella Typhimurium and 30% (24/79) of Salmonella Enteritidis is

45 in S. Paratyphi A that does not occur in S. Typhimurium and demonstrate curious distinctions in moti

46 periplasmic globular domain of PbgA from S. typhimurium and E. coli, which revealed that the globula

47 ), particularly Salmonella enterica serovars Typhimurium and Enteritidis, is responsible for a major

48 In Salmonella typhimurium and Escherichia coli, the virulence determin

49 ers, produced by Salmonella enterica serovar Typhimurium and Escherichia coli.

50 8 in response to NLRC4 activators Salmonella Typhimurium and flagellin, canonical or non-canonical NL

51 neficial to the host during infection with S Typhimurium and possibly other enteric pathogens.

52 seal and cattle populations and that both S. Typhimurium and S.

53 neumophila and three CFU per reaction for S. typhimurium and S. aureus.

54 We evaluated the capacities of S. Typhimurium and Salmonella enterica serovar Enteritidis

55 esting Shigella, Salmonella spp., Salmonella typhimurium and Staphylococcus aureus on E. coli specifi

56 ylation by cytosolic flagellin of Salmonella Typhimurium and Yersinia enterocolitica.

57 s that use T3SS, Salmonella enterica serovar Typhimurium and Yersinia pseudotuberculosis.

58 o flagellins from Salmonella typhimurium (S. typhimurium) and Bacillus subtilis (B. subtilis) were ex

59 y modified nonhalotolerant cells (Salmonella typhimurium) and dead vs. live differentiation of nonhal

60 Typhimurium)] and two extracellular (Vibrio cholerae and

61 intestinalis, Ruminococcus obeum, Salmonella typhimurium, and Clostridium difficile) to quantify, exp

62 endai are all noticeably less motile than S. Typhimurium, and comparative transcriptome sequencing (R

63 in vitro grown S. Typhi, S. Paratyphi A, S. Typhimurium, and E. coli were used to confirm the specif

64 rial pathogens (Escherichia coli, Salmonella typhimurium, and methicillin-resistant Staphylococcus au

65 ng latex beads, Escherichia coli, Salmonella typhimurium, and Mycobacterium tuberculosis in human and

66 etection of model food pathogens, Salmonella typhimurium, and Staphylococcus aureus, in which the flu

67 ane vesicles (OMVs) isolated from Salmonella Typhimurium are potentially useful for developing subuni

68 3SS and fT3SS of Salmonella enterica serovar Typhimurium at ~5 and ~4 nm resolution using electron cr

69 ecies, Pseudomonas putida KT2440, Salmonella Typhimurium ATCC 14028, Staphylococcus epidermidis ATCC

70 ected epithelial cells, a subpopulation of S Typhimurium bacteria damage their internalization vacuol

71 viral nanocontainer based on the Salmonella typhimurium bacteriophage P22 capsid, genetically incorp

72 ceptible to systemic infection by Salmonella Typhimurium because of loss-of-function mutations in Nra

73 d-borne pathogen Salmonella enterica serovar Typhimurium benefits from acute inflammation in part by

74 njugate vaccines against invasive African S. Typhimurium can have profound effects on immunogenicity

75 Salmonella enterica serovar Typhimurium can inject effector proteins into host cells

76 Most lineages of the S. enterica subspecies Typhimurium cause gastroenteritis in humans; however, th

77 Salmonella Typhimurium causes a self-limiting gastroenteritis that

78 Conversely, Salmonella enterica serovar Typhimurium causes gastroenteritis in humans and thrives

79 llular bacterium Salmonella enterica serovar Typhimurium causes persistent systemic inflammatory dise

80 h as that mediated by l-asparaginase II of S Typhimurium causes suppression of activation-induced T c

81 samples were spiked with standard Salmonella typhimurium cells, and detection was done by measuring t

82 Typhimurium chaperone and translocator proteins.

83 ability to kill Salmonella enterica serovar Typhimurium compared to that of macrophages isolated fro

84 T313 pathovar of Salmonella enterica serovar Typhimurium contributes to a high burden of invasive dis

85 Deletion of the S Typhimurium copper exporters, CopA and GolT, was found t

86 nstrate that the Salmonella enterica serovar Typhimurium core promoter is more active than previously

87 S. Typhimurium counters this defense pathway by delivering

88 atory response induced by Salmonella serovar Typhimurium creates a favorable niche for this gut patho

89 The vaccines S. Typhimurium CVD 1931 and S. Enteritidis CVD 1944 were im

90 S. Typhimurium CVD 1931 protected mice against the group B

91 , CVD 1944 did mediate protection against S. Typhimurium D65 (81% efficacy).

92 gesting that cytokinesis failure caused by S Typhimurium delays epithelial cell turnover in the intes

93 introduced into three constructed Salmonella Typhimurium Deltaasd mutants: SLT11 (DeltarfbP), SLT12 (

94 impedimetric aptamer-based biosensor for S. typhimurium detection.

95 ile infection, could also protect against S. typhimurium disease.

96 cles (OMVs) from Salmonella enterica serovar Typhimurium displaying the variable N terminus of PspA (

97 the multidrug-resistant bacterium Salmonella typhimurium DT 104.

98 zation niche for Salmonella enterica serovar Typhimurium during gastrointestinal infections.

99 inoculated Enterococcus spp. and Salmonella typhimurium during the drying of struvite under controll

100 Though several key S Typhimurium effector genes are missing (e.g., avrA, sopB

101 Typhimurium effector, SlrP, prevented anorexia caused by

102 monella enterica subspecies enterica serovar Typhimurium employs a dedicated mechanism, driven by the

103 la pneumoniae or Salmonella enterica serovar Typhimurium, enhanced translocation.

104 al replication, its administration reduced S Typhimurium epithelial cell invasion and lowered the ind

105 orescens, Salmonella Enteritidis, Salmonella Typhimurium, Escherichia coli).

106 r amounts of SPI-1 effector proteins than S. Typhimurium, especially under aerobic growth.

107 tudy showed that Salmonella enterica serovar Typhimurium exhibits sliding motility under magnesium-li

108 Salmonella enterica serovar Typhimurium exploits the host's type I interferon (IFN-I

109 Enteritidis cases have risen and Salmonella Typhimurium fallen.

110 In Salmonella enterica serovar Typhimurium, flagella-mediated motility is repressed by

111 phosphorylation, whereas deletion of the S. Typhimurium flagellin carboxy-terminus prevented caspase

112 isolated Salmonella, 34.50% was confirmed S. Typhimurium, followed by S. Heidelberg (10.86%) and S. E

113 nd II genes competes poorly with wild-type S Typhimurium for colonization of target tissues.

114 creen mutants of Salmonella enterica serovar Typhimurium for their ability to infect and grow in the

115 the aptamer biosensor could discriminate S. typhimurium from 6 other model bacteria strains.

116 Typhimurium from infecting the host.

117 h intraluminal CX3CR1(+) cells preventing S. Typhimurium from infecting the host.

118 to immune serum identified a repertoire of S Typhimurium genes that, when interrupted, result in incr

119 Mutants of three S Typhimurium genes, STM1461 (ydgT), STM2829 (recA), and S

120 The S. Typhimurium genome contains three nitrate reductases, en

121 ore genes within Salmonella enterica serovar Typhimurium genomes reveals a high degree of allelic var

122 The lower levels of S Typhimurium gut colonization and intestinal inflammation

123 r serovars of Salmonella enterica, Typhi and Typhimurium, have evolved a two-component regulatory sys

124 f a small heat shock protein from Salmonella typhimurium in a dimeric form and two higher oligomeric

125 ublished paper-based detection method for S. typhimurium in bird feces and whole milk.

126 persistence of Salmonella enterica serotype Typhimurium in chickens.

127 ally confirmed by capturing and detecting S. typhimurium in ground chicken and ground beef.

128 sed platform was applied for detection of S. typhimurium in inoculated Starling bird fecal samples an

129 in vitro and increased virulence of serovar Typhimurium in mice.

130 and promote virulence of S. enterica serovar Typhimurium in mice.

131 However, the response to S. typhimurium in primary human monocytes has not been stud

132 ated its suitability for the detection of S. typhimurium in spiked (1 x 10(2), 1 x 10(4) and 1 x 10(6

133 unosensor was able to specifically detect S. typhimurium in spiked water and juice samples with a sen

134 The aptasensor detected S. Typhimurium in the concentration range 10(2)-10(8) CFU m

135 required for survival and proliferation of S Typhimurium in the epithelial cell cytosol.

136 ell-dependent opsonophagocytic killing of S. Typhimurium in vitro.

137 ES cell-derived macrophages responded to S. Typhimurium, in a comparable manner to mouse bone marrow

138 SPI-1 expression between S. Paratyphi A andS Typhimurium, indicate that S. Paratyphi A host cell inva

139 at intracellular Salmonella enterica serovar Typhimurium induced the binucleation of a large proporti

140 Additionally, under these conditions S. typhimurium-induced IL-1 release occurred independently

141 Typhimurium-induced inflammation is the production of ox

142 The impaired ability to respond to S Typhimurium-induced oxidative stress results in reactive

143 iently attenuate Salmonella enterica serovar Typhimurium-induced pyroptosis and proinflammatory cytok

144 downstream of IFN-I and RIP3 signaling in S Typhimurium-infected macrophages.

145 model of sepsis, we observed that Salmonella typhimurium-infected mice exhibited simultaneous impaire

146 rest due to its high abundance at loci of S. Typhimurium infection and MLN disruption.

147 and host during Salmonella enterica serovar Typhimurium infection and reveal the molecular impact of

148 In this study, we demonstrate that S Typhimurium infection causes IFN-I-mediated up-regulatio

149 Salmonella typhimurium infection is reported to activate NLRP3 and

150 ity of disease induced by enteric Salmonella Typhimurium infection is strongly modulated by microbiot

151 Salmonella enterica serovar Typhimurium infection of immunocompetent mice results in

152 Typhimurium infection via Ab.

153 Here, we show that Salmonella Typhimurium infection was accompanied by dysbiosis, decr

154 f2 function and antioxidative responses to S Typhimurium infection, eventually leading to cell death.

155 uppressed during Salmonella enterica serovar Typhimurium infection, facilitating increased IL-1beta p

156 During Salmonella Typhimurium infection, intestinal CX3CR1(+) cells can ei

157 and function is severely disrupted during S. Typhimurium infection.

158 analyse MLN tissue from a murine model of S. Typhimurium infection.

159 subsequent phosphorylation in response to S Typhimurium infection.

160 entified to contribute to this step of the S Typhimurium infectious cycle.

161 y Salmonella enterica serovar Typhimurium (S Typhimurium) inhibits T cell responses and mediates viru

162 port that an intestinal pathogen, Salmonella Typhimurium, inhibits anorexia by manipulating the gut-b

163 ted with PBA exhibited significantly lower S Typhimurium intestinal colonization and dissemination to

164 standard DNA and 10(1) copies of Salmonella typhimurium InvA gene sequences (cloned in E. coli and a

165 An S. Typhimurium invA mutant defective in the Salmonella path

166 inactivated the SPI-1 T3SS and attenuated S. Typhimurium invasion.

167 Salmonella enterica serovar Typhimurium is a common cause of food-borne gastrointest

168 Salmonella enterica serotype Typhimurium is a food-borne pathogen that also selective

169 Salmonella typhimurium is a major cause of diarrhea and causes sign

170 ne resistance of Salmonella enterica serovar Typhimurium is attributed to the high-molecular-weight L

171 We demonstrate that while S. Typhimurium is equally invasive under both aerobic and m

172 lls, in which growth of cytosolic Salmonella Typhimurium is inhibited independently or prior to the o

173 Salmonella typhimurium is responsible for about a third of all case

174 ism of FrmR from Salmonella enterica serovar Typhimurium is triggered by metals in vitro, and variant

175 Typhimurium is unclear, although as IgG2a is induced by

176 gative bacterium, Salmonella Typhimurium (S. Typhimurium) is a food borne pathogen responsible for nu

177 s between this lineage and other non-iNTS S. Typhimurium isolates is the presence of prophage BTP1.

178 resistance was noted in 12.7% of Salmonella Typhimurium isolates, appearing in Bas-Congo from 2013 o

179 ease SPI-6 antibacterial activity and that S Typhimurium kills commensal bacteria in a T6SS-dependent

180 ined that, in a murine model of infection, S Typhimurium lacking both l-asparaginase I and II genes c

181 t in the closely related S. enterica serovar Typhimurium LT2 and from a mutant of S Montevideo, each

182 We recode 200 kb of the Salmonella typhimurium LT2 genome through a process we term SIRCAS

183 E. coli O157:H7, Salmonella enterica serovar Typhimurium LT2, Staphylococcus aureus, and Streptococcu

184 In Salmonella enterica serovar Typhimurium, Mg(2+) limitation induces transcription of

185 Following oral inoculation with S. Typhimurium, mice develop a hematopathological syndrome

186 lly found on the Salmonella enetrica serovar Typhimurium multi-resistance plasmid pMG101 from burns p

187 racellular infectious cycle, we screened a S Typhimurium multigene deletion library in Caco-2 C2Bbe1

188 , but avirulent, Salmonella enterica serovar Typhimurium mutant for its ability to compete with wild-

189 An S. Typhimurium mutant strain lacking these two effectors ex

190 In this study, a flagellin-deficient S. Typhimurium mutant was constructed.

191 Typhimurium mutants as surrogates for expression of tran

192 A library of 182 S. Typhimurium mutants each containing a targeted deletion

193 nvestigate the impact of coupling Salmonella typhimurium O-antigen to different amino acids of CRM197

194 actericidal activity can be influenced by S. Typhimurium OAg strain, most likely as a result of diffe

195 is study was to investigate the effect of S. typhimurium on inflammasomes in primary human monocytes.

196 led Nlrc4(-/-) BMDMs in their response to S. typhimurium or flagellin.

197 y IgY from chickens infected with Salmonella Typhimurium or S.

198 against 10,000 50% lethal doses (LD50) of S. Typhimurium or S. Enteritidis, respectively.

199 Salmonella typhimurium or Toxoplasma gondii were administered to kn

200 LRC4 in macrophages infected with Salmonella typhimurium or transfected with flagellin.

201 coli (EHEC) and Salmonella enterica serovar Typhimurium, or the surrogate murine infection model for

202 with IBS, larger numbers of E coli HS and S typhimurium passed through the epithelium than in biopsi

203 the prototypical Salmonella enterica serovar Typhimurium pathogenicity island 1 basal body, determine

204 oid from Scutellaria baicalensis, targets S. Typhimurium pathogenicity island-1 (SPI-1) type III secr

205 rug resistant, whereas a dominant Salmonella Typhimurium pathotype, ST313, was primarily associated w

206 deficient CX3CR1(-/-) mice the numbers of S. Typhimurium penetrating the epithelium were significantl

207 Typhimurium penetrating the epithelium were significantl

208 Typhimurium porins including outer membrane protein OmpD

209 ce IgG1, whereas Th1 Ags, such as Salmonella Typhimurium, predominantly induce IgG2a.

210 Upon entering the host, S Typhimurium preferentially colonizes Peyer's patches, a

211 a detection limit of 10(2) CFU mL(-1) for S. typhimurium, providing an instrument-free quantitative a

212 scherichia coli, Salmonella enterica serovar Typhimurium, Pseudomonas aeruginosa, Bacillus subtilis,

213 in nontyphoidal Salmonella enterica serovar Typhimurium reduced flagellin-induced pyroptosis.

214 Typhimurium, reducing virulence while increasing transmi

215 4.8; 95% CI, 1.1-21.1; P = .039).Salmonella Typhimurium represented 106 of 238 (44.5%) serotyped iso

216 ogenic bacterium Salmonella enterica serovar Typhimurium requires a T6SS encoded within Salmonella pa

217 1) for Staphylococcus aureus, and Salmonella typhimurium, respectively.

218 e II produced by Salmonella enterica serovar Typhimurium (S Typhimurium) inhibits T cell responses an

219 o infection with Salmonella enterica serovar Typhimurium (S Typhimurium).

220 Salmonella enterica serovar Typhimurium (S.

221 onocytogenes and Salmonella enterica serovar Typhimurium (S.

222 dent exposures to flagellins from Salmonella typhimurium (S. typhimurium) and Bacillus subtilis (B. s

223 The Gram-negative bacterium, Salmonella Typhimurium (S. Typhimurium) is a food borne pathogen re

224 In contrast, the S Typhimurium sapA mutants had similar sensitivity to osmo

225 Salmonella (Salmonella enterica serovar Typhimurium) secrete numerous effector proteins, includi

226 antigen extraction was done from Salmonella typhimurium serovars, under the optimized growth conditi

227 Flagellin isolated from the Salmonella Typhimurium SJW1660 strain, which differs by a point mut

228 generated defined mutant derivatives using S Typhimurium SL1344 as the host.

229 sitive immunosensor for detecting Salmonella typhimurium species.

230 we show that infection of host cells with S Typhimurium specifically induces the ubiquitination of t

231 Weight loss, serum cytokine levels, and S. typhimurium splenic translocation were measured.

232 e catalytic triad, is variable, with serovar Typhimurium SpvD having an arginine and serovar Enteriti

233 by a ubiquitin-binding domain in Salmonella Typhimurium SseL.

234 ections of Gram-negative Salmonella enterica Typhimurium (ST), a major source of human food poisoning

235 The B cell response to Salmonella typhimurium (STm) occurs massively at extrafollicular si

236 abbits were orally infected with S. enterica Typhimurium strain chi3987 harboring phagemid NgoPhi6 fm

237 d in vivo killing of the invasive African S. Typhimurium strain D23580.

238 nomes of S. enterica subsp. enterica serovar Typhimurium strain LT2 and Salmonella bongori strain 124

239 tuberculosis, in Salmonella enterica serovar Typhimurium strain SL3261.

240 not affect Ab responses to a noninvasive S. Typhimurium strain that specifically targeted the CX3CR1

241 Typhimurium strain that specifically targeted the CX3CR1

242 ture of metabolically competitive Salmonella typhimurium strains in microfluidic devices.

243 Typhimurium succinate utilization genes contribute to ef

244 ne secretion by S. Paratyphi A but not by S. Typhimurium, suggesting that SPI-1 expression is natural

245 Typhimurium survival mechanisms in macrophages, and can

246 r results reveal a novel strategy in which S Typhimurium T3SS effectors broaden their functions throu

247 w that human NAIP also senses the Salmonella Typhimurium T3SS inner rod protein PrgJ and that T3SS in

248 rene, an indirect mutagen, toward Salmonella typhimurium TA 98 and TA 100.

249 (4-NQO), a direct mutagen toward Salmonella typhimurium TA 98 and TA 100.

250 induced mutagenicity (26%) in the Salmonella typhimurium TA102 strain, as determined by the Ames test

251 genic effect by Ames test against Salmonella typhimurium TA98 and TA100 strains.

252 the highest antimutagenic activity toward S. typhimurium TA98 and TA100.

253 more susceptible to systemic infection by S Typhimurium than wild-type mice.

254 activity against Salmonella enterica serovar Typhimurium that is not shared by the related purine met

255 y labeled Escherichia coli HS and Salmonella typhimurium that passed through from the mucosal side to

256 together with l-asparaginase I to provide S Typhimurium the ability to catabolize asparagine and ass

257 genes, Pseudomonas aeruginosa and Salmonella Typhimurium The geranylated residues are located in the

258 tested for efficacy only against Salmonella Typhimurium, the modified Salmonella strain may be able

259 ions, strains of Salmonella enterica serovar Typhimurium, the murine model of S Typhi, in which vario

260 ne expression during infection by Salmonella typhimurium This occurred in the first 3 d of infection,

261 Typhimurium tissue colonization and consequently disease

262 the response of Salmonella enterica serovar Typhimurium to diverse environmental challenges encounte

263 isotypes specific for the O:4 antigen of S. Typhimurium to effect in vitro and in vivo killing of th

264 Typhimurium to H2O2L and H2O2H, and the results were val

265 ing transposon insertion mutant library of S Typhimurium to immune serum identified a repertoire of S

266 d the ability of Salmonella enterica serovar Typhimurium to infect the central nervous system and cau

267 amily members of Salmonella enterica serovar Typhimurium to link the constitutively expressed CspC an

268 briae (Lpf), which facilitate adherence of S Typhimurium to M cells.

269 Thus, manganese acquisition enables S. Typhimurium to overcome host antimicrobial defenses and

270 In order to assess the ability of Salmonella Typhimurium to replicate in human macrophages, we infect

271 Typhimurium to utilize a variety of carbon sources, incl

272 We observed that the number of S. Typhimurium traversing the epithelium did not differ bet

273 Typhimurium traversing the epithelium did not differ bet

274 ntracellular pathogens, including Salmonella typhimurium, trigger autophagy in host cells, which is w

275 e killing of Escherichia coli and Salmonella typhimurium, two common pathogens, at levels 10- to 20-t

276 solution in situ structure of the Salmonella Typhimurium type III secretion machine obtained by high-

277 Typhimurium undergoes an incomplete tricarboxylic acid (

278 estinal pathogen Salmonella enterica serovar Typhimurium uses specialized metal transporters to evade

279 To pinpoint S Typhimurium virulence factors responsible for these step

280 that asparagine catabolism contributes to S Typhimurium virulence, providing new insights into the c

281 Infection of DR3(-/-) mice with Salmonella typhimurium was associated with defective microbial clea

282 ng this system, the limit of detection of S. typhimurium was found to be 10(2) CFU mL(-1) in culturin

283 Salmonella Typhimurium was infrequent (2.3% pups; 4/175), mostly si

284 ly infected with Salmonella enterica serovar Typhimurium was investigated.

285 ver, in these mice the number of invading S. Typhimurium was significantly reduced after the adoptive

286 Typhimurium was significantly reduced after the adoptive

287 ein secretion in Salmonella enterica serovar Typhimurium, we discovered that several TCMs can attenua

288 Typhimurium, we found that ArtB binds human glycans, ter

289 of designing an effective vaccine against S. Typhimurium, we have synthesized different glycoconjugat

290 Here, by studying Salmonella Typhimurium, we show that the E3 ligase LUBAC generates

291 hin 1 hour, greater numbers of T gondii or S typhimurium were present within mucosae of mice with mig

292 d protection against infection by Salmonella typhimurium were spared.

293 cytogenes V7 and Salmonella enterica serovar Typhimurium were used as model pathogens to evaluate the

294 nse to the intracellular pathogen Salmonella typhimurium, which can disrupt metabolism by uptake of h

295 he phagosome mediates host defense against S Typhimurium, which is counteracted by copper export from

296 available experimental data from Salmonella typhimurium with only a single free parameter.

297 a direct label-free detection of Salmonella Typhimurium with the limit of detection (LOD) of 10(4) C

298 used to capture a food pathogen, Salmonella typhimurium, with starting concentrations as low as 10(

299 od was confirmed to be highly specific to S. typhimurium without interference from other pathogenic b

300 The S Typhimurium yfgA mutant lost the characteristic Salmonel