ライフサイエンスコーパス: typhlocolitis

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1 type type H. hepaticus developed significant typhlocolitis.

2 -kappaB-deficient mice developed spontaneous typhlocolitis.

3 th either cdtB mutant resulted in attenuated typhlocolitis and hyperplasia compared to infection with

4 ection in IL-10(-/-) mice causes subclinical typhlocolitis and that infection with H. trogontum (with

5 role in hepatitis, hepatocellular carcinoma, typhlocolitis, and lower-bowel carcinoma in murine colon

6 H. saguini induced a significant typhlocolitis, associated epithelial defects, mucosa-ass

7 with wild-type H. hepaticus exhibited severe typhlocolitis at 8 months after infection, while animals

8 68, C57BL/6 IL-10(-/-) mice developed severe typhlocolitis best evaluated at the ileocecocolic juncti

9 with Helicobacter rodentium had subclinical typhlocolitis but developed severe diarrhea and loss of

10 ion of Smad3/Rag2-DKO from bacterial-induced typhlocolitis, dysplasia, and tumor development, whereas

11 d progressive, proliferative, and dysplastic typhlocolitis in aging (18- to 24-month-old) Syrian hams

12 ohepatic helicobacters, WT(Hc) did not cause typhlocolitis in B6 mice, but mild to severe lesions dev

13 Other pathogens associated with typhlocolitis in hamsters, Clostridium difficile, Lawson

14 including necrotic enteritis in piglets and typhlocolitis in horses.

15 lar mediators for promoting H. bilis-induced typhlocolitis in mice.

16 urther characterize this finding, we induced typhlocolitis in rederived NF-kappaB-deficient mice usin

17 paticus, which induces chronic hepatitis and typhlocolitis in susceptible mouse strains, produces a c

18 deficient mice by this bacterium resulted in typhlocolitis similar to that observed with other helico

19 C57BL/129 mice developed mild gastritis and typhlocolitis, they had robust immunoglobulin G (IgG) an

20 Acute typhlocolitis was accompanied by signs of sepsis support

21 H. trogontum-induced typhlocolitis was also associated with a significant dec

22 Ten weeks postinoculation, the severity of typhlocolitis was assessed by histologic examination of

23 Proliferative typhlocolitis was characterized by focal to segmental ar

24 By using a murine model of bacteria-induced typhlocolitis, we investigated the role of IL-17A, IL-17

25 induces a rapid-onset, erosive to ulcerative typhlocolitis which impacts the normal anaerobic flora o

26 of body condition with erosive to ulcerative typhlocolitis within 1 to 3 weeks of experimental infect

27 tum also developed severe clinical signs and typhlocolitis within 2 to 4 weeks, whereas B6 mice colon

28 H. trogontum resulted in acute morbidity and typhlocolitis within 8 days.

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