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1 ditions, the most concentrated BA was always tyramine.
2 makes it a promising tool for quantifying of tyramine.
3  process is not facilitated by the substrate tyramine.
4 m the electrooxidation and polymerization of tyramine.
5 es and ratiometric response was obtained for tyramine.
6 nylalanine to phenethylamine and tyrosine to tyramine.
7 rine DHPG/NE ratio and the pressor effect of tyramine.
8 e were achieved with these concentrations of tyramine.
9 phenylethylamine, benzylamine, dopamine, and tyramine.
10 arboxylase (TDC), which converts tyrosine to tyramine.
11 s the plant PPO's acceptance or rejection of tyramine.
12  be reduced and legal limits established for tyramine.
13 anisation specifically through the action of tyramine.
14 sed to 47% with increasing concentrations of tyramine (0.129 to 25.8 mmol/L; P<0.001).
15 along with two natural monomers N-E-caffeoyl tyramine (1) and N-E-feruloyl tyramine (2), using centri
16 ne concentration after the administration of tyramine (12 vs. 56+/-63 pg per milliliter [0.07 vs. 0.3
17  in which the intermediate 4-[(18)F]fluoro-m-tyramine ([(18)F]16) was prepared using a diaryliodonium
18 s N-E-caffeoyl tyramine (1) and N-E-feruloyl tyramine (2), using centrifugal partition chromatography
19  percentage reductions in FVC in response to tyramine (-24 +/- 7 vs. -55 +/- 6 %), phenylephrine (-12
20  young men, the vasoconstrictor responses to tyramine (-25 +/- 1 versus -56 +/- 6%), phenylephrine (-
21  did not significantly blunt the response to tyramine (-30 +/- 2 versus -36 +/- 7%; P = 0.4) or pheny
22         Maximal vasoconstrictor responses to tyramine (-30 +/- 3 versus -41 +/- 3%), phenylephrine (-
23                                              Tyramine (4-hydroxyphenethylamine), which is a monoamine
24 r compared with young men in the presence of tyramine (-6 +/- 7 versus -31 +/- 6%), phenylephrine (-3
25 differentially blunt the pressor response to tyramine, a marker for NE uptake.
26 mpact courtship drive and were controlled by tyramine-a biogenic amine related to dopamine, whose rol
27                                          How tyramine acts on neuronal, cellular and circuit levels i
28 atients supine and standing before and after tyramine administration.
29  artery for intra-arterial administration of tyramine, an agent which displaces noradrenaline from th
30 ulation, we infused graded concentrations of tyramine, an indirect presynaptic norepinephrine release
31 -trans-feruloyl tyramine, and N-cis-feruloyl tyramine, an unreported N-feruloyl tyramine dimer was ch
32       In bovine adrenal chromaffin vesicles, tyramine and (-)erythro-hydroxyephedrine have apparent p
33 rong inducers of bfeA transcription, whereas tyramine and 3,4-dihydroxymandelic acid demonstrated low
34 Use of leaves of some species with prunasin, tyramine and beta-p-arbutin, may be limited in food appl
35 rations of bark lignans, coumarins, proline, tyramine and defensive proteins, and was characterized b
36  oriented approximately perpendicular to the tyramine and directed toward the extracellular surface.
37  used as a carbon and nitrogen source, while tyramine and dopamine can be used only as sources of nit
38 amine (PEA) and its hydroxylated derivatives tyramine and dopamine.
39                                              Tyramine and histamine are the biogenic amines (BA) most
40                               Extremely high tyramine and histamine contents, 1700 and 700 mg/kg, res
41              The synergistic cytotoxicity of tyramine and histamine should be taken into account when
42                            Co-treatment with tyramine and histamine was associated with a stronger cy
43  of dietary BA, the in vitro cytotoxicity of tyramine and histamine was investigated.
44 erivatization with benzoyl chloride, whereas tyramine and histamine were analysed by fluorescence det
45                                              Tyramine and histamine were toxic for HT29 intestinal ce
46 logically active biogenic amines, especially tyramine and histamine, in fermented beverages.
47                                              Tyramine and histamine, the most toxic biogenic amines (
48            When used to analyze transport of tyramine and hydroxyephedrine by the vesicular amine tra
49              To further clarify the roles of tyramine and its metabolite octopamine, we have cloned t
50             Results showed that N-E-feruloyl tyramine and N-E-caffeoyl tyramine exhibited the highest
51                             We conclude that tyramine and octopamine act as neurotransmitters/modulat
52   Flies with a mutation in dTdc2 lack neural tyramine and octopamine and are female sterile due to eg
53                                              Tyramine and octopamine released from neurons expressing
54 ions (CFSs) of lactic acid bacteria (LAB) on tyramine and other biogenic amine production by differen
55 tyrosine and phenylalanine were converted to tyramine and phenethylamine by tyrosine and phenylalanin
56           The reproducibility of delta15N of tyramine and phenethylamine measured by GCC-IRMS average
57 opposite trend was observed, with histamine, tyramine and putrescine being the most abundant in Pigno
58 ere was prevalence of cadaverine followed by tyramine and putrescine; and total amine levels were low
59 re-activity analyses of APP derivatives with tyramine and the neurotoxin 1-methyl-4-phenylpyridinium
60 ctivities elicited by trace amines such as p-tyramine and the psychostimulant amphetamines are manife
61                       Dietary amines such as tyramine and tryptamine also inhibited G34 cleavage.
62                                              Tyramine and tryptamine were found in cheese from high S
63 l SCC categories contained spermine; whereas tyramine and tryptamine were only detected in cheese fro
64 thylamine, putrescine, spermine, spermidine, tyramine and tryptamine) in fish tissues.
65 cadaverine, spermidine, spermine, histamine, tyramine and tryptamine) were determined.
66 osine decarboxylase to convert tyrosine into tyramine and tyramine beta-hydroxylase to convert tyrami
67     Only two biogenic amines (tryptamine and tyramine) and two polyamines (spermidine and spermine) w
68 own serotonin, norepinephrine, dopamine, and tyramine, and because the lung is a major metabolic orga
69                            Phenylethylamine, tyramine, and dopamine are oxidized by TynA to the corre
70 ng, resistance to the NE-releasing effect of tyramine, and increased sensitivity to adrenergic agonis
71 e outward transport or external binding of m-tyramine, and it was prevented by cocaine.
72 ic acid, rutin, scopoletin, N-trans-feruloyl tyramine, and N-cis-feruloyl tyramine, an unreported N-f
73 ve and dietary monoamines such as serotonin, tyramine, and phenylethylamine.
74 mine, naTA, N-acetylserotonin, OA, dopamine, tyramine, and serotonin also have been consistently iden
75 ding L-3,4-dihydroxyphenylalanine, dopamine, tyramine, and serotonin were identified and their relati
76      We conclude that TyrR is a receptor for tyramine, and suggest that it serves to curb high levels
77 noreactive with antisera against octopamine, tyramine, and the enzymes required for their synthesis,
78 yramine, combined the patterns seen with the tyramine- and octopamine antisera.
79 nity to tyramine releasing sites, a specific tyramine antibody was applied.
80 fandel red wines revealed that histamine and tyramine are produced during yeast and malolactic fermen
81 t of the mammalian DATs in that dopamine and tyramine are the preferred substrates, whereas octopamin
82                       Our findings establish tyramine as a neurotransmitter in C. elegans, and we sug
83 sized a series of (p-O-sulfamoyl)-N-alkanoyl tyramines as nonsteroidal estrone sulfatase inhibitors.
84 e legal limit, increases the cytotoxicity of tyramine at concentrations frequently reached in some fo
85 t from one isolate, S. thermophilus produced tyramine at low (47 isolates) and medium (10 isolates) l
86 rovides direct evidence that octopamine- and tyramine-based neuromodulation can be mediated by astroc
87 opic correlations that demonstrated feruloyl tyramines being incorporated into the lignin biopolymers
88                      We found that levels of tyramine beta-hydroxylase (TbetaH), an essential enzyme
89  synthesis, tyrosine decarboxylase (TDC) and tyramine beta-hydroxylase (TBH).
90 ns tyrosine decarboxylase gene, tdc-1, and a tyramine beta-hydroxylase gene, tbh-1.
91 xylase to convert tyrosine into tyramine and tyramine beta-hydroxylase to convert tyramine into octop
92                                              Tyramine beta-monooxygenase (TbetaM) belongs to a family
93                                   The enzyme tyramine beta-monooxygenase (TbetaM) belongs to a small
94                                              Tyramine beta-monooxygenase (TbetaM) catalyzes the synth
95                                              Tyramine beta-monooxygenase (TbetaM), the insect homolog
96 idue, Met471, coordinating the Cu(M) site of tyramine beta-monooxygenase (TbetaM).
97 n of cAMP when exposed to the trace amines p-tyramine, beta-phenethylamine, tryptamine, and octopamin
98                                              Tyramine, beta-phenylethylamine, tryptamine, and octopam
99 with the closely related invertebrate enzyme tyramine-beta-hydroxylase, which converts tyramine to OA
100 pine-induced increase in egg laying requires tyramine biosynthesis.
101                                   Therefore, tyramine can act as a classical inhibitory neurotransmit
102             This has led to a model in which tyramine can bind to either the Cu(I) or Cu(II) forms of
103                                              Tyramine can bind to two characterized receptors in hone
104            Owing to its chemical properties, tyramine can react with nitrite, a common food additive,
105 eir mode of action was also different, while tyramine caused cell necrosis, histamine induced apoptos
106 conformation) with either Na(125)I or (125)I-tyramine cellobiose, also known as the cell-trapping lig
107                          Injection of (125)I-tyramine-cellobiose-HDL demonstrated significantly great
108 g for TDC, the enzyme converting tyrosine to tyramine, combined the patterns seen with the tyramine-
109  synthesis of antimicrobial hydroxycinnamoyl-tyramine conjugates, and prevention of the hypersensitiv
110  increased synthesis of the hydroxycinnamoyl-tyramine conjugates, feruloyl-tyramine (FT) and coumaroy
111 will not cause a rise in blood pressure when tyramine-containing foods are ingested.
112  samples ranged from 3.3 to 30 mg/L, and the tyramine content ranged from 1.0 to 3.0 mg/L.
113                     Increased cadaverine and tyramine contents were found in samples with good sensor
114                      Activation of LGC-55 by tyramine coordinates the output of two distinct motor pr
115 ugates, feruloyl-tyramine (FT) and coumaroyl-tyramine (CT).
116  previously reported to incorporate feruloyl tyramine derivatives.
117                                              Tyramine detection was linearly proportional in response
118 ontrast, two substrates of DAT, dopamine and tyramine, did not significantly impact cross-linking.
119                      The pressor response to tyramine differentially distinguished maprotiline from s
120 -feruloyl tyramine, an unreported N-feruloyl tyramine dimer was characterized as the most abundant po
121 se inhibitor (p-O-sulfamoyl)-N-tetradecanoyl tyramine (DU-14) could reverse scopolamine induced amnes
122 that the vasoconstrictor responses evoked by tyramine during exercise or adenosine were repeatable in
123 ith their sum were putrescine, histamine and tyramine, even if reached levels were below toxicity thr
124                    The low and high doses of tyramine evoked significant reductions in FVC (vasoconst
125 to brachial artery infusions of two doses of tyramine (evokes endogenous noradrenaline release) in 10
126              FVC responses to intra-arterial tyramine (evokes endogenous noradrenaline release), phen
127 es to local intrafemoral artery infusions of tyramine (evokes noradrenaline (NA) release), phenylephr
128  that N-E-feruloyl tyramine and N-E-caffeoyl tyramine exhibited the highest DPPH scavenging activity
129  at the lower end of NET inhibition, whereas tyramine exhibits a linear relation, with NET inhibition
130                                              Tyramine ferulate is a sink for the anticipated build-up
131 ohol-derived units and significant levels of tyramine ferulate.
132 ntained four amines; there was prevalence of tyramine followed by histamine, phenylethylamine and put
133 f 350 nM for (p-O-sulfamoyl)-N-tetradecanoyl tyramine for the inhibition of MDA-MB-231 estrone sulfat
134 te the utility of (p-O-sulfamoyl)-N-alkanoyl tyramines for the inhibition of breast cancer cell estro
135 droxycinnamoyl-tyramine conjugates, feruloyl-tyramine (FT) and coumaroyl-tyramine (CT).
136                               The inhibitory tyramine-gated chloride channel, LGC-55, induces head re
137 rward locomotion through the activation of a tyramine-gated chloride channel, LGC-55.
138 f LGC-55 in C. elegans, the first identified tyramine-gated chloride channel.
139 o inhibit egg laying, in part via the LGC-55 tyramine-gated Cl(-) channel on the HSNs.
140                                Surprisingly, tyramine had a stronger and more rapid cytotoxic effect
141 ontribute to a model in which octopamine and tyramine have distinct and separable neural activities.
142       The two biogenic amines octopamine and tyramine have recently been shown to modulate various pa
143 tants, with altered levels of octopamine and tyramine, have a locomotion deficit.
144 hand, the bacterial-related biogenic amines, tyramine, histamine, tryptamine and beta-phenylethylamin
145                              Octopamine- and tyramine immunostaining in the central complex differed
146                     In each brain hemisphere tyramine immunostaining was found in four neurons innerv
147 These results indicate an essential role for tyramine in cocaine sensitization in Drosophila.
148 not restore the vasoconstrictor responses to tyramine in contracting muscle during heavy rhythmic han
149  HILIC-UPLC-MS approach was elevated urinary tyramine in galN-treated rats, reflecting disturbed amin
150  our technique was demonstrated by analyzing tyramine in spiked serum and milk.
151 flies had reduced amounts of the trace amine tyramine in the brain because of reduced activity of the
152                                The amount of tyramine in the cheeses with the addition of the strain
153 en various potential roles of octopamine and tyramine in the larval brain, such as locomotion, learni
154 nses to endogenous noradrenaline release via tyramine in the skeletal muscle circulation, similar to
155 s, particularly of cadaverine, histamine and tyramine, in low-salt products.
156  a representative of a family of hydrophobic tyramines, in addition to being a potent bactericidal Gr
157                                            m-Tyramine increased the rate of reaction of (2-aminoethyl
158                                       This m-tyramine-induced increase in reactivity appeared to requ
159                       In both protocols, the tyramine-induced increases in deep venous noradrenaline
160                                     However, tyramine-induced mobilization of norepinephrine stores p
161                     Endotoxemia lessened the tyramine-induced reduction in leg vascular conductance (
162 emia + hypoxia (-17% +/- 21%) attenuated the tyramine-induced reduction in leg vascular conductance c
163 al BH(4) supplementation augments reflex and tyramine-induced VC in aged skin, suggesting that reduce
164 O: -17 +/- 2% DeltaCVC(base); P < 0.001) and tyramine infusion (Y: - 33 +/- 4% DeltaCVC(base), O: -15
165 er: 17 +/- 3% Delta CVC(base); P < 0.01) and tyramine infusion (young: 41 +/- 3, older: 21 +/- 4% Del
166 in temperature (T(sk)) = 30.5 degrees C) and tyramine infusion, respectively.
167 conductance) was evaluated by femoral artery tyramine infusion.
168                           Here, we show that tyramine inhibits head movements and forward locomotion
169 s determined by heart rate (HR) change after tyramine injection into the artery that perfused the sin
170 ine and tyramine beta-hydroxylase to convert tyramine into octopamine.
171                                              Tyramine is a biogenic compound derived from the decarbo
172 ansmitter in C. elegans, and we suggest that tyramine is a genuine neurotransmitter in other inverteb
173                                              Tyramine is an important neurotransmitter, neuromodulato
174 been reported; therefore, the measurement of tyramine is an urgent concern.
175 stomach lumen, the most favoured reaction in tyramine is C-nitrosation, which generates mutagenic pro
176 e shown that the product of C-nitrosation of tyramine is mutagenic, in the present article tyramine n
177                     The trace biogenic amine tyramine is present in the nervous systems of animals ra
178 PSS/AuNP/1-m-4-MP) for the quantification of tyramine is presented.
179                         The trace amine para-tyramine is structurally and functionally related to the
180                                              Tyramine is synthesized from tyrosine by the enzyme tyro
181 u(II) form of TbetaM is also reduced at high tyramine, leading us to propose the existence of a bindi
182  using mutants having altered octopamine and tyramine levels and by genetic interference with both sy
183                                    Moreover, tyramine-like immunoreactivity was also found in brain r
184                                              Tyramine-like labeling was observed in AmTAR1-IR positiv
185 nd urocortins, together with the trace amine tyramine, may have a role in the human cardiovascular sy
186  describe the use of MIP-1 alpha in a biotin tyramine-mediated proximity selection to guide the selec
187 atial arrangement in the receptors, with the tyramine moiety of alkaloids or Tyr1 of opioid peptides
188                      However, instead of the tyramine moiety that is present in methanofuran cofactor
189 residues in the bottom of the pocket and the tyramine N+ and OH groups forming ionic interactions or
190                                        After tyramine, NE spillover did not change significantly in p
191 n of Fru(M) by transformer expression in OCT/tyramine neurons changes the aggression versus courtship
192                       The regulation of this tyramine neurotransmitter was found to be linked to pati
193 yramine is mutagenic, in the present article tyramine nitrosation mechanisms have been characterized
194  structurally similar to the bVMAT substrate tyramine, none of them were measurably transported into
195 enzyme capable of synthesizing morphine from tyramine, norlaudanosoline, and codeine.
196                                 Furthermore, tyramine normalized the doubled leg vascular conductance
197 dopamine transport activity; norepinephrine, tyramine, octopamine, and histamine also have high affin
198                               Application of tyramine or octopamine to live preparations silenced dop
199           There was no evidence for feruloyl tyramines, or homologues thereof, accumulating as a chem
200 s could be rescued by feeding the flies with tyramine; other biogenic amines or amine precursors did
201 roup were structural features that increased tyramine oxidase inhibition.
202                          Characterization of tyramine oxidase, carried out prior to the inhibition ex
203  induces transcription of genes encoding the tyramine oxidase, TynA, and the aromatic aldehyde dehydr
204 sayed using commercially available microbial tyramine oxidase.
205 und to be a weak noncompetitive inhibitor of tyramine oxidase.
206  ethyl ester in dry THF with phenethylamine, tyramine, p-methoxyphenethylamine, 2-(p-tolyl)ethylamine
207 d metabolites, such as tyrosine, tryptamine, tyramine, phenethylamine, dopamine, 3-methoxytyramine, s
208 was impaired using iontophoretic delivery of tyramine, phentolamine, and bretylium followed by a nore
209 cadaverine, spermine, spermidine), aromatic (tyramine, phenylethylamine) or heterocyclic (histamine,
210 spermine, spermidine, putrescine, histamine, tyramine, phenylethylamine, cadaverine and serotonin) we
211 onitoring of six biogenic amines (histamine, tyramine, phenylethylamine, tryptamine, putrescine, and
212                                 We show that tyramine plays a specific role in the inhibition of egg
213                       The binding of heparin-tyramine polymer (HT) onto the polycarprolactone (PCL) s
214                           Phenethylamine and tyramine prepared from a mixture of 18 amino acids were
215                              Locally infused tyramine produced dose-dependent pressor responses, pred
216 50% CFS of P. acidophilus was the highest on tyramine production (55%) by Listeria monocytogenes, fol
217                                              Tyramine production by Escherichia coli was also inhibit
218 50% CFS of Pediococcus acidophilus inhibited tyramine production up to 98% by Salmonella paratyphi A.
219                       Significant amounts of tyramine, putrescine, and cadaverine occurred especially
220                          Of the BA examined, tyramine, putrescine, histamine and cadaverine showed hi
221 late Ca(2+) increases through the octopamine/tyramine receptor (Oct-TyrR) and the transient receptor
222 o serotonin receptors (ser-1 and ser-4), one tyramine receptor (ser-2), and two dopamine receptors (d
223 traction-response, whereas in solitary ones, tyramine receptor (TAR) signaling mediated repulsion-res
224 ccurring non-coding polymorphisms in tyra-3 (tyramine receptor 3), which encodes a G-protein-coupled
225 stigated the spatial brain expression of the tyramine receptor AmTAR1 using a specific antibody.
226 ramine, which indicates that this ionotropic tyramine receptor functions in tyramine signaling in viv
227 terns of different splice forms of the ser-2 tyramine receptor locus.
228                             We show that the tyramine receptor SER-2 acts through a Galphao pathway t
229 tly, we analyze a mutant allele of the ser-2 tyramine receptor, a target of the ttx-3 Lhx gene in the
230 dopamine receptor, LGC-55 is a high-affinity tyramine receptor, and LGC-40 is a low-affinity serotoni
231 aviors, we have cloned a putative octopamine/tyramine receptor.
232 obe and in the gnathal ganglion suggest that tyramine receptors are involved in modulating gustatory
233 pressing brain structures are in vicinity to tyramine releasing sites, a specific tyramine antibody w
234                      The dose of intravenous tyramine required to raise systolic blood pressure by 30
235 1 and TYRA-3, for NPY-like neuropeptides and tyramine respectively, do not appear to be involved in o
236 1) and female sex (P=0.02) predicted blunted tyramine responses.
237                      The patients followed a tyramine-restricted diet during the medication trial and
238 al-time cell analysis, whether histamine and tyramine show synergistic toxicity towards intestinal ce
239                    Finally, the N-E-caffeoyl tyramine showed the highest reducing power (EC50=26mug/m
240                   Putrescine, cadaverine and tyramine showed very good correspondence with the irradi
241                   Putrescine, cadaverine and tyramine showed very good correspondence with the level
242 is ionotropic tyramine receptor functions in tyramine signaling in vivo.
243  isolation, the activities of octopamine and tyramine signalings were respectively correlated with at
244 droxylation was observed for the reaction of tyramine solely with Met471Cys TbetaM.
245                             We knocked out a tyramine-specific receptor, TyrR, which was expressed in
246 obes, antennal lobes), indicating that other tyramine-specific receptors may be expressed there.
247 trescine, cadaverine, histamine, serotonine, tyramine, spermidine and spermine), as well as microbiol
248     Here, we report that the biogenic amines tyramine (TA) and octopamine (OA) in Drosophila melanoga
249                   In Caenorhabditis elegans, tyramine (TA) and octopamine (OA) inhibit aversive respo
250                           The biogenic amine tyramine (TA) regulates many aspects of invertebrate phy
251 s ample evidence linking octopamine (OA) and tyramine (TA) to several neurophysiological functions in
252 opamine (OA), L-3, 4-dihydroxyphenylalanine, tyramine (TA), and serotonin as well as metabolites 5-hy
253 e interactions, we have defined the roles of tyramine (TA), octopamine, and neuropeptides in the inhi
254 nvertebrate-specific octopamine-OARalpha and tyramine-TAR signalings respectively mediate attractive
255                                              Tyramine temporally coordinates the different phases of
256 in and liver MAO A and then followed by oral tyramine than was found on administration of tyramine to
257 t common tyrosinase substrates (tyrosine and tyramine), the enzyme is classified as a catechol oxidas
258                               In the case of tyramine, the product release from the oxidized enzyme-i
259 red to FD (from 1mgkg(-1) for putrescine and tyramine to 4mgkg(-1) for histamine); MS/MS method showe
260 tyramine than was found on administration of tyramine to controls.
261 assage of eggs through the vulva and release tyramine to inhibit egg laying, in part via the LGC-55 t
262 me tyramine-beta-hydroxylase, which converts tyramine to OA, suggesting that the OA-ir cells are inde
263 mmunostaining for TBH, the enzyme converting tyramine to octopamine, in contrast, was strikingly simi
264 yme that catalyzes the beta-hydroxylation of tyramine to yield octopamine.
265 reagent for the derivatization of histamine, tyramine, tryptamine and 2-phenylethylamine, which elimi
266 cadaverine, spermidine, spermine, histamine, tyramine, tryptamine and phenylethylamine) were determin
267 cadaverine, spermidine, spermine, histamine, tyramine, tryptamine and phenylethylamine) were determin
268 wine samples analysed had histamine (HIM) or Tyramine (TYM) levels above the limits considered as a p
269 vascular responses to noradrenaline (NA) and tyramine (TYR) are related to SNA such that individuals
270      BAs concentrations increased with time, tyramine (TYR), putrescine (PUT) and cadaverine (CAD) we
271          Cross-linking of DAT did not affect tyramine uptake.
272 ablished that the major product, MftA*, is a tyramine-valine-cross-linked peptide formed by MftC thro
273           AIY memory retrieval neurons sense tyramine via the SER-2 receptor, which is essential for
274 on in FBF in response to the highest dose of tyramine was blunted in older men compared with young me
275 osation of ethylbenzene, phenethylamine, and tyramine was carried out, using UV-visible absorption sp
276                                              Tyramine was found mainly in red wines at <1-3.4 mg/L, w
277 vasoconstriction caused by administration of tyramine was inversely related to workload.
278                                              Tyramine was isolated selectively and pre-concentrated b
279 was not influenced by ageing conditions, and tyramine was only detected in some samples after 43days
280                                     Overall, tyramine was the prevalent amine, followed by putrescine
281 polamine, scopoline, tropine, tropinone, and tyramine, was accomplished by comparison of the in-sourc
282 take and the pressor response to intravenous tyramine were assessed following the initial dose and af
283 asoconstrictor responses to the high dose of tyramine were determined in eight young adults during no
284 genous noradrenaline release (intra-arterial tyramine) were significantly blunted during moderate exe
285 sponses to local intra-arterial infusions of tyramine (which evokes endogenous NE release), phenyleph
286 C) responses to brachial artery infusions of tyramine (which evokes endogenous noradrenaline release)
287 he simulation analysis of phenethylamine and tyramine, which are major monoamine constituents of toba
288  (whole-body cooling) and pharmacologically (tyramine, which displaces noradrenaline from axon termin
289 ues range from 5 to 10 with the exception of tyramine, which exhibited smaller steady-state isotope e
290 enylacetic acid (DOPAC), methyldopamine, and tyramine, which gave negligible signals under conditions
291 ctive in a behavior that requires endogenous tyramine, which indicates that this ionotropic tyramine
292 m biogenic amines, especially histamine, and tyramine, which is an important consideration when selec
293           RIM synthesizes the neuromodulator tyramine, which is required in the L1 stage for learning
294 is associated with a large dietary intake of tyramine while taking MAO inhibitors has been reported;
295 increases in the tyrosine-derived metabolite tyramine, whose exogenous application elicits cell death
296 bly bound to this enzyme and displaceable by tyramine, will be an antidepressant which will not cause
297         In addition to this, co-injection of tyramine with DOPA, the precursor of melanin, had a stro
298 O: -34 +/- 2% DeltaCVC(base); P < 0.001) and tyramine (Y: -38 +/- 4% DeltaCVC(base), O: -35 +/- 3% De
299 ion (cold, young: 38 +/- 4, older: 31 +/- 3; tyramine, young: 36 +/- 3, older: 36 +/- 5 Delta %CVC(ba
300 ine (cold, young: 37 +/- 4, older: 34 +/- 4; tyramine, young: 40 +/- 4, older: 45 +/- 4% Delta CVC(ba
301 (4) (cold, young: 37 +/- 3, older: 36 +/- 3; tyramine, young: 41 +/- 2, older: 36 +/- 3% Delta CVC(ba

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