ライフサイエンスコーパス: tyrosine dephosphorylation

1 2A (PP2A, a Ser/Thr phosphatase activated by tyrosine dephosphorylation).

2 RPTPalpha plays in HCN channel function via tyrosine dephosphorylation.

3 lic forms is prevented in the bloodstream by tyrosine dephosphorylation.

4 ng that MnSOD-BP activity was upregulated by tyrosine dephosphorylation.

5 se phosphatase, MKP-1, is required for STAT1 tyrosine dephosphorylation.

6 itide 3-kinase from Cbl concomitant with Cbl tyrosine dephosphorylation.

7 d-inhibition mechanism that does not involve tyrosine dephosphorylation.

8 se activity of Hog1p is required for its own tyrosine dephosphorylation.

9 peaks at 1 and 9 min in association with Src tyrosine dephosphorylation.

10 e activation of the Cdc2/cyclin B complex by tyrosine dephosphorylation.

11 all STAT family members, is required for its tyrosine dephosphorylation.

12 the Stat1 N-terminal domain inhibited Stat1 tyrosine dephosphorylation.

13 This response preceded the tyrosine dephosphorylation and activation of cdc2 by sev

14 ng amebic adherence with galactose inhibited tyrosine dephosphorylation and amebic lysates had no eff

15 Biochemical analyses show that STAT1 tyrosine dephosphorylation and CRM1-mediated STAT1 nucle

16 14-3-3 and Hsp90 proteins blocked both the tyrosine dephosphorylation and inactivation of Raf-1, su

17 demonstrate here that activated Ras induces tyrosine dephosphorylation and inhibition of FAK mediate

18 HP2 expression exacerbates Cat.G-induced FAK tyrosine dephosphorylation and myocyte apoptosis.

19 .G induces SHP2 activation that leads to FAK tyrosine dephosphorylation and promotes cardiomyocyte an

20 e mutant of SHP2 (SHP2DN) prevented paxillin tyrosine dephosphorylation and Src activation induced by

21 idermal growth factor (EGF) induces paxillin tyrosine dephosphorylation and Src activation, but the s

22 domain couples inositide phosphorylation to tyrosine dephosphorylation and the regulation of intrace

23 P-MEG2 represents a novel connection between tyrosine dephosphorylation and the regulation of secreto

24 study identifies focal adhesion kinase (FAK) tyrosine dephosphorylation as an early mechanism that re

25 P1B also increased the overall rate of IRS-1 tyrosine dephosphorylation by 2.7-3.9-fold (p < 0.01).

26 lves binding of liprin-alpha to LAR-RPTP and tyrosine dephosphorylation by LAR-RPTP.

27 The inactivation of STAT1 occurs through tyrosine dephosphorylation by the tyrosine phosphatase T

28 bitor, pervanadate, blocks Cat.G-induced FAK tyrosine dephosphorylation, caspase-3 activation and DNA

29 actions, by preventing Stat1 deactivation by tyrosine dephosphorylation, cooperate with IFN-gamma/Sta

30 , platelet aggregation, clot retraction, and tyrosine dephosphorylation defects were rescued in the d

31 nic vasculopathy with the failure of PECAM-1 tyrosine dephosphorylation during the formation of blood

32 To avoid nonspecific tyrosine dephosphorylation, HePTP employs Thr106 in the

33 s sufficient and essential to induce protein tyrosine dephosphorylation in H. vermiformis.

34 though cell detachment induced rapid protein tyrosine dephosphorylation in Madin-Darby canine kidney

35 ar mechanism may regulate calpain-1-mediated tyrosine dephosphorylation in other cells.

36 BP itself undergoes functionally significant tyrosine dephosphorylation in response to apoptotic cell

37 ly dynamic, and suggest a potential role for tyrosine dephosphorylation in the insertion and maintena

38 kinase activity that was correlated with its tyrosine dephosphorylation in the synaptic membrane frac

39 SHP-1, which is responsible for JAK2 tyrosine dephosphorylation in VSMC, is completely deacti

40 hatase 1 as the enzyme responsible for STAT1 tyrosine dephosphorylation in VSMCs.

41 y serine phosphorylation, isomerization, and tyrosine dephosphorylation--in the regulation of FAK act

42 negative SHP2 mutant markedly attenuates FAK tyrosine dephosphorylation induced by Cat.G and protects

43 Tyrosine dephosphorylation is a component of this enhanc

44 3 in VSMCs is mediated by p60 c-Src, whereas tyrosine dephosphorylation is mediated by calcineurin.

45 Furthermore, the rate of Cdc2 tyrosine dephosphorylation is reduced by irradiation.

46 This result implicates regulation of Cdc2 tyrosine dephosphorylation, mainly carried out by the Cd

47 addition to protein phosphorylation, protein tyrosine dephosphorylation may play a role in the IL-4-i

48 the sensitive process, implying that protein tyrosine dephosphorylation must occur at or downstream o

49 treatment with pervanadate, indicating that tyrosine dephosphorylation of a critical V7-related subs

50 ens junction formation is accompanied by the tyrosine dephosphorylation of adherens junctions protein

51 at activation of postsynaptic PTP results in tyrosine dephosphorylation of AMPARs and their removal f

52 Geldanamycin also stimulated tyrosine dephosphorylation of beta-catenin and increased

53 PTPRZ1 constitutively promotes the tyrosine dephosphorylation of beta-catenin and, thus, be

54 s (>30 ng/ml), EGF treatment resulted in the tyrosine dephosphorylation of Cas in a time-dependent ma

55 ced by the FcgammaRIIb transmembrane domain: tyrosine dephosphorylation of CD19, inhibition of B cell

56 or GalNAc monomers blocked bacteria-induced tyrosine dephosphorylation of detergent-insoluble protei

57 of a cysteine-to-serine (C-S) mutant, caused tyrosine dephosphorylation of FKBP52, leading to efficie

58 at-shock treatment of HeLa cells resulted in tyrosine dephosphorylation of FKBP52, led to stabilizati

59 verexpress EGFR, EGF treatment induced rapid tyrosine dephosphorylation of focal adhesion kinase (FAK

60 p3p is the major phosphatase responsible for tyrosine dephosphorylation of Fus3p to maintain a low ba

61 Cell-substrate mediated tyrosine dephosphorylation of GAPa-p62 is defective in t

62 and collagen IV, but not laminin, results in tyrosine dephosphorylation of GAPa-p62.

63 DHPG-induced AMPAR internalization and tyrosine dephosphorylation of GluR2 (glutamate receptor

64 Tyrosine dephosphorylation of GSK-3, another putative me

65 n of H. vermiformis were capable of inducing tyrosine dephosphorylation of H. vermiformis proteins.

66 attachment failed to block bacteria-induced tyrosine dephosphorylation of H. vermiformis proteins.

67 ociated with inhibition of bacterial-induced tyrosine dephosphorylation of H. vermiformis proteins.

68 e the major phosphatases responsible for the tyrosine dephosphorylation of Hog1p.

69 Tyrosine dephosphorylation of host proteins was blocked

70 eceptor catalytic activity and by inhibiting tyrosine dephosphorylation of IRS proteins.

71 This physical interaction inhibited tyrosine dephosphorylation of IRS-1 or IRS-2 and increas

72 ptor induced activation of SHP-1 induces the tyrosine dephosphorylation of JAK2 that results in a com

73 vasion were associated with a time-dependent tyrosine dephosphorylation of multiple host cell protein

74 bind p130Cas (PA-PTP1B), causes substantial tyrosine dephosphorylation of p130Cas.

75 ephosphorylation reactions, with the initial tyrosine dephosphorylation of p42 MAPK being the most cr

76 pressing PAG/Cbp, we show that EphB2 induces tyrosine dephosphorylation of PAG/Cbp in a gamma-secreta

77 t cAMP causes rapid (</=1 min) and selective tyrosine dephosphorylation of paxillin, a focal adhesion

78 attachment and invasion was associated with tyrosine dephosphorylation of paxillin, pp125(FAK), and

79 ne precipitate phosphatase assays, there was tyrosine dephosphorylation of PECAM-1.

80 the tyrosine phosphorylation of Cbl and the tyrosine dephosphorylation of pp125(FAK) and p130(cas).

81 n tyrosine phosphatase-1 in the PGE2-induced tyrosine dephosphorylation of PTEN.

82 cultured cell lines and a defect in causing tyrosine dephosphorylation of specific proteins in infec

83 as a model system, we provide evidence that tyrosine dephosphorylation of STAT5 subsequent to IL-2-i

84 ating STAT activities through regulating the tyrosine dephosphorylation of STATs.

85 s integrin exhibited impaired TCPTP-mediated tyrosine dephosphorylation of TbetaRII that led to sever

86 required for invasion and is associated with tyrosine dephosphorylation of the Gal lectin and other h

87 at can be attributed to an inhibition of the tyrosine dephosphorylation of the inactive Cdc2/cyclin B

88 In addition to L. pneumophila-induced tyrosine dephosphorylation of the lectin, bacterial atta

89 To determine whether protein tyrosine dephosphorylation played a role in signaling pa

90 No protein tyrosine dephosphorylation signal was delivered after an

91 ive cells and rescued them from YopH-induced tyrosine dephosphorylation, signaling paralysis, and cel

92 egulation of recombinant NR1/2A receptors by tyrosine dephosphorylation that requires agonist binding

93 in CrkII binding affinity was reversible by tyrosine dephosphorylation with PTP1B and by mutation of