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1 L (category 1: erythema/erosion; category 2: ulcer).
2 ulcers and 6.8% in the helmet group had neck ulcers).
3 e treatment or being diagnosed with a peptic ulcer.
4  infection and a subsequent worsening of the ulcer.
5 thways on the spatial distribution of Buruli ulcer.
6 ortic rupture, and 10 had penetrating aortic ulcer.
7 tantly or previously cultured from the index ulcer.
8 ive in preventing hospital acquired pressure ulcer.
9 ere may have been some effect among Fusarium ulcers.
10 he outcome of treatment of infected pressure ulcers.
11 athy, 4.5% for neuropathy, and 5.7% for foot ulcers.
12 denied experiencing fevers, chills, or mouth ulcers.
13 tained in a commensal state in diabetic foot ulcers.
14 sociated to recurrences of infected pressure ulcers.
15 l status, parenteral nutrition, and pressure ulcers.
16 , ischemic rest pain, gangrene, or extremity ulcers.
17 n the treatment of severe filamentous fungal ulcers.
18 d fractures, failure to thrive, and pressure ulcers.
19 ion and treatment interventions for pressure ulcers.
20  towards prevention or treatment of pressure ulcers.
21  old worldwide and causing recurrent genital ulcers.
22 art bandage' for early detection of pressure ulcers.
23 ermit relief of pain and healing of ischemic ulcers.
24 patient population is patients with pressure ulcers.
25 into the care of diabetic patients with foot ulcers.
26 mina densa blistering, erosions, and chronic ulcers.
27 gainst HD strains from both genital and skin ulcers.
28 n patients with neuroischaemic diabetic foot ulcers.
29 patients with cystic fibrosis and nonhealing ulcers.
30 bundance in HSV-2 PCR-positive than negative ulcers.
31 l treatment for neuroischaemic diabetic foot ulcers.
32 tes are attributed to deep infection of foot ulcers.
33 0] per 1000 person-years; P = .40) or peptic ulcers (10.9 [95% CI, 7.7-15.5] vs 11.4 [95% CI, 8.0-16.
34 al upper gastrointestinal bleeding (125 with ulcers, 19 with Dieulafoy's lesions, and 4 with Mallory
35 he patients were graft rejection (1), shield ulcers (2), graft infection (2), and interface vasculari
36 osal healing (CDEIS <4) with absence of deep ulcers 48 weeks after randomisation.
37 nce after treatment was noted in 44 infected ulcers (63%), after a median interval of 1 year.
38 subscores of >6 in one or more segments with ulcers), a Crohn's Disease Activity Index (CDAI) of 150-
39                                       Buruli ulcer, a debilitating disease, is caused by Mycobacteriu
40                                      Corneal ulcer, a major cause of monocular blindness in developin
41 s of the microbiota colonizing diabetic foot ulcers, a common and costly complication of diabetes, an
42 umber of subjects with large erosions and/or ulcers after 14 days of exposure.
43 ed up their tolerability to alternative anti-ulcer agents, and investigated the T-cell reactivity to
44 or smear-positive filamentous fungal corneal ulcer and a baseline visual acuity of 20/40 to 20/400.
45 n Francisco, who had culture-positive fungal ulcer and baseline visual acuity of 20/400 or worse were
46 othesized relationships between the Pressure Ulcer and Fall Rate Quality Composite Index and other va
47                                 The Pressure Ulcer and Fall Rate Quality Composite Index is a step to
48 ignificantly associated with higher Pressure Ulcer and Fall Rate Quality Composite Index scores.
49 use were not associated with higher Pressure Ulcer and Fall Rate Quality Composite Index scores.
50                                 The Pressure Ulcer and Fall Rate Quality Composite Index was develope
51 ded to examine the usability of the Pressure Ulcer and Fall Rate Quality Composite Index.
52  care quality performance index-the Pressure Ulcer and Fall Rate Quality Composite Index.
53                                 The Pressure Ulcer and Fall Rate Quality Composite Index=100-PUR-FR,
54 n-steroid anti-inflammatory drug use, former ulcer and health behaviours.
55 nts at risk for a hospital-acquired pressure ulcer and higher unit rates of physical restraint use we
56 ely associated with gastric diseases such as ulcer and inflammation.
57 up (ie, 7.6% in the face mask group had nose ulcers and 6.8% in the helmet group had neck ulcers).
58 ndex terms and key words related to pressure ulcers and cost.
59 ed cell proliferation, and eventually peptic ulcers and gastric cancer.
60  forming a recognized risk factor for peptic ulcers and gastric cancer.
61 gs typical of CC complicated by several deep ulcers and perforation.
62 lusion criteria included the presence of leg ulcers and receiving microsclerotherapy or foam scleroth
63 nts with culture-positive filamentous fungal ulcers and visual acuity of 20/40 to 20/400 reexamined 6
64 ients with smear-positive filamentous fungal ulcers and visual acuity of 20/400 (logMAR 1.3) or worse
65  keratinocyte carcinoma, 0.03% for decubitus ulcer, and 0.01% for alopecia areata.
66  40.8% women; 24.6% for rest pain, 37.2% for ulcer, and 38.2% for gangrene).
67 tory distress syndrome, pneumonia, decubitus ulcer, and death) and hospital length of stay were compa
68 other malignancy data, SCC in situ, Marjolin ulcer, and genetic disorders predisposing to cSCC.
69 er gastric pH, a higher incidence of gastric ulcers, and a higher incidence of fecal occult blood los
70 d the self-reported rates of eye infections, ulcers, and abrasions each year.
71 y, dementia, migraine, heart disease, peptic ulcers, and arthritis are up to eight times more common
72 m of disease that includes gastritis, peptic ulcers, and gastric adenocarcinoma.
73 lacebo were headache, peripheral edema, skin ulcer, anemia, upper respiratory tract infection, diarrh
74                   Hospital-acquired pressure ulcers are a serious patient safety concern, associated
75         Chronic wounds such as diabetic (db) ulcers are associated with dysregulation of macrophage f
76                                              Ulcers are deemed neuroischaemic if peripheral neuropath
77 st frequent pathogens that cause the corneal ulcers are P. aeruginosa and S. aureus.
78                                Diabetic foot ulcers are serious and challenging wounds associated wit
79                                     Pressure ulcers are serious, avoidable, costly and common adverse
80                         We defined the index ulcer as that present at the first (index) DFU admission
81 tation, previous gastroesophageal surgery or ulcers as an etiology, and aggressive procedural and sur
82  is the first reported case of recto-sigmoid ulcers associated with use of this phosphate binder.
83 nd stratified according to number of digital ulcers at baseline (</=3 or >3).
84 s with systemic sclerosis and active digital ulcers at baseline.
85 6 (95%CI: dominant to $144,525) per pressure ulcer avoided.
86 y were: >/=18 years old; at risk of pressure ulcer because of limited mobility; expected to stay in h
87 , obesity, type 2 diabetes, gastrointestinal ulcers/bleeds, fractures, and cataracts (odds, 1.21-1.44
88                                       Buruli ulcer (BU) is a necrotizing skin disease most prevalent
89 istory that would suggest a secondary Mooren ulcer, but a chronic hepatitis C infection was detected.
90 with elevated TRV, microalbuminuria, and leg ulcers, but these vascular complications are not indepen
91 positioned it as an unusual cause of corneal ulcer, can be easily recognized in the laboratory, which
92 or eye health (trachoma and onchocerciasis), ulcer care (leprosy), or renal support (schistosomiasis)
93 on per additional hospital acquired pressure ulcer case avoided, estimated using a two-stage cluster-
94 rmine the microbiological profile of corneal ulcer cases diagnosed among patients visiting Tilganga I
95 Gram negative bacteria isolated from corneal ulcer cases.
96 ial role in protection against the cutaneous ulcers caused by Leishmania, and the IL-2 pathway is a p
97 ggest that risk stratification from baseline ulcer characteristics can identify those at highest risk
98            To determine baseline patient and ulcer characteristics that predict a high risk of develo
99 ude radiation gastritis and gastrointestinal ulcers, cholecystitis, radiation pneumonitis, and radioe
100           In patients with infected pressure ulcers, clinical recurrence occurs in almost two-thirds
101             One year later she was well, the ulcers completely healed, and with the autoimmune hepati
102 (TP) are major causative agents of cutaneous ulcer (CU) in the tropics.
103 No satisfactory treatment for neuroischaemic ulcers currently exists, and no evidence supports one pa
104 be asymptomatic or cause mild or severe skin ulcers (cutaneous leishmaniasis [CL]), limited or dissem
105 dmission), the hazard ratio for new pressure ulcers developed (pressure ulcer prevention care bundle
106 ion is an inflammatory disease in which skin ulcer development is associated with mononuclear cell in
107 e of pressure injury (also known as pressure ulcer) development among critical-care patients.
108                                Diabetic foot ulcers (DFUs) are a debilitating complication of diabete
109                                Diabetic foot ulcers (DFUs) are a severe complication of diabetes mell
110                                Diabetic foot ulcers (DFUs) threaten limbs and prompt hospitalization.
111                                Diabetic foot ulcers (DFUs), a life-threatening complication of diabet
112 t of NETosis on the healing of diabetic foot ulcers (DFUs).
113 esophageal varices (57%), followed by peptic ulcer disease (18%) and gastritis (10%).
114                         We evaluated genital ulcer disease (GUD) and HSV-2-associated GUD at quarterl
115 ervicovaginal HSV-2 DNA shedding and genital ulcer disease (GUD) in a cohort of women living with hum
116 pic subgroup has been associated with peptic ulcer disease and an increased bleeding tendency.
117 ) into host cells are associated with peptic ulcer disease and gastric adenocarcinoma.
118                                       Peptic ulcer disease and gastric cancer are caused most often b
119 thelium and is the major causative agent for ulcer disease and gastric cancer development.
120 elicobacter pylori, the main cause of peptic ulcer disease and gastric cancer in adult populations, i
121 is known to prevent the occurrence of peptic ulcer disease and gastric cancer.
122 an stomach and increases the risk for peptic ulcer disease and gastric carcinoma.
123  additional benefits beyond reducing genital ulcer disease and HSV-associated HIV transmission, throu
124 n strains isolated from patients with peptic ulcer disease or gastric cancer, rather than asymptomati
125 diseases including chronic gastritis, peptic ulcer disease, and gastric cancer.
126 ons for PPI use, including history of peptic ulcer disease, gastroesophageal reflux disease, or gastr
127 lecystectomy, operative management of peptic ulcer disease, lysis of peritoneal adhesions, appendecto
128 nfection is the most common cause of genital ulcer disease.
129 tiepileptics, hypertensives, and gastric and ulcer drugs also showed an increase in use (from 2-fold
130                                              Ulcer duration was determined by recording the time take
131                                              Ulcer duration, ulcer size after day 2 and pain levels w
132 s were pain due to the ulcer, ulcer size and ulcer duration.
133 ple treatment or being diagnosed with peptic ulcer during the following 33 months more than twice com
134      Of the 323 patients with smear-positive ulcers enrolled in MUTT-I, 299 (92.6%) were scraped and
135 e provides a clinical overview of venous leg ulcers, focusing on prevention, diagnosis, treatment, an
136 rse events included 3 interface-related skin ulcers for each group (ie, 7.6% in the face mask group h
137 complicated with ischemic colitis, stercoral ulcer formation and subsequent perforation.
138 f the PUABM recapitulated visual patterns of ulcer formation in individuals with SCI.
139               We determined that penetrating ulcer formation in this model resulted from increased hy
140           Critical limb ischemia (CLI), foot ulcers, former amputation, and impaired regeneration are
141           The mean decline in pain from oral ulcers from baseline to week 12 was greater with apremil
142 ial was the cumulative number of new digital ulcers from baseline to week 16.
143  II could not find a benefit for all corneal ulcers, Fusarium keratitis may benefit from the addition
144 types but includes arthralgia, purpura, skin ulcers, glomerulonephritis, and peripheral neuropathy.
145  a non-infected neuroischaemic diabetic foot ulcer greater than 1 cm(2) and of grade IC or IIC (as de
146                Consenting patients with skin ulcers &gt;1 cm in diameter were eligible for this study an
147                     In patients with gastric ulcer (GU) and IM, the GU may have a different healing r
148 U cultures yielding MRSA and protracted open ulcers had a high 24-month cumulative probability of DFU
149                                       Mooren ulcer has been considered as an idiopathic autoimmune ke
150    The association between stress and peptic ulcers has been questioned since the discovery of helico
151  regression modeling showed that complicated ulcer healing (hazard ratio, 3.812; 95% confidence inter
152 essively incorporate treatment to accelerate ulcer healing and address MRSA into the care of diabetic
153 extran sodium sulfate colitis due to delayed ulcer healing and reduced epithelial beta-catenin activa
154  enhancing Wnt/beta-catenin signaling during ulcer healing in IBD.
155 ran sodium sulfate colitis exhibited delayed ulcer healing, more mucosal inflammation, and impaired W
156  60 seconds reduced pain levels and enhanced ulcers' healing by reducing ulcers' size and duration.
157 ong 20,294 GBP patients, diabetes and peptic ulcer history entailed statistically significantly incre
158                          Diabetes and peptic ulcer history seem to be risk factors for MU, but not hy
159 hronic obstructive pulmonary disease (COPD), ulcer history, use of proton pump inhibitors (PPIs), asp
160 e endpoint being diagnosis of a new pressure ulcer, hospital discharge/transfer or 28days; whichever
161 -1) most commonly causes recrudescent labial ulcers; however, it is also the leading cause of infecti
162 ple treatment or being diagnosed with peptic ulcer (HR 2.24; CI 95% 1.16:4.35) after adjustment for a
163                                     Pressure ulcers impose a substantial financial burden.
164  man who was diagnosed with a primary Mooren ulcer in his right eye.
165 d in the microbiological etiology of corneal ulcer in Nepal, which have important public health impli
166 litis in two patients, and neuritis and skin ulcer in one patient.
167 pic finding of a longitudinal mid-esophageal ulcer in the presence of proximal stricture may be indic
168 ed disease chancroid in adults and cutaneous ulcers in children.
169 ureus isolates from uninfected diabetic foot ulcers in French patients harbor a prophage, ROSA-like,
170 nthamoeba cysts in moderate to large corneal ulcers in India.
171               Management of neuropathic foot ulcers in patients with diabetes (DFUs) has changed litt
172                                      Digital ulcers in patients with systemic sclerosis are associate
173 e for many aspects of the management of foot ulcers in people with diabetes is weak, and good-quality
174 with elevated TRV, microalbuminuria, and leg ulcers in SS-Sbeta(0) adults, but these associations wer
175 minuria in the whole population and with leg ulcers in SS-Sbeta(0) adults.
176 tivity Index (CDAI) of 220-450, with mucosal ulcers in the ileum or colon, or both, and a Crohn's Dis
177 e of macitentan for the treatment of digital ulcers in this patient population.
178 larly useful for detecting organisms in deep ulcers in which culture and light microscopy results wer
179 the microbial community structure of genital ulcers in women.
180  being diagnosed in a hospital with a peptic ulcer, in relation to quintiles of stress levels.
181                        A total of 121 peptic ulcer incidents were recorded within 33 months of follow
182 e prevention and management of diabetic foot ulcers, including studies that focus on off-loading, sti
183 stage 2 or worse immobility-related pressure ulcers (&lt;1% vs 2%; P = .001).
184                                   Venous leg ulcer management in the UK varies significantly.
185 ostablation endoscopy documenting esophageal ulcer may identify patients at higher risk for AEF.
186  Secondary outcomes included pain from these ulcers (measured on a 100-mm visual-analogue scale, with
187                                Diabetic foot ulcer microbiota were found to exist in one of four comm
188  assess risk factors for developing marginal ulcer (MU) after gastric bypass (GBP) surgery for obesit
189 ck of suspicious skin lesions or presence of ulcers negative for Treponema pallidum subsp pertenue on
190           As a rule, without treatment, skin ulcers occur and a lengthy healing process may be observ
191 ositivity were having skin wounds, sores, or ulcers (odds ratio [OR], 4.6; 95% confidence interval [C
192 ple treatment or being diagnosed with peptic ulcer of approximately 0.4%, whereas the highest stress
193                                       Fungal ulcers often have worse clinical outcomes than bacterial
194 C with respect to the pathogenesis of Mooren ulcer on this patient remains open, but should be consid
195  of hospital admissions for gastrointestinal ulcer or bleeding was significantly reduced from the pre
196                            Presentation with ulcer or gangrene, age >/=65 years, female sex, large ho
197 adults hospitalized for an infected pressure ulcer or implant-free osteomyelitis and reviewed the lit
198 e stage, antiretroviral regimen, and genital ulcers or cervical tenderness.
199  Acute upper gastrointestinal bleedings from ulcers or esophago-gastric varices are life threatening
200 n at rest or tissue loss, such as nonhealing ulcers or gangrene, related to peripheral artery disease
201 d more commonly in strains that cause peptic ulcers or gastric cancer, rather than asymptomatic infec
202                              None had stomal ulcers or macroscopic inflammation.
203 evere upper gastrointestinal hemorrhage from ulcers or other lesions, Doppler probe guided endoscopic
204 t with macitentan did not reduce new digital ulcers over 16 weeks.
205 ndicating worse pain), the number of genital ulcers, overall disease activity, and quality of life.
206  more based on clinical symptoms, bilirubin, ulcer, pancreatitis, ascites, or radioembolization-induc
207 clude: endophtalmitis, keratitis and corneal ulcers, Parinaud's oculoglandular syndrome, and conjunct
208 tamycin over topical voriconazole for fungal ulcers, particularly among those caused by Fusarium.
209 acterial communities may contribute to HSV-2 ulcer pathogenesis, severity, or prolonged healing.
210        Subgroup analysis of diagnosed peptic ulcer patients revealed the same pattern as the main ana
211      The adjusted mean number of new digital ulcers per patient over 16 weeks was 0.94 in the 3-mg ma
212  further investigated, 11 had attended a leg ulcer/podiatry clinic.
213 tenue on PCR, and active yaws was defined as ulcers positive for T pertenue on PCR.
214 tion or gangrene (PEH) and perforated peptic ulcer (PPU) was analyzed, independent of HV esophageal c
215  and pre-specified covariates (age, pressure ulcer present at baseline, body mass index, reason for a
216  receiving either a patient-centred pressure ulcer prevention care bundle (n=799) or standard care (n
217 -effectiveness of a patient-centred pressure ulcer prevention care bundle compared to standard care.
218                                   A pressure ulcer prevention care bundle consisting of multicomponen
219 andomised within strata to either a pressure ulcer prevention care bundle or standard care.
220  for new pressure ulcers developed (pressure ulcer prevention care bundle relative to standard care)
221                        Although the pressure ulcer prevention care bundle was associated with a large
222 ignificant finding include that the pressure ulcer prevention care bundle was effective but the sampl
223 n partnering with patients in their pressure ulcer prevention care.
224 ages for patients' participation in pressure ulcer prevention care: keep moving; look after your skin
225  reported on direct medical cost of pressure ulcer prevention or treatment, and provided national cos
226 veness and cost-benefit analyses of pressure ulcer prevention performed from the health system perspe
227        Although the cost to provide pressure ulcer prevention to patients at risk can importantly imp
228              The mean (SD) cost for pressure ulcer prevention was euro7.88 (8.21) per hospitalised pa
229 pathy, including pulmonary hypertension, leg ulcers, priapism, chronic kidney disease, and large-arte
230 nt jet velocity [TRV], microalbuminuria, leg ulcers, priapism, stroke, and osteonecrosis) by clinical
231 nt of the neglected tropical disease, Buruli ulcer, produces a cytotoxic macrolide, mycolactone, whos
232        One out of 10 postablation esophageal ulcers progressed to perforation, and no patient without
233 view current controversies related to stress ulcer prophylaxis for critically ill adult patients, inc
234      Selected publications describing stress ulcer prophylaxis in adult patients were retrieved (orig
235   Despite widespread incorporation of stress ulcer prophylaxis into practice around the world, questi
236                                       Stress ulcer prophylaxis is commonly administered to critically
237                                  Many stress ulcer prophylaxis recommendations are based on older stu
238 l cost-effectiveness of the available stress ulcer prophylaxis regimens.
239                                       Stress ulcer prophylaxis should be limited to patients consider
240 s, but also the cost-effectiveness of stress ulcer prophylaxis today.
241  drugs to critically ill patients for stress ulcer prophylaxis warrants further evaluation.
242 r antagonists, the adverse effects of stress ulcer prophylaxis, and overall cost-effectiveness.
243  patients most likely to benefit from stress ulcer prophylaxis, review the comparative efficacy of pr
244 to evaluate the safety of withholding stress ulcer prophylaxis.
245 pithelial sloughing and a large longitudinal ulcer proved fatal.
246                                     Pressure ulcers (PUs) are serious skin injuries whereby the wound
247                                     Fusarium ulcers randomized to oral voriconazole had a 0.43-fold d
248 site Index=100-PUR-FR, where PUR is pressure ulcer rate and FR is total fall rate.
249  stratified in two groups by recent pressure ulcer rates and randomised within strata to either a pre
250                             Whereas pressure ulcer rates were higher in facilities led by DONs with m
251 Practice Advice 5: Patients at high risk for ulcer-related bleeding from NSAIDs should take a PPI if
252                                Diabetic skin ulcers represent a challenging clinical problem with mec
253 ikacin and polymyxin B-trimethoprim, and the ulcer resolved over 3 weeks.
254                                   Esophageal ulcer seems to precede AEF development, and postablation
255 n, and no patient without esophageal thermal ulcers showed the occurrence of perforating esophageal c
256 s with a high salt content developed gastric ulcers significantly more frequently than gerbils consum
257  of the esophagus showed a 9 cm longitudinal ulcer situated 12 cm from the esophago-gastric junction.
258                              Ulcer duration, ulcer size after day 2 and pain levels were more reduced
259                                              Ulcer size and pain were recorded for each participant a
260 outcome measures were pain due to the ulcer, ulcer size and ulcer duration.
261                                              Ulcer size was reduced starting from the second day in t
262 els and enhanced ulcers' healing by reducing ulcers' size and duration.
263                                     Among 49 ulcer specimens from 49 HSV-2 seropositive women, by PCR
264 ract wound information (wound type, pressure ulcer stage, wound size, anatomic location, and wound tr
265 r AEs-subclinical pancreatitis and a mucosal ulcer that had healed by the time of 3-month endoscopy.
266 vement overall, they did seem beneficial for ulcers that were central, deep or large, non-Nocardia, o
267 f we included the participants with improved ulcers, the healing rates increased to 94.7%, 97.3%, and
268 adjunctive therapy in patients with pressure ulcers to accelerate wound healing.
269 s determined by recording the time taken for ulcers to disappear.
270  or foam dressings in patients with pressure ulcers to reduce wound size.
271            The randomized controlled Mycotic Ulcer Treatment Trial (MUTT) I showed a benefit of topic
272 le, 1%, and after the results of the Mycotic Ulcer Treatment Trial (MUTT) II became available, topica
273 icrobiological cure at 6 days in the Mycotic Ulcer Treatment Trial I (MUTT-I).
274 sign, Setting, and Participants: The Mycotic Ulcer Treatment Trial II (MUTT II), a multicenter, doubl
275      This is a secondary analysis of Mycotic Ulcer Treatment Trial II, a multicenter, double-masked,
276 stemic sclerosis and active ischemic digital ulcers, treatment with macitentan did not reduce new dig
277 s from the double-blind Steroids for Corneal Ulcers Trial (SCUT), which included patients at Aravind
278  randomized, controlled Steroids for Corneal Ulcers Trial found that although steroids provided no si
279 e main outcome measures were pain due to the ulcer, ulcer size and ulcer duration.
280               Patients with culture-negative ulcers, viral etiology, coexistent ocular surface diseas
281           Following examination, the corneal ulcer was scanned by IVCM (HRT3/RCM, Heidelberg Engineer
282 L-2, the adjusted mean number of new digital ulcers was 1.44 in the 3-mg macitentan group (n = 88) an
283 t of gastric cancer and duodenal and gastric ulcers, was early associated with gastric disease, but i
284 udgets, the costs to treat a severe pressure ulcer were found to be substantially higher.
285           In a logistic regression analysis, ulcers were identified to be a significant predictor for
286 Long before the cause was discovered, peptic ulcers were known to occur preferentially in individuals
287 ients in India and Nepal with smear-positive ulcers were screened; of the 787 who were eligible, 240
288               Swabs from clinically detected ulcers were tested for HSV-2 and Treponema pallidum by p
289 atures of sevelamer-associated recto-sigmoid ulcers which led to her symptoms.
290 recurrent keratitis presented with a corneal ulcer, which was culture positive for ESBL E coli.
291 ome, incidence of hospital-acquired pressure ulcers, which applied to both the cluster and individual
292 ted treatment, resulting in the formation of ulcers, which are susceptible to increased colonization
293    Apremilast was effective in treating oral ulcers, which are the cardinal manifestation of Behcet's
294 ociated infections, as well as infected foot ulcers, which often lead to amputation.
295 th smear-positive filamentous fungal corneal ulcers who enrolled between May 2010 and August 2015.
296 d quality of life among patients with fungal ulcers who were randomly assigned to natamycin compared
297  have worse clinical outcomes than bacterial ulcers, with no new treatments since the 1960s when topi
298  luminal aggressors, results in erosions and ulcers, with potential complications of bleeding, protei
299 ound closure of neuroischaemic diabetic foot ulcers without affecting safety after 20 weeks of treatm
300  with diabetes mellitus suffer from diabetic ulcers worldwide.

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