ライフサイエンスコーパス: ulcer healing

1 antibiotic therapy with improved venous leg ulcer healing.

2 They received enteric-coated ASA after ulcer healing.

3 n, and proliferation-essential components of ulcer healing.

4 hance mucosal barrier function or accelerate ulcer healing.

5 tivation of SRF is an important component of ulcer healing.

6 oxygenase-2 (COX-2) selective, delay gastric ulcer healing.

7 nced angiogenesis and accelerated esophageal ulcer healing.

8 increased neovascularization and accelerated ulcer healing.

9 xpression, significantly accelerates gastric ulcer healing.

10 l role for VEGF and enhanced angiogenesis in ulcer healing.

11 broblast growth factor and a reduced rate of ulcer healing.

12 c, Grb2, and Sos during experimental gastric ulcer healing.

13 n to be essential for cell proliferation and ulcer healing.

14 oblasts to evaluate their role in venous leg ulcers healing.

15 Acyclovir improved ulcer healing--61% of those receiving acyclovir healed b

16 essively incorporate treatment to accelerate ulcer healing and address MRSA into the care of diabetic

17 encoding recombinant human VEGF(165) affects ulcer healing and angiogenesis.

18 They resumed ASA after ulcer healing and H pylori eradication.

19 seen over the course of a month to evaluate ulcer healing and HIV-1 RNA shedding.

20 Overall, strength of evidence was low for ulcer healing and low or insufficient for time to comple

21 on of Helicobacter pylori expedites duodenal ulcer healing and prevents recurrences.

22 gnificantly accelerated experimental gastric ulcer healing and promoted re-epithelialization and musc

23 hostin A46 treatment significantly inhibited ulcer healing and reduced EGF-R levels, EGF-R phosphoryl

24 extran sodium sulfate colitis due to delayed ulcer healing and reduced epithelial beta-catenin activa

25 ked increase in colonic H2S synthesis drives ulcer healing and resolution of inflammation.

26 h celecoxib significantly delayed esophageal ulcer healing and suppressed ulceration-triggered increa

27 tudy, we examined the role of SRF in gastric ulcer healing and the mechanisms involved.

28 role of VEGF and angiogenesis in esophageal ulcer healing, and 3) demonstrate the feasibility of gen

29 ated receptor tyrosine kinase to ERKs during ulcer healing are as yet unknown.

30 elective inhibitor, celecoxib, on esophageal ulcer healing as well as on the cellular and molecular e

31 ficantly changed at week 12 in segments with ulcer healing, based on endoscopic examination (CDEIS: 2

32 blood vessels, is crucial for gastroduodenal ulcer healing because it enables delivery of oxygen and

33 asis of solid tumors, but also for wound and ulcer healing, because without the restoration of blood

34 o this strategy has not been shown to affect ulcer healing, but does reduce ulcer recurrence.

35 rvicovaginal HSV-2 DNA and slightly improved ulcer healing, but it did not decrease genital and plasm

36 We examined the role of SRF in ulcer healing by local injection of an SRF expression pl

37 in gastric ulcer margin accelerates gastric ulcer healing by promoting cell re-epithelization, proli

38 gs indicate that celecoxib delays esophageal ulcer healing by reducing ulceration-induced esophageal

39 eutic strategy that expedites chronic venous ulcer healing by treating the underlying ulcer pathophys

40 60 seconds reduced pain levels and enhanced ulcers' healing by reducing ulcers' size and duration.

41 ial agents associated with better venous leg ulcer healing compared with usual care (dressings and ba

42 Tayside, Scotland, receiving both NSAIDs and ulcer-healing drugs appeared to experience a large rise

43 ) to determine the efficacy of one exposure (ulcer-healing drugs) in preventing the serious upper gas

44 regression modeling showed that complicated ulcer healing (hazard ratio, 3.812; 95% confidence inter

45 tain whether episodic acyclovir will enhance ulcer healing if delivered at primary health care settin

46 resolution in 14 of 32 patients and complete ulcer healing in 5 of 18 patients.

47 creased crypt cell proliferation and delayed ulcer healing in GM-CSF(-/-) mice were rescued by exogen

48 , caused marked colon ulceration and delayed ulcer healing in GM-CSF(-/-) mice.

49 enhancing Wnt/beta-catenin signaling during ulcer healing in IBD.

50 cilitating epithelial cell proliferation and ulcer healing in response to injury of the colon crypt e

51 findings indicate that experimental gastric ulcer healing involves activation of EGF-R-ERK signal tr

52 ndicate that Raf-1 activation during gastric ulcer healing is Ras mediated, involves Shc-Grb2-Sos, an

53 nsulin-like growth factor (IGF)-1 in gastric ulcer healing is unknown.

54 These surrogate markers for venous leg ulcer healing may allow for early clinical trials to be

55 ran sodium sulfate colitis exhibited delayed ulcer healing, more mucosal inflammation, and impaired W

56 with bleeding and/or perforations) and delay ulcer healing, presumably by blocking prostaglandin synt

57 regulating VEGF expression during esophageal ulcer healing remain unknown.

58 Whether they affect esophageal ulcer healing remains unexplored.

59 dy significantly reduced the acceleration of ulcer healing resulting from the treatment.

60 g HCO3- secretion, the role of mechanisms in ulcer healing, the interaction of trefoil factors with t

61 or Ang1 into the ulcer base could accelerate ulcer healing through enhanced angiogenesis.

62 as candidate surrogate markers of venous leg ulcer healing using the Curative Health Services populat

63 Median time to ulcer healing was 99 days (95% CI 84-126) in the hosiery

64 Complete ulcer healing was achieved in 16 (53%) patients, whereas

65 in the bandage group, and the proportion of ulcers healing was much the same in the two groups (70.9

66 efficacy of VEGF gene therapy in esophageal ulcer healing, we studied whether a single local injecti

67 MRE determined ulcer healing with 90% accuracy and endoscopic remission

68 MRE evaluates ulcer healing with a high level of accuracy when ileocol

69 The primary endpoint was time to ulcer healing, with a maximum follow-up of 12 months.