ライフサイエンスコーパス: uncoupling agent

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1 ation by administration of the mitochondrial uncoupling agent 2,4-dinitrophenol (DNP) in a large anim

2 ere readily resolved without pharmacological uncoupling agents, allowing identification of 4 major un

3 ps that modulate bacterial susceptibility to uncoupling agents and certain antibiotics.

4 tion were detected with blunted responses to uncoupling agents and reduced membrane potentials.

5 dependent on respiration and is prevented by uncoupling agents, and the Vmax for K+ is 1.2-1.5 microm

6 development of liver-targeted mitochondrial uncoupling agents as a potential novel therapy for lipod

7 Treatment with 2 microM AGA, an uncoupling agent, blocked the spread of Lucifer yellow i

8 cal blockade of adrenal gap junctions by the uncoupling agent carbenoxolone reduces nerve stimulation

9 tes, as well as in those treated with TNF or uncoupling agent carbonyl cyanide m-chlorophenyl hydrazo

10 Continuous superfusion with the uncoupling agent carbonyl cyanide-p-trifluoromethoxy-phe

11 ontrast, uptake of Cu-Mb is inhibited by the uncoupling agents carbonyl cyanide m-chlorophenylhydrazo

12 by hyperosmotic stress and the mitochondrial uncoupling agent, dinitrophenol, both of which lead to i

13 Oscillations were also eliminated by the uncoupling agents heptanol, halothane or acidification.

14 cells with suramin (100 microM), a receptor-uncoupling agent, inhibited LPA-stimulated proliferation

15 mitochondrial membrane by treatment with an uncoupling agent inhibits import of the tandem tRNA subs

16 channels indicating that the actions of such uncoupling agents, like voltage gating, are intrinsic he

17 importantly, treatment with a mitochondrial uncoupling agent or adenosine triphosphate synthesis inh

18 gonists and antagonists or by treatment with uncoupling agents such as pertussis toxin or N-ethyl mal

19 mapping in the absence of electromechanical uncoupling agents, the method relieves a long-standing l

20 Rotenone, NMDA and uncoupling agents were added to the brain slice superfus

21 he hypothesis that oxidative phosphorylation uncoupling agents would antagonize the effect of rotenon

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