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1 ve ACET proteins which are unglycosylated or underglycosylated.
2 solated from beta4GalT5 morphant embryos are underglycosylated and are unable to bind recombinant Bmp
3 ly transmitted subtype B viruses, and it was underglycosylated but no different in length compared to
4 ulator (CFTR) expressed in Sf9 insect cells, underglycosylated CFTR expressed in yeast is not effecti
5 in solubilizing the notoriously intractable underglycosylated CFTR suggest that this detergent may b
6 affects the immune response to CII, so that underglycosylated CII is less effective in the induction
7 Palpha1, but in the astrocytoma cells, it is underglycosylated compared with SIRPalpha1 produced in t
8 but underhydroxylated at lysyl residues and underglycosylated compared with tissue-derived CII, wher
9 pressed on tumor cells is characteristically underglycosylated, creating peptide and glycopeptide neo
10 n MUC1 is expressed on adenocarcinomas in an underglycosylated form that serves as a tumor antigen in
11 pithelial origin MUC1 is overexpressed in an underglycosylated form with truncated O-glycans and accu
12 , or 22L, results in the accumulation of two underglycosylated, full-length PrP species and an N-term
18 cific contrast agent (MN-EPPT) targeting the underglycosylated MUC-1 (uMUC-1) tumor antigen, found on
20 gly, the initial rate of endocytosis of this underglycosylated MUC1 was stimulated by twofold compare
24 ks of tumorigenesis and is overexpressed and underglycosylated on almost all human epithelial cell ad
25 ins in these transgenic plants accumulate as underglycosylated protein species with apparent molecula
27 UPR regulator Ire1p, which detects misfolded/underglycosylated proteins in the ER, controlled Msb2p c
28 ansmitted viruses were, on average, modestly underglycosylated relative to the viruses from chronical
29 c breast tissue, the extracellular domain is underglycosylated, resulting in the exposure of a highly
30 ncing of TbNST4 in the procyclic form caused underglycosylated surface glycoprotein EP-procyclin.
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