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1 iated with HLA class I-DSA could improve our understanding of ABMR and be useful for diagnostic or th
6 ochar use highlight the need for an improved understanding of biochar's impacts on soil NO emissions.
14 rofiling with great potential to improve our understanding of cellular heterogeneity through discover
15 fill gaps in trait coverage will improve our understanding of changes in fine-root traits across spac
16 tions related to the application of SERS for understanding of chemical processes at the nanoscale, wi
17 e alone remains poor, reflecting our limited understanding of cis-regulatory grammar and hampering th
24 human pancreatic beta-cells, broadening our understanding of cytokine-induced beta-cell apoptosis in
27 ch is absolutely essential for improving our understanding of disease mechanisms contributing to risk
28 ing pathways, with implications for improved understanding of disease recurrence and therapeutic resp
31 emarkably common in early human embryos, our understanding of early embryonic somatic mutations is ve
33 bon budget in China's forests and for better understanding of effects of climate and stand characteri
34 bstrates, as foundational to the mechanistic understanding of emerging PBP2a resistance mutations.
35 pect this insight will decisively change our understanding of essential phenomena occurring within th
36 des a structural framework for a mechanistic understanding of eukaryotic ribosome assembly in the mod
38 e exploited as a model system towards better understanding of F. pseudograminearum-wheat interaction.
39 requently begin in childhood/adolescence, an understanding of fear-extinction learning in children is
42 PCA for clinical genetic testing, expanding understanding of genetic contribution to aggressive PCA,
45 e efforts will contribute to a more complete understanding of global carbon and nitrogen cycling and
46 have important implications for our current understanding of healthy and adaptive brain processing.
47 These findings may have implications for our understanding of how altered folate availability causes
49 ng cellular ATP production, and advances our understanding of how defective FA signaling contributes
50 eighboring mesenchymal partners provides new understanding of how different cell types are maintained
51 ng cardiac specification is critical for our understanding of how heart formation is initiated during
52 tuberculosis enzyme, augmenting our current understanding of how M. tuberculosis meets its nutrient
53 ctin-mitochondria interaction, enhancing our understanding of how mitochondria properly localize in a
54 either FTD or NCL, in part because of a poor understanding of how mutations in genes such as GRN cont
56 this single-cell approach provides a better understanding of how prostate cancer cells respond heter
59 behavior and pathology, but we lack a basic understanding of how sex differences in the human cerebe
60 Together, these models provide a biophysical understanding of how stem cell scaling is maintained dur
64 individual isoforms, necessitating a deeper understanding of how the distinct transcriptional progra
65 Here, we highlight recent advances in our understanding of how the genome folds inside the 3D nucl
67 across different temperatures would advance understanding of how thermally activated processes contr
68 r interpretations of forest dynamics and our understanding of how these forests are responding to glo
70 protein acetylation dynamics is critical for understanding of how this modification influences protei
71 esistance that would be helpful for a better understanding of how to manage antibiotic-resistant bact
75 le and has begun making contributions to the understanding of human intestinal transport in normal ph
78 roptosis in macrophages, and revises current understanding of independent and collaborative functions
82 of centriole components have accelerated our understanding of its assembly, function, evolution, and
83 e drug reaction will further our mechanistic understanding of its development and potentially lead to
86 lasticity or TGP) may occur, we have limited understanding of key aspects of TGP, such as the environ
87 is critical to health, making furthering our understanding of L-R development an important concern.
89 It is demonstrated that SERS provides a deep understanding of living cells as well as their microenvi
90 ines recent work that has contributed to our understanding of LOS-deficiency and compares it to studi
91 ing Alzheimer's disease (AD) requires a deep understanding of mechanisms affecting complex brain syst
93 ors and coregulators, can profoundly improve understanding of mechanisms underlying genetic associati
97 se questions will provide a greater level of understanding of miRNA biology and critical insights int
98 a has been implicated in aging, but a deeper understanding of mitochondrial dynamics and mitophagy du
99 the Drosophila visual system, where a deeper understanding of molecular mechanisms allows for the gen
105 erstanding of complex networks; however, our understanding of natural microecologies is limited.
106 y their differential actions may advance our understanding of nervous system disorders and suggest st
108 wledge in these areas lag behind our current understanding of neuroprotection and vascular biology in
109 dimers as the key antigenic target, and deep understanding of neutralizing mechanisms, multiple vacci
114 mitations, there has been an increase in the understanding of pathophysiology and important risk fact
115 nt paradigm shifts that have transformed our understanding of pediatric ocular motor disease at the p
117 ctly in a single nanostructure, limiting our understanding of phonon coupling with photons and plasmo
118 ans.This study sought to advance the current understanding of placental vitamin D metabolism and its
119 h sufficiently long values of T2 requires an understanding of precisely how the position of nuclear s
120 e and rhizosphere of plants, a more detailed understanding of predator-prey interactions, changes in
122 s to simian immunodeficiency virus (SIV), an understanding of processes that promote successful cross
123 s, significant progress has been made in the understanding of proteasome assembly, structure, and fun
129 predominant one concerning questions on the understanding of science, the main goal, the stage of in
130 Over the past 50 years in pharmacology, an understanding of seven transmembrane (7TMR) function has
131 Nevertheless, significant gaps remain in our understanding of several crucial aspects of MP exposure
134 FEM) modeling has been undertaken to enhance understanding of SICM as an electrochemical cell and to
135 e about RTK interactions and can further our understanding of signal transduction across the plasma m
136 has significant global implications for our understanding of soil N cycling pathways and N2 O produc
139 ingly, the physiological and pharmacological understanding of specific motoneuronal contributions to
140 pulating the epigenome to facilitate further understanding of stem cell biology and engineering of st
141 e manner, with the aim of obtaining a deeper understanding of stem cell fate computation, in order to
144 A-4 pathway are therefore required to inform understanding of such immune dysregulation syndromes.
146 that can contribute to a more comprehensive understanding of symbiosis as a major driving force of e
148 formation has prevented a detailed molecular understanding of the assorted ANT-associated diseases, i
149 n, it is imperative that we develop a better understanding of the basic biology of this parasite and
150 model mice in vivoSIGNIFICANCE STATEMENT Our understanding of the basis for intellectual impairment i
151 ivo functions of ADAR enzymes, informing our understanding of the biological importance of A-to-I edi
152 ion of gene models will not only further our understanding of the biological processes of this plant
153 physiological measurements, has improved our understanding of the biological responses during drought
155 ing deconstruction not only provide a better understanding of the cell wall architecture but also is
156 thcare costs inherent in device use, a clear understanding of the clinical benefits relative to costs
157 ation and transmigration, which advances our understanding of the complex contribution of CtsB to ang
158 ents, are paving the way for a more complete understanding of the complex events that help build the
163 Work with this model system has enriched our understanding of the cyclopropanation-Cope rearrangement
168 results suggest large biases in our current understanding of the distribution, area and volumes of t
169 ial to provide epidemiologists with a deeper understanding of the divergent pathways via which neighb
171 ingle-molecule approaches have increased our understanding of the dynamic behavior of complex multipr
172 cial and ecological factors, and enhance our understanding of the dynamics of natural populations.
175 e effects in social species will improve our understanding of the ecological and evolutionary implica
176 eralisation and the development of a broader understanding of the ecology and evolution of mutualisms
177 genes duplicated in Trisomy 21 and a lack of understanding of the effect of disease pathology on the
179 ecent studies that advance the knowledge and understanding of the effects of various environmental fa
180 void this issue is hindered by an inadequate understanding of the electrically-induced wrinkling defo
182 1):1-9) was an important contribution to the understanding of the epidemiology of cardiovascular dise
188 these findings contribute to our fundamental understanding of the functional interplay between host E
189 ved issue, even though it is critical to the understanding of the functional properties of MSMAs.
191 during development, contributing to improved understanding of the genetic etiology of familial tooth
192 on sequencing has substantially enhanced our understanding of the genetics of primary brain tumors by
193 ications may be accomplished through a sound understanding of the hemodynamic and physiological conse
195 w the EBV life cycle and discuss our current understanding of the immune response to EBV in healthy,
196 AR1-AtZED1 protein complex provides a better understanding of the immune signaling hub controlled by
198 described by Glorius et al., builds a broad understanding of the impact of individual functional gro
199 s, patients and their families, for a better understanding of the impact this disease has on a person
201 scover new catalysts by obtaining a detailed understanding of the initial steps of CO2 electroreducti
202 cidating cerebellar function will require an understanding of the interactions, both short- and long-
203 logy provides a better and more quantitative understanding of the intracellular behavior of drug-load
204 ant use in biotechnology, we lack a detailed understanding of the kinetics of nucleic acid hybridizat
206 aking place throughout the tropics, improved understanding of the magnitude and spatial variation in
208 ular homeostasis and therapy, our structural understanding of the MBS CC interaction with LZ PKG-1alp
210 tural products contain chlorine and thus, an understanding of the mechanism of its introduction into
217 es have not significantly contributed to our understanding of the molecular basis of trainability.
221 linical challenge and may relate to the poor understanding of the molecular mechanisms involved.
224 However, until recently, deficiencies in our understanding of the nature of these cell populations, c
228 dedness, implying a fundamental shift in our understanding of the ontogenesis of hemispheric asymmetr
231 past decade has seen significant advances in understanding of the pathogenesis and progression of lun
232 light how such improvements enable a greater understanding of the pathogenesis of disease and the rea
237 he lower urinary tract and contribute to our understanding of the pathophysiology of urinary retentio
238 explore several new ideas that could improve understanding of the phenotypic and genotypic difference
239 This gap in knowledge stems from a lack of understanding of the physics of surface structures inter
240 omputation devices will be aided by a better understanding of the physics underlying material behavio
241 nst antithrombin polymerization, an improved understanding of the polymerogenic intermediates is cruc
242 xpression/function, could be improved by the understanding of the properties of the cancer secretome,
243 This article reviews recent advances in our understanding of the proteasome's multistep ATP-dependen
245 postmortem human brain cells and further our understanding of the regulome and the impact of neuropsy
247 ndividual variability, necessitates a deeper understanding of the relationship between stimulation pa
248 egions throughout plant genomes will advance understanding of the relationship between TF binding, ch
249 es in photosynthetic infrastructure, but our understanding of the relative effects of these factors a
250 our simple model contribute a more intimate understanding of the response and consequence of rotatin
251 brain structures which are essential to the understanding of the result statistics from the analyses
252 ng survivors of explosions, but with limited understanding of the resulting retinal pathologies.
254 s lack effective therapies despite increased understanding of the role factors such as an overactive
255 novel strategy could greatly facilitate our understanding of the role of BMP in biological systems.
258 ted with patient survival would increase the understanding of the role of glioma cancer stem cells (G
259 Our work has the potential to improve our understanding of the role of global methylation in human
261 ere is a fundamental gap in our quantitative understanding of the role of local ECM size and arrangem
262 n and repair, the mechanistic and functional understanding of the role of PARPs in different biologic
266 of theoretical mathematical models toward an understanding of the system's regulatory properties.
267 nits and antigenic epitopes to gain a better understanding of the technology at a molecular level.
268 e climates, which will require a mechanistic understanding of the trade-offs that determine trait div
269 dings have enabled a deeper and more precise understanding of the transcriptional mechanisms that ind
270 unity-based surveillance allows for a better understanding of the true burden and seasonality of dise
271 th, is an important yield component, but our understanding of the underlying genes and mechanisms is
273 cant advantages that allow for a fundamental understanding of the underlying physics of a system.
280 hiadiazoles are likely to result if a deeper understanding of their preferred patterns of molecular a
281 reported RGGT inhibitors, rationally improve understanding of their structure-activity relationship.
286 utionary and genetic approaches will improve understanding of these symbiotic associations and, in th
287 h over the previous decades has improved our understanding of this complex arrhythmia while unravelin
290 aminergic neurons and contribute to a better understanding of this functionally complex group of neur
291 hange is a promising new approach but better understanding of this mechanism of action is needed.
293 e epidemiology of SJS/TEN contributes to the understanding of this still underinvestigated severe ski
294 work opens the door to a detailed atomistic understanding of transformation reactions in even larger
295 toward our expanding cellular and molecular understanding of type-2-cell-mediated immunity, as well
296 ation of these interactions could expand our understanding of viruses and be exploited for epidemiolo
298 n be experimentally validated to improve our understanding of what triggers and maintains the growth
299 m(6)A) RNA modifications in mRNA requires an understanding of when and where in the life of a pre-mRN
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