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1 0, for stunting and 22.1%, 19.4 to 24.8, for underweight).
2 y as separate outcomes (compared with normal/underweight).
3 reater severity of hypertension and low BMI (underweight).
4 se districts more women were obese than were underweight.
5 in which Indian women are most likely to be underweight.
6 42% (34-50) for wasting and 54% (49-59) for underweight.
7 eight for age, weight for age, stunting, and underweight.
8 this risk comparable to never being obese or underweight.
9 ented >/= 3 mo before conception and was not underweight.
10 , 35% overweight, 36% normal weight, and 12% underweight.
11 19.6) in those who were stunted, wasted, and underweight.
12 re anemia were malaria, poor sanitation, and underweight.
13 derweight; and 9.40 (8.02, 11.03) for severe underweight.
14 er determinant of mortality than stunting or underweight.
15 , 52,006 had an optimum BMI, and 13,602 were underweight.
16 t, 7% were underweight, and 9% were severely underweight.
17 32% were overweight, and 38% were normal or underweight.
18 tatus included in this study were anemia and underweight.
19 n child-growth outcomes such as stunting and underweight.
20 eeks, 37% were overweight/obese, and 3% were underweight.
21 t failure if they had severe disease or were underweight.
22 z score or on rates of stunting, wasting, or underweight.
23 compare incidences of stunting, wasting, and underweight.
24 zard ratios were 1.06 (95% CI, 0.44-2.28) in underweight, 1.27 (95% CI, 0.87-1.85) in overweight, and
25 .34, 3.02) and 2.60 (95% CI: 2.25, 3.00) for underweight, 1.39 (95% CI: 1.23, 1.57) and 1.73 (95% CI:
26 confidence interval [95% CI], 0.97-6.50) in underweight, 1.68 (95% CI, 0.92-3.06) in overweight, and
29 I at the time of transplantation: BMI <18.5 (underweight), 18.5 to 24.99 (normal weight), 25 to 29.99
30 th Organization definitions: less than 18.5 (underweight), 18.5 to 29.9 (normal weight), 30 to 34.9 (
31 eight (kg)/height (m)(2)) of less than 18.5 (underweight), 18.5-24.9 (normal weight), 25.0-29.9 (over
33 95% Confidence Interval 1.28, 1.81) for mild underweight; 2.63 (2.20, 3.14) for moderate underweight;
34 Prepregnancy BMI was distributed as follows: underweight, 3.2%; normal weight, 47.5%; overweight, 25.
35 criteria for stunting (26% [6-54]) or being underweight (36% [3-61]) versus those who did not (25% [
37 information on BMI was available, 9.7% were underweight, 39.0% were of normal weight, 34.5% were ove
38 at the region had high rates of stunting and underweight (40% and 31% of children aged <5 years had a
39 31 patients met inclusion criteria; 12% were underweight, 50% normal weight, 11% overweight, and 27%
41 e sizes were 2,661,519 (mortality), 587,096 (underweight), 558,347 (stunting), and 568,609 (wasting)
42 categories were 2.4% at BMI less than 18.5 (underweight), 61.8% at BMI of 18.5 to 24.9 (normal weigh
43 In 1990, the leading risks were childhood underweight (7.9% [6.8-9.4]), household air pollution fr
44 00, 1.06; P = 0.04), 5% higher odds of being underweight (95% CI: 1.02, 1.09; P < 0.01), and 9% highe
46 ed the prevalences of stunting, wasting, and underweight among children and of underweight, overweigh
49 ols; this pattern was partially preserved in underweight AN patients but not in weight-restored ones.
50 e significantly higher in hedonic eating; in underweight AN patients, 2-arachidonoylglycerol concentr
53 stunting analysis, 397,080 children for the underweight analysis, 384,163 children for the wasting a
54 I included in this cohort, 5,678 (9.8%) were underweight and 51,896 (90.2%) were normal weight at bas
56 and palmitoylethanolamide were measured in 7 underweight and 7 weight-restored AN patients after eati
59 relationship, with the highest rates in the underweight and morbidly obese extremes and the lowest r
60 atients, and (2) is the relationship between underweight and mortality also observed in patients with
61 tween combinations of stunting, wasting, and underweight and mortality among children <5 y of age.
62 the LVRS arm than in the medical arm in the underweight and normal weight groups at all follow-up ti
63 to compare short- and long-term mortality in underweight and normal weight patients (n = 57,574).
65 predictive value (PPV) of 72.1% and 68.3% in underweight and normal-weight mothers, respectively.
66 idelines, inadequate GWG can be predicted in underweight and normal-weight mothers, whereas excessive
71 known about the socioeconomic patterning of underweight and overweight as economies move through the
72 e objective was to assess whether burdens of underweight and overweight coexist among lower socioecon
73 a has yet to experience a situation in which underweight and overweight coexist in the low-SES groups
79 ng, nonsmoking, test for trend p = 0.002 for underweight and p = 0.009 for normal weight) after adjus
81 blend (UNIMIX) porridge on the prevalence of underweight and stunting among infants in South Kivu Pro
83 In addition, 13.8% of women start pregnancy underweight and the rate of obesity increases during pre
89 ren was 9.1%, 29.1% were stunted, 18.6% were underweight, and 2.4% were overweight; among the women,
92 in seven of 15 countries assessed for being underweight, and in those who participated in few early-
93 imate-related increases in the prevalence of underweight, and most climate-related deaths were projec
94 f global acute malnutrition (GAM), stunting, underweight, and overweight in children; and stunting, u
95 ., breast milk substitutes, infection rates, underweight, and pubertal timing) differ between these s
101 ure to first-line ART, entry to adolescence, underweight, and/or undetectable drug levels were at hig
102 underweight; 2.63 (2.20, 3.14) for moderate underweight; and 9.40 (8.02, 11.03) for severe underweig
104 f LOS >3 days were higher in adults who were underweight (aOR, 1.6; 95% CI, 1.1-2.4), and odds of mec
105 ratio [aOR], 2.2), unvaccinated (aOR, 3.7), underweight (aOR, 6.3), and too young to be immunized (a
106 95% confidence interval [CI], 0.987-0.988), underweight (ARD, 0.0068; RR, 0.968; 95% CI, 0.968-0.969
108 adults, overweight and obesity (and possibly underweight) are associated with increased all-cause mor
109 reduction of the prevalence of stunting and underweight at 12 mo of age among rural Congolese infant
110 ldren (5.23, 2.61-10.5), fewer children were underweight at 18 months (0.81, 0.66-0.99), and fewer in
112 ng the F2 generation, IUGR lineage rats were underweight at birth (6.7 vs. 8.0 g, P < 0.0001) and obe
113 =0.0092), chronic active disease (p=0.0148), underweight at diagnosis (p=0.0271) and during follow-up
115 e offspring was strongest for women who were underweight before pregnancy (P for interaction < 0.01).
119 782 acute ischemic stroke patients, 282 were underweight (BMI < 18.5 kg/m(2)), 2306 were normal-weigh
122 s 0.86 (0.80-0.93) for participants who were underweight (BMI < 18.5), 1.43 (1.36-1.52) for those who
124 with STEMI by BMI category were as follows: underweight (BMI <18.5 kg/m(2)) 1.6%, normal weight (18.
127 nfections at multiple and unspecified sites (underweight (BMI <18.5): hazard ratio (HR) = 4.26, 95% c
128 a nonlinear fashion, with patients who were underweight (BMI <18.5; HR, 2.65; 95% CI, 1.63-4.31) and
130 ing to prepregnancy body mass index (BMI) in underweight (BMI<18.5 kg/m(2)), normal weight (BMI=18.5-
131 as found between maternal BMI categorized as underweight [BMI (kg/m(2)) <18.5], healthy BMI (BMI: 18.
132 tegorized based on standard criteria (normal/underweight, BMI<25 kg/m(2) [n=486]; overweight, 25</=BM
133 nesthesiologists risk classification 4 or 5, underweight body mass index, noncardiac surgery, history
134 DSM-IV eating disorder but were not markedly underweight (body mass index over 17.5), were enrolled i
135 .9 kg/m, hospital mortality was higher among underweight (body mass index, < 18.5; relative risk, 1.3
136 % CI: 3.1, 7.1) in those who were wasted and underweight but not stunted; and 12.3 (95% CI: 7.7, 19.6
137 d and underweight but not wasted; wasted and underweight but not stunted; and stunted, wasted, and un
138 .6, 4.3) among children who were stunted and underweight but not wasted; 4.7 (95% CI: 3.1, 7.1) in th
139 ; wasted only; underweight only; stunted and underweight but not wasted; wasted and underweight but n
141 ified relationship among age, fertility, and underweight; childbearing is concentrated in the narrow
142 ed the higher risk of death in obese but not underweight children (HR, 1.09; 95% CI, 0.96 to 1.24).
147 that 42.2% of prepregnant women in India are underweight compared with 16.5% of prepregnant women in
149 eased from 14% to 18%, whereas prevalence of underweight decreased from 12% to 9% during this period.
152 a simultaneous decrease in the prevalence of underweight (estimated decrease of 0.06% per year, 95% C
153 of the following characteristics: clinically underweight, exhaustion, low energy expenditure, slow wa
156 trition on lung function was observed in the underweight group and in pancreatic- insufficient patien
157 djusted HR of coronary heart disease for the underweight group was 1.25 (1.05-1.49) in women and 1.09
158 t increment in BMI, 1.10 (0.91-1.32) for the underweight group, 0.99 (0.92-1.07) for the overweight g
159 e regression analyses showed that within the underweight group, an increase in BMI resulted in improv
161 ver, in most of sub-Saharan Africa childhood underweight, HAP, and non-exclusive and discontinued bre
163 tios for hospital discharge were lower among underweight (hazard ratio, 0.71; p < 0.001) and obese (h
166 an age of onset dependent influence towards underweight, higher disease activity and a more intensiv
168 fidence interval [95% CI], 1.03 to 1.32) and underweight (HR, 1.26; 95% CI, 1.09 to 1.47) children th
169 no association was observed between maternal underweight (HR, 1.46; 95% CI, 0.90-2.35), overweight (H
171 terization of I-Rai1 mice showed significant underweight, hyperactivity and impaired learning and mem
177 s in Georgia, Chile, and China, and the most underweight in rural areas of Timor-Leste, India, Niger,
178 experimental approaches suggest that Tyr is underweighted in the prediction algorithm due to the abs
179 nted as obese, overweight, normal weight, or underweight, in contrast to patients who had none of the
180 tricts, more women were overweight than were underweight; in 49 of these districts more women were ob
181 l-weight patients and increased mortality in underweight individuals (odds ratio, 1.51; 95% confidenc
184 robiota from healthy or severely stunted and underweight infants; age- and growth-discriminatory taxa
185 eficiency, malaria, breastfeeding, stunting, underweight, inflammation, low socioeconomic status, and
186 ndency to overweight small probabilities and underweight large probabilities, and those for the motor
188 variables of stunting (low height-for-age), underweight (low weight-for-age), wasting (low weight-fo
189 Prevention body mass index z score criteria: underweight (< -1.89), normal weight (-1.89 to +1.04), o
190 line body mass index (BMI) was classified as underweight (<18.5 kg/m(2)), normal (18.5-24.9 kg/m(2)),
192 eight in meters squared) categories included underweight (<18.5), normal BMI (18.5-24.9), overweight
193 e grouped into predefined weight categories: underweight (<1st percentile), reference (1st-74th perce
195 according to baseline body mass index (BMI): underweight (<21 kg/m(2)), normal weight (21-25 kg/m(2))
196 ain a percentile ranking and then grouped as underweight (<5th percentile), normal weight (5th percen
200 5; 95% CI: 1.00, 1.10); in 2011, children of underweight mothers had a 21% higher risk of being stunt
204 types defined by body mass index categories (underweight, normal weight, overweight, and obesity) and
205 Survival was estimated by BMI category (underweight, normal weight, overweight, class 1 obesity,
206 that were stratified by BMI in 5 categories, underweight, normal weight, overweight, obese, and morbi
209 urements as a function of gestational age in underweight, normal-weight, overweight, and obese class
211 ith increased early preterm birth risk among underweight (odds ratio (OR) = 2.94, 95% confidence inte
212 11, 95% confidence interval: 1.04, 1.18) and underweight (odds ratio = 1.21, 95% confidence interval:
213 Obese (odds ratio, 1.28; P=0.008), severely underweight (odds ratio, 1.29; P<0.0001), and underweigh
214 nderweight (odds ratio, 1.29; P<0.0001), and underweight (odds ratio, 1.39; P=0.002) subjects were as
216 hospital-acquired infections were higher in underweight (odds ratio, 1.88; p = 0.008) and obese (odd
218 rience dependent overweighting of small, and underweighting of large, probabilities whereas ventral f
220 ons: no deficits; stunted only; wasted only; underweight only; stunted and underweight but not wasted
221 0.56 (95% CI: 0.43, 0.73) in adults who were underweight or normal weight, 0.67 (95% CI: 0.57, 0.79)
222 on the basis of measured height and weight (underweight or normal weight, overweight, and obesity).
223 ry interventions in low-income countries and underweight or nutritionally at-risk populations increas
225 ysical activity guidelines, and being either underweight or obese were associated with poor health st
227 ardiovascular disease, diabetes, depression, underweight, or activity limitations is associated with
228 t, and overweight in children; and stunting, underweight, overweight, and central obesity in women.
229 ces in 5-year cumulative incidence of CKD in underweight, overweight, and obese participants compared
232 sting, and underweight among children and of underweight, overweight, and obesity in women for all 14
233 (P = .05), while each 1-unit BMI gain among underweight participants was associated with a 9.32-mg/L
235 mortality, with risks quickly increasing for underweight patients (body mass index < 18.5 kg/m).
242 remained separate over 17 y, suggesting that underweight patients remained at a significant survival
243 obese and overweight patients and higher in underweight patients than in those with normal body mass
244 Crude mortality was significantly higher for underweight patients than normal weight patients at 30 d
245 tter volume and cortical thinning in acutely underweight patients to normalize following successful t
247 ht, 116 were obese, and 16 were underweight; underweight patients were excluded from the analyses bec
250 es explain the higher mortality after AMI in underweight patients, and (2) is the relationship betwee
253 Compared with people of a healthy weight, underweight people (BMI <20 kg/m(2)) had a 34% higher (9
254 l BMI, national percentage of overweight and underweight people) obtained from publicly available dat
255 being 0.33% (95% CI 0.24-0.42; p<0.0001) for underweight people, 0.50% (0.47-0.53; p<0.0001) for norm
256 children that a person will have: obese and underweight persons are hypothesized to have fewer child
257 ed increased inflammation, weight gain among underweight persons predicted reduced inflammation.
260 had a greater effect on preterm births among underweight pregnant women (BMI <18.5 kg/m(2); RR 0.84,
261 49.1-53.1] in Niger), 22.7% (22.5-22.9) were underweight (ranging from 1.8% [1.3-2.3] in Jordan to 41
265 dence interval [CI], 0.970-0.987; P < .001), underweight (RR, 0.971; 95% CI, 0.968-0.974; P < .001),
266 UNIMIX: 46.4%; P = 0.31), the prevalence of underweight (RUCF: 20.4%; UNIMIX: 18.2%; P = 0.42), or w
267 stis jirovecii infection, moderate or severe underweight, severe acute malnutrition, disease duration
270 ween groups defined by anemia, stunting, and underweight status to identify optimal recall periods fo
272 bined Zn+MV did not reduce the incidences of underweight, stunting, or wasting in Tanzanian infants.
276 a plethora of clinical symptoms ranging from underweight to nutrient-, vitamin- and electrolyte defic
279 d normal weight, 116 were obese, and 16 were underweight; underweight patients were excluded from the
280 djusted body mass index (BMI) percentiles as underweight (UW), at risk of UW (RUW), normal, overweigh
285 eater caudate prediction error response when underweight was associated with lower weight gain during
288 his large cohort of critically ill patients, underweight was independently associated with a higher h
289 multilevel Cox proportional hazard analysis, underweight was independently associated with a higher h
291 ) times, respectively, more likely to become underweight (weight-for-age z score <-2) after adjustmen
292 months; and a recuperative model, targeting underweight (weight-for-age Z score <-2) children aged 6
293 f age, prevalences of stunting, wasting, and underweight were 19.8%, 6.0%, and 10.8%, respectively.
294 (69-83); the lowest figures for wasting and underweight were both less than 2.5% and the highest wer
295 We also calculated a ratio of the number of underweight women (<18.5) divided by the number of overw
297 ormal BMI) were 28.8 (95% CI, 12.2-47.2) for underweight women, 17.6 (95% CI, 10.5-25.1) for overweig
300 Adjusted ORs were 1.2 (95% CI, 1.0-1.3) for underweight women; 1.1 (95% CI, 1.1-1.2) for overweight
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