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1 le elements that have expanded presumably by unequal crossing over.
2 receding meiotic recombination, resulting in unequal crossing over.
3 enotypes through infrequent recombination or unequal crossing over.
4 rgoing concerted evolution by the process of unequal crossing over.
5 tion slippage or, more likely, intra-allelic unequal crossing over.
6 ve elements that undergo increased levels of unequal crossing-over.
7 d strand slippage during DNA replication and unequal crossing-over.
8  of the gene cluster by a process of meiotic unequal crossing-over.
9 heir origins through replication slippage or unequal crossings over.
10 at substitutions can alter the mechanisms of unequal crossing-over, altering the way concerted evolut
11 ene RPP8 and two RPP8 homologs indicate that unequal crossing over and gene conversion may have contr
12 nvolves several genetic mechanisms including unequal crossing over and gene conversion.
13 e occurred as a result of repeated events of unequal crossing over and pericentric inversions during
14  region, however, is evolving rapidly due to unequal crossing over and/or gene conversion.
15   Reticulate evolutionary processes, such as unequal crossing-over and gene conversion, are known to
16 g a low rate of sequence homogenization from unequal crossing-over and gene conversion.
17                                   Intergenic unequal crossing-over and gene conversions are important
18 viously had been thought to involve frequent unequal crossing-over and gene conversions.
19  mechanisms, including replication slippage, unequal crossing-over, and gene conversion.
20 s directly measured the frequency of meiotic unequal crossing-over (approximately 3 x 10(-6)), leadin
21                     Both gene conversion and unequal crossing over are attractive mechanisms to effec
22 array's formation, and propose a model using unequal crossing-over as the primary mechanism of array
23  for its instability, because it facilitates unequal crossing-over at the locus.
24 of the deletions are shown to have arisen by unequal crossing over between CLCNKB and the nearby rela
25 bsequent repair, and cannot be attributed to unequal crossing over between resistance gene homologs.
26 chimeric gene, which was likely derived from unequal crossing over between RPP8-Ler and RPH8A ancesto
27                      In African populations, unequal crossing over between the 3DL1 and 3DL2 genes pr
28 -Tooth disease type 1A (CMT1A), results from unequal crossing over between two >98% identical 24 kb r
29 for the derivation of this haplotype invokes unequal crossing over between two known ancestral KIR ha
30 independent events that likely resulted from unequal crossing-over between segmental duplications.
31 irectly the frequency of meiotic, intergenic unequal crossing-over between sister chromatids.
32 ique P450 enzyme, CYP337B3, which arose from unequal crossing-over between two parental P450 genes, r
33 eferentially paternal in origin, arises from unequal crossing over due to homologous recombination be
34 HNPP) deletion are reciprocal products of an unequal crossing-over event between misaligned flanking
35 ng DNA was used to estimate the frequency of unequal crossing-over events (6.3 x 10(-5) events per ge
36 cial syndrome/DiGeorge syndrome results from unequal crossing-over events between two 240-kb low-copy
37 GGT, and GGTLA, consistent with Alu-mediated unequal crossing-over events.
38 of the ubiquitin-C gene in rodents is due to unequal crossing-over events.
39 f alleles and recombination, most likely via unequal crossing-over events.
40 ization of molecular genetic events, such as unequal crossing over, gene conversion and crossover asy
41 ions, duplication of the KIR3DL1/S1 locus by unequal crossing over has enabled individuals to carry a
42 xchange of flanking markers, consistent with unequal crossing over; however, although the number of D
43 ese lines allowing us to measure the rate of unequal crossing over in the PF exon.
44 o the high probability of misreplication and unequal crossing-over in the repeated segment of the gen
45 riants suggested that most of them arose via unequal crossing over, indicating that rp3 is a complex
46        These data raise the possibility that unequal crossing over may be responsible in part for the
47  concerted evolution, although the degree of unequal crossing over may differ among complex satellite
48 epeats in a given gene, which influences how unequal crossing over may occur.
49    Thus, if these reactions occur in humans, unequal crossing-over or gene conversion may also contri
50  CNVs and disease-associated CNCs is meiotic unequal crossing over, or nonallelic homologous recombin
51 loss of Rp1-D resistance can be explained by unequal crossing over that occurred mostly within coding
52      In contrast to most measures of meiotic unequal crossing-over that require the deletion of a gen
53 ave been diversified by gene duplication and unequal crossing over, thereby generating haplotypes wit
54 re for the evolution of a T-box subfamily by unequal crossing over to form a two-gene cluster that wa
55 ons caused by illegitimate recombination and unequal crossing over were major driving forces in the e
56 lication) or interallelic recombination with unequal crossing over, whereas both mechanisms appear to
57 t proximal and distal CMT1A-REPs and promote unequal crossing over, which occurs 10 times more freque
58 ents duplicating two to five LRRs because of unequal crossing-over within or between RGC2 genes at on

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