1 affected by progressive stimulation of sugar
uniport.
2 ectly, (i) the rate and selectivity of SGLT1
uniport activity and (ii) the apparent affinities of SGL
3 the other site 1 variants showed substantial
uniport activity relative to exchange.
4 K(m)(app) for 3-O-methylglucose
uniport and antiport are unaffected by metabolic poisoni
5 or obligatory exchange (also referred to as
uniport and antiport mechanisms, respectively).
6 )nucleoside mono-, di-, and triphosphates by
uniport and antiport.
7 ization of K321 dramatically changed the Na+
uniport and Na+/glucose cotransport kinetics.
8 de and FCCP stimulation of 3-O-methylglucose
uniport are associated with increased AMP-activated prot
9 This carrier catalyzed substantial
uniport besides a counter-exchange transport, exhibited
10 etely blocked by a potent mitochondrial Ca2+
uniport blocker, Ru360.
11 ial inhibitors) stimulates erythrocyte sugar
uniport but not sugar antiport.
12 Ki(app) for inhibitions of 3-O-methylglucose
uniport by cytochalasin B and forskolin (sugar export si
13 ppression results from inhibition of carrier
uniport function.
14 The maximum rate of
uniport in K321A increased 3-5-fold with a decrease in t
15 FCCP stimulation of 3-O-methylglucose
uniport in resealed erythrocyte ghosts requires cytosoli
16 An energy landscape for symport and
uniport is presented.
17 hanisms: Na+ transport occurs by a saturable
uniport mechanism, and water permeation is through a low
18 er envelope occurs by a potential-stimulated
uniport mechanism.
19 envelope can occur by a potential-stimulated
uniport mechanism.
20 urnover rate of NIS was >/=22 s-1 in the Na+
uniport mode and >/=36 s-1 in the Na+/I- cotransport mod
21 as the blockade of the mitochondrial calcium
uniport,
prevents the resveratrol-induced augmentation i
22 on, allow K+ to cross the cell membrane by a
uniport process.
23 Sugar
uniport (
sugar uptake or exit in the absence of sugar at
24 Uniport suppression is not mediated by interaction with
25 be mediated by reversal of SbSUT1 and/or by
uniporting SWEETs.
26 The antiport to
uniport switch mechanism requires ATP hydrolysis, is ass
27 and is taken up by the mitochondria via the
uniport transporter, causing mitochondrial depolarizatio
28 n, ryanodine receptor, and the mitochondrial
uniport transporter.
29 Phosphate also inhibits reverse
uniport under some conditions (EC50 approximately 20 mic