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1 in receptor terminals of both myelinated and unmyelinated fibers.
2 i-GluR4 or anti-GluR2/4 stains predominantly unmyelinated fibers.
3 rs, but only on Schwann cells of sympathetic unmyelinated fibers.
4 of myelinated fibers but relative sparing of unmyelinated fibers.
5 al role in the mediation of sensory input by unmyelinated fibers.
6 ch matches the time for the peak response in unmyelinated fibers.
7 se to cowhage is different in myelinated and unmyelinated fibers, (5) the time of peak itch sensation
8 tion of the sciatic nerve revealed a loss of unmyelinated fibers and extensive ultrastructural damage
9  which is devoid of synapses and composed of unmyelinated fibers and glial cells.
10 ites that serve local high-energy demands of unmyelinated fibers and signal transmission.
11 ning for tyrosine hydroxylase (TH), were all unmyelinated fibers and some of them ended as growth con
12 rficial laminae: C1, likely to be endings of unmyelinated fibers, and C2, of small myelinated fibers.
13 ted to small-diameter myelinated axons, thin unmyelinated fibers, and small terminals that contained
14                            It is argued that unmyelinated fibers are not blocked by the neurotoxins b
15 ated axons in the dorsal roots, although the unmyelinated fibers are preserved.
16 ements of conduction velocity indicated that unmyelinated fibers are responsible for glutamatergic si
17 resent on axons and Schwann cells of sensory unmyelinated fibers, but only on Schwann cells of sympat
18 ngs in hypertonic P18 kits decreased only in unmyelinated fibers, despite a loss in both myelinated a
19 es revealed early preferential loss of small unmyelinated fibers followed by prominent demyelination
20 ined the effects of C-peptide replacement on unmyelinated fiber function in the hind paw, sural nerve
21 old reduction in the density of regenerating unmyelinated fibers in LAR-/- nerves distal to the crush
22 l sprouting occurs, producing an increase in unmyelinated fibers in the corpus callosum.
23 1.2 channels were predominately localized in unmyelinated fibers in the cortex, hippocampus, spinal c
24 vier along adult CNS and PNS axons and along unmyelinated fibers in the PNS.
25 ever, although 65% of the AEN is composed of unmyelinated fibers, it has not been determined whether
26                     There was no evidence of unmyelinated fiber loss or a decrease in the number of m
27 III, and IV are now known and there is clear unmyelinated fiber loss.
28 otion that altered potassium homeostasis and unmyelinated fibers may represent a potential vehicle fo
29 d restored the diabetes-induced reduction of unmyelinated fiber number (P < 0.01) and mean axonal siz
30                               Myelinated and unmyelinated fibers of the lingual nerve were increased
31  the function of the adhesion molecule L1 in unmyelinated fibers of the peripheral nervous system (PN
32 eripherin (a marker of thinly myelinated and unmyelinated fibers) or calcitonin gene-related peptide
33 n of the labeling patterns suggests that AEN unmyelinated fibers project primarily to the ventral tip
34 c locations known to be innervated by small, unmyelinated fibers, suggesting that NGF modulated senso
35 he role of degeneration of myelinated versus unmyelinated fibers, we investigated the effects of an L
36 amatic proliferation in the Schwann cells of unmyelinated fibers, which resulted in the segregation o

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