ライフサイエンスコーパス: unresponsiveness

1 treatment (referred to as possible sustained unresponsiveness).

2 receptor blockade partially reversed T cell unresponsiveness.

3 months and were considered to have sustained unresponsiveness.

4 gesting a role for CD4+ T cells in enforcing unresponsiveness.

5 reas the loss of consciousness is defined by unresponsiveness.

6 imal T cell signaling events from functional unresponsiveness.

7 t may function to mediate TNF-induced T cell unresponsiveness.

8 M cells via psigma1 could induce a state of unresponsiveness.

9 and resulted in the induction of Ag-specific unresponsiveness.

10 transactivation activity, leading to T cell unresponsiveness.

11 tokine signal pathways and leading to T cell unresponsiveness.

12 se 3 is required for the induction of T cell unresponsiveness.

13 o examine the mechanism of in vivo rituximab unresponsiveness.

14 CAV and demonstrated in vitro donor-specific unresponsiveness.

15 T (T reg) cells to sites where they maintain unresponsiveness.

16 cyte reaction was used as standard to detect unresponsiveness.

17 nical trials and, in some studies, sustained unresponsiveness.

18 pe and associates with poor prognosis and RA unresponsiveness.

19 e system employs to produce antigen-specific unresponsiveness.

20 l-established method of inducing immunologic unresponsiveness.

21 ipients demonstrated in vitro donor-specific unresponsiveness.

22 re equal in their ability to induce specific unresponsiveness.

23 unosuppressive molecules that promote T cell unresponsiveness.

24 cing febrile illness that leads to IFN-gamma unresponsiveness.

25 oreceptor CD28 will prevent the induction of unresponsiveness.

26 omplex alone are sufficient to induce T cell unresponsiveness.

27 lls has been suggested as the basis for this unresponsiveness.

28 l activation followed by T cell deletion and unresponsiveness.

29 lly associated with acquisition of sustained unresponsiveness.

30 030 children [0.2%]) and not associated with unresponsiveness.

31 in T-cell exhaustion, a state of functional unresponsiveness.

32 feeding of peanut butter to assess sustained unresponsiveness.

33 domain-only protein (Hopx) to mediate T cell unresponsiveness.

34 peanut powder, and all 4 achieved sustained unresponsiveness.

35 d reports about conscious experiences during unresponsiveness.

36 nd only 10.8% of subjects achieved sustained unresponsiveness.

37 delta power increased from responsiveness to unresponsiveness.

38 th after stopping OIT and achieved sustained unresponsiveness.

39 , and occurs through the induction of B cell unresponsiveness.

40 ited their activation and resulted in T-cell unresponsiveness.

41 d with the likelihood of achieving sustained unresponsiveness.

42 nuation, indicating development of beta-cell unresponsiveness.

43 le of Treg cells in allergen-specific T-cell unresponsiveness.

44 owder and a cooked egg to test for sustained unresponsiveness.

45 e primary outcome was induction of sustained unresponsiveness 2 to 5 weeks after discontinuation of t

46 ial-precoeruleus complex produced behavioral unresponsiveness, a monotonous sub-1-Hz cortical EEG, an

47 allenge with alloantigen failed to break the unresponsiveness, a transient recovery from tolerance wa

48 3 years, (3) percentage attaining sustained unresponsiveness after 3 years, (4) immunologic end poin

49 Sustained unresponsiveness after 4 weeks of avoidance was seen in

50 urther work is required to confirm sustained unresponsiveness after a longer period of secondary pean

51 This is the first demonstration of sustained unresponsiveness after peanut OIT, occurring in half of

52 mine whether peanut OIT can induce sustained unresponsiveness after withdrawal of OIT.

53 To identify a mechanism for T cell unresponsiveness against mycobacterial lipid Ags in lepr

54 The Full Outline of UnResponsiveness, an emerging alternative, is more sensi

55 was associated with attainment of sustained unresponsiveness and a reduction in adverse reactions.

56 Although anesthesia undoubtedly induces unresponsiveness and amnesia, the extent to which it cau

57 aled a biphasic pattern of MBP proliferative unresponsiveness and an induction of Th1 cytokines.

58 Immunosuppression and T-lymphocyte unresponsiveness and apoptosis are typical of acute T. g

59 nating from these MSCs retain their TGF-beta unresponsiveness and become inflammatory.

60 ically susceptible mice induced PLP-specific unresponsiveness and completely protected the majority (

61 Parasite Ag-specific T cell unresponsiveness and diminished IFN-gamma production are

62 podocytes of diabetic mice, causing insulin unresponsiveness and DN.

63 of key inhibitory molecules reversed T cell unresponsiveness and enabled maximal T cell functions, e

64 mechanism(s) responsible for this functional unresponsiveness and eventual depletion of CD4(+) T cell

65 was effective in inducing possible sustained unresponsiveness and immune changes that suggest modulat

66 ents, tested in vitro, showed donor-specific unresponsiveness and in specimens from allograft biopsie

67 ft survival and profound alloreactive T-cell unresponsiveness and overcame the opposite effects of ca

68 halitogenic determinant induced PLP-specific unresponsiveness and protected mice from induction of EA

69 h anti-idiotype 3H1 or CEA could reverse CEA unresponsiveness and result in the induction of CEA-spec

70 to those with well-documented growth hormone unresponsiveness and severe short stature.

71 ne animal maintained donor-specific cellular unresponsiveness and stable anti-alphaGal antibody level

72 o baboons can induce donor-specific cellular unresponsiveness and stable anti-alphaGal antibody level

73 of protein induce both mucosal and systemic unresponsiveness and that use of mucosal adjuvants that

74 mechanisms, PD-L1-dependent T cell-intrinsic unresponsiveness and the activation of T regulatory cell

75 t partial TCR signaling that leads to T cell unresponsiveness and tolerance in vivo.

76 grams of activation or tolerance (functional unresponsiveness and/or physical elimination of antigen-

77 tures of the human disease, including T cell unresponsiveness, and thus represent an appropriate mode

78 tress, erythema, decreased body temperature, unresponsiveness, and, often, death.

79 tion drives responding cells into functional unresponsiveness (anergy) that can be followed by their

80 Immunological unresponsiveness-anergy- of CD4(+) T cells is characteri

81 p27-differentiated macrophages induce severe unresponsiveness/anergy in T cells.

82 Antiestrogen unresponsiveness appears to be the major acquired resist

83 ugh the molecular mechanisms underlying this unresponsiveness are unknown, some models of B cell aner

84 respond and/or relapse and the mechanisms of unresponsiveness are unknown.

85 l patients (0 of 8) displayed donor specific unresponsiveness as gauged by IFN-gamma expression and T

86 were evaluated for continuation of sustained unresponsiveness at 30 months and 36 months.

87 The primary end point, sustained unresponsiveness at 4 weeks after stopping early interve

88 Four subjects had sustained unresponsiveness at study completion.

89 olerance is manifest as a bias toward immune unresponsiveness, both in the context of a major histoco

90 ne is remarkable in that it induces profound unresponsiveness, but subjects often report "ketamine dr

91 suggest that CD154 blockade induces humoral unresponsiveness by a mechanism that involves the indire

92 ted lympholysis assays showed donor-specific unresponsiveness by day 30 across MHC class I barriers.

93 ote the induction of antigen-specific T-cell unresponsiveness by DC.

94 f B7/CD28 may facilitate induction of T-cell unresponsiveness by generating AAMphi.

95 The induction of immunologic unresponsiveness by i.v. administration of Ag-coupled ly

96 animals demonstrated in vitro donor-specific unresponsiveness by MLR and CML and did not demonstrate

97 then slowly induced longer lasting mast cell unresponsiveness by removing membrane FcepsilonRI.

98 cells persist in the periphery in a state of unresponsiveness called anergy.

99 tive at old than at young age, due to T cell unresponsiveness, caused by various age-related changes

100 (PPOIT) was effective at inducing sustained unresponsiveness compared with placebo in a double-blind

101 Anergic features and chemokine unresponsiveness could be simultaneously reversed by cul

102 toms or memory problems; 76 had seizures, 88 unresponsiveness (decreased consciousness), 86 dyskinesi

103 In unresponsiveness, delta waves propagated from frontal to

104 This IL-2 unresponsiveness did not require the continuous presence

105 lts unveil a fundamental mechanism of T cell unresponsiveness different from anergy or exhaustion, dr

106 ediates this dynamic antigen-specific T cell unresponsiveness differs from previously described forms

107 ect thalamocortical activity contributing to unresponsiveness during sleep.

108 cell engraftment and in vitro donor-specific unresponsiveness, enabling prolonged survival of subsequ

109 he induction of chimerism and donor-specific unresponsiveness following pig SpTx.

110 se, and thus help maintain the proliferative unresponsiveness found in the anergic Th1 cells.

111 The syndrome was characterized by coma/unresponsiveness (four episodes), dilated pupils (four e

112 in addition to being markers for immunologic unresponsiveness, gene expression levels of TCL1A and CD

113 ildren with egg allergy and induce sustained unresponsiveness in a clinically significant subset.

114 n primates and induced long-term Ag-specific unresponsiveness in a memory T cell-mediated inflammator

115 Treatment may result in sustained unresponsiveness in a proportion of patients, whereas ot

116 f immunological tolerance (long-term antigen unresponsiveness in an immunocompetent host) presents th

117 e characterized a common mechanism of T cell unresponsiveness in cancer driven by the upregulation of

118 fe and effective method of inducing specific unresponsiveness in CD4+ T cells for the prevention and

119 latory molecules and induce antigen-specific unresponsiveness in CD4+ T helper cells.

120 (+)Ifngr1(f/f) mice with selective IFN-gamma unresponsiveness in CD8alpha(+) dendritic cells displaye

121 An initial assessment of IL-2 unresponsiveness in cells from selected HIV-infected ind

122 Attempts to identify growth hormone unresponsiveness in children with idiopathic short statu

123 iotic and peanut OIT and assessing sustained unresponsiveness in children with peanut allergy.

124 gether, our findings indicate that chemokine unresponsiveness in CLL lymphocytes results from failure

125 ergy, an acquired state of T cell functional unresponsiveness in Foxp3(-) cells, have both been impli

126 The mechanisms of this unresponsiveness in healthy subjects are not fully under

127 e to peripheral self-Ags is the induction of unresponsiveness in mature specific T cells.

128 de (alpha-GalCer) induces long-term NKT cell unresponsiveness in mice.

129 lood indicated donor-specific posttransplant unresponsiveness in micro-cell-mediated lympholysis (m-C

130 ients tested so far exhibited donor-specific unresponsiveness in MLR (7/17) and CML (6/13) reactions

131 f the VTL grafts demonstrated donor-specific unresponsiveness in MLR assays, development of periphera

132 effective in achieving donor-specific immune unresponsiveness in models of organ transplantation.

133 acute rejection and produces donor-specific unresponsiveness in murine recipients of heterotopic hea

134 otocol and evidence for early donor-specific unresponsiveness in one of these patients.

135 d accompany the acquisition of food allergen unresponsiveness in oral immunotherapy.

136 regulatory T cells, and through induction of unresponsiveness in precursors of T effector cells, beta

137 anti-CD3 directly induces a state of immune unresponsiveness in primed pathogenic autoreactive effec

138 All recipients showed donor-specific unresponsiveness in standard cell-mediated lympholysis a

139 gnaling induces a state of anergy or antigen unresponsiveness in T cells, mediated through calcineuri

140 e mediated through deletion and induction of unresponsiveness in targeted memory T-cell populations.

141 the induction of immunological tolerance (or unresponsiveness in the absence of exogenous immunosuppr

142 n the recipients and donor-specific cellular unresponsiveness in vitro.

143 one of the animals displayed donor-specific unresponsiveness in vitro.

144 DCs and the promotion of DC-mediated T cell unresponsiveness in vivo.

145 as Hopx) in iT(reg) cells to mediate T cell unresponsiveness in vivo.

146 ans of inducing sustained Ag-specific T cell unresponsiveness in vivo.

147 novel category is characterized by their ABA unresponsiveness in Ws and activation in rop10-1 at 1 mi

148 ergy, an acquired state of T cell functional unresponsiveness, in natural peripheral tolerance remain

149 shares characteristics with antigen-specific unresponsiveness induced by other routes, yet there are

150 ss to irrelevant, nontolerizing Ag, and this unresponsiveness is associated with significant apoptosi

151 This unresponsiveness is associated with the constitutive act

152 ely inhibited in autoreactive cells in which unresponsiveness is maintained by anergy.

153 e presence and persistence of donor-specific unresponsiveness is not available.

154 ponse in newborns and the mechanism for this unresponsiveness is not clear.

155 The mechanism of rituximab unresponsiveness is not known.

156 A similar state of unresponsiveness is observed when the proliferative resp

157 This unresponsiveness is rapidly reversible, requiring contin

158 (TCR) with antigen alone will induce T-cell unresponsiveness; ligation of the coreceptor CD28 will p

159 ss of UCB lymphocytes is attributable to pan-unresponsiveness, lymphocyte repressive or recipient-spe

160 a TLR agonist LPS, suggesting that NKT cell unresponsiveness may be a major mechanism of terminating

161 This serotype-specific unresponsiveness may reflect immune paralysis due to lar

162 early development of antigen-specific T-cell unresponsiveness mediated by BM-derived antigen-presenti

163 eed to be treated for 7 to achieve sustained unresponsiveness (number needed to treat, 1.27; 95% CI,

164 s in mice given Ag via the a.c. results from unresponsiveness of Ag-specific CD8(+) T cells.

165 These results suggest that the Ag unresponsiveness of anergic B cells can be overcome by c

166 hus, BCR destabilization may underlie the Ag unresponsiveness of anergic B cells.

167 Thus maintenance of the unresponsiveness of anergic cells is critical for preven

168 on, responsible for lipopolysaccharide (LPS) unresponsiveness of C3H/HeJ mice, into the TIR domain of

169 Unresponsiveness of COX-2 to IL-10 is due to the deficie

170 th CO(2)), based on previous studies showing unresponsiveness of Glu deprotonation to CO(2).

171 on and induced a variable period of relative unresponsiveness of IgG anti-dsDNA-producing B cells, as

172 The surprising unresponsiveness of Inhba expression to hypoxia was conf

173 tumor cells, which stands in contrast to the unresponsiveness of KRAS-mutant cancers to EGFR-directed

174 ssion of FoxP3N/NLS sufficiently induces the unresponsiveness of mouse primary CD4+ CD25- T cells, wh

175 These data indicate that there is an early unresponsiveness of neonatal alveolar macrophages to Pne

176 bodies also was induced, indicating that the unresponsiveness of NZB/NZW mice to these antigens can b

177 n vivo and in vitro, fully rescues the ppGpp-unresponsiveness of RNAP lacking omega, likely explainin

178 gehog pathway inhibitor, cyclopamine, and 4) unresponsiveness of Smoothened-/- mouse embryonic fibrob

179 Immunological unresponsiveness of T cells to alloantigen can be induce

180 the transcription factor NFATc2, as well as unresponsiveness of T cells.

181 on, our findings explain the profound immune unresponsiveness of the Aly mouse.

182 The unresponsiveness of the memory cells from the nonpersist

183 ch dramatically contrasted with the complete unresponsiveness of the MSG-derived hepatocytes, also as

184 The unresponsiveness of Thy-1 (+) cells is not because of de

185 In contrast to the in vitro unresponsiveness of Treg cells when cultured alone, subs

186 echanisms underlying how FoxP3 maintains the unresponsiveness of Tregs.

187 duced TACI expression is responsible for the unresponsiveness of XID mouse to TI-2 Ags and BCR activa

188 d (APRIL) or BAFF and their receptors in the unresponsiveness of XID mouse to TI-2 Ags.

189 highlighted the critical role of functional unresponsiveness or 'anergy'.

190 n the mature repertoire, but that functional unresponsiveness or anergy exists in the mature B-cell r

191 T cell unresponsiveness or anergy is one of the mechanisms that

192 jections of alpha-GalCer result in long-term unresponsiveness or anergy of iNKT cells, severely limit

193 l killer T (NKT) cells, results in long-term unresponsiveness or anergy, which severely limits its cl

194 ipheral T-cell tolerance, which manifests as unresponsiveness or death through apoptosis.

195 nce that DCs in situ induce antigen-specific unresponsiveness or tolerance in central lymphoid organs

196 not related to inadequate nutrition, insulin unresponsiveness, or growth hormone deficiency.

197 -positive periphery with no signs of anergy, unresponsiveness, or prior activation.

198 ted specifically to acquisition of sustained unresponsiveness rather than to receiving PPOIT treatmen

199 This restorable form of splenic unresponsiveness referred to as IFN-gamma-dependent aner

200 The induction of alloantigen-specific unresponsiveness remains an elusive goal in organ transp

201 ytokines responsible as a mechanism for this unresponsiveness, restimulation assays revealed increase

202 evidence of heterogeneity in Full Outline of Unresponsiveness score agreement across hospitals.

203 This demonstrates that the Full Outline of Unresponsiveness score can be utilized reliably in criti

204 et of patients for which the Full Outline of Unresponsiveness score could be particularly beneficial

205 The Full Outline of Unresponsiveness score has emerged as an alternative to

206 ter-rater reliability of the Full Outline of Unresponsiveness score in five intensive care units.

207 linked to Glasgow Coma Scale/Full Outline of UnResponsiveness score information.

208 The Full Outline of UnResponsiveness score may be a better predictor of mort

209 The Full Outline of UnResponsiveness score might be a better prognostic tool

210 tem reflex components of the Full Outline of UnResponsiveness score showed a much wider range of mort

211 The Full Outline of Unresponsiveness score showed excellent inter-rater agre

212 , 0.663-0.768) and using the Full Outline of UnResponsiveness score was 0.742 (95% CI, 0.694-0.790),

213 In multivariable models, the Full Outline of UnResponsiveness score was more useful than the Glasgow

214 Glasgow Coma Scale and Full Outline of UnResponsiveness score were recorded within 1 hour of ad

215 xes and respiration into the Full Outline of UnResponsiveness score.

216 a new coma score, the FOUR (Full Outline of UnResponsiveness) score.

217 ts that the successful induction of specific unresponsiveness secondary to intrathymic transplantatio

218 upon activation, and CD39-/- DCs showed ATP unresponsiveness (secondary to P2-receptor desensitizati

219 mediate some of the parasite-specific T cell unresponsiveness seen in patent filarial infection.

220 n which 27.5% of subjects achieved sustained unresponsiveness (SU) after 2 years.

221 nduced desensitization in most and sustained unresponsiveness (SU) in a smaller subset.

222 rechallenge at month 32 to assess sustained unresponsiveness (SU).

223 ific Abs in external secretions and systemic unresponsiveness termed oral or mucosal tolerance.

224 or the maintenance of specific immunological unresponsiveness that can reduce the severity of the det

225 Furthermore, it induced a profound state of unresponsiveness that could be overcome only by prolonge

226 ays and consider the potential for reversing unresponsiveness through stimulatory signals or replacem

227 memory T cells that was able to break T cell unresponsiveness to a nonmutated tumor Ag and provide pr

228 ucocorticoid deficiency (FGD), or hereditary unresponsiveness to adrenocorticotropin (ACTH; OMIM 2022

229 in seven of nine animals, including complete unresponsiveness to Ag challenges in two animals.

230 -cell activation was followed by full T-cell unresponsiveness to allergen after 1 year in the MAT-Fel

231 , can be a powerful way of inducing specific unresponsiveness to alloantigens in vivo.

232 w-dose UVB exposure induces antigen-specific unresponsiveness to antigen(s) introduced through UV-irr

233 to deletion of the corresponding T cells and unresponsiveness to antigenic rechallenge with strong ad

234 telet function testing and presumed clinical unresponsiveness to aspirin, has been previously reporte

235 ion arrest in STAT5b-RARalpha(+) APL and its unresponsiveness to ATRA, we examined the effect of STAT

236 leukaemia cells, which refers to a state of unresponsiveness to B cell receptor stimulation.

237 ved poorly after adoptive transfer, and were unresponsiveness to BCR stimulation in vitro.

238 ne profile provides a mechanism that ensures unresponsiveness to commensal bacteria while maintaining

239 PC unresponsiveness to CXCL12 results in a marked accumulat

240 localized in splenic follicles despite their unresponsiveness to CXCL13.

241 mice complemented these defects and reversed unresponsiveness to DEN-induced liver injury and maligna

242 gely T cell mediated, that promotes specific unresponsiveness to donor alloantigens.

243 a key mechanism for inducing and maintaining unresponsiveness to donor alloantigens.

244 oping novel strategies for inducing specific unresponsiveness to donor alloantigens.

245 T cells, and those off drugs showed specific unresponsiveness to donor alloantigens.

246 ented the development of OAD, and maintained unresponsiveness to donor antigen for more than 42 days

247 cterized by a more advanced presentation and unresponsiveness to H. pylori eradication therapy.

248 trinitrophenyl-OVA failed to induce systemic unresponsiveness to hapten or protein.

249 Unresponsiveness to harmless antigens is established thr

250 of intracellular signaling may contribute to unresponsiveness to IFNalpha/beta in HIV-1 disease.

251 finding that was associated with lymphocyte unresponsiveness to IL-12 stimulation in vitro.

252 emagglutinin fusion peptide along with their unresponsiveness to inducers of IFN as measured in vitro

253 ting as disturbed lymph node homeostasis and unresponsiveness to inflammatory stimuli.

254 itamin A deficiency could be associated with unresponsiveness to interferon-based antiviral therapy.

255 onse to antiviral therapy are due to greater unresponsiveness to intracellular actions of interferon

256 These CD8+ T cells transferred unresponsiveness to naive recipients.

257 The observed unresponsiveness to OVA-psigma1 could be adoptively tran

258 CD154 blockade, therefore, induced humoral unresponsiveness to pig cells.

259 tagenesis led to constitutive activation and unresponsiveness to PIP3 in vitro or insulin in vivo.

260 her the manipulation of PKB can overcome the unresponsiveness to protection of the diabetic myocardiu

261 myocardium and, importantly, it reversed the unresponsiveness to protection of the diabetic myocardiu

262 x, proliferated and failed to mediate T cell unresponsiveness to rechallenge with antigen.

263 al dendritic cells (DCs) that mediate T cell unresponsiveness to rechallenge with antigen.

264 kdown in any of the mechanisms that maintain unresponsiveness to self (a state known as self-toleranc

265 ndritic cells may play a role in maintaining unresponsiveness to self-Ag during chronic inflammation.

266 series of discrete checkpoints that maintain unresponsiveness to self.

267 es, a critical process in the maintenance of unresponsiveness to self.

268 These data suggest that the unresponsiveness to T-dependent Ags displayed by hCR2-po

269 mechanisms; apoptotic contraction and marked unresponsiveness to TCR stimulation, as a synchronized h

270 At day 120, MLR demonstrated unresponsiveness to the host and donor antigens but stro

271 veloping in SW thymus grafts showed specific unresponsiveness to the major histocompatibility complex

272 ns are effective immunogens to overcome host unresponsiveness to the nominal antigen, the structural

273 lating altered peptide ligand, L144, induced unresponsiveness to the self peptide, proteolipid protei

274 To overcome unresponsiveness to the self-high molecular weight melan

275 the SW but not the HU grafts showed specific unresponsiveness to the SW donor.

276 s associated with the development of in vivo unresponsiveness to the transplanted spleen.

277 o alloantigen develops, either activation or unresponsiveness to the triggering antigen, will enable

278 However, costs and unresponsiveness to therapy in a sizeable proportion of

279 patients at higher risk of GVHD progression, unresponsiveness to therapy, or death.

280 tolerance can be harnessed to induce humoral unresponsiveness to transplanted alloantigens.

281 One mechanism contributing to immunologic unresponsiveness toward tumors may be presentation of tu

282 ested by newborn mice, stimulates a state of unresponsiveness toward viral antigens.

283 Additionally, immunological 'unresponsiveness' towards the resident commensal microfl

284 baseline, transition into unresponsiveness, unresponsiveness, transition into responsiveness, and re

285 Mechanisms underlying this acquired unresponsiveness, typified by diminished functional resp

286 into five states: baseline, transition into unresponsiveness, unresponsiveness, transition into resp

287 e attempted to induce specific immunological unresponsiveness using donor bone marrow cell infusions

288 to proteins, whereas haptens elicit systemic unresponsiveness via the intestinal epithelial cell barr

289 Possible sustained unresponsiveness was achieved in 82.1% receiving PPOIT a

290 T cell unresponsiveness was associated with defects in TCR prox

291 Unresponsiveness was partially overcome using high-dose,

292 CD19 unresponsiveness was partially reversible, where nonresp

293 The tolerant T-cell unresponsiveness was reversed by the addition of IL-2.

294 This unresponsiveness was reversible by treatment with anti-C

295 The unresponsiveness was unable to be overcome by supplement

296 ndpoint of general anesthetics is behavioral unresponsiveness, which is commonly associated with loss

297 Three animals demonstrated donor-specific unresponsiveness, while maintaining normal alloresponses

298 ffects are seen that can distinguish between unresponsiveness with and without consciousness.

299 and language disintegration into a state of unresponsiveness with catatonic features often associate

300 is characterized by antigen-specific T-cell unresponsiveness with diminished IFN-gamma and IL-2 prod