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1 n tests for routinely encountered agents are unrevealing.
2 Gynecological investigation was unrevealing.
3 outine microbiological investigations remain unrevealing.
4 ormed by the primary care physician that was unrevealing.
5 causes of inherited and acquired ataxia were unrevealing.
6 ing of common cancer susceptibility genes is unrevealing.
7 rior clinical cardiac gene panel testing was unrevealing.
8 aluation for alternative causes of fever was unrevealing.
9 or OMM, especially when tissue histology is unrevealing.
10 Past history was unrevealing.
11 Metabolic testing was unrevealing.
12 Diagnostic workup including brain biopsy was unrevealing.
13 nown genetically mediated arteriopathies was unrevealing.
14 Serum and cerebrospinal fluid studies were unrevealing and a biopsy of the cystic lesions was perfo
15 stigated because diagnostic evaluations were unrevealing, and in each a common donor was recognized.
16 ronchoscopy with bronchoalveolar lavage were unrevealing, and open lung biopsy revealed active small
17 atients with MDS and a normal-karyotype were unrevealing, and sequencing the IER3 coding and proximal
23 ory, examination, and genetic evaluation are unrevealing, NCS/EMG and muscle biopsy together provide
25 nylpropanoids along with lignin accumulation unrevealing possible mechanistic basis of EB resistance
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