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1 y revascularization, and hospitalization for unstable angina).
2 -segment elevation myocardial infarction; 6% unstable angina).
3 nts (composite of CAD death, nonfatal MI, or unstable angina).
4 he trial of death, myocardial infarction, or unstable angina.
5 t (within 3 months) myocardial infarction or unstable angina.
6 vation myocardial infarction, and 23.6% with unstable angina.
7 events, including 141 MIs and 84 episodes of unstable angina.
8 -STEMI (NSTEMI), and 19 777 (40% women) with unstable angina.
9 ns exist regarding the use of angiography in unstable angina.
10 non-ST-elevation myocardial infarction, and unstable angina.
11 cardial infarction and rehospitalization for unstable angina.
12 ups and do not include subgroup analysis for unstable angina.
13 ndromes, including myocardial infarction and unstable angina.
14 her vascular events, and hospitalization for unstable angina.
15 8 months after receiving 2 Cypher stents for unstable angina.
16 hemic stroke, transient ischemic attack, and unstable angina.
17 ome, including patients with enzyme-negative unstable angina.
18 ath, myocardial infarction, or admission for unstable angina.
19 onset, while in 52% it was class II to IV or unstable angina.
20 iagnosis in 1 man was downgraded from AMI to unstable angina.
21 , and stroke, as well as hospitalization for unstable angina.
22 non-fatal stroke) or hospital admission for unstable angina.
23 ion, nonfatal stroke, or hospitalization for unstable angina.
24 the primary outcome plus hospitalization for unstable angina.
25 l infarction, stroke, or hospitalization for unstable angina.
26 classified as myocardial infarction (MI) and unstable angina.
27 rwent PCI for acute myocardial infarction or unstable angina.
28 ients), myocardial infarction (17 patients), unstable angina (10 patients), or stroke (3 patients).
29 reatments for acute myocardial infarction or unstable angina (10%), treatments for heart failure (9%)
31 rdial infarction, 105,708 [21.1%]; high-risk unstable angina, 146,464 [29.3%]), and 144,737 (28.9%) f
32 ial infarction, 13.1% (95% CI, 1.1-23.7) for unstable angina, 16.4% (95% CI, 15.1-17.7) for heart fai
33 non-ST-elevation myocardial infarction, 218 unstable angina, 163 sudden cardiac death); 188 (9%) per
35 her in the MI group (7.5%) compared with the unstable angina (2.7%) and non-ACS (2.6%) groups (P < .0
36 mized 360 patients presenting with stable or unstable angina (28% of patients) and negative Troponin
37 and RITA-3 (Randomized Intervention Trial of Unstable Angina 3) non-ST-elevation acute coronary syndr
38 al infarction, 83.8% [95% CI, 83.3-84.4] for unstable angina, 30.5% [95% CI, 29.3-31.6] for heart fai
40 e rupture occurred not only in patients with unstable angina (46%) or myocardial infarction (MI, 33%)
41 0.1+/-15.4% versus 45.4+/-23.3%; P<0.01) and unstable angina (67.4+/-12.9% versus 45.4+/-23.3%; P>0.0
43 infarction, coronary revascularisation, and unstable angina (active treatment HR 0.89, 95% CI 0.79-0
44 admitted with acute myocardial infarction or unstable angina (acute coronary syndromes) were randomiz
45 mic stroke, myocardial infarction, new-onset unstable angina, acute coronary interventions, and vascu
46 acute coronary syndromes (ACS) present with unstable angina, acute myocardial infarction, and sudden
47 e for coronary thrombosis, the main cause of unstable angina, acute myocardial infarction, and sudden
48 over the initial year of treatment and more unstable angina admissions (hazard ratio=2.8 [1.1-7.5]).
49 evascularisation alone (0.84, 0.75-0.94) and unstable angina alone (0.81, 0.67-0.97) during full foll
51 ith men, women were older and more often had unstable angina and congestive heart failure, lower phys
53 Trial participants had typical symptoms of unstable angina and frequently had a positive electrocar
55 ased risk of acute ischemic coronary events (unstable angina and myocardial infarction) equal to 4.5%
56 n CRP and SAA were observed in patients with unstable angina and non-Q-wave myocardial infarction, wi
59 initiative included high-risk patients with unstable angina and non-ST-segment elevation myocardial
60 ed risk of adverse outcomes in patients with unstable angina and non-ST-segment elevation myocardial
61 t elevation acute coronary syndromes include unstable angina and non-ST-segment elevation myocardial
62 ctrum of clinical presentations ranging from unstable angina and non-ST-segment elevation myocardial
63 h poorer clinical outcomes in the setting of unstable angina and non-STEMI and after percutaneous cor
64 s significant reduction in revascularization/unstable angina and nonsignificant reductions in other c
66 ent depression in patients with recent MI or unstable angina and without other life-threatening medic
67 ry artery disease, 2806 hospitalizations for unstable angina, and 1029 fatal or nonfatal strokes occu
72 cularization procedures, hospitalization for unstable angina, and diagnosis of new ischemic heart dis
73 hypertension, diabetes, renal insufficiency, unstable angina, and heart failure, but less smoking.
74 on, non-fatal stroke, hospital admission for unstable angina, and hospital admission for heart failur
75 atal stroke, urgent revascularisation due to unstable angina, and hospital admission for heart failur
76 MLD, longer stent length, diabetes mellitus, unstable angina, and hypertension were independent predi
77 chemic attack, intracerebral hemorrhage, and unstable angina, and inverse (0.69) for subarachnoid hem
80 dial infarction, coronary revascularisation, unstable angina, and new angina during active treatment
81 -ST-segment-elevation myocardial infarction, unstable angina, and non-acute coronary syndrome) in The
85 -segment elevation myocardial infarction and unstable angina, and venous thromboembolism prophylaxis.
86 xamined: (1) myocardial infarction (MI); (2) unstable angina; and (3) revascularization not associate
87 stive failure, hypertension, prior CABG, and unstable angina; and had higher body mass index and lowe
89 rction [MI], congestive heart failure [CHF], unstable angina, arrhythmia, symptomatic hypotension, or
90 nfarction, stroke, admission to hospital for unstable angina, arterial revascularisation, or cardiova
91 al infarction or stroke, hospitalisation for unstable angina, arterial revascularisation, or cardiova
92 infarction, non-fatal stroke, admission for unstable angina, arterial revascularisation, or cardiova
93 al infarction or stroke, hospitalization for unstable angina, arterial revascularization, or cardiova
94 ardial infarction, stroke, hospital stay for unstable angina, arterial revascularization, or confirme
95 scular disease (CVD) (myocardial infarction, unstable angina, arterial revascularization, stroke, or
96 s with recent acute myocardial infarction or unstable angina as a prognostic indicator for eventual c
98 on Therapy) were used to report rates of MI, unstable angina, cardiac arrest, and cardiac death and t
100 ained ACS, defined as myocardial infarction, unstable angina, cardiac arrest, or death due to ischemi
101 s included acute myocardial infarction (MI), unstable angina, cardiac catheterization, percutaneous t
102 MACE defined as acute myocardial infarction, unstable angina, cardiogenic shock, ventricular arrhythm
103 dial infarction, stroke, hospitalization for unstable angina, coronary revascularization, or heart fa
104 ents including death, myocardial infarction, unstable angina, coronary revascularization, stroke, tra
105 (fatal and nonfatal myocardial infarctions, unstable angina, deaths from coronary heart disease, fat
106 Patients with myocardial infarction and unstable angina demonstrate increased total binding of p
109 o be male, younger, and with higher rates of unstable angina, emergency operation, recent or transmur
111 s severe types of ACS at presentation (e.g., unstable angina > non-ST-segment elevation MI > ST-segme
112 002), 0.41 and 0.77 for revascularization or unstable angina (hazard ratio, 0.53; 95% CI, 0.40 to 0.7
113 Reductions in risk of hospitalization for unstable angina (hazard ratio, 0.55; 95% confidence inte
114 non-fatal stroke, admission to hospital for unstable angina, heart failure, and all-cause mortality.
115 , myocardial infarction, hospitalization for unstable angina, heart failure, or a peripheral vascular
116 condary analysis, additional events included unstable angina, heart failure, or stroke at five years.
117 cular disease events (myocardial infarction, unstable angina, heart failure, or stroke) in relation t
118 or non-fatal stroke, hospital admission for unstable angina, hospital admission for heart failure, d
119 or non-fatal stroke, hospital admission for unstable angina, hospital admission for heart failure, o
120 s all-cause death, myocardial infarction, or unstable angina hospitalization over a median follow-up
121 rtality, nonfatal myocardial infarction, and unstable angina hospitalization was similar and fair for
123 d point was death, myocardial infarction, or unstable angina hospitalizations over a median follow-up
124 d toward an increase in hospitalizations for unstable angina (HR, 1.37; 95% CI, 0.99 to 1.91; P = 0.0
126 nonfatal myocardial infarction, stroke, and unstable angina in models adjusted for age, sex, hyperte
127 all patients treated with PCI for stable or unstable angina in small native coronary vessels (refere
128 ss the effect of angiography on mortality in unstable angina, incorporating the results of additional
129 rdiac death (3), myocardial infarction (12), unstable angina/ischemic ventricular fibrillation (2), s
130 tion, stroke, coronary revascularisation, or unstable angina; key secondary endpoint was the composit
131 all-cause mortality, myocardial infarction, unstable angina leading to hospitalization, and revascul
132 te (within 6 months), age, gender, stable or unstable angina, lesion location, and additional treatme
133 female gender, body mass index <25 kg/m(2), unstable angina, moderate or poor ejection fraction, and
134 I (adjusted OR, 0.77; 95% CI, 0.63-0.95) and unstable angina, mortality was lower among women (adjust
136 evels), major adverse cardiovascular events (unstable angina, myocardial infarction, death), and safe
137 /=65 years of age who were hospitalized with unstable angina, myocardial infarction, heart failure, i
139 et, with acute coronary syndromes defined as unstable angina (n=12) and acute myocardial infarction (
140 sion [n = 7], myocardial infarction [n = 5], unstable angina [n = 3], pericarditis [n = 2], arrhythmi
141 mia (ie, acute myocardial infarction [MI] or unstable angina; n = 329), there were no differences in
142 gible patients were older than 18 years with unstable angina, non-ST segment elevation myocardial inf
143 ndrome encompasses three clinical diagnoses: unstable angina, non-ST-segment elevation myocardial inf
144 in comparison with UFH in the management of unstable angina/ non-ST-segment elevation myocardial inf
145 clinical trials for acute coronary syndrome (unstable angina/non-ST segment elevation myocardial infa
147 asymptomatic/mild angina, stable angina, and unstable angina/non-ST-elevation myocardial infarction b
148 -elevation myocardial infarction, 35527 with unstable angina/non-ST-elevation myocardial infarction,
149 rican College of Cardiology for treatment of unstable angina/non-ST-elevation myocardial infarction.
151 al presentation, diagnosis, and treatment of unstable angina/non-ST-segment elevation myocardial infa
152 ce of these agents in treating patients with unstable angina/non-ST-segment elevation myocardial infa
153 n scintigraphy (SPS) in patients with recent unstable angina/non-ST-segment elevation myocardial infa
154 n (GP) IIb/IIIa inhibitors are beneficial in unstable angina/non-ST-segment elevation myocardial infa
155 s and DAPT in the postdischarge treatment of unstable angina/non-ST-segment-elevation myocardial infa
156 s among specific PPI agents in patients with unstable angina/non-ST-segment-elevation myocardial infa
157 yocardial infarction [STEMI] and 15,459 with unstable angina/non-STEMI [UA/NSTEMI]), of whom 10 613 (
158 enoxaparin in improving clinical outcomes in unstable angina/NSTEMI patients has led to investigation
159 ry revascularization, or hospitalization for unstable angina, occurred in 446 of the participants who
161 ypertension and 28 cardiovascular events (17 unstable angina or arrhythmia, 3 nonfatal stroke, 3 hear
162 ypertension and 31 cardiovascular events (11 unstable angina or arrhythmia, 8 nonfatal myocardial inf
163 se, coronary arterial revascularization, and unstable angina or congestive heart failure requiring ho
164 rdiac death, myocardial infarction (MI), and unstable angina or congestive heart failure warranting h
166 rs) and most patients presenting either with unstable angina or for percutaneous coronary interventio
167 reference group and had the highest risk of unstable angina or myocardial infarction (HR, 5.84 [3.43
168 (PCI) Access Site Approach in Patients With Unstable Angina or Myocardial Infarction Managed With an
169 sified platelet inhibition for patients with unstable angina or myocardial infarction without ST-segm
171 n=375,886) or acute coronary syndromes (ACS; unstable angina or myocardial infarction, n=450,329) at
174 In the MIRACL trial, 3,086 patients with unstable angina or non-Q-wave myocardial infarction were
176 d, placebo-controlled trial of patients with unstable angina or non-Q-wave myocardial infarction.
178 ND PATIENTS: Patients with medically managed unstable angina or non-ST-segment elevation myocardial i
179 nts with multivessel disease presenting with unstable angina or non-ST-segment elevation myocardial i
180 -eluting stent implantation in patients with unstable angina or non-ST-segment elevation myocardial i
181 rction (TIMI) 18 trial, patients with either unstable angina or non-ST-segment elevation myocardial i
182 baseline and in patients with stable angina, unstable angina, or a history of myocardial infarction.
183 , myocardial infarction, hospitalization for unstable angina, or any coronary revascularization).
184 adverse events of congestive heart failure, unstable angina, or arrhythmia--with the use of pooled d
185 atal myocardial infarction, ischemic stroke, unstable angina, or cardiac arrest with resuscitation.
190 erial revascularization, hospitalization for unstable angina, or death from cardiovascular causes).
191 erial revascularization, hospitalization for unstable angina, or death from cardiovascular causes.
192 and non-fatal stroke, hospital admission for unstable angina, or hospital admission for heart failure
195 )-myocardial infarction, hospitalization for unstable angina, or urgent/emergency coronary revascular
196 olecule 1 (P<0.001), and previous history of unstable angina (P=0.01) were independent predictors of
198 tive protein levels were reduced (P=0.03) in unstable angina patients receiving amoxicillin, and fibr
199 ta from the Can Rapid Risk Stratification of Unstable Angina Patients Suppress Adverse Outcomes With
200 ta from the Can Rapid Risk Stratification of Unstable Angina Patients Suppress Adverse Outcomes With
201 he CRUSADE (Can Rapid Risk Stratification of Unstable Angina Patients Suppress Adverse Outcomes with
202 lled in the Can Rapid risk stratification of Unstable angina patients Suppress ADverse outcomes with
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205 08 CRUSADE (Can Rapid risk stratification of Unstable angina patients Suppress ADverse outcomes with
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207 lled in the Can Rapid Risk Stratification of Unstable Angina Patients Suppress Adverse Outcomes With
208 on, and the Can Rapid risk stratification of Unstable angina patients Suppress ADverse outcomes with
209 he CRUSADE (Can Rapid risk stratification of Unstable angina patients Suppress ADverse outcomes with
210 he CRUSADE (Can Rapid Risk Stratification of Unstable Angina Patients Suppress Adverse Outcomes With
211 he CRUSADE (Can Rapid Risk Stratification of Unstable Angina Patients Suppress Adverse Outcomes With
212 00 CRUSADE (Can Rapid risk stratification of Unstable angina patients Suppress ADverse outcomes with
213 he CRUSADE (Can Rapid Risk Stratification of Unstable Angina Patients Suppress Adverse Outcomes with
214 he CRUSADE (Can Rapid Risk Stratification of Unstable Angina Patients Suppress Adverse Outcomes with
215 ting in the Can Rapid Risk Stratification of Unstable Angina Patients Suppress Adverse Outcomes With
216 ts from the Can Rapid Risk Stratification of Unstable Angina Patients Suppress Adverse Outcomes with
217 ears in the Can Rapid Risk Stratification of Unstable Angina Patients Suppress Adverse Outcomes With
218 We linked Can Rapid Risk Stratification of Unstable Angina Patients Suppress Adverse Outcomes With
219 The crude all-cause 1-year mortality for unstable angina patients with H-FABP <5.8 microg/l was 2
222 ligible patients had stable angina pectoris, unstable angina pectoris, or non-ST-elevation myocardial
223 ents presenting with stable angina pectoris, unstable angina pectoris,and ST-segment elevation myocar
225 oronary artery disease, history of stable or unstable angina, previous multi-vessel percutaneous coro
226 hypertension; diabetes mellitus; nonsmokers; unstable angina; previous coronary artery bypass graftin
227 ts (acute myocardial infarction, arrhythmia, unstable angina, pulmonary embolism) and little or no as
228 , neurological disease, active endocarditis, unstable angina, recent myocardial infarction, and pulmo
229 e PURSUIT (Platelet Glycoprotein IIB/IIIA In Unstable Angina: Receptor Suppression Using Integrilin T
230 USTO)-IIb, Platelet Glycoprotein IIb/IIIa in Unstable Angina: Receptor Suppression Using Integrilin T
232 , death, reinfarction, rehospitalization for unstable angina, repeat coronary revascularization (targ
233 rate of all-cause mortality, nonfatal MI, or unstable angina requiring hospitalization at 4 years.
234 and either an acute myocardial infarction or unstable angina requiring hospitalization within the pre
235 l myocardial infarction, ischemic stroke, or unstable angina requiring hospitalization) in multivaria
236 ction, fatal or nonfatal ischemic stroke, or unstable angina requiring hospitalization) was lower wit
237 site of all-cause mortality, nonfatal MI, or unstable angina requiring hospitalization; the secondary
238 cular death, nonfatal myocardial infarction, unstable angina requiring rehospitalization, coronary re
239 ath due to any cause, myocardial infarction, unstable angina requiring rehospitalization, revasculari
240 ll causes, myocardial infarction, documented unstable angina requiring rehospitalization, revasculari
241 any cause, myocardial infarction, documented unstable angina requiring rehospitalization, revasculari
242 ary end point (death, myocardial infarction, unstable angina requiring rehospitalization, stroke, or
243 ch additionally included hospitalisation for unstable angina requiring unplanned revascularisation) a
244 rction, nonfatal stroke, hospitalization for unstable angina requiring urgent revascularization, or c
245 osites of death, myocardial infarction (MI), unstable angina, revascularization >30 days, and stroke
247 end point was death, myocardial infarction, unstable angina, revascularization, or stroke (mean foll
248 The third Randomized Intervention Trial of unstable Angina (RITA-3) evaluated early IS (n = 895) ve
249 , the third Randomized Intervention Trial of unstable Angina (RITA-3) recently reported a halving of
250 ere was a reduction in rehospitalization for unstable angina (RR = 0.69, 95% CI 0.65 to 0.74, p < 0.0
251 art failure (CHF) within 4 weeks before CEA; unstable angina; steroid-dependent chronic obstructive p
252 h, acute MI, new congestive heart failure or unstable angina, stroke, and significant ventricular arr
253 dial infarction, coronary revascularization, unstable angina, stroke, or congestive heart failure.
254 subsequent vascular events in patients with unstable angina, the cost-effectiveness of this combinat
255 ugh early invasive therapy reduces recurrent unstable angina, the magnitude of benefit on other impor
256 The diagnosis in 2 women was upgraded from unstable angina to AMI, and the diagnosis in 1 man was d
257 155 to 271 mg/dl 3 to 36 months after MI or unstable angina to placebo or pravastatin 40 mg per day.
258 ere bleeding according to the Clopidogrel in Unstable Angina to Prevent Recurrent Events (CURE) and T
259 l alone versus aspirin alone, Clopidogrel in Unstable Angina to Prevent Recurrent Events (CURE) in fa
261 Patients (n=5059) from the Clopidogrel in Unstable Angina to Prevent Recurrent Events (CURE) rando
262 r ACS was demonstrated in the Clopidogrel in Unstable angina to prevent Recurrent Events (CURE) trial
263 lopidogrel and placebo (CURE [Clopidogrel in Unstable Angina to Prevent Recurrent Events], CREDO [Clo
265 entional Treatment Strategy in Patients with Unstable Angina) trials for a collaborative meta-analysi
267 an acute myocardial infarction (MI) (n = 8), unstable angina (UA) (n = 15), stable coronary artery di
268 routine invasive approach for patients with unstable angina (UA) and non-ST-segment elevation myocar
269 ne early use of clopidogrel in patients with unstable angina (UA) and non-ST-segment elevation myocar
271 creased CHD death/myocardial infarction (MI)/unstable angina (UA) event rate in carriers of the GP1BA
272 real-world, medically managed patients with unstable angina (UA) or non-ST-segment elevation myocard
273 ent elevation myocardial infarction (NSTEMI)/unstable angina (UA) who were managed medically without
274 oronary intervention for stable angina (SA), unstable angina (UA), or acute myocardial infarction (AM
278 re 2 trials of an early invasive strategy in unstable angina (UA)/non-ST-elevation myocardial infarct
279 ) bleeding among patients with elective PCI, unstable angina (UA)/non-ST-segment elevation myocardial
280 nder and cardiac biomarkers in patients with unstable angina (UA)/non-ST-segment elevation myocardial
282 aphic and clinical outcomes in patients with unstable angina (UA)/non-ST-segment elevation myocardial
283 ] death, myocardial infarction [MI], stroke, unstable angina [UA] leading to hospitalization, coronar
284 infarction; three patients with progressive unstable angina underwent diagnostic coronary angiograph
285 g angiography within 2 months of their index unstable angina versus 0.097 (CI, 0.090 to 0.105) for th
287 ard ratio, 4.06; 95% CI, 1.84-8.94; P<0.001; unstable angina versus non-ACS: adjusted hazard ratio, 1
288 iovascular event plus hospital admission for unstable angina was greater than 1.3, a dedicated study
289 (n=8) specimens from patients with stable or unstable angina were classified as complicated or uncomp
291 ndomized trial, 2008 patients with stable or unstable angina were randomly assigned in a 2:1 ratio to
292 d recurrent ischemic events (recurrent MI or unstable angina) were identified through Olmsted County,
293 MRS was primarily due to hospitalization for unstable angina, which is associated with repeat cathete
295 cardial infarction (MI), nonfatal stroke, or unstable angina with evidence of ischemia requiring hosp
296 -segment elevation myocardial infarction, or unstable angina with high-risk features) or nonacute ind
297 spitalized for ACS (myocardial infarction or unstable angina) with type 2 diabetes occurred between F
299 infarction or who had been hospitalized for unstable angina within the previous 180 days to receive
300 ninvasive stress results (n = 1906; 57%) and unstable angina without high-risk features (n = 902; 27%
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