ライフサイエンスコーパス: upper limb

1 rol over very short time scales (<100 ms for upper limb).

2 rceived self-attribution and position of the upper limb.

3 n about natural, multijoint movements of the upper limb.

4 mpany gains in motor function of the paretic upper limb.

5 arterial network in TSP2-null muscles of the upper limb.

6 cribed by others in quiescent muscles of the upper limb.

7 task dependency in vestibular control of the upper limb.

8 of the left and right distal muscles of the upper limb.

9 ormally innervated face and the deafferented upper limb.

10 plementary roles in patterning the mammalian upper limb.

11 lly identified motoneurons projecting to the upper limb.

12 wing mechanical perturbations applied to the upper limb.

13 ivity patterns for different segments of the upper limb.

14 hboring regions that represent the trunk and upper limbs.

15 ons can directly influence movements of both upper limbs.

16 at commenced and remained predominant in the upper limbs.

17 electrical burn requiring amputation of his upper limbs.

18 presented with mild myoclonus affecting the upper limbs.

19 on by re-engaging functional activity of the upper limbs?

20 re on the head and/or neck (435 [33.4%]) and upper limbs (455 [34.9%]).

21 were more obvious in the lower limb than the upper limb (59% versus 29%).

22 identify how sensorimotor adaptation of the upper limb, a cerebellar-dependent process restoring mov

23 haracterized by congenital heart defects and upper limb abnormalities.

24 y and motor functions of the stroke-affected upper limb, accompanied by cortical plasticity.

25 ent with a compensatory response to maintain upper-limb accuracy in the face of body movement.

26 nticipate, predict, comprehend, and memorize upper limb actions, which they cannot simulate, as effic

27 outflow is the most important determinant of upper limb akinesia in Parkinson's disease.

28 lidotomy on previously described measures of upper limb akinesia in Parkinson's disease.

29 Various measures of upper limb akinesia were assessed in 6 patients with bil

30 in the design of prosthetic arms have helped upper limb amputees achieve greater levels of function.

31 ing functional MRI (fMRI) we investigated 13 upper limb amputees with phantom limb pain (PLP) during

32 reassess its relationship to phantom pain in upper limb amputees.

33 ed collateral growth (arteriogenesis) in the upper limb and capillary formation and vessel maturation

34 ls originating near the ligation site in the upper limb and connecting to the ischemic calf muscle re

35 workers handling a mine, resulting in severe upper limb and facial injuries.

36 improvement in functional use of the paretic upper limb and quality of life 2 years after a 2-week CI

37 king system to track markers placed on their upper limb and recorded single-unit activity from a micr

38 or as well a cognitive performance, posture, upper limb and working memory function were assessed in

39 with sensory ataxia, severe weakness of the upper limbs and axial muscles with distinctly preserved

40 significant neurological amelioration in the upper limbs and trunk with more-modest amelioration in t

41 rt symptoms experienced in every part of the upper limbs and vertebral spine.

42 urophysiologically active involvement of the upper limbs and were compared with MR images in 12 age-m

43 n between shape elements, which characterize upper limbs, and upper-limb-specific motion patterns.

44 ngruent visuoproprioceptive signals from the upper limb are essential for successful VT integration.

45 tures, as non-object directed actions of the upper limb are not well characterized in non-human prima

46 , we selected all patients in whom bilateral upper limb arterial anatomy was assessed based on the pe

47 An ABC upper limb arterial axis variant was detected in 156 (25

48 Upper limb ataxia was evident in 75%, lower limb ataxia

49 th a subsequent good or poor recovery of the upper limb based solely on a T1-weighted structural brai

50 r cortex (PMv) represent the position of the upper limbs based on visual and proprioceptive informati

51 minimum level of grip is required before the upper limb becomes active in balance control and also th

52 The types of cut marks on upper limb bones indicate simple flesh removal activitie

53 hals were more heavily muscled, had stronger upper-limb bones, and possessed unusual shapes and orien

54 ction velocity was moderately reduced in the upper limbs but unobtainable in the legs.

55 e interventions, including evaluation of the upper limbs by Fugl-Meyer Assessment (FMA; primary outco

56 uce bidirectional changes in excitability in upper limb CM pathways.

57 A significant group x time interaction in upper limb (combined hand and modified arm) Fugl-Meyer a

58 with the long-standing cortical network for upper limb control.

59 4.0; 95% confidence interval 1.6-10) but not upper-limb deep venous thrombosis (odds ratio 0.6; 95% c

60 autosomal dominant disorder characterized by upper limb defects, apocrine-gland defects including mam

61 In patients with long-term upper-limb deficits after stroke, robot-assisted therapy

62 haracterized by congenital heart defects and upper limb deformations.

63 no demonstrable effects on the occurrence of upper limb digital ulcers or on other vascular manifesta

64 subcortical stroke patients with unilateral upper limb disability.

65 of losing clinically meaningful mobility and upper limb disease progression milestones across the lif

66 movements is to maintain the accuracy of the upper limb during unpredictable body movement, but only

67 ed by their involvement in the processing of upper-limb dynamics.

68 th absent or severely shortened upper limbs (upper limb dysplasia), despite some variability, could p

69 viduals born without hands (individuals with upper limb dysplasia), who use tools with their feet.

70 Upper limb EMGs were recorded to control for covert musc

71 -like) and derived (Homo-like) features, the upper limbs (excluding the hand and wrist) of the Malapa

72 lude that the motor networks controlling the upper limb exhibit an intrinsic periodicity at submoveme

73 inal projections to the motor neuron pool of upper-limb extensor muscles have been reported to differ

74 itate responsiveness of the motor pathway to upper limb flexor muscles.

75 In monkeys with deafferented upper limbs for 12 to 20 years, it was found that the br

76 eas and the transformation of the use of the upper limbs from a purely locomotor function to one incl

77 vical cord area was associated with impaired upper limb function and increased activations with handg

78 al research, few are available for measuring upper limb function post-stroke.

79 Upper limb function was generally less impaired, but 54%

80 for the primary outcome measure of impaired upper limb function, as measured with the Wolf motor fun

81 point to a more consistent effect on distal upper limb function, especially for inhibitory (cathodal

82 e cervical CST also correlated with impaired upper limb function, independent of cord area (p = 0.03)

83 in clinically significant gains in tests of upper limb function.

84 limb was assessed with clinical measures of upper limb function.

85 te axonal changes within the cranial CST and upper limb function.

86 ence for low-level, hardwired constraints on upper-limb function.

87 er-limb training program designed to improve upper-limb functional motor capacity.

88 cognitive retardation; abnormalities of the upper limbs; gastroesophageal dysfunction; cardiac, opht

89 l malformations, in particular craniofacial, upper limb, heart, and urinary system defects that are p

90 ent study we show that selectivity to static upper-limb images and motion processing go hand in hand.

91 We propose that the selectivity to static upper-limb images in the OTC may be a result of experien

92 chronic stroke who had presented with severe upper limb impairment and asked whether it was possible

93 hypothesized that proportional resolution of upper limb impairment depends on ipsilesional corticomot

94 For most patients, resolution of upper limb impairment during the first 6 months poststro

95 By 12 weeks, upper limb impairment resolved by 70% in patients with M

96 These findings indicate that upper limb impairment resolves by 70% of the maximum pos

97 Upper limb impairment was measured in 93 patients at 2,

98 In one form of CIMT to treat upper limb impairment, use of the less severely affected

99 volving 127 patients with moderate-to-severe upper-limb impairment 6 months or more after a stroke, w

100 ent therapy (CIMT) was developed to overcome upper limb impairments after stroke and is the most inve

101 Those with upper limb impairments completed the DASH again at 6 wee

102 n 17 patients, lower limb in 31 patients and upper limb in 28 patients.

103 After long-term denervation of an upper limb in macaque monkeys, the representation of the

104 n provided via TMSR affected the maps of the upper limb in primary motor (M1) and primary somatosenso

105 it into a multisensory representation of the upper limb in space.

106 f peri-hand neurons in the perception of the upper limbs in any species.

107 the region of the jaw but also included the upper limbs in five of them.

108 ces dynamically represent the space near the upper limbs in hand-centered coordinates.

109 ty to static images of human body parts, and upper limbs in particular, with respect to other object

110 rs characterized by progressive gait ataxia, upper limb incoordination, and dysarthria.

111 The evolution of the human upper limb involved a change in function from its use fo

112 ss began between the ages of 8 and 16 years, upper limb involvement beginning between 10 and 43 years

113 een proposed that functional selectivity for upper limbs is driven by processing of their distinctive

114 ing consisted of three 5-min cycles of right upper limb ischaemia, induced by an automated cuff-infla

115 e ischaemic preconditioning, using transient upper limb ischaemia.

116 e human forearm was induced by 20 minutes of upper limb ischemia (inflation of a blood pressure cuff

117 adial access despite several risk factors of upper limb ischemia - diabetes, end-stage renal failure,

118 One of them is upper limb ischemia caused by radial artery thrombosis.

119 IR injury was induced by 20 minutes of upper limb ischemia followed by reperfusion.

120 te ischemic preconditioning, using cycles of upper limb ischemia-reperfusion as a conditioning stimul

121 yperparathyroidism, or even symptoms of left upper limb ischemia.

122 ssed unusual shapes and orientations of some upper-limb joint complexes relative to the Skhul/Qafzeh

123 ributed neural representation of ipsilateral upper limb kinematics in both monkey and man.

124 ential in restoring function for people with upper-limb loss.

125 ral nervous system region defined as bulbar, upper limb, lower limb or diaphragmatic), diagnosis, fun

126 s demonstrate that vestibular control of the upper limb maintains reaching accuracy during unpredicta

127 heart-hand" disorders, with a combination of upper limb malformations and cardiac defects.

128 Holt-Oram syndrome is characterized by upper limb malformations and cardiac septation defects.

129 abnormalities; others might cause extensive upper limb malformations but less significant cardiac ab

130 and Arg237Gln and Arg237Trp caused extensive upper limb malformations but less significant cardiac ab

131 disorder characterized by facial dysmorphia, upper limb malformations, growth and cognitive retardati

132 tional connectivity in TMSR patients between upper limb maps in M1 and S1 was comparable with healthy

133 cate areas for improvement of the DASH as an upper limb measure for acute stroke.

134 associated with faster disease progression (upper-limb Medical Research Council scale progression ra

135 of mobility milestones by 2.1-4.4 years and upper limb milestones by 2.8-8.0 years compared with tre

136 lated and clinically meaningful mobility and upper limb milestones.

137 easured in intracellular recordings from 206 upper limb motoneurones in ten chloralose-anaesthetized

138 information from the corticospinal tract to upper limb motoneurons.

139 oad but most patients with E90K and N98S had upper limb motor conduction velocities <38 m/s.

140 contribution weight of lesion likelihood to upper limb motor deficit.

141 INTERPRETATION: Our results suggest that upper limb motor execution, and particularly dexterous c

142 Upper limb motor function scores, electromyography from

143 l in elderly humans experiencing declines in upper limb motor functions.

144 ential of dopaminergic treatments to improve upper limb motor movements was tested in 7 aged rhesus m

145 Upper limb motor nerve conduction velocity was 19.9 m/s

146 L-Dopa or GBR-12909 improved upper limb motor performance by up to 40% in the aged an

147 tomical structures in supporting post-stroke upper limb motor recovery and points towards methodologi

148 r imagery in stroke patients with persistent upper limb motor weakness.

149 In addition, upper-limb motor performance times of the aged GDNF-trea

150 1 may promote proximal, and possibly distal, upper-limb motor recovery following frontal and frontopa

151 correlate with poorer levels of recovery of upper limb movement.

152 s involved in 4 weeks of mental rehearsal of upper limb movements during 45-min supervised sessions t

153 lliculus (SC) are involved in the control of upper limb movements.

154 Upper-limb movements are often composed of regular submo

155 score (a composite measure of walking speed, upper-limb movements, and cognition; for this z score, n

156 Here, we recorded SLRs from an upper limb muscle while humans reached toward (pro-reach

157 Latencies to upper limb muscles correlated with cervical MRI lesion l

158 fects of abobotulinumtoxinA injection in the upper limb muscles on muscle tone, spasticity, active mo

159 nA at doses of 500 U or 1000 U injected into upper limb muscles provided tone reduction and clinical

160 st that the corticospinal drive to lower and upper limb muscles shows significant developmental chang

161 of homologous left and right pairs of distal upper limb muscles was performed.

162 These PTNs facilitate EMG activity in upper limb muscles, contributing to corticomuscular cohe

163 pinal input to the spinal segments supplying upper limb muscles, whilst leaving intact excitation tra

164 logically exaggerated common drive in distal upper limb muscles.

165 tant disparities between proximal and distal upper limb muscles.

166 sponsiveness of corticospinal projections to upper limb muscles.

167 e recorded myoelectric signals from multiple upper-limb muscles in subjects with cortical lesions.

168 Distal upper limb myopathy/cachexia is not previously described

169 unrelated patients in whom a distal, mainly upper limb, myopathy was the predominant and early clini

170 ncompassed small and large insertions in the upper limb (n = 21), lower limb (n = 27), and spine (n =

171 severity scale scores, greater reduction of upper limb nerve conduction study amplitudes, more frequ

172 e analysis of the axonal components of human upper limb nerves based on highly specific molecular fea

173 bly increase the dexterity and embodiment of upper-limb neuroprostheses and will constitute an import

174 To restore motor function with upper-limb neuroprostheses requires that somatosensory f

175 r synergist and non-synergist muscles of the upper limb occur in the newborn baby and become restrict

176 s in promoting motor function of the paretic upper limb of stroke patients.

177 re post-depositional, but those in the right upper limb of the adult hominin strongly indicate active

178 from the spinal cord to innervate the human upper limb, of which 10% are motor neurons.

179 racterized by predominant myoclonic jerks of upper limbs, often provoked by cognitive activities, and

180 Subgroups with bulbar, lower and upper limb onset showed different decline rates of ALSFR

181 tation (TFD) of parts of the stroke-affected upper limb or of the less-affected contralateral limb mi

182 jects received RIPC (or placebo) stimuli (x3 upper limb (or dummy arm), 5-minute cycles of 200 mm Hg

183 developed to restore function to people with upper-limb paralysis.

184 tional capacity in people with long-standing upper-limb paresis poststroke.

185 The upper limb perimeter decreased in 7 of 9 patients.

186 inimum level of grip is necessary before the upper limb plays an active role in vestibular-evoked bal

187 Significant improvements in contralateral upper limb postural (P2) and kinetic tremors, spiral sco

188 ered in the differential diagnosis of distal upper limb predominant myopathy.

189 ions, known to be important for multisensory upper limb processing.

190 However, control of upper limb prostheses is limited by the lack of sensory

191 s to conclude that significant between-group upper-limb-related behavioral differences must have been

192 vanic vestibular stimulation (GVS) can evoke upper limb responses to maintain balance.

193 Upper-limb RIPC performed while patients were under prop

194 The trial compared upper-limb RIPC with a sham intervention.

195 ty in multivoxel activation patterns in both upper-limb-selective OTC regions and the hMT+.

196 s suggest that the functional specificity of upper-limb-selective regions may be partially determined

197 hV4 or LO-1, suggesting a tight link between upper-limb selectivity and motion processing.

198 Sympathetic denervation of the upper limb significantly improves forearm skeletal muscl

199 tematically perceived the position of moving upper limbs slightly ahead of their real position but on

200 On the hairy skin of the upper limb, spatial acuity for pain and touch followed o

201 lements, which characterize upper limbs, and upper-limb-specific motion patterns.

202 Upper limb strength is only mildly affected and cardiac

203 gs in motor cortex as rats learned a skilled upper-limb task.

204 velopmental disorder affecting the heart and upper limb, the gene for which was mapped to chromosome

205 naturalistic movements involving the entire upper limb, the results also suggest that achieving high

206 ical anatomy of nerves innervating the human upper limbs, the definite quantity of sensory and motor

207 In relation to the upper limbs, the main predictor of outcome is initial se

208 Resolution of impairment was insensitive to upper limb therapy dose.

209 bility in this pathway, and is unaffected by upper limb therapy dose.

210 n individualized, progressive, task-specific upper-limb training program designed to improve upper-li

211 A 3 Hz 'filter' for postural (P2) upper limb tremor was detected by accelerometry/spectral

212 hronization and independence with respect to upper limb tremor.

213 duals born with absent or severely shortened upper limbs (upper limb dysplasia), despite some variabi

214 of consciousness, effort-associated vertigo, upper limb weakness and temporary vision problems.

215 included 121 stroke patients with a residual upper limb weakness within 6 months following stroke (on

216 t subject (aged 57 years), and marked distal upper limb weakness.

217 At an individual level, those patients whose upper limbs were most affected had a higher incidence of

218 Upper limbs were procured from brain-dead organ donors.

219 of these tremors, the action tremors of the upper limbs were subclassified according to the predomin