ライフサイエンスコーパス: upper lip

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1 al maxillary excess and hypermobility of the upper lip.

2 al maxillary excess and hypermobility of the upper lip.

3 capacity of cells contributing to the future upper lip.

4 ferents, was activated by stimulation of the upper lip (5-30 V; 10 Hz), and BFcrb was recorded at the

5 ease of IQR, P-value 0.0371), and wrinkle on upper lip (7.7% more wrinkle on upper lip per increase o

6 minent eyes, a narrow nasal bridge, a tented upper lip, a high palate, an open mouth, tightly adheren

7 al examination revealed hypermobility of the upper lip and absence of generalized altered passive eru

8 ilateral swellings, and midline notch in the upper lip and alveolus.

9 face was represented from chin/lower lip to upper lip and neck/upper face in a rostrocaudal sequence

10 rther understand how retinoic acid regulates upper lip and palate morphogenesis we searched for genes

11 hat retinoic acid is a critical regulator of upper lip and primary palate development in Xenopus laev

12 better understand the complex nature of the upper lip and primary palate development which will lead

13 The upper lip and primary palate form an essential separatio

14 growth and differentiation necessary for the upper lip and primary palate morphogenesis.

15 nction also results in a median cleft in the upper lip and primary palate.

16 and RARgamma result in a median cleft in the upper lip and primary palate.

17 that fate mapping revealed contribute to the upper lip and primary palate.

18 representation correspond to the upper face, upper lip, and chin plus lower lip, whereas three or fou

19 crest cell (cNCC) mesenchyme is required for upper lip closure.

20 In the fetal specimens, the upper lip (coronal plane), alveolar ridge, tooth sockets

21 olved in regulating mid-face development and upper lip fusion and are therefore strong candidates for

22 Somatosensory stimuli were applied to the upper lip (glabrous skin) and the chin (hairy skin).

23 ed in response to tactile stimulation of the upper lip in 69% of cases, the lower lip in 85% of cases

24 res reorganizes after deafferentation of the upper lip in neonatal rats (postnatal day [PND] 1-30).

25 displacement) to the glabrous surface of the upper lip in order to index the modulation and specifici

26 rtently affect facial structures such as the upper lip, lateral aspect of the nose, and lower eyelid.

27 Stimulation of the nose, upper lip, lower lip, and chin caused a somatotopic late

28 drives cNCC mesenchyme proliferation during upper lip morphogenesis, and that disruption of this seq

29 The upper lip of the chick embryo forms by confluence of pri

30 reflects maxillary versus mandibular origin, upper lip origin and whisker rostrocaudal arc.

31 d wrinkle on upper lip (7.7% more wrinkle on upper lip per increase of IQR, P-value 0.0218).

32 bitemporal narrowing, broad nasal tip, thin upper lip, posteriorly rotated or low-set ears, and micr

33 izing enzyme is ectopically expressed in the upper lip primordia of Lrp6-deficient embryos, indicatin

34 The upper lip region has the most significant change in HDX

35 ectoderm of the medial nasal processes, the upper lip remained intact in mutant mice.

36 The field potentials evoked in crus II by upper lip stimulation did not differ between wild-type a

37 In wild-type mice, upper lip stimulation increased BFcrb in crus II by 32 +

38 somatosensory cortex blood flow produced by upper lip stimulation was not attenuated in D2-null mice

39 eatures that include a prominent maxilla and upper lip that readily reveal the upper gingiva, widely

40 2) Upper lip whisker axons had more boutons than those from

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