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1 in the presence of antibody specific for the upstream stimulatory factor.
2 the binding of Sp1, an Sp1-like protein, and upstream stimulatory factor.
3 existence of two E-like boxes known to bind upstream stimulatory factors.
4 ter is regulated by tandem E-boxes that bind upstream stimulatory factors.
7 ors, revealed that the transcription factors upstream stimulatory factor 1 (USF-1) and USF-2 are indi
8 s interferon regulatory factor 1 (IRF-1) and upstream stimulatory factor 1 (USF-1) in gamma interfero
10 sic helix-loop-helix leucine zipper proteins upstream stimulatory factor 1 (USF1) and USF2 were shown
11 tor (Inr) and a recognition site (E-box) for upstream stimulatory factor 1 (USF1), and to stimulate U
12 was found, whereas for rs8076131, changes in upstream stimulatory factor 1 and 2 transcription factor
16 p-helix leucine zipper transcription factors upstream stimulatory factors 1 and 2 (USF1 and USF2) did
17 1 as well as an E-box motif that is bound by upstream stimulatory factors 1 and 2 (USF1 and USF2).
18 , CaRE2, within BDNF promoter III that binds upstream stimulatory factors 1 and 2 (USF1/2) and show t
19 ologue-2 (Mash-2), which increases levels of upstream stimulatory factors 1 and 2 (USF1/2) and their
20 eriments demonstrated that overexpression of upstream stimulatory factors 1 and 2 increased cGMP-depe
21 te that two basic helix-loop-helix proteins, upstream stimulatory factors 1 and 2, are major proteins
22 strated that this region was occupied by the upstream stimulatory factors 1/2 (USF1/USF2), similar to
23 r binding the E-C4 element was identified as upstream stimulatory factor-1 (USF-1), a basic helix-loo
24 lucidation of a novel mechanism of action of upstream stimulatory factor-1 (USF-1), which involves it
25 s interactions of the basic helix-loop-helix upstream stimulatory factor-1 (USF1) with an E-box1-cont
28 -loop-helix-leucine-zip transcription factor upstream stimulatory factor 2 (USF2) is required for the
29 y identifies the human transcription factor, upstream stimulatory factor 2 (USF2), as a nuclear facto
30 ously determined that the regulatory factor, upstream stimulatory factor 2 (USF2), can stimulate tran
31 he DOR promoter contains an E-box that binds upstream stimulatory factor and is crucial for the DOR p
32 on assays indicated that members of both the upstream stimulatory factor and Sp families of transcrip
34 s and cotransfection experiments showed that upstream stimulatory factors and CCAAT/enhancer-binding
35 activating protein-2, nuclear factor 1, and upstream stimulatory factor, and together, they constitu
36 g luciferase reporter gene assays identified upstream stimulatory factors as the transcription factor
37 -2 gene in rat granulosa cells and binds the upstream stimulatory factor (as do E-box regions of othe
38 o binding studies suggest a function for the upstream stimulatory factor at both the -65 and -332 E-b
39 with 3-fold activation or repression and one upstream stimulatory factor binding site associated with
43 promoter elements demonstrated a functional upstream stimulatory factor/E box in the TATA box-proxim
45 at -444 to -278 with an E-box at -332 where upstream stimulatory factor functions for maximal induct
46 the cell-specific enhancer, which binds the upstream stimulatory factor helix-loop-helix protein and
47 tivates the DOR promoter by synergizing with upstream stimulatory factor in specific DNA binding.
48 Moreover, an E box (-185 to -180) that binds upstream stimulatory factor is essential for the dor pro
49 ich binds the basic helix-loop-helix protein upstream stimulatory factor, is required for PIGR promot
50 e BRCA2 promoter by inhibiting binding of an upstream stimulatory factor protein complex to the promo
51 esis of the E-boxes abolished the binding of upstream stimulatory factor proteins and decreased the a
52 ted with Sp1/Sp3 and USF1/USF2 (where USF is upstream stimulatory factor), respectively, were require
54 thermore, we have demonstrated that Ik-2 and upstream stimulatory factor synergize in trans-activatin
56 that two cellular transcription factors, an upstream stimulatory factor USF, and a member of the E2F
57 show that FIRE3 (-68/-58) binds not only the upstream stimulatory factors USF-1/USF-2 but also the CC
58 , we now demonstrate that two such proteins, upstream stimulatory factor (USF) 1 and 2, readily assoc
59 Previous in vitro studies demonstrated that upstream stimulatory factor (USF) 1 and USF2 bind to the
60 we showed that the E-box specifically binds upstream stimulatory factor (Usf) 1 and Usf2, which are
61 ansactivators specificity factor 1 (Sp1) and upstream stimulatory factor (USF) and an open reading fr
63 (FAS), we examined the relationship between upstream stimulatory factor (USF) and SREBP-1c, two tran
64 has been shown to bind transcription factor upstream stimulatory factor (USF) and to contribute to e
65 cipitation assay, we show that Myc, Max, and upstream stimulatory factor (USF) bind to the chromosoma
66 ur finding that the depolarization-sensitive upstream stimulatory factor (USF) binds to a composite C
67 (COUP-TF) binds specifically to AF3 and that upstream stimulatory factor (USF) binds to an E-box moti
68 ch members of the nuclear factor 1 (NF1) and upstream stimulatory factor (USF) families bind to and r
72 on -226 to -194 of the ADH promoter, whereas upstream stimulatory factor (USF) was shown previously t
74 ding activator proteins 1 and 4, CAAT, GATA, upstream stimulatory factor (USF), estrogen receptor (ER
76 H complex as a heterodimer of the ubiquitous upstream stimulatory factor (USF)-1 and USF-2 proteins.
77 primed 5'Dbeta2 promoter requires binding of upstream stimulatory factor (USF)-1 to a noncanonical E-
80 l signaling and OPN promoter activity via an upstream stimulatory factor (USF)-activated cognate inhi
84 The gene encoding the transcription factor upstream stimulatory factor (USF)1 influences susceptibi
85 unctional consequences of binding of Myc and upstream stimulatory factor (USF)1 to endogenous cellula
86 and gel mobility supershift assays identify upstream stimulatory factor (USF; USF1:USF2) and activat
89 In this study, we investigated the role of upstream stimulatory factors (USF) in the regulation of
90 ne analysis, with evidence of recruitment of upstream stimulatory factors (USF) transcription factors
92 iched (BETA2:E47) and generally distributed (upstream stimulatory factor [USF]) protein-DNA complexes
97 Gel mobility shift assays identified the upstream stimulatory factors (USFs) as a major E-box-bin
98 mal promoter region from -71 to -50 and that upstream stimulatory factors (USFs), including USF1 and
99 a consensus E-box (CACGTG) was shown to bind upstream stimulatory factor using nuclear extracts from
102 racteristics of the helix-loop-helix protein upstream stimulatory factor, whereas a factor binding th
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