1 crystals but not in response to LPS or other
urate crystals.
2 al mechanism for the removal of inflammatory
urate crystals.
3 rbated the inflammatory response elicited by
urate crystals and abrogated the anti-inflammatory effec
4 abbit knee joints with 10 mg of pyrogen-free
urate crystals and analyzed IL-8 levels, leukocyte influ
5 ent understanding of the interaction between
urate crystals and key cellular components of the gouty
6 ytidylic acid or a combination of monosodium
urate crystals and Mycobacterium smegmatis was effective
7 ent-activating structures such as monosodium
urate crystals and zymosan was not affected by BCD.
8 in the response of mononuclear phagocytes to
urate crystals,
and these events can be distinguished at
9 and the inflammatory response to monosodium
urate crystals are translating into potential new treatm
10 ion of the NLRP3 inflammasome in response to
urate crystals,
ATP and lipotoxic fatty acids.
11 The noninflammatory removal of
urate crystals by mature macrophages defines a new pathw
12 Intrarenal
urate crystal deposition was absent in all groups.
13 rol disease manifestations related to tissue
urate crystal deposition.
14 Although
urate crystal diagnosis remains the gold standard for di
15 itis, caused by the deposition of monosodium
urate crystals in and around the joints.
16 The role of formalin in dissolving
urate crystals in pathologic specimens was further clari
17 s that results from deposition of monosodium
urate crystals in the joint.
18 ymorphonuclear cell accumulation elicited by
urate crystals in the murine peritoneal cavity.
19 eta release and PMN accumulation elicited by
urate crystals in the murine peritoneal cavity.
20 ute neutrophilic inflammation in response to
urate crystals in the subcutaneous air pouch model.
21 Urate crystals induce different classes of neutrophil ch
22 In normal mice,
urate crystals induced a 10-fold increase (P < 0.01) in
23 In mIL-8RH(-/-) mice,
urate crystals induced a proteinaceous leukocyte-poor ex
24 AC component C6 to determine if MAC mediated
urate crystal-
induced arthritis.
25 tential role for interleukin-1 inhibition in
urate crystal-
induced inflammation.
26 Analogously, monosodium
urate crystal-
induced neutrophil migration to the tibiof
27 familial cold autoinflammatory syndrome and
urate crystal-
induced peritonitis.
28 s was also observed in a model of monosodium
urate crystals-
induced inflammation.
29 ndent neutrophil recruitment in a monosodium
urate crystal inflammatory murine peritonitis model.
30 odel of gouty arthritis, direct injection of
urate crystals into the rat joint provoked a marked infl
31 In contrast, LPS, monosodium
urate crystals,
or M. smegmatis alone had no activity.
32 wever, tophi and tissue stores of monosodium
urate crystals resolve slowly, particularly in patients
33 inflammation, NLRP3 activation by monosodium
urate crystals similarly increased IL-6.
34 Monosodium
urate crystals stimulate IL-1beta secretion via cryopyri
35 peritonitis model of gout, using monosodium
urate crystals to activate NLRP3, 15d-PGJ2 caused a sign
36 The potential for antibodies against
urate crystals to potentiate further crystallization may
37 Likewise, release of the chemokine KC by
urate crystals was attenuated by [d-Trp(8)]-gamma-MSH, a
38 Urate crystals were injected into subcutaneous air pouch