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1 crystals but not in response to LPS or other urate crystals.
2 al mechanism for the removal of inflammatory urate crystals.
3 rbated the inflammatory response elicited by urate crystals and abrogated the anti-inflammatory effec
4 abbit knee joints with 10 mg of pyrogen-free urate crystals and analyzed IL-8 levels, leukocyte influ
5 ent understanding of the interaction between urate crystals and key cellular components of the gouty
6 ytidylic acid or a combination of monosodium urate crystals and Mycobacterium smegmatis was effective
7 ent-activating structures such as monosodium urate crystals and zymosan was not affected by BCD.
8 in the response of mononuclear phagocytes to urate crystals, and these events can be distinguished at
9  and the inflammatory response to monosodium urate crystals are translating into potential new treatm
10 ion of the NLRP3 inflammasome in response to urate crystals, ATP and lipotoxic fatty acids.
11               The noninflammatory removal of urate crystals by mature macrophages defines a new pathw
12                                   Intrarenal urate crystal deposition was absent in all groups.
13 rol disease manifestations related to tissue urate crystal deposition.
14                                     Although urate crystal diagnosis remains the gold standard for di
15 itis, caused by the deposition of monosodium urate crystals in and around the joints.
16           The role of formalin in dissolving urate crystals in pathologic specimens was further clari
17 s that results from deposition of monosodium urate crystals in the joint.
18 ymorphonuclear cell accumulation elicited by urate crystals in the murine peritoneal cavity.
19 eta release and PMN accumulation elicited by urate crystals in the murine peritoneal cavity.
20 ute neutrophilic inflammation in response to urate crystals in the subcutaneous air pouch model.
21                                              Urate crystals induce different classes of neutrophil ch
22                              In normal mice, urate crystals induced a 10-fold increase (P < 0.01) in
23                        In mIL-8RH(-/-) mice, urate crystals induced a proteinaceous leukocyte-poor ex
24 AC component C6 to determine if MAC mediated urate crystal-induced arthritis.
25 tential role for interleukin-1 inhibition in urate crystal-induced inflammation.
26                      Analogously, monosodium urate crystal-induced neutrophil migration to the tibiof
27  familial cold autoinflammatory syndrome and urate crystal-induced peritonitis.
28 s was also observed in a model of monosodium urate crystals-induced inflammation.
29 ndent neutrophil recruitment in a monosodium urate crystal inflammatory murine peritonitis model.
30 odel of gouty arthritis, direct injection of urate crystals into the rat joint provoked a marked infl
31                 In contrast, LPS, monosodium urate crystals, or M. smegmatis alone had no activity.
32 wever, tophi and tissue stores of monosodium urate crystals resolve slowly, particularly in patients
33 inflammation, NLRP3 activation by monosodium urate crystals similarly increased IL-6.
34                                   Monosodium urate crystals stimulate IL-1beta secretion via cryopyri
35  peritonitis model of gout, using monosodium urate crystals to activate NLRP3, 15d-PGJ2 caused a sign
36         The potential for antibodies against urate crystals to potentiate further crystallization may
37     Likewise, release of the chemokine KC by urate crystals was attenuated by [d-Trp(8)]-gamma-MSH, a
38                                              Urate crystals were injected into subcutaneous air pouch

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