ライフサイエンスコーパス: uremic toxin

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1 n, stiff vessels, coronary heart disease, or uremic toxins).

2 excreted by the kidneys, which are potential uremic toxins.

3 d is being elucidated through the effects of uremic toxins.

4 relatively wide molecular weight spectrum of uremic toxins.

5 ts stresses exerted by as yet poorly defined uremic toxins.

6 acid, gut microbiome products, and so-called uremic toxins accumulating in chronic kidney disease.

7 These records described 32 previously known uremic toxins and 56 newly reported solutes.

8 kidney proximal tubule (PT) transporters of uremic toxins and solutes (e.g., indoxyl sulfate, p-cres

9 OAT1 and/or OAT3 in the handling of over 35 uremic toxins and solutes, including those derived from

10 s (including tryptophan-derivatives that are uremic toxins), and lipids.

11 P) are now recognized as a distinct class of uremic toxins, and numerous compounds in this category h

12 rogram suggested that increased clearance of uremic toxins by intensified hemodialysis improves pregn

13 emodialysis, although beneficial in terms of uremic toxin clearance, also contributes to cognitive de

14 Uremic toxins could modify the expression and/or activit

15 hanisms, including direct neuronal injury by uremic toxins, could also be involved, especially in the

16 d review of the existing knowledge regarding uremic toxins facilitates the design of experimental stu

17 date, assessment of the biologic activity of uremic toxins has focused primarily on in vitro effects,

18 bacterial endotoxins, or adsorb gut-derived uremic toxins have been developed.

19 In CKD, uremic toxins, hyperparathyroidism and Klotho deficiency

20 Emerging evidence suggests that uremic toxins, in particular indoxyl sulfate (IS) and p-

21 We tested whether the uremic toxin indoxyl sulfate (IS), an endogenous ligand

22 icrobiome and demonstrate that levels of the uremic toxin indoxyl sulfate can be modulated in vivo by

23 The highly protein-bound uremic toxin indoxyl sulfate has emerged as a potent tox

24 Accumulation of uremic toxins is a hallmark of renal excretory dysfuncti

25 n that reuse could decrease the clearance of uremic toxins, leading to a decrease in the delivered do

26 This dysfunction has been ascribed to uremic toxins, malnutrition, and dialysis.

27 levels of phosphorus and/or other potential uremic toxins may play an important role by transforming

28 is review, we demonstrate that protein-bound uremic toxins may play an important role in progression

29 uggest that indoxyl sulfate, a protein-bound uremic toxin, may induce vascular dysfunction and thromb

30 dialysis and hence varying concentrations of uremic toxins on CYP3A4 activity using the 14C-erythromy

31 However, the impact of these uremic toxins on the crosstalk between endothelium and l

32 P and history of cardiovascular disease; and uremic toxins p-cresyl sulfate and indoxyl sulfate.

33 Protein-bound uremic toxins (PBUTs) are difficult to remove by convent

34 dequacy, the roles of urea and creatinine as uremic toxins remain controversial.

35 Altogether, these results suggest that uremic toxins, such as IS, through effects on drug trans

36 nce of I3S lies in the fact that it is a key uremic toxin that accumulates to high micromolar concent

37 d is p-cresyl sulfate (PCS), a protein-bound uremic toxin that originates from tyrosine metabolism by

38 Specific uremic toxins that are removed by protein-leaking membra

39 elevation of numerous previously identified uremic toxins, we identified several additional markers

40 of resistin, a proinflammatory cytokine and uremic toxin, were significantly elevated during both fo

41 cess of protein leads to the accumulation of uremic toxins, whereas a diet insufficient in protein co

42 Uremic toxins with potent effects have been identified.

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