ライフサイエンスコーパス: ureteral obstruction

1 ry angiogenesis in the cornea and unilateral ureteral obstruction).

2 ed renal tubular epithelium after unilateral ureteral obstruction.

3 ding to tubular atrophy following unilateral ureteral obstruction.

4 elial cell (TEC) apoptosis during unilateral ureteral obstruction.

5 ent of fibrosis in a rat model of unilateral ureteral obstruction.

6 odel of renal fibrosis induced by unilateral ureteral obstruction.

7 the upper urinary tract following unilateral ureteral obstruction.

8 ntly found in patients with renal disease or ureteral obstruction.

9 was also increased in p27-/- mice following ureteral obstruction.

10 and macrophage infiltration after unilateral ureteral obstruction.

11 ve other common causes for cholecystitis and ureteral obstruction.

12 eys significantly increased after unilateral ureteral obstruction.

13 er in kidneys from CLOCK-deficient mice with ureteral obstruction.

14 chymal damage were significantly worse after ureteral obstruction.

15 exacerbated inflammation and fibrosis after ureteral obstruction.

16 ently available for the cancer patients with ureteral obstruction.

17 kidneys closely parallels that observed with ureteral obstruction.

18 ey injury induced by ischemia reperfusion or ureteral obstruction.

19 c cystinosis, in a mouse model of unilateral ureteral obstruction.

20 d sterile inflammation induced by unilateral ureteral obstruction.

21 ion on kidney fibrosis induced by unilateral ureteral obstruction.

22 sis, cisplatin nephrotoxicity, and bilateral ureteral obstruction.

23 d levels of active TGF-beta after unilateral ureteral obstruction.

24 uropathy (19%), bowel obstruction (14%), and ureteral obstruction (12%).

25 the protective effects in a 3-day unilateral ureteral obstruction (3dUUO) mouse model.

26 We report a case of ureteral obstruction 5 years after cadaveric renal trans

27 shared several clinical features, including ureteral obstruction (5/7 patients), lymphocele (3/7), b

28 on also occurred in kidneys after unilateral ureteral obstruction, a model of tubulointerstitial fibr

29 f a single dose of suramin immediately after ureteral obstruction abolished the expression of fibrone

30 After unilateral ureteral obstruction, all members of the Wnt family exce

31 Endotoxemia and ureteral obstruction also increased NGAL and MCP-1 gene

32 n1alpha1-eGFPL10a mice subject to unilateral ureteral obstruction and analyzed and validated the resu

33 The area under the curve diminished during ureteral obstruction and correlated well with mean corti

34 Patients were clinically stable with known ureteral obstruction and had been referred for antegrade

35 Ureteral obstruction and high ureteral pressure reduced

36 18 induces TLR4 expression during unilateral ureteral obstruction and induces TLR4 expression in HK-2

37 reatment of idiopathic RPF aims at relieving ureteral obstruction and inducing disease regression, an

38 ow that renal fibrosis induced by unilateral ureteral obstruction and metastasis of human cancer xeno

39 retroperitoneum, where it frequently causes ureteral obstruction and renal failure.

40 s and renal fibrosis in mice with unilateral ureteral obstruction, and also attenuates ER stress, pro

41 rile renal inflammation following unilateral ureteral obstruction, and in experimental pyelonephritis

42 studied: Maleate nephrotoxicity, unilateral ureteral obstruction, and LPS preconditioning.

43 ificantly at 14 and 21 days after unilateral ureteral obstruction, and renal oxidized protein levels

44 endoureterotomy is useful for other types of ureteral obstruction, and we aimed to assess its long-te

45 inflammation in murine models of unilateral ureteral obstruction, antimembrane basal GN, and infusio

46 aradigm for immune mediated inflammation and ureteral obstruction as a model for non-immune mediated

47 interstitial fibrosis in mouse kidney after ureteral obstruction, as demonstrated by a reduced inter

48 Ureteral obstruction caused significant increases in int

49 cantly more fibrosis after 7 d of unilateral ureteral obstruction compared with wild-type mice, despi

50 Of 442 patients with ureteral obstruction confirmed at CT, 200 had documented

51 3 hours after reperfusion; after unilateral ureteral obstruction (day 7) in mice; and after gentamic

52 ys of diabetic rats and mice with unilateral ureteral obstruction depicted significant loss of SCAI e

53 diagnosis was established, and the degree of ureteral obstruction determined on urograms was compared

54 at day 21, relative to untreated unilateral ureteral obstruction disease model.

55 is, we subjected VDR-null mice to unilateral ureteral obstruction for 7 days.

56 In mice subjected to unilateral ureteral obstruction, genetic deletion or pharmacologic

57 enges, namely, fistulae, abscesses, bowel or ureteral obstruction, hemorrhage, cancer and thickened m

58 In animals exposed to unilateral ureteral obstruction, Hh pathway suppression by expressi

59 allowed accurate prediction of the degree of ureteral obstruction in 44 (94%) of 47 patients with acu

60 nowledge, this is the first reported case of ureteral obstruction in a transplant kidney caused by an

61 images can be used to predict the degree of ureteral obstruction in acute ureterolithiasis.

62 in the fibrotic kidney induced by unilateral ureteral obstruction in mice, whereas renal Smad abundan

63 voluted tubule cells and those of unilateral ureteral obstruction in the afflicted mouse kidney sugge

64 n highly selected children with intraluminal ureteral obstruction in the hands of a very experienced

65 Ureteral obstruction in the transplant recipient can pro

66 ed inflammation and fibrosis associated with ureteral obstruction in vivo Therefore, domain 4 of CTGF

67 After unilateral ureteral obstruction in wild-type mice, we observed a pr

68 We present evidence that: i) ureteral obstruction induced cell-specific but transient

69 Ureteral obstruction induced Shh, predominantly in the r

70 Reversing the unilateral ureteral obstruction-induced inflammatory microenvironme

71 In a novel mouse model, unilateral ureteral obstruction-induced inflammatory milieu activat

72 ysyl oxidase inhibitor alleviated unilateral ureteral obstruction-induced tubular dilatation and prol

73 tubulointerstitial region of the unilateral ureteral obstruction-injured kidney in mice correlating

74 Unilateral ureteral obstruction injury was induced in Snai1 knockou

75 eate), prerenal (endotoxemia), or postrenal (ureteral obstruction) injury.

76 ession in the afflicted kidney by unilateral ureteral obstruction is accompanied by changes in Usf1 a

77 we report that renal nerve stimulation after ureteral obstruction is the primary profibrotic signal a

78 Unilateral ureteral obstruction kidney Mphis form three distinct su

79 ependent murine fibrosis model of unilateral ureteral obstruction, kidney fibrosis was unexpectedly m

80 ul division of the isthmus, and avoidance of ureteral obstruction, long-term data revealed good graft

81 hen subjected to the normotensive unilateral ureteral obstruction model of endogenous RAS activation,

82 We used the unilateral ureteral obstruction model of progressive kidney fibrosi

83 In the unilateral ureteral obstruction model of renal fibrosis, let-7c upr

84 Here, in the unilateral ureteral obstruction model of renal fibrosis, tubular ep

85 that led to severe fibrosis in a unilateral ureteral obstruction model of renal fibrosis.

86 ers of disease progression in the unilateral ureteral obstruction model of renal interstitial fibrosi

87 In the unilateral ureteral obstruction model, ICG-001 ameliorated renal in

88 placement were performed in 45 patients with ureteral obstruction (n = 40), leak (n = 3), or both (n

89 he normal and injured kidneys, we found that ureteral obstruction not just blocked the NP elimination

90 In one additional control animal, ureteral obstruction occurred at day 10, and was associa

91 , compound 8, starting the day of unilateral ureteral obstruction operation, inhibited collagen depos

92 +) C57BL/6 mice were subjected to unilateral ureteral obstruction or sham surgery (n = 8/group; sham,

93 ed to 5/6 subtotal nephrectomy or unilateral ureteral obstruction, plasma levels of angiopoietin-2 al

94 A signature of unilateral ureteral obstruction, proximal tubular atrophy leads to

95 Finally, in vivo model of unilateral ureteral obstruction revealed that matrix stiffness-regu

96 abetic rat and mouse kidneys with unilateral ureteral obstruction showed SMA expression, as evidenced

97 in treatment of mice subjected to unilateral ureteral obstruction similarly reduced YAP/TAZ levels an

98 injecting oleic acid followed by unilateral ureteral obstruction surgery in mice resulted in enhance

99 and then performed either sham or unilateral ureteral obstruction surgery.

100 tion of folic acid nephropathy or unilateral ureteral obstruction, TbetaRII(endo+/-) mice exhibited l

101 In mice with ureteral obstruction that were treated with the pan anti

102 In the kidneys with unilateral ureteral obstruction, there were significantly more apop

103 th bone marrow from coll-GFP mice, underwent ureteral obstruction to induce fibrosis.

104 In vivo, using ureteral obstruction to model renal fibrosis, we observe

105 After 21 d of unilateral ureteral obstruction, total kidney collagen was signific

106 renal fibrosis disease induced by unilateral ureteral obstruction, transforming growth factor-beta ty

107 ts or wild-type mice subjected to unilateral ureteral obstruction, TRPC3 expression increased in the

108 mean age, 45 years; P = .54) with unilateral ureteral obstruction underwent contemporaneous urinalysi

109 s of renal interstitial fibrosis, unilateral ureteral obstruction, unilateral ischemia-reperfusion, C

110 nancy should not be interpreted as a sign of ureteral obstruction unless the finding persists in the

111 schemic kidney injury is afforded by 24 h of ureteral obstruction (UO) applied 6 or 8 days prior to t

112 s, we subjected the hypomorphs to unilateral ureteral obstruction (UUO) and again found significant p

113 , in two models of kidney injury: unilateral ureteral obstruction (UUO) and ischemia reperfusion.

114 After unilateral ureteral obstruction (UUO) and ischemia/reperfusion, Nog

115 thelial cells of a mouse model of unilateral ureteral obstruction (UUO) and related cell models using

116 ction in renal fibrosis following unilateral ureteral obstruction (UUO) in mice, a model of progressi

117 Unilateral ureteral obstruction (UUO) is a well-characterized murin

118 aling and renal fibrosis in a rat unilateral ureteral obstruction (UUO) model by transferring a doxyc

119 Using the established unilateral ureteral obstruction (UUO) model of kidney fibrosis as o

120 The unilateral ureteral obstruction (UUO) model of kidney injury induce

121 d JNK targets were activated in a unilateral ureteral obstruction (UUO) model of renal fibrosis in vi

122 In a mouse unilateral ureteral obstruction (UUO) model of renal fibrosis, inju

123 tivation in renal fibrosis in the unilateral ureteral obstruction (UUO) model.

124 tubular fibrosis by using a mouse unilateral ureteral obstruction (UUO) model.

125 Two-day-old mice underwent unilateral ureteral obstruction (UUO) or sham operation; 28 days la

126 stigated between 3 and 14 d after unilateral ureteral obstruction (UUO) or sham surgery (n = 8 mice p

127 stigated between 3 and 14 d after unilateral ureteral obstruction (UUO) or sham surgery.

128 of impaired renal growth: chronic unilateral ureteral obstruction (UUO) results in greater injury to

129 ne expression in a mouse model of unilateral ureteral obstruction (UUO) revealed significant inductio

130 l tubules of kidneys subjected to unilateral ureteral obstruction (UUO) using Cre-loxP-mediated gene

131 injury in vivo, a mouse model of unilateral ureteral obstruction (UUO) was employed.

132 g this type of injury, modeled by unilateral ureteral obstruction (UUO), cells undergo epithelial-to-

133 rteen days after the induction of unilateral ureteral obstruction (UUO), wild-type mice reconstituted

134 al tubular autophagy in mice with unilateral ureteral obstruction (UUO).

135 ar epithelial injury initiated by unilateral ureteral obstruction (UUO).

136 murine renal fibrosis induced by unilateral ureteral obstruction (UUO).

137 d in C57BL/6 mice with a model of unilateral ureteral obstruction (UUO).

138 e interstitial fibrosis caused by unilateral ureteral obstruction (UUO).

139 stered to rats that had undergone unilateral ureteral obstruction (UUO).

140 us angiotensin-(1-7) in mice with unilateral ureteral obstruction (UUO).

141 rbates kidney fibrosis induced by unilateral ureteral obstruction (UUO).

142 interstitial fibrosis induced by unilateral ureteral obstruction (UUO).

143 acrophages upregulated Mrc2 after unilateral ureteral obstruction (UUO).

144 the tubulointerstitium (TI) after unilateral ureteral obstruction (UUO).

145 II (Ang II) and mice subjected to unilateral ureteral obstruction (UUO).

146 the fate of these cells following unilateral ureteral obstruction (UUO).

147 mia-reperfusion (I/R, days 1-56), unilateral ureteral obstruction (UUO, days 1-10), and Alport mice (

148 ng protein were subjected to left unilateral ureteral obstruction versus sham operation.

149 arterial pressure, and in six other piglets ureteral obstruction was released.

150 ssue collagen in the kidneys after sustained ureteral obstruction, when compared with their wild-type

151 fibrogenesis in mice subjected to unilateral ureteral obstruction, whereas activation of Hif in myelo

152 monstrated no evidence of ureteral reflux or ureteral obstruction, whereas an immediate prior cohort

153 antly in injured epithelium after unilateral ureteral obstruction, whereas downstream signaling from

154 Kidney hydronephrosis in C2RD was caused by ureteral obstruction, which was, in turn, induced by SCF

155 men, 137 women; mean age, 59 years) without ureteral obstruction who underwent unenhanced scanning a

156 tion may present with acute cholecystitis or ureteral obstruction without its classical clinical symp