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1 RRP = resorufin phosphate, DUT = Dyomics-647 uridine triphosphate).
2              Application of the P2Y agonists uridine triphosphate (15 microM) and adenosine 5'-0-(Z-t
3 bstantial levels of 2'-deoxy-2'-spirooxetane uridine triphosphate (8), a potent inhibitor of the HCV
4           Inserted U's are derived from free uridine triphosphate and are added to the 3' terminus of
5 ed overflow metabolism allows homeostasis of uridine triphosphate and cytidine triphosphate levels at
6 (UGP) alternatively makes UDP-galactose from uridine triphosphate and galactose-1-phosphate.
7 , epidermal growth factor increased cellular uridine triphosphate and this increase was reversed by i
8 s feedback inhibition (which is dependent on uridine triphosphate) and became more sensitive to activ
9  uridine monophosphate, uridine diphosphate, uridine triphosphate, and their nonphosphate compound-ur
10                               An analogue of uridine triphosphate containing a cationic functional gr
11 onists, such as lysophosphatidic acid (LPA), uridine triphosphate, growth hormone, vascular endotheli
12 ate (ATP) > or = 5'adenosine-diphosphate > 5'uridine-triphosphate > or = 5'uridine-diphosphate, suppo
13                                We found that uridine triphosphate-mediated (UTP-mediated) Cl(-) secre
14                                We found that uridine triphosphate-mediated (UTP-mediated) Cl- secreti
15 situ terminal deoxynucleotidyl transferase d-uridine triphosphate nick end labeling (TUNEL) assay, DN
16                     Increased terminal deoxy uridine triphosphate nick end labeling positivity was ob
17        Using both terminal deoxy transferase uridine triphosphate nick end-labelling and poly ADP-rib
18                                   Two analog uridine triphosphates tethering additional functionality
19                             5-(3-Aminopropyl)uridine triphosphate (UNH(2)) as a uridine substitute wa
20 or T7 RNA polymerase than 5-(2-mercaptoethyl)uridine triphosphate (USH).
21 last growth factor (bFGF), by the pyrimidine uridine triphosphate (UTP) and by hypotonicity.
22  was mediated by elevations in intracellular uridine triphosphate (UTP) and cytidine triphosphate (CT
23 ate (ADP), adenosine triphosphate (ATP), and uridine triphosphate (UTP) dose dependently increased th
24       In view of recent data indicating that uridine triphosphate (UTP) is the physiologic phosphate
25                                       Apical uridine triphosphate (UTP) stimulation was shown to incr
26              However, vasoconstrictors (e.g. uridine triphosphate (UTP)) and growth factors, which el
27 to approximately 79% of the peak response to uridine triphosphate (UTP), a P2Y agonist (n = 19).
28                            Two sets of (35)S-uridine triphosphate (UTP)-labeled antisense and sense r
29 hanced the [Ca(2+)](i) increase generated by uridine triphosphate (UTP).
30 s that were similar in magnitude to those of uridine triphosphate (UTP).
31 phate (2MeSADP) or the P2Y2/P2Y4 R agonist 5'uridine-triphosphate (UTP) were reduced but not abolishe

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