ライフサイエンスコーパス: urinary bladder

1 liver and kidney, and was excreted into the urinary bladder).

2 arances suggested chronic obstruction in the urinary bladder.

3 on in isolated detrusor muscle of guinea pig urinary bladder.

4 ich result from developmental defects of the urinary bladder.

5 ediate different sensations arising from the urinary bladder.

6 IR) structures in the neonatal and adult rat urinary bladder.

7 bladder, left and right kidneys, spleen, and urinary bladder.

8 ging agent into the submucosa of the porcine urinary bladder.

9 (TL) sensory neurons that innervate the rat urinary bladder.

10 Primary elimination was via kidneys to the urinary bladder.

11 cral spinal neurons receiving input from the urinary bladder.

12 s and mechanosensory transduction within the urinary bladder.

13 f visceral nociception between the colon and urinary bladder.

14 arkable permeability barrier function of the urinary bladder.

15 rience sensory and motor dysfunctions of the urinary bladder.

16 t to cause hyperplasia and carcinomas in the urinary bladder.

17 lusions in the epithelium of the urethra and urinary bladder.

18 inal afferent nerve endings in the mammalian urinary bladder.

19 V and radioactivity concentration within the urinary bladder.

20 scle (detrusor) and urothelium layers of the urinary bladder.

21 ubset of unmyelinated fibres innervating the urinary bladder.

22 e and aggressive neuroendocrine tumor of the urinary bladder.

23 ts on apoptotic cell death in the developing urinary bladder.

24 ing transitional cell carcinoma (TCC) of the urinary bladder.

25 ressed in kidney, spleen, brain, ureter, and urinary bladder.

26 ological) mechanosensory transduction in the urinary bladder.

27 cancers of the head and neck, esophagus and urinary bladder.

28 of various cancers, including cancer of the urinary bladder.

29 invasive transitional cell carcinoma of the urinary bladder.

30 matory pain from visceral organs such as the urinary bladder.

31 observed with porcine submucosa derived from urinary bladder.

32 tion, induces EGFR and ERK activation in the urinary bladder.

33 l system with contiguous extension up to the urinary bladder.

34 nter the brain and was not excreted into the urinary bladder.

35 s critical for enhancing UPEC fitness in the urinary bladder.

36 ignaling of ketamine-induced cystitis in rat urinary bladder.

37 PERIOD 2 (PER2) in a contractile organ, the urinary bladder.

38 f the oesophagus, stomach, larynx, lung, and urinary bladder.

39 pump urine from the renal pelvis toward the urinary bladder.

40 rm the presence of c-kit positive ICs in pig urinary bladder.

41 and three distinct histological areas of the urinary bladder.

42 y and tumor tissue and clearance through the urinary bladder.

43 ignificant increases above that in wild-type urinary bladders.

44 ladder (0.10 mGy/MBq [380 mrad/mCi]) and the urinary bladder (0.05 mGy/MBq [190 mrad/mCi]).

45 fmol/mg of tissue, respectively), and murine urinary bladder (1.4 +/- 0.1 and 6.2 +/- 2.4 fmol/mg of

46 0.8 fmol/mg of tissue, respectively), canine urinary bladder (1.8 +/- 0.5 and 9.0 +/- 6.0 fmol/mg of

47 invasive transitional cell carcinoma of the urinary bladder (a model of human invasive bladder cance

48 otransmission was characterized in the mouse urinary bladder, a model for the pathological or ageing

49 tracer administration, at which time minimal urinary bladder activity was present to interfere with t

50 the neuropeptide, PACAP, may play a role in urinary bladder afferent pathways after visceral (urinar

51 Similar findings were also observed in the urinary bladder, although the inflammatory infiltrate wa

52 ents with transitional-cell carcinoma of the urinary bladder and 1149 controls in Spain; all the part

53 20 patients with small-cell carcinoma of the urinary bladder and concurrent urothelial carcinoma.

54 athy characterized by severe dilation of the urinary bladder and defective intestinal motility.

55 The sensory neurons innervating the urinary bladder and distal colon project to similar regi

56 lomeningocele typically possess a neurogenic urinary bladder and exhibit varying degrees of bladder d

57 ith a higher decorin expression, than in the urinary bladder and heart, two tissues with a lower deco

58 ole in the activation of Akt and MAPK in the urinary bladder and in bladder hypertrophy during cystit

59 role of mGluR5 in the neural control of the urinary bladder and in the inhibition of the micturition

60 ystem, of virus-labeled neurons from the rat urinary bladder and the external urethral sphincter simu

61 exists for vagal innervation of the male rat urinary bladder and to assess whether those vagal affere

62 In primary tumors of lung, urinary bladder and tongue, induced by different carcino

63 eurally mediated plasma extravasation in the urinary bladder and urethra were examined in urethane-an

64 hird of all cancers of the pancreas, kidney, urinary bladder and uterine cervix.

65 Urinary bladder and uterine guidelines did not have any

66 ucing the volume-induced contractions of rat urinary bladder and was devoid of cardiovascular effects

67 r the liver and kidneys, 1 for spleen, 1 for urinary bladder, and 1 generic compartment accounting fo

68 chloride channels in intestine, gallbladder, urinary bladder, and airway epithelia in various animals

69 ssues and was highest in the salivary gland, urinary bladder, and distal intestinal tract.

70 ive cultures were obtained from the urethra, urinary bladder, and epididimydes, and the ID(50) was de

71 ically significant higher uptake in kidneys, urinary bladder, and lacrimal gland.

72 ormin was primarily taken up by the kidneys, urinary bladder, and liver but also to a lesser extent i

73 s after injection were found in the kidneys, urinary bladder, and liver.

74 FS of canine mesenteric artery (CMA), canine urinary bladder, and murine urinary bladder in the amoun

75 eased risk of human cancers, including skin, urinary bladder, and respiratory tract cancers.

76 rest (ROIs, 50 pixels) were placed in liver, urinary bladder, and tumor tissue in both image sets.

77 Papillary and invasive cancers of the urinary bladder appear to evolve and progress through di

78 The sensory components of the urinary bladder are responsible for the transduction of

79 small bowel, lumbar vertebra, psoas muscle, urinary bladder) as well as the noise-equivalent countin

80 the neurons processing information from the urinary bladder at this level of the neural axis are lik

81 lays a role in the development of TCC of the urinary bladder by acting as a bona fide tumor suppresso

82 blot analysis and in 60 primary TCCs of the urinary bladder by immunohistochemistry.

83 ale rat urinary bladder tumors arise through urinary bladder calculi formation but is insufficient to

84 at desensitization of TRPV1 receptors in the urinary bladder can minimize the effects of cross-sensit

85 SIR, 9.78; 95% CI, 1.18 to 35.30; P = .009), urinary bladder cancer (SIR, 9.51; 95% CI, 1.15 to 34.37

86 Genome-wide association studies (GWAS) of urinary bladder cancer (UBC) have yielded common variant

87 Urinary bladder cancer (UBC) patients at muscle invasive

88 reast cancer (BC), prostate cancer (PC), and urinary bladder cancer (UBC), and is therefore an import

89 Most deaths from urinary bladder cancer are owing to metastatic disease.

90 50% of candidate TumiD targets in T24 human urinary bladder cancer cells is augmented by UPF1.

91 ortant as the effect of NAT2 polymorphism on urinary bladder cancer differs dramatically between mono

92 are continued efforts in the development of urinary bladder cancer markers.

93 Efforts continue in the development of urinary bladder cancer markers.

94 hough efforts continue in the development of urinary bladder cancer markers.

95 COX-2 has also been strongly implicated in urinary bladder cancer primarily by studies with nonsele

96 s) infer a consistent and robust increase in urinary bladder cancer risk following exposures to aroma

97 d for various cancers, this paper focuses on urinary bladder cancer, a cancer in which a role for NAT

98 However, for some malignancies such as urinary bladder cancer, the ability to accurately assess

99 developing various cancer types, especially urinary bladder cancer.

100 d sensitive telomerase activity detection in urinary bladder cancer.

101 ns, 5.8 mm vs 11.9 mm; P < .001), and in the urinary bladder compared with PET/CT (means, 5.9 mm vs 1

102 Afferent pathways innervating the urinary bladder consist of myelinated Adelta- and unmyel

103 Afferent pathways innervating the urinary bladder consist of myelinated Adelta-fibers and

104 Liver, spleen, urinary bladder contents, and total-body activities were

105 comes primarily (97.2%) from activity in the urinary bladder contents.

106 Consistent with increased urinary bladder contractility caused by the absence of B

107 Here, the contributions of each pathway to urinary bladder contraction and the underlying electrica

108 We propose a concept of urinary bladder control involving a previously unidentif

109 Results: The urinary bladder (critical organ), liver, kidney, and spl

110 The urinary bladder (critical organ), liver, kidney, and spl

111 The urinary bladder demonstrated the largest peak uptake (18

112 t 6 to 8 weeks, either 4-layer multilaminate urinary bladder-derived extracellular matrix or expanded

113 t, galanin expression in nerve fibers in the urinary bladder detrusor and urothelium was decreased or

114 Transgenic expression of survivin in the urinary bladder did not cause histologic abnormalities o

115 rom interstitial cystitis, a chronic painful urinary bladder disorder characterized by thinning or ul

116 sponsive MRF neurons that did not respond to urinary bladder distension (i.e. out of the 46 remaining

117 r neurons was recorded in response to graded urinary bladder distension (UBD) in rats pretreated with

118 ims of this study were to examine effects of urinary bladder distension (UBD) on T(3)-T(4) spinal neu

119 of 283 spinal dorsal horn neurons (DHNs) to urinary bladder distension (UBD).

120 neurons exist which encode for a stimulus of urinary bladder distension.

121 Urinary bladder distention (0.5-2.0 mL; 20 seconds) was

122 ficantly increased for both viscerovisceral (urinary bladder distention and colorectal distention) co

123 55 of 152 (36%) spinal neurons responded to urinary bladder distention in dextran sulfate sodium-tre

124 an background activity of neurons excited by urinary bladder distention in rats with dextran sulfate

125 threshold volume for excitatory responses to urinary bladder distention in rats with inflamed colon (

126 nse curves of excitatory responses to graded urinary bladder distention were significantly increased

127 olorectal distention) convergent neurons and urinary bladder distention-receptive neurons in rats wit

128 These changes may contribute to urinary bladder dysfunction after SCI.

129 ecessive disease characterized by congenital urinary bladder dysfunction, associated with a significa

130 condition which characterizes many types of urinary bladder dysfunctions including urinary incontine

131 Positive cultures from the urethra, urinary bladder, epididymides, and testes were obtained

132 4) IFU had positive cultures of the urethra, urinary bladder, epididymides, and/or testes.

133 rvoirs (QIRs) in Lamp1+ endosomes within the urinary bladder epithelium.

134 ng revealed only low levels of F-Dapa in the urinary bladder, even after displacement of kidney bindi

135 PK uptake was highest in the gallbladder and urinary bladder, followed by the liver, kidney, bone mar

136 The highest uptake was in the urinary bladder, followed by the liver, kidneys, and spl

137 implications for treatment of dysfunctional urinary bladder, for which this atropine- and P2X1 antag

138 lts reveal a central role for BK channels in urinary bladder function and indicate that BK channel dy

139 The role of SK3 channels in urinary bladder function was assessed using cystometry i

140 To address the role of the BK channel in urinary bladder function, the gene mSlo1 for the pore-fo

141 ot of Homer2 or Homer3) resulted in impaired urinary bladder function, which was associated with high

142 and treatment of small cell carcinoma of the urinary bladder has become evident.

143 Traditionally, sensory signaling in the urinary bladder has been attributed to activation of bla

144 ing frequency and altered sensation from the urinary bladder has been suggested.

145 xplore the specific autoimmune mechanisms of urinary bladder has long been hindered due to a lack of

146 tic or unresectable urothelial cancer of the urinary bladder has traditionally been treated with syst

147 dings of spinal afferents that innervate the urinary bladder have never been unequivocally identified

148 st, B1R mRNA was not detected in control rat urinary bladder; however, following acute (24 h) and chr

149 PV1, is under investigation for treatment of urinary bladder hyper-reflexia and chronic pain conditio

150 ltered visceral sensation (allodynia) and/or urinary bladder hyperreflexia in the clinical syndrome,

151 rcinoma (TCC), the most common cancer of the urinary bladder in dogs, is usually diagnosed at an adva

152 and greater accumulation of (18)F-FLT in the urinary bladder in male than female mice.

153 pannexin channels into the lumen of the rat urinary bladder in response to distension or stimulation

154 ry (CMA), canine urinary bladder, and murine urinary bladder in the amounts of 7.1 +/- 0.7, 26.5 +/-

155 ion (UTI), manifested by inflammation of the urinary bladder, in humans and is a major global public

156 in human transitional cell carcinomas of the urinary bladder including 36 primary tumors and 1 recurr

157 rogenic contractions of guinea pig and mouse urinary bladder, indicating a second mode of action of n

158 t uptake of 18F-annexin V in the kidneys and urinary bladder, indicating rapid renal clearance of 18F

159 d bacterial adhesion to epithelial cells and urinary bladder infection in mice.

160 esistance, vasculitis, diabetic nephropathy, urinary bladder infections, prostatitis, gastric paresis

161 dder afferent nerve hyperexcitability during urinary bladder inflammation or irritation.

162 llular ATP during pathological conditions of urinary bladder inflammation or irritation.

163 eceptors (Trks) in micturition reflexes with urinary bladder inflammation.

164 ry bladder afferent pathways after visceral (urinary bladder) inflammation.

165 termine the pathway involved in transmitting urinary bladder inputs to thoracic spinal segments.

166 Small cell carcinoma of the urinary bladder is a rare and aggressive neuroendocrine

167 In most cases, small-cell carcinoma of the urinary bladder is admixed with other histological types

168 The urinary bladder is innervated by small afferent neurons

169 An abnormal urinary bladder is no longer a contra-indication to rena

170 2110 to guinea pig cardiac (KD = 5.8 nM) and urinary bladder (KD = 4.9 nM) membranes were of high aff

171 en found in all colorectal, ovarian, breast, urinary bladder, kidney, lung, and pancreatic tumors stu

172 ed in the prostate, breast, colon, pancreas, urinary bladder, kidney, lung, liver, stomach, testis, a

173 , which confirmed the conclusion that in the urinary bladder, low P(CO2) is due to the lack of CA.

174 Urinary bladder malformations associated with bladder ou

175 muscle actin-positive cells were present in urinary bladder matrix (UBM) than in ePTFE (22.2+/-3.3%

176 Urinary bladder matrix-BM (UBM-BM) was found to be a rig

177 sion of neurotrophic factors in the inflamed urinary bladder may contribute to this increased express

178 Both male and female 5-HT3A mutant mice had urinary bladder mucosal and smooth muscle hyperplasia an

179 The epithelium of the urinary bladder must maintain a highly impermeable barri

180 revious studies have demonstrated changes in urinary bladder neurotrophic factors after bladder dysfu

181 sual case of a foreign body removed from the urinary bladder of a 63-year-old male which mimicked a p

182 ateral sprouting in the descending colon and urinary bladder of adult transgenic mice (i.e., those ti

183 veral years because of the presence in their urinary bladder of large, yellowish stones.

184 nstillation of [(99m)Tc]Tat-peptide into the urinary bladder of living rats in vivo, no transepitheli

185 ng water reduces E. coli colonization in the urinary bladder of mice.

186 axonal densities in the descending colon and urinary bladder of NGF transgenic mice with and without

187 root ganglion (DRG) neurons innervating the urinary bladder of rats.

188 cells of Cajal (ICC) have been identified in urinary bladder of several species, but their presence i

189 cognitive impairment, nystagmus, and spastic urinary bladder of varying severity.

190 e fiber immunoreactivity was observed in the urinary bladders of 5-HT3Avs/vs compared with 5-HT3A wil

191 Phenotypically, the urinary bladders of all transgenic lines developed simpl

192 The urinary bladders of SK3T/T had significantly greater bla

193 for calcitonin gene-related peptide) in the urinary bladders of transgenic mice, fibers undergo spro

194 determined that the epithelial lining of the urinary bladder, or urothelium, expresses two subtypes o

195 d effective dose was 0.017 mSv/MBq, with the urinary bladder, osteogenic cells, and red marrow receiv

196 r types were analyzed: stomach, small bowel, urinary bladder, other urothelial, breast, ovarian, and

197 thrography evaluation of the urethra and the urinary bladder plays a very important role in the diagn

198 ildren with small capacity, defunctionalized urinary bladders present unique operative challenges.

199 During chronic inflammation, the urinary bladder presented a predominance of monocyte/mac

200 owever, activation of TRPV1 receptors in the urinary bladder prior to acute colitis increased the num

201 ed ovarian, uterine, and vaginal tumors, and urinary bladder proliferative lesions, including three t

202 The urinary bladder received the highest radiation dose with

203 The urinary bladder receives the highest OD in all investiga

204 2-/-, and P2X2/P2X3(Dbl-/-) mice had reduced urinary bladder reflexes and decreased pelvic afferent n

205 endogenous nerve growth factor (NGF) in the urinary bladder regulated type I collagen expression bec

206 days postsurgery, mice were euthanized, the urinary bladder removed, then fresh-fixed and stained fo

207 nal cell death and functional changes in the urinary bladder, resulting in bladder hyperdistension, u

208 erative, other organs, such as the mammalian urinary bladder, shift from near-quiescence to a highly

209 The synergy between intrinsic urinary bladder signalling mechanisms and an inflammator

210 le synovium and perisynovial adipose tissue, urinary bladder, skeletal muscle, myocardium, and viscer

211 let obstruction (PBOO) induces remodeling of urinary bladder smooth muscle (detrusor).

212 Contraction of urinary bladder smooth muscle (UBSM) is caused by the re

213 ion of nerve transmission to Ca2+ signals in urinary bladder smooth muscle (UBSM) is incompletely und

214 annels are key regulators of excitability in urinary bladder smooth muscle (UBSM) of guinea-pig.

215 tant role in controlling the excitability of urinary bladder smooth muscle (UBSM).

216 ling membrane potential and contractility of urinary bladder smooth muscle (UBSM).

217 Urinary bladder smooth muscle cells of Slo(-/-) mice lac

218 Urinary bladder smooth muscle contraction is triggered b

219 ght to have a particularly prominent role in urinary bladder smooth muscle function and therefore are

220 A 50% decrease in MLCK in urinary bladder smooth muscle had no effect on RLC phosp

221 s, increased phasic contractions of isolated urinary bladder smooth muscle strips and exposed high af

222 a2+](i), MLCK activation, and contraction in urinary bladder smooth muscle strips neurally stimulated

223 mining the excitability and contractility of urinary bladder smooth muscle.

224 s of NGF protein in the descending colon and urinary bladder, so these tissues display increased dens

225 In the absence of BK channels, urinary bladder spontaneous and nerve-evoked contraction

226 In contrast, an abnormal urinary bladder stores urine at high pressure, leaks, or

227 A normal urinary bladder stores urine at low pressure, does not l

228 evoked by beta-adrenoceptor (AR) agonists in urinary bladder strips and cultured bladder urothelial c

229 xponential fitting of activity overlying the urinary bladder suggested that approximately 12% of acti

230 n introduced for the treatment of overactive urinary bladder syndrome.

231 -invasive transitional cell carcinoma of the urinary bladder (TCC-UB) and identified a spectrum of ge

232 nterest in developing methods to enlarge the urinary bladder that avoid bringing the urine in contact

233 s investigated the sensory components of the urinary bladder that may underlie the pathophysiology of

234 In the urinary bladder, the capsaicin-gated ion channel TRPV1 i

235 organs, and more accurate than PET/CT in the urinary bladder; the alignment of thoracic MR/PET with e

236 n were primarily inhibited by input from the urinary bladder through either supraspinal structures or

237 d UPEC attenuation of PMN trafficking to the urinary bladder through pathogen-specific local inductio

238 the peak neurogenic contraction of the mouse urinary bladder to 30-40% of control.

239 Exposure of the lumen of winter flounder urinary bladder to the CaR agonists, Gd(3+) and neomycin

240 when attempting to smile and failure of the urinary bladder to void completely despite a lack of ana

241 ectional area of DRG neurons innervating the urinary bladder trigone (UBT) were evaluated by coupling

242 idence supports the hypothesis that male rat urinary bladder tumors arise through urinary bladder cal

243 s, based on increased incidences of male rat urinary bladder tumors at high exposure levels and on fe

244 Urinary bladder undergoes dramatic volume changes during

245 Surprisingly, sAPs in the mouse urinary bladder, unlike those from other species, are tr

246 y transduction can initiate visceral pain in urinary bladder, ureter, gut and uterus.

247 r responses to mechanical stimulation of the urinary bladder, urethra, colon and penis, and electrica

248 TNP-ATP) on pelvic afferents innervating the urinary bladder using an in vitro mouse bladder-pelvic n

249 investigation of small cell carcinoma of the urinary bladder using novel methods to understand the ge

250 Hodgkin lymphoma, ovary, pancreas, prostate, urinary bladder, uterus, and thyroid) were estimated, wh

251 ively active in lymphatic endothelium in the urinary bladder, uterus, intestine, heart, and airways.

252 During filling, urinary bladder volume increases dramatically with littl

253 (0.094 +/- 0.03), pancreas (0.066 +/- 0.01), urinary bladder wall (0.047 +/- 0.02), and adrenals (0.0

254 upper large intestinal wall (0.08 +/- 0.07), urinary bladder wall (0.08 +/- 0.02), and liver (0.07 +/

255 e human reference adult were received by the urinary bladder wall (0.262 mGy/MBq), kidneys (0.0296 mG

256 ans with the highest absorbed doses were the urinary bladder wall (0.62 mSv/MBq) and the pancreas (0.

257 ses (for 150 MBq injected) were found in the urinary bladder wall (12.2 mGy), spleen (8.1 mGy), kidne

258 the kidneys (14.3 +/- 3.6 muSv/MBq) and the urinary bladder wall (13.5 +/- 3.7 muSv/MBq).

259 e from the (11)C-nicotine injection were the urinary bladder wall (14.68 +/- 8.70 muSv/MBq), kidneys

260 estimate of radiation dose equivalent to the urinary bladder wall (ca. 180 +/- 30 mSv/MBq or 0.7 rem/

261 10 studies, 0.0151 cGy/MBq, occurred for the urinary bladder wall (with hydration and 1- to 2-h voidi

262 tracer, the absorbed dose was highest in the urinary bladder wall and kidneys, followed by the pancre

263 the critical organ (33 mGy), followed by the urinary bladder wall and spleen (10 mGy each), salivary

264 Human radiation dose estimates indicated urinary bladder wall as the dose-limiting organ (0.200 m

265 voiding interval to 60 or 90 min lowered the urinary bladder wall dose to 0.0885 mGy/MBq (0.327 rad/m

266 ffective dose was 31 +/- 1 muSv/MBq, and the urinary bladder wall had the highest absorbed dose at 37

267 fective dose was 38 +/- 4 muSv/MBq, with the urinary bladder wall having the highest absorbed dose at

268 The urinary bladder wall is the critical organ for 11C-WAY10

269 The dose to the urinary bladder wall of the reference patient varies bet

270 e in children younger than 12 y, whereas the urinary bladder wall received the highest dose in older

271 The urinary bladder wall received the highest radiation dose

272 he urine (biological half-life ca. 4 h), the urinary bladder wall received the highest radiation dose

273 The urinary bladder wall received the highest radiation dose

274 Hypertrophy occurs in urinary bladder wall smooth muscle (BSM) in men with par

275 The urinary bladder wall was the organ with the highest esti

276 The critical organ for dosimetry is the urinary bladder wall with a dose of 0.0586 +/- 0.0164 mG

277 doses (mSv/MBq) were 0.40 +/- 0.058 for the urinary bladder wall, 0.11 +/- 0.011 for the kidneys, 0.

278 Peak organ doses were to the urinary bladder wall, 0.258 mGy/MBq (0.955 rad/mCi), and

279 he highest absorbed radiation doses, was the urinary bladder wall, at 0.047 +/- 0.008 and 0.067 +/- 0

280 rbed organ doses were seen in the spleen and urinary bladder wall, followed by kidney, adrenals, and

281 the highest mean dose coefficients were the urinary bladder wall, kidneys, and spleen.

282 upper large intestine wall, small intestine, urinary bladder wall, kidneys, and thyroid had the highe

283 th high radiation burden included the lungs, urinary bladder wall, kidneys, gallbladder wall, heart w

284 6.6 and 60.6 times higher in the kidneys and urinary bladder wall, respectively, than estimates from

285 pt two--the excretory organs gallbladder and urinary bladder wall, which had peak activities at 32 an

286 The critical organ was the urinary bladder wall, with a dose of 0.406 mGy/MBq.

287 +/- 4 muGy/MBq for the absorbed dose in the urinary bladder wall.

288 allbladder wall, small intestine, liver, and urinary bladder wall.

289 the gallbladder, liver, lungs, kidneys, and urinary bladder wall.

290 duced only modest effects on the dose to the urinary bladder wall.

291 The critical organ was the urinary bladder wall.

292 ddition, the radioactivity signal within the urinary bladder was lower at 3 h after injection, especi

293 can be derived from the kidneys, ureter, or urinary bladder, we evaluated whether measurement of int

294 n and radioactivity concentration within the urinary bladder were analyzed in both scans.

295 ident cancer of liver, colorectal, lung, and urinary bladder were included as cases and up to four ag

296 Dynamic PET scans of the urinary bladder were obtained in 6 subjects; 2 subjects

297 se rate at 1 m (EDR-1m) from the sternum and urinary bladder were obtained.

298 Kidney pairs and urinary bladders were cultured 48 h after infection.

299 wed a markedly-contracted and small-capacity urinary bladder with a thickened, irregular and edematou

300 including pain and discomfort related to the urinary bladder, without infection or other identifiable