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1 ll maintained the increased water intake and urinary output.
2 mode of iron excretion, favoring fecal over urinary output.
3 ne and lidocaine group had three-fold higher urinary output (2.1 mL//kg/hr [95% CI, 1.2-3.8] vs 0.7 m
4 d and was the only reliable predictor of the urinary output after furosemide (area under the receiver
5 been shown to improve glomerular filtration, urinary output, and renal histopathology in laboratory a
10 rticularly compelling because measurement of urinary output is inexpensive and routinely performed in
12 venous blood flow, decreases intraoperative urinary output, lowers respiratory compliance, increases
13 omes (KDIGO) stage 2 (>/=2 times baseline or urinary output <0.5 mL/kg/h for >/=12 hours) and plasma
14 ined subgroup of 120 patients with oliguria (urinary output, < 400 ml per day), dialysis-free surviva
15 th respect to oxidative biomarkers, the 24-h urinary output of F2-Iso and 8-OHdG had median values of
17 +/- 0.3 mL/kg; p < 0.001), body weight, and urinary output remained stable under decision assist and
18 , and was the only reliable predictor of the urinary output response to furosemide in acute kidney in
22 educed while patients were maintained within urinary output targets a higher percentage of the time.
26 rinary furosemide concentrations, and hourly urinary output were used to assess furosemide pharmacoki
27 refore, we evaluated the predictive value of urinary output within 24 hours after extracorporeal memb
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