ライフサイエンスコーパス: urine output

1 xtraction ratio; hematocrit; hemoglobin; and urine output.

2 on (APACHE) III score, serum creatinine, and urine output.

3 , serum and urine sodium and osmolality, and urine output.

4 increased metabolic acidosis, and decreased urine output.

5 pectively, with abdominal pain and decreased urine output.

6 er and had significantly higher minimum 4-hr urine output.

7 fferences in donor age and lowest total 4-hr urine output.

8 ney injury in 67.2% of the patients with low urine output.

9 bitors stimulate AQP2 trafficking and reduce urine output.

10 lated, PDSinh-C01 produced a 60% increase in urine output.

11 nd classified AKI using serum creatinine and urine output.

12 macroscopic appearance, renal blood flow and urine output.

13 ase in serum creatinine level or decrease in urine output.

14 Diuretic dose was a strong predictor of urine output.

15 miR-192 blunted the adaptational increase of urine output.

16 The addition of nesiritide did not change urine output.

17 output and the factors associated with high urine output.

18 c) Pearson correlations were quantified with urine output.

19 higher proteinuria and a marked reduction in urine output.

20 es associated with a significant increase in urine output.

21 inine (1.91 [1.06 to 3.44] per 1 mg/dl), and urine output (0.12 [0.03 to 0.44] L/d) were independentl

22 pital stay or by study day 14, and a greater urine output (2,926 +/- 1,179 ml vs. 2,350 +/- 1,066 ml;

23 7 years, respectively) and lowest total 4-hr urine output (327+/-208 versus 507+/-437 cc, respectivel

24 atients by levels of serum creatinine and/or urine output according to Kidney Disease Improving Globa

25 In early AKI, urine output after a furosemide stress test (FST), which

26 AUC)+/-SEM of 0.87+/-0.09 (P<0.0001), 2-hour urine output after FST was significantly better than eac

27 sily detected in the urine in the very first urine output after ischemia in both mouse and rat models

28 patients who developed acute kidney injury, urine output alone was a better mortality predictor than

29 alGuard Therapy, which aims to maintain high urine output; alpha-melanocyte-stimulating hormone, with

30 deletion resulted in a threefold increase in urine output and a 500-fold increase in glucosuria, as w

31 ived an FST and evaluated the ability of FST urine output and biomarkers to predict the development o

32 In CoCl2-treated animals, acute urine output and endogenous lithium clearance increased

33 on provoked a three- to fourfold increase in urine output and endogenous lithium clearance, 33% inhib

34 of 13 different commonly consumed drinks on urine output and fluid balance when ingested in a euhydr

35 in-2 knockdown in the hypothalamus decreases urine output and fluid intake, and increases urine osmol

36 luding ACE inhibitors, BG9719 increased both urine output and GFR.

37 Changes in urine output and glomerular filtration rate are therefor

38 on of fluctuation of vasopressin and nightly urine output and its role in patient selection for desmo

39 Both stimuli increased urine output and lithium clearance three- to four-fold a

40 These LUTS may be masked by low urine output and may pose risk to renal allografts after

41 UTB(inh)-14 also increased urine output and reduced urine osmolality in mice given

42 inine were all significantly improved, while urine output and serum lactate had beneficial trends.

43 ly increased solute-free water clearance and urine output and significantly decreased urinary osmolal

44 BG9719 alone caused an increase in urine output and sodium excretion (P<0.05).

45 Water consumption, urine output and sodium excretion were higher in HS rats

46 00032-0.01 mg/kg) dose-dependently increased urine output and the diuretic effect reached a plateau a

47 dels to identify the impact of nesiritide on urine output and the factors associated with high urine

48 Secondary compensation returned urine output and urinary Na(+) excretion to control leve

49 ) was seen for continuous items (e.g., 24-hr urine output) and coded items requiring judgment (e.g.,

50 (2) lower donor age, (3) higher minimum 4-hr urine output, and (4) more HLA matches in recipients of

51 itioned base excess, 2) maintained a greater urine output, and 3) received furosemide; and slower in

52 Heart rate, arterial blood pressure, urine output, and base deficit (as a reflection of sever

53 ents, arterial and mixed venous blood gases, urine output, and biochemical and hematologic analyses w

54 crit, improves mean systemic blood pressure, urine output, and cardiac function, and decreases the ne

55 n, there were no differences in body weight, urine output, and fluid balance.

56 ted with deteriorating acid-base status, low urine output, and hyperkalemia.

57 mpt to normalize blood pressure, heart rate, urine output, and mental status, which are the tradition

58 owed a dramatic improvement in hemodynamics, urine output, and metabolic acidosis, as well as a perce

59 al congestion score, dyspnea assessment, net urine output, and net weight change.

60 etter preserved gas exchange, renal flow and urine output, and prolonged survival.

61 recipient and donor age, cold ischemia time, urine output, and Scr.

62 First, ensuing hypertension decreases urine output, and second, guanylyl cyclase-A (GC-A), the

63 ven when blood pressure, cardiac output, and urine output are within clinically acceptable ranges.

64 m creatinine concentration and a decrease in urine output, are considered tantamount to the injury of

65 de of circulatory pressures, cardiac output, urine output, arterial blood gases, ventilation:perfusio

66 Mean arterial blood pressure, heart rate, urine output, arterial blood oxygen, and PCO2 values, ar

67 eart rate, a lower troponin T, and a greater urine output as compared with norepinephrine (p < .05).

68 After volume expansion, urine output as well as urinary sodium and cyclic GMP ex

69 Cumulative urine output at 4 h after ingestion of cola, diet cola,

70 Urine output at 65 mm Hg was 49+/-18 mL/hr and was 43+/-

71 mated glomerular filtration rates, and daily urine output at days 1, 7, 15, and 30 after kidney trans

72 In these same patients, furosemide increased urine output at the expense of decreased GFR.

73 patients who were nonoliguric (>400 ml/d of urine output) at initiation of the study.

74 er normal values of systolic blood pressure, urine output, base deficit, hemoglobin, and cardiac inde

75 We also assessed 10 creatinine and urine output-based SGF definitions relative to 12-month

76 pressure (CPP), central venous pressure, and urine output before and after the administration of hype

77 Validated questionnaires and low urine output before transplantation may be used to ident

78 ently with HBOC, but consistent patterns for urine output, blood urea nitrogen, and creatinine, were

79 espectively, when serum creatinine level and urine output both indicated stage 3 AKI.

80 reversed the changes in renal blood flow and urine output, but impaired creatinine clearance persiste

81 uretic dose was a strong predictor of higher urine output, but neurohormonal activation (as evidenced

82 Tolvaptan increased urine output by 3 h in a dose-dependent manner (p < 0.00

83 sultambenzosulfonamide at 20 mg/kg increased urine output by 3-5-fold and reduced urine osmolality by

84 ral venous pressure, mean arterial pressure, urine output, central venous (or mixed venous) oxygen sa

85 A treatment results in a further increase in urine output compared with diabetes alone, suggesting a

86 For all 15 animals, physiologic data (urine output, core temperature, arterial pressure, heart

87 Urine output correlated with cortical ADC with furosemid

88 Urine output, creatinine clearance, and sodium and potas

89 ned the significance of oliguria meeting the urine output criteria for AKI (AKI-UO) and examined its

90 nts meet both the serum creatinine level and urine output criteria for AKI and when these abnormaliti

91 Thus, their hyperglycemia, hyperphagia and urine output declined significantly.

92 AP), systemic vascular resistance index, and urine output did not demonstrate any significant changes

93 severity and duration across creatinine and urine output domains with the risk for RRT and likelihoo

94 arrhythmias, peak lactate, inotropic scores, urine output, duration of mechanical ventilation, intens

95 ht provide useful information in addition to urine output during decongestive treatment in heart fail

96 On day 1, KW-3902 monotherapy increased urine output during the first 6 h (445, 531, 631, and 57

97 nnaire detailing their fluid consumption and urine output during the race.

98 one did not significantly affect the rate of urine output, endogenous lithium clearance (an inverse m

99 e downstream advantages, including increased urine output, enhanced plasma volume, reduced weight los

100 significantly greater bladder capacity, and urine output exceeded the infused saline volume.

101 ht loss had occurred and the patient's daily urine output exceeded their daily fluid intake.

102 envalues [lambda(i)]) imaging parameters and urine output from serial bladder volumes were calculated

103 Conivaptan significantly increased urine output in a dose-dependent manner (-11+/-17, 68+/-

104 te clearance), urinary sodium excretion, and urine output in a double-blind, placebo-controlled, cros

105 Daily urine output in AQP1/AQP3 double knockout mice (15 ml) w

106 e significant changes in sodium excretion or urine output in GC-A-deficient mice.

107 Nesiritide did not increase urine output in patients with ADHF.

108 lowed by the mesenteric and celiac arteries, urine output increased 4.2-fold in wild-type mice compar

109 Urine output increased by >80% (p < .01) during the firs

110 The urine output increased from 70.7 (IQR: 70) ml/day to 1,2

111 n be precipitated by dehydration and reduced urine output, increased protein intake, heavy physical e

112 The change in urine output is consistent with the pharmacokinetics of

113 dent on dialysis treatments, oliguric with a urine output less than 650 ml per day, and uremic with a

114 w hemoglobin levels were associated with low urine output, low serum albumin, high parathyroid hormon

115 mg/dL or 50% from baseline within 48 hrs or urine output < 0.5 mL/kg/hr for > 6 hrs despite fluid re

116 2) hyponatremia; or 3) diuretic resistance (urine output </=125 ml/h following intravenous furosemid

117 nd Cl(-)/Cr ratios were both associated with urine output </=1500 mL (area under the curve, 0.830 and

118 inine level >/=2 times the baseline level or urine output <0.5 ml per kilogram of body weight per hou

119 DGF was defined by urine output <30 cc/hour, decline in serum creatinine of

120 Accurate urine output measurements as well as serum creatinine va

121 Hourly urine output measurements were used to guide the infusio

122 Volumes of study fluid administered, 24-hour urine output, measures of resource utilization, and mort

123 e net iron excretion (measured by faecal and urine output minus food input).

124 The time of temperature and urine output nadir was used as a marker of circadian rhy

125 ameters of perfusion (pulse, blood pressure, urine output, normal base deficit).

126 ality was associated independently with 24-h urine output (odds ratio [OR], 5.6; 95% confidence inter

127 /dL in serum creatinine within 48 hrs and/or urine output of < or = .5 mL/kg/hr for > 6 hrs.

128 A minimum hourly urine output of 0.5 mL/kg is a key target guiding periop

129 tated Ringer's solution, infused to a target urine output of 1 mL x kg(-1) x hr(-1) throughout the 24

130 Low urine output of less than 250 mL per day was also predic

131 When subtle reductions in urine output or a rising creatinine are observed postope

132 lood urea nitrogen (OR, 1.02 per mg/dl), log urine output (OR, 0.64 per log ml/d), and heart rate (OR

133 on of 1400W did not change renal blood flow, urine output, or creatinine clearance, whereas infusion

134 erences in GFR, effective renal plasma flow, urine output, or sodium excretion for any time interval

135 etabolism, skin microcirculatory blood flow, urine output, or splanchnic perfusion.

136 m and urine pharmacokinetics, did not affect urine output, osmolality, salt excretion, or acid-base b

137 Overall, in the setting of early AKI, FST urine output outperformed biochemical biomarkers for pre

138 sorption, and urine osmolality and increased urine output (P </= 0.03 except for urine output with ra

139 sorption, and urine osmolality and decreased urine output (P </= 0.04, except for sodium reabsorption

140 After 6 hrs, intestinal wet/dry ratios, urine output, peritoneal fluid, and intraluminal fluid w

141 The treated dogs had excellent urine output posttransplant, with peak serum creatinine

142 tween nesiritide and predictors affected the urine output prediction.

143 y high blood pressure, enhanced thirst, high urine output, proteinuria, and kidney damage.

144 ody treatment: blood pressure; C(3a) levels; urine output; proteinuria; blood urea nitrogen; and kidn

145 ted with arterial pH (r2 = .28; p = .01) and urine output (r2 = .21; p = .03) when analyzed by log-li

146 ient's sCr value decreased to 1.4 mg/dl, and urine output returned to greater than 2000 ml per day.

147 We measured stroke work, urine output, serum creatinine, among other parameters,

148 Graft function was monitored by urine output, serum creatinine, and renal biopsy.

149 Body weight, water intake, urine output, solute and urea excretion, serum and urine

150 etermine whether a low perioperative minimum urine output target is safe and fluid sparing when compa

151 y undergoing elective colectomy to a minimum urine output target of 0.2 mL/kg/h (low group) or 0.5 mL

152 A perioperative urine output target of 0.2 mL/kg/h is noninferior to the

153 Rather than the absolute dose of diuretic or urine output, the primary signal of interest when evalua

154 ed severe salt wasting and sharp increase in urine output under basal conditions.

155 he kidney via the renal artery would restore urine output (UO) and glomerular filtration rate (GFR) i

156 Concerns have been raised about the use of urine output (UO) criteria in CLD.

157 ypertension, systemic hypotension, decreased urine output (UOP), and metabolic acidosis.

158 >/=50% from previous lowest value, and/or if urine output was <1 mL/kg/h on postnatal days 2 to 7.

159 Daily urine output was 1.5-fold greater in UT-B- deficient mic

160 For 24 hours, urine output was 113+/-51 mL/h with placebo and 110+/-56

161 Total urine output was 1294+/-1024 mL and 3492+/-1613 mL durin

162 Median (25th, 75th percentiles) 24-h urine output was 2,280 (1,550, 3,280) ml with nesiritide

163 tions were near normal; lactate was cleared; urine output was adequate.

164 When FST urine output was assessed in patients with increased bio

165 All urine output was collected daily.

166 Urine output was higher during the first when compared w

167 Urine output was higher for groups 2 and 3 than for grou

168 In treated animals, urine output was higher, metabolic acidosis was attenuat

169 Urine output was measured daily in the 75 patients who r

170 Urine output was measured in 5,864 subjects; of these, 5

171 Blood was sampled, total urine output was measured, and the animal was then kille

172 FST urine output was the only biomarker to significantly pre

173 Urine output was the only clinical index of cardiac func

174 Urine output was then collected for the subsequent 4 h.

175 The mean renal blood flow and total urine output were 68.0 mL/min/100 g and 560 mL in the le

176 Arterial pressure and urine output were also attenuated in recombinant human a

177 Hourly temperature and urine output were ascertained from the patient records.

178 f age, serum creatinine, and intra-operative urine output were compared.

179 Flow, resistance, and urine output were measured serially for 4 hr.

180 Blood pressure, heart rate, and urine output were recorded, as well as blood urea nitrog

181 Urine outputs were collected over 3 h subsequent to i.m.

182 lary blood flow and red blood cell velocity, urine output) were measured.

183 sorption, and urine osmolality and increased urine output, while raxibacumab, a PA-directed monoclona

184 Regardless of the end point, combining FST urine output with individual biomarkers using logistic r

185 ncreased urine output (P </= 0.03 except for urine output with raxibacumab [P = 0.17]).

186 ion of PDSinh-C01 produced a 30% increase in urine output, with increased Na(+) and Cl(-) excretion.

187 ted in favorable changes in hemodynamics and urine output without affecting blood pressure or heart r

188 sopressin, and decreases in H(2)O intake and urine output without any effects on mean arterial pressu