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1  is inadequate to abolish the serial 24-hour urine protein.
2 nt interaction was detected between baseline urine protein and ACEI therapy (P = 0.003).
3                                              Urine protein and creatinine ratio (uPr/uCr) was used to
4 eak, which was assessed by measuring 24-hour urine protein and Evans blue dye, was used as a marker o
5                Clinical parameters including urine protein and glomerular filtration rate were measur
6                                              Urine protein and microalbumin did not change in either
7 othalamate glomerular filtration rate [GFR], urine protein, and microalbumin) in 148 living kidney do
8 septal thickness, nerve involvement, 24-hour urine protein, and serum alkaline phosphatase.
9                              The majority of urine proteins appear as cleavage products that are foun
10            IgE antibodies to alphaGal, swine urine proteins, beef and pork meat, serum albumin protei
11                           The fingerprint of urine protein charge forms identifies the glomerular dis
12 inical rejection (n = 6), underwent in-depth urine protein compositional analysis with LC-MS/MS, and
13 croalbumin varied from 3 to 34% of the total urine protein concentration.
14 desonide, or placebo, stratified by baseline urine protein creatinine ratio (UPCR).
15  (median age, 51 vs 53 years) and had higher urine protein-creatinine ratios (median, 98 vs 66 mg/g)
16  age and disease matched through analysis of urine (protein/creatinine) to generate 12 treatment pair
17 n before AMR, and many have proteinuria with urine protein/creatinine more than 0.5 in 41% and more t
18 ary end point was a prespecified decrease in urine protein/creatinine ratio and stabilization or impr
19                                              Urine protein/creatinine ratio at day 7 was elevated in
20 holesterol (209.1 vs. 204.3 mg/dL, P=0.973), urine protein/creatinine ratios (0.398 vs. 0.478 mg/dL,
21                            Serum creatinine, urine protein/creatinine ratios, severity of histologic
22       At 52 weeks there was no difference in urine protein/creatinine, mean arterial pressure or scor
23 tes aged 18 years or older with proteinuria (urine protein:creatinine ratio [UPCR] 500-5000 mg/g) and
24 quantifying the spontaneous variation in the urine protein:creatinine ratio in SLE GN patients who ar
25 32,110 person-years) from seven studies with urine protein dipstick measurements.
26 r kidney donors, and correlated results with urine protein dipstick readings and multiple other param
27                             Twenty-four-hour urine protein electrophoresis (UPEP) was normal.
28 noclonal protein (>/=200 mg per 24 hours) in urine protein electrophoresis (UPEP).
29 tration up to 266 micromol/L (3.0 mg/dl) and urine protein excretion >900 mg/d.
30                        Average baseline 24-h urine protein excretion (g/d) for treated and nontreated
31  measured protein-creatinine ratio and 24-hr urine protein excretion (n=192) and albumin-creatinine r
32 cial after adjustment for blood pressure and urine protein excretion (relative risk, 0.67 [95% CI, 0.
33  n-3 LCPUFA supplementation on the change in urine protein excretion (UPE) and on glomerular filtrati
34 e (ACE) inhibitors reduce blood pressure and urine protein excretion and slow the progression of chro
35 pressure levels was greater in patients with urine protein excretion greater than 1.0 g/d (P < 0.006)
36 129 mm Hg may be beneficial in patients with urine protein excretion greater than 1.0 g/d.
37         We quantitated preprocurement, timed urine protein excretion in 23 "normal" cadaver kidney do
38 tolic blood pressure of 110 to 129 mm Hg and urine protein excretion less than 2.0 g/d were associate
39 children (n = 103) had significantly greater urine protein excretion rates than the non-ADPKD childre
40 e levels increased to 4.3 +/- 0.8 mg/dl, and urine protein excretion rose to 0.64 +/- 0.28 mg/mg crea
41 creases in serum creatinine concentration or urine protein excretion were detected.
42 creatinine), but serum creatinine levels and urine protein excretion were not different from normal.
43 of systolic and diastolic blood pressure and urine protein excretion with kidney disease progression
44 r, income, education, previous CVD, baseline urine protein excretion, and baseline estimated GFR.
45 r increasing serum creatinine concentration, urine protein excretion, and diastolic blood pressure, a
46 n restriction on the rate of decline in GFR, urine protein excretion, and onset of end-stage renal di
47 </=90 mL/min/1.73 m(2) (odds ratio, 2.0) and urine protein &gt;200 mg/g (odds ratio, 2.3).
48 ange, up to 266 micromol/L (3.0 mg/dL), 24-h urine protein &gt;900 mg/d, and at least 6 mo of follow-up.
49                          At higher levels of urine protein, Hispanics had a significantly lower risk
50 ant into rats caused a threefold increase in urine protein in collections from 6 to 24 h after inject
51 teins) of 0.92 (reference range, 0.8-2.0), a urine protein level of 15 mg/dL (normal level, <20 mg/dL
52  with lupus nephritis, correlating well with urine protein levels and systemic lupus erythematosus di
53 n after kidney transplantation and typically urine protein levels are below 500 mg/d.
54 assessed by serial serum creatinine and 24-h urine protein measurements.
55                 The patients with an initial urine protein/osmolality ratio >0.13 mg/L per mosmol per
56 er comparison cohort noninferiority study of urine protein screening for specific indications compare
57 We assessed FEPR (FEPR = [serum creatinine x urine protein] / [serum protein x urine creatinine], %)
58 ed frequency of hemoglobin A(1c), lipid, and urine protein testing; blood pressure measurement; and f
59 c analysis were used to identify patterns of urine proteins that are characteristic of the diseases.
60 rate (<40 mL/min/1.73 m(2)) and proteinuria (urine protein to creatinine ratio >/=0.55 mg/mg) were si
61  interviewed and tested for proteinuria-spot urine protein to creatinine ratio (abnormal: >/=0.20 mg/
62 ck positive level (approximately 300 mg/d or urine protein to creatinine ratio of 0.22), aggressive B
63 adequately anticoagulated thromboembolism; a urine protein to creatinine ratio of less than 1; and me
64                                         Spot urine protein to creatinine ratio, spot urine albumin to
65                           Early detection of urine protein to slow progression of chronic kidney dise
66 f 20-60 ml/min per 1.73 m(2)), and a 24-hour urine protein-to-creatinine ratio >/=800 mg/g to TGF-bet
67  95% CI, 1.48 to 7.23; P<0.001); >/=0.30 g/g urine protein-to-creatinine ratio (HR, 2.44; 95% CI, 1.4
68 (FEPR) may better reflect kidney damage than urine protein-to-creatinine ratio (PCR).
69 an eGFR =38 ml/min per 1.73 m(2), and median urine protein-to-creatinine ratio [UPCR] =0.20 g/g).
70                                     The mean urine protein-to-creatinine ratio decreased by 2% (95% C
71 or predictive of unsuccessful conversion was urine protein-to-creatinine ratio more than 1.
72  rate, urine albumin-to-creatinine ratio and urine protein-to-creatinine ratio) did not (Rho = -0.222
73 s per year; higher BP, serum phosphorus, and urine protein-to-creatinine ratio; lower serum albumin a
74 uria significantly decreased: mean change in urine protein-to-creatinine ratios was -2.53+/-3.76, P =
75                                     The mean urine protein (Up)-to-creatinine (c) ratio increased fro
76 r filtration rate was 38.7 mL/min, and 24-hr urine protein was 1.37 g.
77                               Measurement of urine protein was the most common method of determining
78 g hypertensive donors if kidney function and urine protein were normal.
79                                              Urine proteins were separated by two-dimensional electro
80 +/-2,900 mg (mean+/-SD) of quantitated daily urine protein, which did not correlate with creatinine c

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