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1 ator, plasminogen activator inhibitor-1, and urokinase-plasminogen activator receptor.
2 toxin activation critically depended on both urokinase plasminogen activator receptor and plasminogen
4 r receptor, urokinase plasminogen activator, urokinase plasminogen activator receptor, and vascular e
5 llagen receptors mannose receptor (Mrc1) and urokinase plasminogen activator receptor-associated prot
6 icits in intracellular collagen degradation (urokinase plasminogen activator receptor-associated prot
8 gesting that RoBo-1 is a novel member of the urokinase plasminogen activator receptor/CD59/Ly-6/snake
9 with its relationship to the multifunctional urokinase plasminogen activator receptor/CD59/Ly-6/snake
12 ons were examined by Western immunoblotting, urokinase plasminogen activator receptor, fibronectin, a
13 in hemangioma-derived stem cells, including urokinase plasminogen activator receptor, interleukin-6,
14 nfiltration in the absence of either stromal urokinase plasminogen activator receptor or stromal plas
16 te the potential association between soluble urokinase plasminogen activator receptor (suPAR) and inc
19 We have previously observed that soluble urokinase plasminogen activator receptor (suPAR) prevent
21 , we report the crystal structure of soluble urokinase plasminogen activator receptor (suPAR), which
23 le functional domain similar to the Ly-6 and urokinase plasminogen activator receptor superfamily of
24 apoptotic cells) and pathologic roles of the urokinase-plasminogen activator/receptor system (leads t
28 on promotes the induction of T cell membrane urokinase plasminogen activator receptor (uPAR) and that
31 erence analysis procedure now identifies the urokinase plasminogen activator receptor (UPAR) as a gen
34 Western blot analysis were used to quantify urokinase plasminogen activator receptor (uPAR) expressi
37 aims of this study were to determine whether urokinase plasminogen activator receptor (uPAR) gene amp
41 vated protein kinase 2 (p38(MAPK)), and that urokinase plasminogen activator receptor (uPAR) is an im
43 large ( approximately 70%) reduction in the urokinase plasminogen activator receptor (uPAR) level in
44 ported to mediate the internalization of the urokinase plasminogen activator receptor (uPAR) via liga
48 ith a targeted mutation of the gene encoding urokinase plasminogen activator receptor (uPAR), a key c
49 ing strategy: 1) an elevated tumor receptor, urokinase plasminogen activator receptor (UPAR), and 2)
50 r localization of human cytokeratin 1 (CK1), urokinase plasminogen activator receptor (uPAR), and gC1
51 s identify the subcellular relocalization of urokinase plasminogen activator receptor (uPAR), LIM and
57 ing cathepsin D; matrix metalloproteinase 2; urokinase plasminogen activator receptor (uPAR); fibrone
58 P)-2; urokinase plasminogen activator (uPA); urokinase plasminogen activator receptor (uPAR); plasmin
59 ssue-type plasminogen activator (tPA-/-) and urokinase plasminogen activator receptor (uPAR-/-), we i
60 morphisms in the asthma susceptibility gene, urokinase plasminogen activator receptor (uPAR/PLAUR) ha
62 ercellular adhesion molecule-1 [ICAM-1], and urokinase plasminogen activator receptor [uPAR]) increas
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