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1 fter receiving intravesical inoculation with uropathogenic Escherichia coli.
2 fimbria-mediated haemagglutination assay of uropathogenic Escherichia coli.
3 1 fimbriae (FimH) are positively selected in uropathogenic Escherichia coli.
4 in two different pilus biogenesis systems in uropathogenic Escherichia coli.
5 flammatory markers typically associated with uropathogenic Escherichia coli.
6 mice, but not humans, and known to recognize uropathogenic Escherichia coli.
7 ired for assembly and secretion of P pili in uropathogenic Escherichia coli.
8 of P pili by the chaperone/usher pathway in uropathogenic Escherichia coli.
9 apC usher required for P pilus biogenesis by uropathogenic Escherichia coli.
10 P pili are important virulence factors in uropathogenic Escherichia coli.
11 essential for the expression of Pap pili by uropathogenic Escherichia coli.
12 PapG adhesins mediate the binding of uropathogenic Escherichia coli.
13 plicated as a preferred binding receptor for uropathogenic Escherichia coli.
14 l-binding adhesin molecules of P fimbriae of uropathogenic Escherichia coli.
15 prototypic self-associating AT protein from uropathogenic Escherichia coli.
18 e show that purified CdiA-CT(536) toxin from uropathogenic Escherichia coli 536 translocates into bac
19 in-containing protein C (TcpC) from virulent uropathogenic Escherichia coli, a common human pathogen.
20 ArdB encoded on a pathogenicity island from uropathogenic Escherichia coli and a KlcA from an IncP-1
21 o interfere with adhesive fiber formation in uropathogenic Escherichia coli and oligomerization of am
22 ow that the coupling of fimbrial adhesins of uropathogenic Escherichia coli and pathogenic Neisseria
23 antivirulence strategies aimed at targeting uropathogenic Escherichia coli and potentially other Qse
25 natonium), umami (monosodium glutamate), and uropathogenic Escherichia coli; and release acetylcholin
30 ytotoxic necrotizing factor type 1 (CNF1) of uropathogenic Escherichia coli belongs to a family of ba
31 side (a ligand that promotes the adhesion of uropathogenic Escherichia coli by binding to the FimH pr
32 nt communication pathway by which strains of uropathogenic Escherichia coli can inhibit the growth of
34 ic necrotizing factor type 1 (CNF1)-positive uropathogenic Escherichia coli caused more inflammation-
35 ), but the distribution of Dr subtypes among uropathogenic Escherichia coli causing UTI among otherwi
38 ladder epithelial cells were challenged with uropathogenic Escherichia coli (CFT073) and microbial PA
40 iae are homopolymeric adhesive organelles of uropathogenic Escherichia coli composed of DraE subunits
42 ession and attenuates enterohaemorrhagic and uropathogenic Escherichia coli (EHEC and UPEC), Salmonel
43 ration of different pathogenicity islands in uropathogenic Escherichia coli, enterohaemorrhagic E. co
44 The secreted autotransporter toxin (Sat) of uropathogenic Escherichia coli exhibits cytopathic activ
46 he otherwise healthy host is the movement of uropathogenic Escherichia coli from the intestinal tract
47 expression occurred in clinical isolates of uropathogenic Escherichia coli grown in media with diffe
49 nized variants of PapG, the major adhesin of uropathogenic Escherichia coli, have been deduced in par
53 (pyelonephritis- associated pilus) operon of uropathogenic Escherichia coli is a functional homolog o
55 n of pyelonephritis-associated pili (Pap) in uropathogenic Escherichia coli is epigenetically control
56 ephritis-associated pili (Pap) expression in uropathogenic Escherichia coli is regulated by a complex
60 nsporter toxin (Sat), found predominantly in uropathogenic Escherichia coli, is a member of the SPATE
61 t infections (UTIs), predominantly caused by uropathogenic Escherichia coli, is the adhesion of bacte
62 h this platform, we observed the growth of a uropathogenic Escherichia coli isolate, with an initial
65 he assembly of type 1 pili on the surface of uropathogenic Escherichia coli proceeds via the chaperon
68 1Cer) monoclonal antibody, and P fimbriae on uropathogenic Escherichia coli (specific for Galalpha1-4
70 s study was to determine whether OMVs from a uropathogenic Escherichia coli strain can induce cardiac
73 e gene encoding d-serine deaminase, dsdA, in uropathogenic Escherichia coli strain CFT073 results in
75 ntly, we identified a fimbrial usher gene in uropathogenic Escherichia coli strain CFT073 that is abs
77 K5(-) mutants from an O75(+) K5(+) wild-type uropathogenic Escherichia coli strain in phagocytosis as
78 vic pain behavior elicited by infection with uropathogenic Escherichia coli strain NU14 and ASB strai
79 ide triggers rugose biofilm formation by the uropathogenic Escherichia coli strain UTI89 and by enter
84 ry tract infections are caused by strains of uropathogenic Escherichia coli that encode filamentous a
85 of fimbriae are the type 1 and P fimbriae of uropathogenic Escherichia coli, the major causative agen
87 the P pilus, a key virulence factor used by uropathogenic Escherichia coli to adhere to the host uri
88 onserved chaperone/usher pathway and used by uropathogenic Escherichia coli to attach to bladder cell
89 Pyelonephritis-associated pili (pap) allow uropathogenic Escherichia coli to bind to epithelial cel
92 f adhesion resulting from the interaction of uropathogenic Escherichia coli to molecularly well defin
93 mH, which would otherwise mediate binding of uropathogenic Escherichia coli to the host urothelium to
95 polymeric structures that mediate binding of uropathogenic Escherichia coli to the surface of the kid
96 p of the P pilus that mediates attachment of uropathogenic Escherichia coli to the uroepithelium of t
99 copolymer nanoparticles are conjugated with uropathogenic Escherichia coli type 1 pilus adhesin FimH
101 Urinary tract infections (UTIs) caused by uropathogenic Escherichia coli (UPEC) affect 150 million
102 Urinary tract infections (UTIs) caused by uropathogenic Escherichia coli (UPEC) are a significant
109 ction of the prostate by clinically relevant uropathogenic Escherichia coli (UPEC) can initiate and e
115 Invasion of bladder epithelial cells by uropathogenic Escherichia coli (UPEC) contributes to ant
117 describe the whole bladder transcriptome of uropathogenic Escherichia coli (UPEC) cystitis in mice u
118 e insights into the transcriptional state of uropathogenic Escherichia coli (UPEC) during infection.
121 The metV genomic island in the chromosome of uropathogenic Escherichia coli (UPEC) encodes a putative
122 While in transit within and between hosts, uropathogenic Escherichia coli (UPEC) encounters multipl
123 Here, we show in a murine model of UTI that uropathogenic Escherichia coli (UPEC) established quiesc
124 elial cells (BECs) that expels intracellular uropathogenic Escherichia coli (UPEC) from their intrace
125 binding, invasion, and biofilm formation of uropathogenic Escherichia coli (UPEC) in the host urothe
126 s bladder epithelial binding and invasion by uropathogenic Escherichia coli (UPEC) in the initial sta
130 Urinary tract infection (UTI) caused by uropathogenic Escherichia coli (UPEC) is a substantial e
131 The pathogenesis of pyelonephritis caused by uropathogenic Escherichia coli (UPEC) is not well unders
144 m of initiating innate host defenses against uropathogenic Escherichia coli (UPEC) is the production
145 is isolate TOP52 was compared to that of the uropathogenic Escherichia coli (UPEC) isolate UTI89 in a
146 Here, we show that Hfq is critical for the uropathogenic Escherichia coli (UPEC) isolate UTI89 to e
150 urinary tract infections (UTI), cystitis by uropathogenic Escherichia coli (UPEC) occurs through an
152 tandem mass spectrometry to characterize the uropathogenic Escherichia coli (UPEC) outer membrane sub
156 ated urinary tract infection (UTI) caused by uropathogenic Escherichia coli (UPEC) represents a preva
158 ified two chromosomal open reading frames in uropathogenic Escherichia coli (UPEC) strain CFT073 whic
161 ed transurethrally with the cystitis-derived uropathogenic Escherichia coli (UPEC) strain UTI89.
170 own that 36% (5 of 14) of mice infected with uropathogenic Escherichia coli (UPEC) will have at least
171 usly shown to contribute to the virulence of uropathogenic Escherichia coli (UPEC) within the urinary
173 (UTIs), the majority of which are caused by uropathogenic Escherichia coli (UPEC), afflict nearly 60
174 tract infections (UTIs), primarily caused by uropathogenic Escherichia coli (UPEC), annually affect o
175 tract infections (UTI), primarily caused by uropathogenic Escherichia coli (UPEC), are one of the le
176 t infections (UTIs), predominantly caused by uropathogenic Escherichia coli (UPEC), belong to the mos
177 re fundamental for keeping kidneys free from uropathogenic Escherichia coli (UPEC), but we have shown
178 pe 1 fimbria is a proven virulence factor of uropathogenic Escherichia coli (UPEC), causing urinary t
179 (CNF1), a toxin produced by many strains of uropathogenic Escherichia coli (UPEC), constitutively ac
183 cal for colonization of the urinary tract by uropathogenic Escherichia coli (UPEC), mediate opposing
184 nfections (UTIs) have complex dynamics, with uropathogenic Escherichia coli (UPEC), the major causati
189 haracterize the adaptive immune responses to uropathogenic Escherichia coli (UPEC), the predominant u
193 ulator of stress resistance and virulence in uropathogenic Escherichia coli (UPEC), the principal cau
196 1 pili (T1P) are major virulence factors for uropathogenic Escherichia coli (UPEC), which cause both
197 pe 1 pili are important virulence factors in uropathogenic Escherichia coli (UPEC), which cause the m
198 a key event in the pathogenesis mediated by uropathogenic Escherichia coli (UPEC), yet the mechanism
211 ciation of 3 putative genes for virulence of uropathogenic Escherichia coli; uropathogenic specific p
213 Using the P and type 1 pilus systems of uropathogenic Escherichia coli, we show that a conserved
214 s from Hib pili and from P-pili expressed on uropathogenic Escherichia coli were used to predict the
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