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1 ents, shifted voltage dependence and reduced use dependence.
2 red for channel activation and showed strong use dependence.
3 was increased, and repetitive pulsing showed use dependence.
4 agonists, (ii) voltage dependence, and (iii) use dependence.
5 id in onset, reversible, and did not display use dependence.
6 at activation of the channel exhibits strong use dependence.
7 ndence and nonalcohol psychoactive substance use dependence.
8 )3.2 T currents exhibited little voltage- or use-dependence.
9 heat and agonist activations differ in cross use-dependence.
11 inactivation could be partly responsible for use dependence, a more stringent test would require that
12 s (1 s Vh = -46 mV), showed substantial 1 Hz use dependence and had inactivation (60 ms pulse) recove
13 standing factors that determine specificity, use dependence and other properties of K(+) channel drug
15 t in IVS5 did not mimic the complete loss of use dependence as observed for the replacement of the wh
16 s had a 1 s Vh of -29 mV, showed little 1 Hz use dependence at -67 mV and recovered from the inactiva
17 cited by short depolarizations showed strong use dependence at frequencies as low as 1 Hz, although r
18 y critically shape the dynamic character and use dependence for cranial afferent transmission at the
19 role of inactivated channel conformation and use dependence for diltiazem, a specific benzothiazepine
20 ion attenuate lidocaine (lignocaine)-induced use dependence; however, the pharmacological role of slo
22 ines conduction in peripheral nerves and its use dependence in tetrodotoxin-resistant (TTXr) sodium c
26 eived a primary (DSM-IV) diagnosis of heroin use/dependence (n = 2797) and (2) data from unstructured
27 recovery from inactivation and decrease the use dependence of channel activity with and without the
34 s implications for gating, drug affinity and use dependence of their respective channel complexes.
42 for the 2 binding conformations can control use-dependence, the hallmark of successful antiarrhythmi
43 ngle channel properties of mutants that lost use dependence toward diltiazem were characterized by dr
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